ライフサイエンスコーパス: sensory cortex

1 ion enhances stimulus coding in the relevant sensory cortex.

2 interactions with the sensory thalamus, not sensory cortex.

3 is stored via synaptic plasticity in primary sensory cortex.

4 ntoparietal regions and travelled to FEF and sensory cortex.

5 ral, somatotopic organization of the sheep's sensory cortex.

6 medial thalamic nucleus axons in the whisker sensory cortex.

7 s affect the responses of neurons throughout sensory cortex.

8 sed principally postnatally, and enriched in sensory cortex.

9 naptic plasticity via ECM removal within the sensory cortex.

10 odel originally formulated to describe early sensory cortex.

11 itutes functional microcircuits in the mouse sensory cortex.

12 hanism for the control of motor decisions by sensory cortex.

13 in supragranular pyramidal cells in primary sensory cortex.

14 motor cortex influence sensory processing in sensory cortex.

15 ivity across neuronal populations in primary sensory cortex.

16 ulations such as those found in layer 2/3 of sensory cortex.

17 tations of sounds are formed at the level of sensory cortex.

18 ough a spatially global recruitment of early sensory cortex.

19 ning and evaluate the impact of attention on sensory cortex.

20 representation, even at the level of primary sensory cortex.

21 ation of specific sensory content in primary sensory cortex.

22 orking memory processes in PFC and posterior sensory cortex.

23 fields, again has more of the appearance of sensory cortex.

24 ctor stimuli were abruptly suppressed beyond sensory cortex.

25 ypical and granular areas of association and sensory cortex.

26 characterized as a higher-order, multimodal sensory cortex.

27 SP and NO signaling pathways in rat somatic sensory cortex.

28 a basic organizational feature of mammalian sensory cortex.

29 nsidered an enteroceptive area within limbic sensory cortex.

30 e phase-locked between primary and secondary sensory cortex.

31 spiking, and superficial layer sinks in the sensory cortex.

32 the structure of population activity in the sensory cortex.

33 e to learning-evoked synaptic changes in the sensory cortex.

34 ed by decreases in correlated variability in sensory cortex.

35 thought to be encoded by neurons outside of sensory cortex.

36 ions of increased thalamic connectivity with sensory cortex.

37 l areas before they become apparent in early sensory cortex.

38 N computations might be a generic feature of sensory cortex.

39 little is known about how stress affects the sensory cortex.

40 ey to determining the response properties in sensory cortex.

41 including abnormal neural signaling in human sensory cortex.

42 lly in the whisker barrel region of parietal sensory cortex.

43 e role for cross-sensory influences in early sensory cortex.

44 nt to generate attractor dynamics in primary sensory cortex.

45 creases in beta band power in prefrontal and sensory cortex.

46 that this information is shared early in the sensory cortex.

47 completely understood, especially outside of sensory cortex.

48 that allows for greater flexibility than in sensory cortex.

49 apping thalamic representations than primary sensory cortex.

50 omputational role of inhibitory cells in the sensory cortex.

51 in the primary motor cortex (-25%; p<0.001), sensory cortex (-15%, p<0.001) and frontal cortex (-12%;

52 ive coding, high-level cortical areas inform sensory cortex about incoming sensory signals, a compari

53 r study is the first to show that postreward sensory cortex activity meets these two key criteria of

54 These results support the idea that early sensory cortex activity reflects perceptual experience,

55 ration and learning; they receive input from sensory cortex and excite deep layer neurons, which cont

56 Although there is no sensory cortex and hence no conscious vision, some corti

57 Furthermore, replay events in the sensory cortex and hippocampus were coordinated to refle

58 ether sensory cholinergic signals target the sensory cortex and how they relate to local functional t

59 o GAP-43 is present in primary and secondary sensory cortex and more so in secondary areas.

60 ed coordinates, broadcast throughout primary sensory cortex and provides strong modulation of signals

61 essing of attended acoustic stimuli in early sensory cortex and reduced processing of distracting inp

62 reveal distinct interactions between primary sensory cortex and rIFC in humans and suggest that synch

63 ular recording to reveal connections between sensory cortex and striatum.

