ライフサイエンスコーパス: septic arthritis

1 tilage from damage in a murine model of knee septic arthritis.

2 nhanced eradication of causative bacteria in septic arthritis.

3 nts a promising new therapeutic strategy for septic arthritis.

4 idural abscess, vertebral osteomyelitis, and septic arthritis.

5 is, and the emm type 1.0 strains which cause septic arthritis.

6 mote chronic inflammatory conditions such as septic arthritis.

7 icile infection occasionally misdiagnosed as septic arthritis.

8 sponse and host defense in a murine model of septic arthritis.

9 s infective endocarditis, osteomyelitis, and septic arthritis.

10 by 10 days of oral therapy is sufficient for septic arthritis.

11 hort-term clinical outcomes in patients with septic arthritis.

12 s in murine models of systemic infection and septic arthritis.

13 for the diagnosis and treatment of bacterial septic arthritis.

14 is and management of suspected and confirmed septic arthritis.

15 ynovial fluid laboratory data for diagnosing septic arthritis.

16 eral, monoarticular arthritis who might have septic arthritis.

17 95% CI, 15%-24%) are less common findings in septic arthritis.

18 on significantly increase the probability of septic arthritis.

19 inflammatory diseases such as rheumatoid and septic arthritis.

20 ons, including endocarditis, bacteremia, and septic arthritis.

21 al method to increase the yield in suspected septic arthritis.

22 g complications: osteomyelitis (6 patients), septic arthritis (1 patient), infective endocarditis (4

23 01), black (100% vs 53%; P < .001), and have septic arthritis (35% vs 1%; P < .001).

24 Hi infection from 2008-2016 (n = 24) to have septic arthritis (35% vs 4%, respectively; P = .01).

25 ird of cases, including endocarditis (~12%), septic arthritis (7%), vertebral osteomyelitis (~4%), sp

26 ins were identified and were associated with septic arthritis among black men who have sex with men a

27 cuss various risk factors for development of septic arthritis and examine host factors (tumour necros

28 The clinical course of septic arthritis and histopathological and radiological

29 ds to infective endocarditis, osteomyelitis, septic arthritis and metastatic abscess formation.

30 , and of joint and bone infections including septic arthritis and osteomyelitis (acute, subacute, and

31 Early diagnosis and prompt treatment of septic arthritis and osteomyelitis are essential.

32 A case of Bordetella petrii septic arthritis and osteomyelitis in an elbow resulted

33 being recognized increasingly as a cause of septic arthritis and osteomyelitis in young children.

34 mens can optimize the treatment of pediatric septic arthritis and osteomyelitis.

35 the most up to date literature on pediatric septic arthritis and osteomyelitis.

36 e respiratory tract and is a common cause of septic arthritis and osteomyelitis.

37 f a variety of pediatric diseases, including septic arthritis and osteomyelitis.

38 results in a reduced capacity to cause both septic arthritis and osteomyelitis.

39 e of a man with Erysipelothrix rhusiopathiae septic arthritis and possible infective endocarditis.

40 A case of septic arthritis and possible osteomyelitis as sequelae

41 oppler sonograms does not allow exclusion of septic arthritis and should not preclude aspiration when

42 nia in 27 (18%), meningitis in 22 (15%), and septic arthritis and/or osteomyelitis in 3 (2%).

43 pital readmission relating to pneumonia, one septic arthritis, and one suspected venous thrombosis),

44 s (infective endocarditis, epidural abscess, septic arthritis, and osteomyelitis), the mean age was 4

45 ia or sepsis, endocarditis, osteomyelitis or septic arthritis, and skin or soft tissue infection.

46 more likely to develop meningoencephalitis, septic arthritis, and spinal infection.

47 Symptoms and signs of septic arthritis are an important medical emergency, wit

48 The short-term and long-term consequences of septic arthritis are uncertain.

49 In 10 195 (84%) patients with septic arthritis as the primary admitting diagnosis, 90-

50 sis are required to assess the likelihood of septic arthritis before the Gram stain and culture test

51 lternative to surgical incision/drainage for septic arthritis, but this practice has not been widely

52 -binding function of CNA in a mouse model of septic arthritis by comparing the virulence of isogenic

53 oint, prompt identification and treatment of septic arthritis can substantially reduce morbidity and

54 ptic arthritis of the lumbar facet joint and septic arthritis caused by direct inoculation of bacteri

55 mes of 28 reported cases of osteomyelitis or septic arthritis caused by Scedosporium species in immun

56 n the English-language medical literature of septic arthritis caused by this organism.

57 Septic arthritis caused by uncommon microorganisms and s

58 iated with polyarthralgia and renal failure, septic arthritis, classic erysipeloid, and peritonitis.

59 receiving CTLA4-Ig treatment had more-severe septic arthritis, compared with controls and mice receiv

60 Secondary septic arthritis diagnosis versus primary diagnosis was

61 tients with a primary admitting diagnosis of septic arthritis (ICD-10) was compared with that in pati

62 s who received arthroscopic knee washout for septic arthritis in England between April 1, 1997, and M

63 s aureus is the most common pathogen causing septic arthritis in humans.

64 hritis caused by uncommon microorganisms and septic arthritis in immunocompromised hosts are other no

65 e isolated from cases of bovine mastitis and septic arthritis in lambs.

