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1 on leading to protein isoforms with internal sequence deletions.
2 point mutations in topA increase the rate of sequence deletion and duplication events.
3 h p16 silencing in the absence of p16 coding sequence deletion and MAX induction restores p16 express
4 he A genome include retroelement insertions, sequence deletions, and mutations causing in-frame stop
5           Furthermore, we identified a large sequence deletion ( approximately 0.58 kb) in the C-term
6  that transfected RNAs harboring 5'-terminal sequence deletions are able to direct the synthesis of v
7                          PolyA simple repeat sequence deletions are common in tumors with microsatell
8 of the break points associated with all four sequenced deletions are consistent with this model.
9 ated error-prone end-joining and restricting sequence deletion at the break junction during repair.
10                               53BP1 supports sequence deletion during MMEJ consistent with a putative
11 d deletions in terms of the size and rate of sequence deletion, evolutionary factors can reconcile th
12                                              Sequence deletions, histochemical B-glucuronidase (GUS)
13 cially in the fruit pedicel through a 579-bp sequence deletion in its 5' upstream region, resulting i
14                                   Additional sequence deletion in order to shorten the internal stand
15 relia, mating type O is determined by coding-sequence deletions in a different gene, mtB, which is sp
16 siological effects of in-frame actin-binding sequence deletions in the context of a highly functional
17  for known coreceptor polymorphisms or large sequence deletions in the nef gene associated with delay
18    Experiments with antibody competition and sequence deletion indicated that His657-Lys676 of FAD-AP
19                                   The signal sequence deletion mutant, phoZDeltass, was used to ident
20  nucleus, as the nuclear localization signal sequence-deletion mutant of MDM2, which localized in the
21 p1-binding site in HEG1 contains a 9 residue sequence, deletion of which abrogates HEG1's ability to
22 ty arises in the LMP-1 gene: point mutation, sequence deletion or duplication, and homologous recombi
23  deletions or duplications within the coding sequence, deletion or duplication frameshifts, insertion
24 ever, we did identify the 21-bp MEF2A coding sequence deletion originally implicated in adCAD1 in 1 o
25                                        Small sequence deletions resulted in a novel class of heteroch
26 However, deviations from the model for short sequence deletions suggest that RAM contains sequence-sp
27  constructed a series of point mutations and sequence deletions throughout Cdc19p, which allow us to
28 T domain, as a G18A/G20A double mutant and a sequence deletion within the conserved motif impaired Ad