64 n neural spiking activity in primary somatic sensory cortex and the frequency of whisker stimulation.

65 s (SEP) were recorded from the contralateral sensory cortex and the sensory nerve action potential (S

66 effects of dopaminergic neurotransmission in sensory cortex and their possible roles in perception, l

67 encoding is subsequently reinstated in early sensory cortex and what the role of the hippocampus is i

68 or overlapping representations in a primary sensory cortex and whether learning can modulate these r

69 maps obtained from optogenetic activation of sensory cortex and wide-field imaging revealed topograph

70 trinsic properties of FS interneurons in the sensory cortex, and a deficit in the formation of excita

71 c transmission in the hippocampus and in the sensory cortex, and are found at somatodendritic as well

72 less activation of forebrain, inhibition of sensory cortex, and behavioral arrest.

73 rophy, cortical atrophy of primary motor and sensory cortex, and cortical reorganization of the senso

74 ebral cortex, in neuronal development in the sensory cortex, and in reward.

75 ory input can remodel representations in the sensory cortex, and this effect is heavily influenced by

76 s-induced modulation of beta oscillations in sensory cortex, and whole-brain connectivity, showing th

77 Neuronal responses of sensory cortex are highly variable, and this variability

78 Growing evidence indicates that responses in sensory cortex are modulated by factors beyond direct se

79 lus location and identity representations in sensory cortex are modulated by reward learning.

80 One hypothesis is that regions of sensory cortex are reactivated during retrieval of senso

81 Cortical neurons, even in primary sensory cortex, are heterogeneous in their selectivity,

82 and adaptable threat representations in the sensory cortex, arising from experience-based sculpting

83 nt evidence suggests that neurons in primary sensory cortex arrange into competitive groups, represen

84 stimulus-specific patterns of activation in sensory cortex as a result of expectation, but this meth

85 supported by postencoding interactions with sensory cortex associated with reward.

86 m inclusive of the medial geniculate; 3) the sensory cortex barrel field and cell bodies of the ventr

87 f plaques in the vibrissae-receptive primary sensory cortex (barrel cortex), in which the cortical co

88 have revealed traveling waves of activity in sensory cortex, both following sensory stimulation and d

89 cause linear changes in activity patterns in sensory cortex but cause dynamic, nonlinear changes in b

90 ctivity between thalamus and lateral primary sensory cortex but reduced connectivity between thalamus

91 forward projection from primary to secondary sensory cortex, but also a route through specific higher

92 ses, not only in their corresponding primary sensory cortex, but in other primary sensory cortices.

93 rmance depends on the activity of neurons in sensory cortex, but little is known about the brain's ca

94 te and chronic investigations of the sheep's sensory cortex by characterizing its exact position, its

95 ents in the SC similar to those generated in sensory cortex by specific thalamic afferents.

96 or developmental lesions, responsiveness of sensory cortex can be converted from the deprived modali

97 hared variability of neuronal populations in sensory cortex can be largely explained by two factors t

98 ve shown that the firing of neurons in early sensory cortex can be modulated by multisensory interact

99 ical or transcranial magnetic stimulation of sensory cortex can temporarily disrupt these phantom sen

100 s demonstrate that feedforward activation of sensory cortex can underlie attentional priority.

101 d the groundwork for the notion of a modular sensory cortex, canonical cortical circuits and an under

102 oral choice means that even neurons in early sensory cortex carry information about an upcoming decis

103 the patterns of neural activity decoded from sensory cortex change as a function of load, as one woul

104 d past measurements, which show that primary sensory cortex codes the whisking envelope as a motor co

105 prevailing ideas regarding coding schemes in sensory cortex: columnar populations can efficiently enc

106 kness with lower values in primary motor and sensory cortex compared with association cortex.

107 ing in the leg area of the primary motor and sensory cortex compared with controls.

108 reased incidence of SDs in parietal/temporal sensory cortex compared with other regions.