66 ly increased the susceptibility to S. aureus septic arthritis in mice, whereas anti-TNF therapy deter

67 Mycoplasma arthritidis causes a severe septic arthritis in rats under natural and experimental

68 These include septic arthritis in rheumatoid arthritis, rheumatic mani

69 uced in DBA/1 mice and Staphylococcus aureus septic arthritis in Swiss mice.

70 cing articular inflammation, particularly in septic arthritis, in which antiinflammatory effects may

71 nal intensity, and contrast enhancement) and septic arthritis (indicated by synovial enhancement and

72 Septic arthritis is a chronic inflammatory disorder caus

73 Septic arthritis is a severe and rapidly debilitating di

74 Research using experimental murine models of septic arthritis is also generating novel immunotherapeu

75 ase of a control patient with staphylococcal septic arthritis, it is not clear from the present study

76 e report herein the draft genome sequence of septic arthritis K. kingae strain PYKK081.

77 , vertebral infections, transient synovitis, septic arthritis, Legg-Calve-Perthes disease, lower extr

78 The septic arthritis literature of 2000 revisited several to

79 t in vivo mouse models of sepsis and a novel septic arthritis model, we found that the amount and act

80 2), cellulitis (n = 2), peritonitis (n = 1), septic arthritis (n = 1), otitis media (n = 10), and sin

81 ntations including pneumonia (n = 15 [12%]), septic arthritis (n = 9 [7%]), and epiglottitis/supraglo

82 Native joint septic arthritis (NJSA) is definitively diagnosed by a p

83 Native joint septic arthritis (NJSA) is poorly studied.

84 depict increased flow in most patients with septic arthritis, normal flow on power Doppler sonograms

85 Candida septic arthritis occurred concomitantly in 21%.

86 We report a case of septic arthritis of a native knee joint due to Corynebac

87 We review the changing epidemiology of septic arthritis of native joints in adults, encompassin

88 terium striatum, a rare and unusual cause of septic arthritis of native joints.

89 The diagnoses of septic arthritis of the lumbar facet joint and septic ar

90 rtality and adverse joint outcomes following septic arthritis of the native knee.

91 Evidence of septic arthritis on MR images was present in 53 (33%) fe

92 th advanced pedal infection show evidence of septic arthritis on MR images.

93 es [26; discitis (10), osteomyelitis (nine), septic arthritis (one), cellulitis (six)], vascular syst

94 Of 11 patients with septic arthritis, one had asymmetric increased flow, and

95 The diagnosis, treatment, and monitoring of septic arthritis or osteomyelitis in children has become

96 is series includes 13 cases of F. tularensis septic arthritis or osteomyelitis in the United States d

97 alitis, cellulitis or soft tissue infection, septic arthritis or osteomyelitis, and endocarditis duri

98 docarditis, osteomyelitis, epidural abscess, septic arthritis, or bloodstream infection (ie, SIRI) be

99 sis/bacteremia, endocarditis, osteomyelitis, septic arthritis, or epidural/brain abscess).

100 or infective endocarditis, epidural abscess, septic arthritis, or osteomyelitis.

101 zed increasingly as an important etiology of septic arthritis, osteomyelitis, and bacteremia, especia

102 enous implant-related infection comprised of septic arthritis, osteomyelitis, and biofilm formation o

103 In the case of septic arthritis, permanent destruction of articular car

104 ients with invasive extraurinary infections (septic arthritis/pyomyositis, nontraumatic meningitis/he

105 Ultimately, the diagnosis of septic arthritis rests on the opinion of a clinician exp

106 iously healthy child whose osteomyelitis and septic arthritis resulted in unusually extensive photope

107 conducted using a murine model of S. aureus septic arthritis revealed that, in contrast to an agr mu

108 gery for trauma (RR 1.9 [1.4-2.6]), previous septic arthritis (RR 4.9 [2.7-7.6]) or inflammatory arth

109 Septic arthritis (SA) is a rheumatologic emergency assoc

110 ts with rheumatoid arthritis (RA) developing septic arthritis (SA).

111 3 met the gold standard for the diagnosis of septic arthritis, satisfied all inclusion criteria.

112 ic retinoid derivative, in a mouse model for septic arthritis shows significant reduction of proinfla

113 rsion, pulmonary embolism, pyloric stenosis, septic arthritis, sinus venous thrombosis, slipped capit

114 ting such imbalance during S. aureus-induced septic arthritis still requires detailed investigation.

115 ysipelas, cellulitis, pneumonia, bacteremia, septic arthritis, streptococcal toxic shock syndrome, an

116 milar to more significant disorders, such as septic arthritis, the diagnosis should remain one of exc

117 bacteremia, endocarditis, osteomyelitis, and septic arthritis typically require prolonged intravenous

118 itis (OM); V, highly suggestive of OM and/or septic arthritis; VI, known OM; and NOS (not otherwise s

119 ) was compared with that in patients in whom septic arthritis was a secondary diagnosis.

120 system/spine infections, osteomyelitis, and septic arthritis were labeled as IDRIs if discharge code

121 ling activates S. aureus infection-triggered septic arthritis, which results in inflammation of the s

122 onuclear cell count of at least 90% suggests septic arthritis with an LR of 3.4 (95% CI, 2.8-4.2), wh