ライフサイエンスコーパス: seminal vesicle

1 trol expression in liver, eye, intestine and seminal vesicle.

2 ults in obstruction of the proximally placed seminal vesicle.

3 the ductal region and the ampulla of the rat seminal vesicle.

4 the incidence of cancer present only in the seminal vesicle.

5 ducts into the epididymis, vas deferens and seminal vesicle.

6 soybean, and cyclooxygenase (COX) from sheep seminal vesicle.

7 ecretory proteins produced by the guinea pig seminal vesicle.

8 erm, testis, epididymis, prostate gland, and seminal vesicle.

9 atic cells at the testis base and around the seminal vesicle.

10 in 20 fractions in 5 weeks) to prostate and seminal vesicles.

11 ity and disconnection between the testes and seminal vesicles.

12 argin) to the prostate, and proximal 1 cm of seminal vesicles.

13 ses of the rat urogenital sinus and neonatal seminal vesicles.

14 omote prostate growth and antagonizes DHT in seminal vesicles.

15 eoplasia as well as hyperplasia/neoplasia in seminal vesicles.

16 dependent growth of the ventral prostate and seminal vesicles.

17 rone and precocious maturation of testis and seminal vesicles.

18 phogenesis defects in the prostate gland and seminal vesicles.

19 t ductal outgrowth in the prostate gland and seminal vesicles.

20 phogenesis and this was absent in svs mutant seminal vesicles.

21 oids while having no effect on the uterus or seminal vesicles.

22 ulting in an absence of mature sperms in the seminal vesicles.

23 ulbocavernosus (BC) muscle, scent gland, and seminal vesicles.

24 e fertility, accompanied by hypogonadism and seminal vesicle agenesis/hypodysplasia.

25 e weights) were observed in the prostate and seminal vesicles, along with minimal repression of circu

26 Therefore, we propose OSVIRA (Obstructed Seminal Vesicle and Ipsilateral Renal Agenesis) as an ac

27 ificity were low signal intensity within the seminal vesicle and lack of preservation of seminal vesi

28 on of Kgf mRNA during development of the rat seminal vesicle and prostate, both in vitro and in vivo.

29 mimic androgen action in explant cultures of seminal vesicle and prostate.

30 portant role in the development of the mouse seminal vesicle and rat ventral prostate.

31 tor of epithelial growth in the prostate and seminal vesicle and that the FGF10 gene is not regulated

32 or seminal actin-binding protein (SABP) from seminal vesicles and as extraparotid glycoprotein (EP-GP

33 rin was expressed in epithelial cells of the seminal vesicles and ejaculatory ducts.

34 The reverse is true in the seminal vesicles and fetal liver.

35 Furthermore, the involvement of seminal vesicles and other extracapsular extension were

36 Cultures from aorta, vas deferens, seminal vesicle, and kidney tissue were characterized wi

37 ymphocytic infiltration into the epididymis, seminal vesicle, and prostate gland was evident.

38 thelia of the adult mouse stomach, prostate, seminal vesicle, and the developing choroid plexus by in

39 dentified in amyloid deposits in the cornea, seminal vesicles, and brain.

40 ts; bilateral occlusion of the vas deferens, seminal vesicles, and ejaculatory ducts by calcification

41 l epithelial cells of human salivary glands, seminal vesicles, and the collecting tubules of the kidn

42 uantity of sperm released from the testes to seminal vesicles, and these tissues displayed rhythmic a

43 ntly lower, but not the weights of prostate, seminal vesicles, and uterus.

44 seminal vesicle and lack of preservation of seminal vesicle architecture.

45 The seminal vesicles are paired organs of the male reproduct

46 For example, the testes and seminal vesicles are relatively large in species with hi

47 Although congenital anomalies of seminal vesicles are usually asymptomatic, they may lead

48 roductive system, including the prostate and seminal vesicles, are derived from epithelial precursors

49 ls of the testis, vas deferens, prostate, or seminal vesicles) as a most likely source of the sexuall

50 ally all males exhibited enormously enlarged seminal vesicles because of pronounced hyperplasia of th

51 in uterus, lung, pancreas, salivary glands, seminal vesicles, bone marrow cells, and cecum, where it

52 and inhibition of ERK1/2 activation blocked seminal vesicle branching morphogenesis.

53 other mutations that reduce prostatic and/or seminal vesicle branching, the svs mutation dramatically

54 absent or atrophic, including the prostate, seminal vesicle, bulbourethral gland, and caudal ductus

55 gulated by androgen in both the prostate and seminal vesicles but not in other organs.

56 he development of prostate and possibly also seminal vesicle cancer.