109 al extracellular current in vibrissa primary sensory cortex contained oscillatory components at the s

110 The sensory cortex contains a wide array of neuronal types,

111 nhibition.SIGNIFICANCE STATEMENT The primary sensory cortex contains six distinct layers that interac

112 isrupted synaptic and circuit development in sensory cortex contributes to these deficits.

113 ehavior, assessing how neural populations in sensory cortex cooperate to transmit information.

114 Y, n=9) in the frontal cortex, motor cortex, sensory cortex, corpus callosum, hippocampus, thalamus,

115 and spatial extent of activation of vibrissa sensory cortex critically depend on behavioral context a

116 The neural circuitry within sensory cortex determines its functional properties, and

117 al sensorimotor regions of interest (primary sensory cortex, dorsal and ventral premotor cortex) and

118 on became progressively worse, including the sensory cortex, dorsal striatum and thalamus.

119 These results illustrate that changes in sensory cortex during associative learning extend to the

120 level dependent functional MR imaging of rat sensory cortex during forepaw stimulation.

121 nced hippocampal connectivity to the primary sensory cortex during retrieval of extinguished stimuli

122 re selectivity within specific subregions of sensory cortex (e.g., orientation selectivity in primary

123 For instance, sensory cortex encodes stimuli that fluctuate over few t

124 full complement of mechanistic influences on sensory cortex even as it interacts with more automatic

125 tion about stimulus occurrence is encoded in sensory cortex, evidence from neuronal recordings has no

126 ances states of high, persistent activity in sensory cortex evoked by behaviorally relevant stimuli.

127 reorganization has not yet been found in any sensory cortex except AI of the gerbil.

128 ition but did not modulate corticospinal and sensory cortex excitability or sensorimotor integration.

129 We conclude that the primary sensory cortex exerts a significant influence on both sp

130 In conclusion, the superficial layers of sensory cortex exhibit a high degree of learning-depende

131 Different subtypes of GABAergic neurons in sensory cortex exhibit diverse morphology, histochemical

132 etwork in top-down modulation of activity in sensory cortex, expectation-related activity in several

133 level in humans, local desynchronization in sensory cortex (expressed as time-series entropy) versus

134 th increased activations in the left primary sensory cortex face area due to median nerve stimulation

135 eg area during handgrip and the left primary sensory cortex face area during median nerve stimulation

136 e same set of animals, since most studies of sensory cortex focus on a single sensory modality.

137 e demonstrated the importance of the primary sensory cortex for the detection, discrimination, and aw

138 lators in the spinal cord, motor cortex, and sensory cortex from clinically and neuropathologically d

139 dorsal attention network and ventral visual sensory cortex [frontal-sensory synchrony (FSS)] signifi

140 a different role for spontaneous activity in sensory cortex: gating of sensory inputs.

141 o explains why a transient thalamic input to sensory cortex gives rise to responses with amplitudes i

142 es in connectivity between motor centers and sensory cortex guide subsequent sensorimotor learning.

143 stacle avoidance, lesions of primary whisker sensory cortex had minimal impact.

144 The sensory cortex has been interpreted as coding informatio

145 influences on neuronal responses in primary sensory cortex has been observed previously using severa

146 Recently, the study of sensory cortex has focused on the context-dependent evol

147 Neurons in primary sensory cortex have diverse response properties, whereas

148 ocorticography arrays implanted on motor and sensory cortex, high-frequency power (65-95 Hz) was extr

149 lling these attention-related modulations of sensory cortex, however, are poorly understood.

150 cement and suppression of neural activity in sensory cortex (i.e., top-down modulation).

151 ion biases stimulus representations in early sensory cortex, i.e., whether the integration of prior k

152 In primary sensory cortex, IEG expression can label neurons that ar

153 ssible explanation for these results is that sensory cortex implements attractor dynamics, although t

154 pyramidal neurones of layer II/III of somato-sensory cortex in acutely isolated slices obtained from

155 vides a foundation to understand the role of sensory cortex in combining sensory and cognitive variab

156 ol operates in part by biasing processing in sensory cortex in favor of expected target stimuli.

