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1 tivating TRPA1, an excitatory ion channel on sensory nerve endings.
2 activating capsaicin-sensitive perivascular sensory nerve endings.
3 tussive mediators and activation of afferent sensory nerve endings.
4 e-1 (COX-1) or COX-2 produce hyperalgesia in sensory nerve endings.
5 detector of environmental cold in mammalian sensory nerve endings.
6 activating specific (vanilloid) receptors on sensory nerve endings.
7 Capsaicin is a specific activator of sensory nerve endings.
8 s and modulate their sensitivity directly in sensory nerve endings.
9 robiome and by its unusually high density of sensory nerve endings.
10 ng at CB1 cannabinoid receptors localized on sensory nerve endings.
11 ds, arrector pili muscles, Merkel cells, and sensory nerve endings.
12 been reported to be released from cutaneous sensory nerve endings after hapten application, we deter
14 mily of ion channels, which are expressed in sensory nerve endings and in skin, respond to distinct t
15 estored the branching of diabetes-suppressed sensory nerve endings and regeneration in the diabetic c
16 there is increased pulpal NGF, sprouting of sensory nerve endings, and increased immunoreactivity fo
17 In knockout mice lacking synapsin I and II, sensory nerve endings are normally developed but not sti
21 TRPM3, and indicate that TRPM3 activation in sensory nerve endings can contribute to neurogenic infla
22 alpha-motor axons in SOD1(G93A) mice, Ia/II sensory nerve endings degenerate in the absence of obvio
23 ent with capsaicin, which depletes SP in the sensory nerve endings, eliminates stress-control differe
24 nates following the activation of peripheral sensory nerve endings following damage or exposure to in
27 gnaling regulates density and maintenance of sensory nerve endings in the skin and may have important
28 ation results from the excitation of primary sensory nerve endings in the skin, but the underlying mo
29 strate that BC signaling stimulates adjacent sensory nerve endings in the trachea to release the neur
32 suggest that endogenous CGRP concentrated in sensory nerve endings may regulate locally the immune re
33 el, DRASIC, in several different specialized sensory nerve endings of skin, suggesting it might parti
35 dermal epidermal junction next to peripheral sensory nerve endings, suggesting that viral reactivatio
36 A raises blood pressure by stimulating renal sensory nerve endings that contain synapsin-positive mic
37 tivates TRPM8, a cation channel expressed in sensory nerve endings that serves as the primary cold se
38 hnique to selectively label spinal afferent (sensory) nerve endings that innervate the periosteum and
39 hiocyanate) to the skin activates underlying sensory nerve endings, thereby producing pain, inflammat
40 ent strategies that target TRPA1 channels on sensory nerve endings to achieve chemical deterrence.
42 nociceptive effect starts after entering the sensory nerve endings, where these agents are axonally t
43 , lipids, opioids, and ions excite/sensitize sensory nerve endings, which not only induces itch but f
44 another group, we blocked 5-HT3 receptors on sensory nerve endings with tropisetron (300 microg kg(-1