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1 complaint (balance disturbance, headache and sensory loss).
2 espread alterations in neural activity after sensory loss.
3 prove recovery in an animal model of chronic sensory loss.
4  and drinking, and how these are affected by sensory loss.
5 icity related to chronic, persistent partial sensory loss.
6 to upper extremity paresis, paresthesia, and sensory loss.
7      Three individuals had additional distal sensory loss.
8  characterized by distal muscle weakness and sensory loss.
9 tions that are specifically uncovered during sensory loss.
10 e majority of patients are left with lasting sensory loss.
11 of adult primates reactivate over time after sensory loss.
12 onsidered, rather than a model primarily for sensory loss.
13 ce and six times more than the subgroup with sensory loss.
14 ay be restored in individuals suffering from sensory loss.
15 phy, often with foot deformity, weakness and sensory loss.
16  quality of life of the individual with dual sensory loss.
17 em of sound localization in people with dual sensory loss.
18 kle joint pain, tenderness to palpation, and sensory loss.
19 ned, accompanied by hypotonia, and motor and sensory loss.
20 o pain, paralysis, autonomic involvement and sensory loss.
21 at reflects cerebellar motor dysfunction and sensory loss.
22 nerve disorder characterised by weakness and sensory loss.
23 tectomy, nerves are transected, resulting in sensory loss.
24 and after recovering from different types of sensory loss.
25 r axon conduction and thermal and mechanical sensory loss.
26 re characterized by progressive weakness and sensory loss.
27 nteraction terms between these variables and sensory loss.
28 culon homology domain and is associated with sensory loss(3), interacts with FAM134B and participates
29                                  Adults with sensory loss across multiple systems may be an important
30 ssion comparing participants with vs without sensory loss, adjusting for demographic and clinical cov
31 sses of hearing and vision function, or dual sensory loss, affect a large number of individuals of al
32       With the aim of determining how recent sensory loss affects cerebral morphology and functional
33 sory loss and little is known how the recent sensory loss affects the human brain.
34                                    Following sensory loss, alterations in tongue kinematics were acco
35 d apraxia with dystonia, myoclonus, cortical sensory loss and alien limb phenomenon.
36                                        After sensory loss and deafness, the brain's effective connect
37 tentials (CMAPs and SNAPs), distal weakness, sensory loss and decreased reflexes.
38 y and region may modify associations between sensory loss and depression is unknown.
39 effects from either early onset or long-term sensory loss and little is known how the recent sensory
40 ng sensory information to motor cortex, such sensory loss and representational reorganization could a
41                                The region of sensory loss and visual analog pain scale improved in th
42 th-dependent axonal degeneration with distal sensory loss and weakness, deep-tendon-reflex abnormalit
43  distribution of both large- and small-fiber sensory loss and which approaches and techniques may be
44 xtent of the atrophy is greater with greater sensory loss and with longer recovery times.
45 strongly associated with obesity(1), causing sensory loss and, in some patients, neuropathic pain(2,3
46 lated diplopia, dysarthria, vertigo, ataxia, sensory loss, and bilateral visual disturbance), with mu
47 f neurotoxic chemotherapy resulting in pain, sensory loss, and decreased quality of life.
48 akness and atrophy, foot deformities, distal sensory loss, as well as diminished tendon reflexes.
49 e used as a surrogate measure of small-fiber sensory loss but appear not to correlate closely with se
50                                              Sensory loss can thus confer cross-modal plasticity by c
51 s of the frailty index (morbidity, function, sensory loss, cognition and mood, and other) were assess
52                                    Following sensory loss, compensatory crossmodal reorganization occ
53  differences in the association between dual sensory loss (concurrent hearing and vision loss) and de
54 c pain and itching defined as distal thermal sensory loss (confirmed by thermal roller), score of 4 o
55                      As a result, congenital sensory loss could be thought of as a connectome disease
56 es for which this explanation of the partial sensory loss could not be maintained.
57 osensory deficits in OA, since the extent of sensory loss directly correlated with the radiographic s
58                   Hearing loss (HL) and dual sensory loss (DSL) are prevalent, disabling, and associa
59 changes, as well as patterns of weakness and sensory loss due to transverse myelitis or peripheral ne
60                               The pattern of sensory loss following transection of the anterolateral
61 sive neuropathy with distal muscle weakness, sensory loss, gait disturbances, foot deformities, reduc
62                             Sensory loss (no sensory loss, hearing loss only, vision loss only, and d
63 'asleep numbness' in 20, mild pain in 13 and sensory loss in 23.