157 describe the neuroanatomical organization of sensory cortex in four rodents: laboratory Norway rats (

158 elium may be a functional feature of primary sensory cortex in general.

159 noninvasive neural stimulation on the human sensory cortex in inhibiting aversive memory in a labora

160 SDs are inducible preferentially in primary sensory cortex in mice and most likely in humans.

161 cubic millimeter regions of vibrissa primary sensory cortex in mouse.

162 image tissue oxygenation in the rat primary sensory cortex in response to sensory stimulation.

163 and/or disturbed organization of the primary sensory cortex in schizophrenia.

164 ng animal data evince a critical role of the sensory cortex in the long-term storage of aversive cond

165 ces supports the conserved role of the human sensory cortex in the long-term storage of aversive cond

166 ge of research into threat processing in the sensory cortex in the past decade has generated particul

167 e sensory/motor divide by showing a role for sensory cortex in the selection of motor behavior.

168 TEMENT We have used calcium imaging in mouse sensory cortex in vivo to reconstruct the onset of focal

169 e uniquely regulated compared to the primary sensory cortex in ways that render them vulnerable to ca

170 s temporal rate is represented by neurons in sensory cortex, in this issue of Neuron, new evidence fr

171 asticity have been well characterized within sensory cortex, in which the ability of altered sensory

172 ous stimuli may result from neural biases in sensory cortex induced by recent perceptual history.

173 ntracortical recurrent circuits in mammalian sensory cortex influence dynamics of sensory representat

174 Neurons in sensory cortex integrate multiple influences to parse ob

175 expansion of trunk motor cortex and forepaw sensory cortex into the deafferented hindlimb cortex, as

176 eory that cortical recurrent connectivity in sensory cortex is a substrate for sensory memories.

177 Sensory cortex is able to encode a broad range of stimul

178 s on post-perceptual changes (integration in sensory cortex is adult-like, but higher-level decision

179 e maturation of the GABAergic circuit in the sensory cortex is altered during a critical developmenta

180 The mammalian sensory cortex is composed of multiple types of inhibito

181 ctional organization and plasticity of adult sensory cortex is derived from animals housed in standar

182 We show, in mouse and human, that sensory cortex is more susceptible to SDs.

183 cognitive and motivational systems influence sensory cortex is not well understood.

184 population responses and network activity in sensory cortex is not well understood.

185 ontribute to map plasticity and stability in sensory cortex is not well-understood.

186 Sensory cortex is ordered into columns, each tuned to a

187 Although early sensory cortex is organized along dimensions encoded by

188 The primary sensory cortex is positioned at a confluence of bottom-u

189 l variability in the responses of neurons in sensory cortex is shared between pairs of neurons.

190 lore the possibility that a core function of sensory cortex is the generation of an internal simulati

191 e response properties that define columns in sensory cortex is thought to begin early in cortical mat

192 Early sensory cortex is typically investigated in response to

193 ual neurons and local population activity in sensory cortex is unclear.

194 t choice-correlated neural activity in early sensory cortex is unstable across observers and tasks, u

195 ons, including cortical pyramidal neurons in sensory cortex, is characterized by strong attenuation t

196 If sensory prediction error neurons exist in sensory cortex, it is unknown whether they manifest as g

197 Growing evidence suggests sensory cortex itself flexibly encodes value beyond labe

198 ty so that changes could occur in regions of sensory cortex lacking activity.

199 In sensory cortex, layer 4 (the granular layer) is the targ

200 l hyperexcitation in supragranular layers in sensory cortex, likely through reduced inhibition.

201 by an integrated neural system involving the sensory cortex, limbic system, and striatum.

202 Such expectancy-driven modulation of primary sensory cortex may affect perceptions of external events

203 e, the nature of aura symptoms suggests that sensory cortex may be preferentially susceptible.

204 al learning, suggest that changes in primary sensory cortex may mediate learning.