57 Signal intensity in the peripheral zone and seminal vesicles decreased on T2-weighted images in 42 (

58 omized males without affecting the uterus or seminal vesicles, demonstrating that the classical genot

59 the BMP family, Gdf7, is required for normal seminal vesicle development.

60 ans in cases of microscopic transcapsular or seminal vesicle disease.

61 istal male reproductive tract (vas deferens, seminal vesicles, ejaculatory ducts).

62 ctivation also resulted in transformation of seminal vesicle epithelial cells in Pten-null mice.

63 from autologous benign prostatic epithelium, seminal vesicle epithelium, or fibroblasts.

64 ashion to control the differentiation of the seminal vesicle epithelium.

65 rowth, branching, and differentiation of the seminal vesicle epithelium.

66 Proteins expressed from the seminal vesicles evolve more rapidly than those from oth

67 bryos were transferred to females mated with seminal vesicle-excised males.

68 mi showed a reduction of spermatozoa and the seminal vesicles exhibited a dramatic reduction of semin

69 of prostatic glands, ejaculatory ducts, and seminal vesicles expressed E-cadherin but not N-cadherin

70 enetically marked Adh1 additionally promotes seminal vesicle expression suggesting downstream or intr

71 reoperative PSA, Gleason sum, stage, margin, seminal vesicle, extra-prostatic extension (EPE), HA, HY

72 -2 ligands in the seminal vesicle, we probed seminal vesicle fluid with 125I-labeled LRP-2 in a gel-b

73 h divergence in major protein composition of seminal vesicle fluid, suggesting that changes in gene e

74 nd to the prostasin-binding protein in mouse seminal vesicle fluid.

75 ly linked 82-kDa complex when incubated with seminal vesicle fluid.

76 ng protein was identified in mouse and human seminal vesicle fluid.

77 EBRT (45 Gy in 25 fractions) to prostate and seminal vesicles followed by BT prostate boost (110 Gy i

78 es such as the epididymis, vas deferens, and seminal vesicle from a straight Wolffian duct.

79 tion of fructose in human semen, a marker of seminal vesicle function.

80 AGs), which, like the mammalian prostate and seminal vesicles, generate most of the seminal fluid con

81 of seminal fluid by surgical excision of the seminal vesicle gland.

82 n were associated with cancer involvement of seminal vesicles, higher Gleason sum, and a positive RT-

83 ous species, including bigger testes, larger seminal vesicles, higher sperm counts, richer mitochondr

84 (68)Ga-PSMA-11 localized in a lymph node and seminal vesicle in a patient with no abnormal (68)Ga-RM2

85 nd retrovesical in 17 (40%), within retained seminal vesicles in nine (22%), and at anterior or later

86 serum testosterone levels and enlargement of seminal vesicles in SIRT1 heterozygous males.

87 Congenital anomalies of seminal vesicles include agenesis, hypoplasia, duplicati

88 tection of extracapsular extension (ECE) and seminal vesicle infiltration (SVI) in patients with pros

89 100 [75%] vs 84 [63%]; P = .01), but not for seminal vesicle invasion (122 [91%] vs 115 [85%]; P = .0

90 ess (HR, 1.7; 95% CI, 1.2-2.2; P =.001), and seminal vesicle invasion (HR, 1.4; 95% CI, 1.1-1.9; P =.

91 0001), positive surgical margins (P = .028), seminal vesicle invasion (P < .0001), lymph node involve

92 capsular extension (P <.01), and presence of seminal vesicle invasion (P <.01) were independent predi

93 ariate analysis, preoperative PSA (P = .04), seminal vesicle invasion (P = .02), PSA velocity (P < .0

94 extracapsular extension (all P < or = .005), seminal vesicle invasion (P = .07), and biochemical prog

95 /m(2)) had a significantly decreased risk of seminal vesicle invasion (P =.039).

96 ive margins, extracapsular extension, and no seminal vesicle invasion (P =.24).

97 level greater than 10 ng/mL (P: < or =.01), seminal vesicle invasion (P: =.02), prostatectomy Gleaso

98 in non-neoplastic prostates correlated with seminal vesicle invasion (rho = 0.275, P = 0.0169) and i

99 plasmic expression in tumors correlated with seminal vesicle invasion (rho = 0.282, P = 0.0098).

