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1 vealed at least 13 well segregated variants (serovars).
2  genetically similar nontyphoidal Salmonella serovars.
3 identification of closely related Salmonella serovars.
4 y both nontyphoidal and typhoidal Salmonella serovars.
5 uld likely protect against only one or a few serovars.
6  the antigenic basis of the various Listeria serovars.
7 fertility caused by different C. trachomatis serovars.
8 more robust against the most closely related serovars.
9 and a PCR was formulated that differentiated serovar 16 isolates from all 15 known serovars and other
10 used whole-genome sequencing of the proposed serovar 16 reference strain A-85/14 to confirm the prese
11  existence of a sixteenth serovar-designated serovar 16-of A. pleuropneumoniae.
12 t forward that they comprised a new serovar, serovar 16.
13 re, we demonstrate that in a highly virulent serovar 4b strain, two genes gtlB and gttB are responsib
14 nd host interaction, a capsule mutant of the serovar 5 strain HS069 was generated (HS069Deltacap) thr
15 fied between serovars, with the exception of serovars 5 and 12.
16        Because 87% of NTS belonged to only 4 serovars, a multivalent vaccine may be an effective stra
17             Enteritidis, the most common NTS serovar, accounted for 38.5% of all NTS isolates (n = 25
18 serovar Typhimurium was the most predominant serovar, accounting for 41.8% (188/450) of NTS isolates.
19  Control and Prevention (CDC), several other serovars also contribute to clinical cases of salmonello
20 ghly lethal challenge dose of the homologous serovar and determined protection against other group B
21 e of the software in querying for a pathogen serovar and for genome sequence identifiers.
22 tiated serovar 16 isolates from all 15 known serovars and other common respiratory pathogenic/commens
23 ive agent of leptospirosis includes multiple serovars and species of pathogenic leptospires that are
24 icity between two highly abundant Salmonella serovars and suggest that native variation in the expres
25 s is found sporadically throughout different serovars, and several inhibit activation of the innate i
26  demonstrate that a simple pathogen-defining serovar antigen, that mediates bacteriophage susceptibil
27 ritidis, and 3% (3) were Salmonella enterica serovar Arizonae.
28 asmid of Salmonella enterica subsp. enterica serovar Bareilly and Escherichia coli O157:H7.
29 ighly conserved across different S. enterica serovars, but residue 161, located close to the catalyti
30           Isolates are characterized into 15 serovars by their capsular polysaccharide, which has sho
31                                         Both serovars can adhere to and invade M cells and enterocyte
32             Only a small group of Salmonella serovars cause this systemic infection, known as invasiv
33 mined protection against other group B and D serovars circulating in sub-Saharan Africa.
34 lyses were restricted to cases attributed to serovars commonly isolated from wildlife/environment (e.
35  isolation and propagation of L. interrogans serovar Copenhageni strain IC:20:001 in semi-solid media
36  of C3H/HeJ mice with Leptospira interrogans serovar Copenhageni using an enzootic mode of transmissi
37 be applied to humans, we used C. trachomatis serovar D (strain UW-3/Cx) to induce infertility in mice
38 osal and systemic routes with C. trachomatis serovar D (UW-3/Cx) rMOMP and challenged in the ovarian
39 oculations of rhesus macaques with wild-type serovar D strain D/UW-3/Cx or a plasmid-deficient deriva
40  mice were protected against challenges with serovars D (UW-3/Cx), D (UCI-96/Cx), and E (IOL-43) but
41 OMP and challenged in the ovarian bursa with serovars D (UW-3/Cx), D (UCI-96/Cx), E (IOL-43), or F (N
42 he conserved LNPTIAG epitope and neutralized serovars D, E, and F.
43 and their surrounding constant segments from serovars D, E, and F.
44 s of the distribution of Salmonella enterica serovar Derby (S.
45 gical data show the existence of a sixteenth serovar-designated serovar 16-of A. pleuropneumoniae.
46       S. Typhimurium was the only Salmonella serovar detected in L. olivacea, and phylogenetic analys
47 gnificant variation and do not contribute to serovar determination.