64 pmental synesthesia and plasticity following sensory loss in acquired synesthesia.
65 ting the potential of artemisinin to prevent sensory loss in CLRN1 (c.144T>G) patients.
66 and weight loss are followed by weakness and sensory loss in the distribution of a single anatomical
67 bacute meningomyeloradiculitis was left with sensory loss in the feet.
68 tosensory function in a rat model of chronic sensory loss in the forelimb.
69                                              Sensory loss induces cross-modal plasticity, often resul
70                             Prominent distal sensory loss involved all modalities, resulting in neuro
71                                              Sensory loss is associated with behavioural changes, but
72 e results demonstrate that recently acquired sensory loss is associated with both changed cerebral mo
73                 These findings indicate that sensory loss is associated with depression in older adul
74                          The area of partial sensory loss is due to transection of the fibres that ar
75 anding cross-modal plasticity in response to sensory loss is essential to maximize patient susceptibi
76                              This pattern of sensory loss is explained as follows.
77                                          The sensory loss is proportional to the food protein additio
78                                     Unimodal sensory loss leads to structural and functional changes
79                                              Sensory loss leads to widespread cross-modal plasticity
80 ypti mosquitoes, despite suffering olfactory sensory loss, maintain the overall effectiveness of thei
81                                         Dual sensory loss may present at any age as a result of genet
82 europsychiatric conditions, vision and other sensory loss, musculoskeletal disorders, and genitourina
83                                              Sensory loss (no sensory loss, hearing loss only, vision
84 primary motor cortex (M1), especially if the sensory loss occurs early in development.
85 portunity to prevent progressive symptoms of sensory loss, pain, autonomic dysfunction, ulcerations,
86 e reactivation of somatosensory cortex after sensory loss produced by spinal cord lesions in the comm
87                          The TRPA1-dependent sensory loss produced by STZ occurs before the onset of
88 f differences in species, type and extent of sensory loss, recovery time after injury, and age at the
89 leading to progressive gait and limb ataxia, sensory loss, reduced tendon reflexes, dysarthria, absen
90 n loss) and depression may highlight gaps in sensory loss research and health care services, and by s
91 t the functional reorganization that follows sensory loss results from changes in synaptic strength a
92 stations of the neuropathic state-allodynia, sensory loss, shooting pains, etc, that can manifest lon
93 hin nondeprived sensory areas as a result of sensory loss.SIGNIFICANCE STATEMENT Early blindness has
94 esia [TH], mechanical hyperalgesia [MH], and sensory loss [SL]) at every time point was based on QST
95 esia [TH], mechanical hyperalgesia [MH], and sensory loss [SL]) identified through quantitative senso
96 professionals can meet the challenge of dual sensory loss that awaits us with the aging of the popula
97 rd injury (SCI) leads to permanent motor and sensory loss that is exacerbated by intraspinal inflamma
98 d autonomic neuropathy type 1 (HSAN1) causes sensory loss that predominantly affects the lower limbs,
99 imilar, including distal muscle weakness and sensory loss, their molecular pathogenesis is likely to
100 rom these studies of the cortical effects of sensory loss to evaluate the histological consequences i
101 atic, neurological examination showed distal sensory loss to pain, or vibration or distal loss of ref
102 ual variability in the brain's adaptation to sensory loss underpinning much of the observed variation
103 showed that the odds of depression with dual sensory loss (vs no loss) was higher in urban (OR, 3.16;
104                             Prominent distal sensory loss was a consistent feature in one family, res
105 earing loss only, vision loss only, and dual sensory loss) was determined by respondents' self-report
106 oa mutation as a mouse model for mixed motor-sensory loss when the entire neuraxis is considered, rat
107 d at a later age, we find these mice develop sensory loss with a distal small fiber neuropathy and pe
108 ave slowly progressive weakness, wasting and sensory loss, with an axonal neuropathy typical of CMT2,
109         Blindness is a common cause of major sensory loss, with an estimated 39 million people worldw

 
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