100 ocation, extraprostatic extension (EPE), and seminal vesicle invasion (SVI) of prostate cancer foci w

101 elation of extracapsular extension (ECE) and seminal vesicle invasion (SVI) was evaluated in 445 surg

102 traprostatic extension (EPE), 452 (18%) with seminal vesicle invasion (SVI), 1,434 (58%) with positiv

103 ikelihoods of extracapsular extension (ECE), seminal vesicle invasion (SVI), and adjacent organ invas

104 inimisation algorithm stratifying by risk of seminal vesicle invasion and centre to either the contro

105 xpression in prostate cancer cells decreased seminal vesicle invasion and distant metastases.

106 cantly correlated with human prostate cancer seminal vesicle invasion and lymph node metastasis.

107 athohistological analysis revealed extensive seminal vesicle invasion and necrosis in CF tumours, rec

108 rapy PSA level, surgical margins, PSADT, and seminal vesicle invasion are prognostic variables for a

109 es, and rates of extracapsular extension and seminal vesicle invasion compared with cancers not invol

110 Extraprostatic extension and seminal vesicle invasion were assessed by using five-poi

111 leason score > or = 7, positive margins, and seminal vesicle invasion were associated with significan

112 tic extension, positive surgical margins, or seminal vesicle invasion) were randomly assigned to adju

113 ilaterally, with extracapsular extension, no seminal vesicle invasion, a 2-mm positive margin at the

114 ors such as the prostatectomy Gleason score, seminal vesicle invasion, absolute pre-RT PSA level, and

115 level, primary Gleason grade greater than 3, seminal vesicle invasion, and higher number of removed a

116 ports, the risks of extracapsular extension, seminal vesicle invasion, and lymph node metastasis were

117 c capsular invasion, surgical margin status, seminal vesicle invasion, and lymph node status.

118 iptional signature score was associated with seminal vesicle invasion, androgen-independent progressi

119 rgical margin, extraprostatic extension, and seminal vesicle invasion, as well as lymph node metastas

120 ed, along with 5 clinicopathologic features (seminal vesicle invasion, biopsy Gleason score, extracap

121 ositive margins, extraprostatic extension or seminal vesicle invasion, but interpretation of these an

122 , vascular invasion, lymph node involvement, seminal vesicle invasion, capsular penetration, positive

123 after radical prostatectomy include men with seminal vesicle invasion, Gleason score 8 to 10, extensi

124 ical margin status, extracapsular extension, seminal vesicle invasion, lymph node invasion, and andro

125 se pathologic findings at prostatectomy (ie, seminal vesicle invasion, positive surgical margins, ext

126 extracapsular extension, lymph node status, seminal vesicle invasion, post-radical retropubic prosta

127 ive prostate-specific antigen concentration, seminal vesicle invasion, surgical margin status, extrac

128 Gleason 4 or 5 patterns or extracapsular or seminal vesicle invasion.

129 05), transcapsular tumor spread (P < .0001), seminal vesicle involvement (P = .0012), and tumors of a

130 ), extraprostatic extension (P = 0.003), and seminal vesicle involvement (P = 0.002) at prostatectomy

131 with extracapsular extension (P = 0.044) and seminal vesicle involvement (P = 0.024).

132 ndom permuted blocks were used, with risk of seminal vesicle involvement and radiotherapy-treatment c

133 3aN0M0 prostate cancer, an estimated risk of seminal vesicle involvement less than 30%, prostate-spec

134 67.3%; isolated capsular penetration, 59.6%; seminal vesicle involvement, 79.6%; pelvic lymph node in

135 ed in patients with extracapsular extension, seminal vesicle involvement, higher prostatectomy Gleaso

136 ined disease, isolated capsular penetration, seminal vesicle involvement, or pelvic lymph node involv

137 logic features, such as a positive margin or seminal vesicle involvement, will develop biochemical fa

138 ifferentiated nonprostatic mouse epithelium (seminal vesicle) is sufficient for respecification to pr

139 specified for anatomic locations (prostate, seminal vesicles, local lymph nodes, distant lymph nodes

140 ated males also showed significantly smaller seminal vesicles, lower circulating androgens, and decre

141 The prostate lobes, seminal vesicles, lungs, and periaortic lymph nodes were

142 r urogenital organs, congenital anomalies of seminal vesicles may accompany other urinary or genital

143 d into prostate organoids using neonatal rat seminal vesicle mesenchyme in vitro.

144 examines the mechanism by which PHS from ram seminal vesicle microsomes catalyzes the oxidation of th

145 s, testes, and hormonal profile, and dilated seminal vesicles, midline cyst, or calcifications on TRU

146 Studies with pig prostate and seminal vesicle mitochondrial preparations also revealed

147 PET/CT correctly detected invasion of seminal vesicles (n = 11 of 21 patients; 52%) with 86% a

148 nce of apoptosis as they transition into the seminal vesicle near the end of spermatogenesis, pointin

149 a more locally invasive phenotype and causes seminal vesicle obstruction at high penetrance.