205 ore likely stimulus modality and the primary sensory cortex may participate in the redistribution of

206 havioural task, post-stimulatory activity in sensory cortex may play a functional role in processes s

207 Our results suggest that sensory cortex may play a previously unrecognized role i

208 i, indicating that populations of neurons in sensory cortex may reflect prediction errors (PEs), mism

209 neuronal representation as early as primary sensory cortex mediate the perceptual advantage conferre

210 in IDBs, it remains largely unclear whether sensory cortex modulates IDBs and what the underlying ne

211 Spike trains from sensory cortex neurons can predict trial-to-trial variab

212 Action Potential (APs) patterns of sensory cortex neurons encode a variety of stimulus feat

213 ure selectivity is a fundamental property of sensory cortex neurons, yet the mechanisms underlying it

214 For sensory cortex, neurophysiological and psychophysical fi

215 of NR2A and NR2B (NR2A/B) protein in somatic sensory cortex of adult rats.

216 e analyzed multineuron word distributions in sensory cortex of anesthetized rats and cats, and found

217 iphery may shape the organization of primary sensory cortex of other modalities as well.

218 of neuronal activity in the vibrissa primary sensory cortex of rat, a region that receives intrinsic

219 c integration after transplantation into the sensory cortex of stroke-injured adult rats.

220 In the thalamus and sensory cortex of the anesthetized rats, where the lCBF

221 ation and spontaneous neural activity in the sensory cortex of the awake monkey.

222 e of alterations in neuronal circuits in the sensory cortex of the mouse model of FXS (Fmr1 KO).

223 circuit function have been uncovered in the sensory cortex of the mouse model of FXS (Fmr1 KO).

224 ic resonance imaging (fMRI) studies of early sensory cortex often measure stimulus-driven increases i

225 neuronal oscillations in stimulus-receiving sensory cortex) only accounts for the accuracy time cour

226 Due to the diversity of tuning properties in sensory cortex, only a fraction of neurons are engaged i

227 nocellular visual pathway beginning in early sensory cortex or even subcortically.

228 s drives the communication of predictions to sensory cortex or receives prediction signals from elsew

229 such disturbances are present in any primary sensory cortex other than the auditory.

230 We investigated the effects on motor and sensory cortex parameters after a model-free learning ta

231 Models of learning-dependent sensory cortex plasticity require local activity and rei

232 Neuronal variability in sensory cortex predicts perceptual decisions.

233 ndent functional connectivity within the MDT/sensory cortex/prefrontal cortex network.

234 ggered increases of activity in the auditory sensory cortex prior to the occurrence of an auditory ta

235 e a theory explaining how the speed at which sensory cortex processes incoming information is adjuste

236 Lesions of primary sensory cortex produce impairments in brain function as

237 Neuronal populations in sensory cortex produce variable responses to sensory sti

238 bgranular layers (layers 5 and 6) of primary sensory cortex provide corticofugal output to thalamus a

239 elative impairment of potassium clearance in sensory cortex, providing a potential mechanism for the

240 at D1/D5-mediated dopaminergic modulation in sensory cortex regulates positive recurrent corticoeffer

241 n conscious awareness associated with sleep, sensory cortex remains highly active during the differen

242 onsistent with growing evidence that primary sensory cortex remains plastic into adulthood, and they

243 Neuronal populations in sensory cortex represent time-changing sensory input thr

244 The fine-tuning of circuits in sensory cortex requires sensory experience during an ear

245 bottom-up signals arise from the frontal and sensory cortex, respectively, and different modes of att

246 Psilocybin increased sensory cortex responses during emotional recollection,

247 ation of the body part somatotopy in primary sensory cortex (S1 complex, hereafter S1) [1, 2], and th

248 erior cingulate cortex (ACC) and the primary sensory cortex (S1) in rats with inflammatory pain.

249 However, we show here that, like sensory cortex (S1), mouse M1 also has the canonical cir

250 the mouse compare with those in neighboring sensory cortex (S1).