150 t reductions (~40%) of spermatozoa stored in seminal vesicles of males, resulting in decreased egg vi

151 ra (two cases, both with reflux) or into the seminal vesicle (one case); one case was contralateral a

152 or prostatectomy Gleason score of 8 to 10 or seminal vesicle or lymph node involvement.

153 s, 74 Gy in 37 fractions to the prostate and seminal vesicles or the equivalent using hypofractionate

154 n 4+3 tumours might overestimate the risk of seminal-vesicle or lymph-node invasion.

155 lesion, region (prostate, including bed and seminal vesicle, or extraprostatic, including all lymph

156 basal cell layer, stroma, ejaculatory ducts, seminal vesicles, or transitional epithelium.

157 -independent mechanism according to in vitro seminal vesicle organ cultures.

158 ated the development of ventral prostate and seminal vesicle organ rudiments in serum-free organ cult

159 yme activity in epididymis and low levels in seminal vesicle, ovary and uterus compared to other stra

160 sion of 76% for ventral prostate and 64% for seminal vesicle (P < 0.05 for both).

161 signal generated by reaction of purified ram seminal vesicle PGHS with arachidonic acid, suggesting t

162 ent in human tissues and highly expressed in seminal vesicles, pituitary, thyroid, pancreas, renal co

163 in the central tail artery, vasa deferentia, seminal vesicles, prostate, and uterus, with the latter

164 toxicity while inhibiting the development of seminal vesicle/prostate cancers in male rats by >50%.

165 pithelium were secretory proteins, including seminal vesicle protein secretion 2 and 5.

166 c autoantibodies against the human SVS2-like seminal vesicle protein semenogelin.

167 yocardium, adrenal cortex, epithelium of the seminal vesicles, proximal tubules and the collecting du

168 the GP1G gene was also active outside of the seminal vesicle, RNA from a variety of guinea pig tissue

169 immune responses to a prostate autoantigen, seminal vesicle secretory protein 2 (SVS2), which we bel

170 igh similarity to the coding exon of a human seminal vesicle secretory protein gene, semenogelin II.

171 ase gene and comparison with other mammalian seminal vesicle secretory protein genes reveals a common

172 t, the 100-kDa protein was identified as the seminal vesicle secretory protein II (SVS-II), a major c

173 These data suggest that these seminal vesicle secretory proteins may have functional r

174 nea pig codes for three of the four abundant seminal vesicle secretory proteins produced in this spec

175 The mouse seminal vesicle shape (svs) mutation is a spontaneous re

176 In the developing seminal vesicles, sustained activation of ERK1/2 was ass

177 ne is expressed at highest efficiency in the seminal vesicle (SV) from a promoter that contains a can

178 ilbestrol (DES) leads to feminization of the seminal vesicle (SV) in male mice, as illustrated by tis

179 ne production was assessed by measuring host seminal vesicle (SV) weights as an indirect measure over

180 lopment of the rat ventral prostate (VP) and seminal vesicle (SV).

181 Volumes of the prostate and seminal vesicles (SV) were calculated by using whole-vol

182 's AR agonist actions on the levator ani and seminal vesicle target tissues.

183 he capsule and low signal intensity within a seminal vesicle that has lost its normal architecture we

184 sia of the male genital tract, including the seminal vesicle, the vas deferens and the prostate.

185 HIP1R mRNA levels were decreased in seminal vesicle tissue from mice bearing miR-23b/-27b-tr

186 in 12 patients after needle puncture of the seminal vesicle to inject contrast material for radiogra

187 ) and coding region identical to that of the seminal vesicle transcript.

188 1 (11.2%) patients; obstructing cysts of the seminal vesicles, vas deferens, ejaculatory ducts, or pr

189 ush and seminal plasma from the prostate and seminal vesicle was obtained from ZIKV inoculated and sh

190 To identify LRP-2 ligands in the seminal vesicle, we probed seminal vesicle fluid with 12

191 of recipient mice reduced androgen-dependent seminal vesicle weight (8.3 vs 26.7 mg; p < 0.05), but d

192 es of activation, i.e. LH levels, testes and seminal vesicle weights were not altered.

193 eks were much reduced; however, SC-SF-1(-/-) seminal vesicles weights were comparable suggesting inta

194 nd 30 weeks of age, and prostate tissues and seminal vesicles were harvested.

195 inical importance of congenital anomalies of seminal vesicles with images of some rare and previously

196 nhibiting growth of rat ventral prostate and seminal vesicles, without accompanying increases in seru