251 nsic network properties, microstimulation of sensory cortex should evoke activity patterns resembling

252 Neurons in early sensory cortex show weak but systematic correlations wit

253 temporal context-dependent responses in the sensory cortex.SIGNIFICANCE STATEMENT Our perception of

254 ects represent an efficient process by which sensory cortex simultaneously enhances relevant informat

255 Sensory cortex, such as the auditory cortex, is involved

256 um concentrations showed larger increases in sensory cortex, suggesting a mechanism of susceptibility

257 ression of multiple types of gamma rhythm in sensory cortex suggests a mechanistic substrate for comb

258 between perception and the activity in early sensory cortex suggests that consciously perceived posit

259 pharmacological inactivation of rat somatic sensory cortex suppresses peripheral information transmi

260 th glutamatergic inputs from the association sensory cortex (Te3) that drive BLA projection neurons,

261 nclear why there are so many more neurons in sensory cortex than in the sensory periphery.

262 known translaminar inhibitory circuit in the sensory cortex that acts to enhance the feature selectiv

263 sponse to an instruction to attend, areas of sensory cortex that code the attributes of the expected

264 Layer (L)2 is a major output of primary sensory cortex that exhibits very sparse spiking, but th

265 re encoded in layer 2/3 pyramidal neurons of sensory cortex that feed forward.

266 aring them to the functional organization of sensory cortex that is activated by naturalistic stimuli

267 ng accounts for the resting hyperactivity in sensory cortex that leads to hallucinations.

268 al circuit for pursuit to identify a part of sensory cortex that provides instructive signals for mot

269 al modeling and analysis of the mapped mouse sensory cortex that the perfusive efficiency of the netw

270 vity distributed across regions of low-level sensory cortex that univariate methods cannot detect.

271 ow the gamma oscillations are constrained to sensory cortex, that they occur independently in auditor

272 g during sleep.SIGNIFICANCE STATEMENT In the sensory cortex, the balance between excitation and inhib

273 imulus representations undergo plasticity in sensory cortex, thereby automatically capturing attentio

274 put is necessary to maintain organisation in sensory cortex, thereby reopening the question what happ

275 sociated with circuit-specific plasticity in sensory cortex, this switch in LTS cell synaptic inhibit

276 The dynamic interaction of lateral OFC with sensory cortex thus implements computations critical for

277 ning reconfigures neural circuits in primary sensory cortex to "learn" associative attributes of a st

278 s an enhancement of memory processing in the sensory cortex to achieve desired forgetting of recent v

279 plified the intensity percept, demonstrating sensory cortex to be the common gateway both to time and

280 We performed a study of rat somatic sensory cortex to confirm and extend these observations,

281 ex parallels the retinotopic organization of sensory cortex to enable an efficient interface between

282 It spreads beyond sensory cortex to frontoparietal association areas, whic

283 sensorimotor cortical circuit (i.e., primary sensory cortex to primary motor cortex).

284 l circuits that transmit behavioral state to sensory cortex to produce this modulation are unknown.

285 The response of neurons in sensory cortex to repeated stimulus presentations is hig

286 directly impacts the piriform, the olfactory sensory cortex, to mediate social learning.

287 ANCE STATEMENT During postnatal development, sensory cortex undergoes functional refinement, through

288 How is information in sensory cortex used to guide behavior?

289 specificity of endocannabinoid signalling in sensory cortex using whole-cell recordings from layer 2/

290 cal areas, secondary and cross-modal, of the sensory cortex (visual, auditory, and somatosensory), as

291 (visual or auditory) induces LTP within the sensory cortex (visual/auditory, respectively) and can b

292 , we examined neuronal activity in vibrissal sensory cortex, vS1, together with vibrissal motor corte

293 ated with neural codes particularly in early sensory cortex, we have so far no understanding of the n

294 We propose that this regime is relevant for sensory cortex when it extracts complex features from li

295 calcium-binding proteins are upregulated in sensory cortex when thalamocortical afferents arrive.

296 to an adaptive and flexible role for primary sensory cortex, where function is shaped by experience a

297 rocess (preparatory excitability increase in sensory cortex), whereas the effect on RT is explained b

298 ery to static representations in the primary sensory cortex, with downstream regions supporting decis

299 ntion can act directly on neural activity in sensory cortex without involving attentional modulation

300 Extending the analyses beyond sensory cortex yielded some evidence for memory content-