48 iming to better understand the epidemiology, serovar distribution, antimicrobial resistance (AMR), an
49 ful epidemiological tool to characterize the serovar diversity and AMR profiles in NTS.
50 inst the group D serovar Salmonella enterica serovar Dublin (85% vaccine efficacy).
51 were decidualised in-vitro, infected with Ct serovar E, and changes in expression of genes of interes
52 omparative genomic analysis reveals that all serovars encode a subset of "core" effectors, suggesting
53 ovar Typhimurium SpvD having an arginine and serovar Enteritidis a glycine at this position.
54 es of S. Typhimurium and Salmonella enterica serovar Enteritidis DeltaguaBA DeltaclpX live oral vacci
55                          Salmonella enterica serovar Enteritidis is a common cause of foodborne illne
56                          Salmonella enterica serovar Enteritidis is a major cause of foodborne diseas
57  chick colonization with Salmonella enterica serovar Enteritidis requires a virulence-factor-dependen
58     Overall this work shows that S. enterica serovar Enteritidis strains circulating in Uruguay have
59 phimurium, 10% (10) were Salmonella enterica serovar Enteritidis, and 3% (3) were Salmonella enterica
60 ogical paradox surrounds Salmonella enterica serovar Enteritidis.
61  to MLST sequence type 11 the major ST among serovar Enteritidis.
62                          Salmonella enterica serovars Enteritidis and Kentucky differ greatly in epid
63                         Although S. enterica serovars Enteritidis and Typhimurium are responsible for
64 stinguished between all previously described serovars except 5 and 12, which were detected by the sam
65 were protected but not those challenged with serovar F (N.I.1) from a different subcomplex.
66 isolation of Salmonella of a wide variety of serovars, from an array of animal feeds, food animals, a
67 LH supported the growth of L. borgpetersenii serovar Hardjo strain HB15B203 at 29 degrees C.
68 ne serovar provides protection against other serovars has not been well studied.
69 sults of 2 patients, each infected only with serovar Ia strains, revealed multiple same-serovar infec
70 nce of international lineages of S. enterica serovars in food production chain is supported by conser
71 o investigate the distribution of Salmonella serovars in MCL and their products, a total of 1287 pre-
72  Most international lineages belonging to 28 serovars, including, S. enterica serovars S.
73                          Salmonella enterica serovar Infantis (S.
74 increase (95% CI: 20%, 49%) in environmental serovar infection.
75 h serovar Ia strains, revealed multiple same-serovar infections over 1-5 years.
76             The age distribution and limited serovars involved make control of NTS disease by vaccine
77 rovide protection against another Salmonella serovar is determined by the accessibility of shared O A
78    The host range for YSD1 across Salmonella serovars is broad, but not comprehensive, being limited
79 175) and compared with strains from the same serovars isolated from human clinical cases, livestock,
80                                              Serovar J strains isolated from 1 patient 3 years apart
81                        Chlamydia trachomatis serovar L2 and Chlamydia muridarum, which do not express
82 nella at the genus, species, subspecies, and serovar levels of specificity.
83 stics and genomes of 10 atypical S. enterica serovars linked to multistate foodborne outbreaks in the
84 lmonella strains were evaluated as potential serovar markers.
85                                     Four NTS serovars (Mbandaka, Bredeney, Infantis and Virchow) were
86 show that CBA120 infects Salmonella enterica serovar Minnesota, and this host range expansion is like
87 , Gram-negative bacteria Salmonella enterica serovar Montevideo.
88 charide O-antigen of Yersinia enterocolitica serovars O:5/O:5,27.
89 ) in attenuating infectivity across multiple serovars of C. trachomatis without host cell toxicity.
90 primary isolation of two diverse species and serovars of pathogenic leptospires directly from host ki
91 sing standard microbiological techniques and serovars of S. enterica were determined by PCR and/or ag
92                                              Serovars of Salmonella enterica cause both gastrointesti
93                                    Two major serovars of Salmonella enterica, Typhi and Typhimurium,
94 onses in poultry to infections with distinct serovars of Salmonella enterica.
95 resistance in 135 typhoidal and nontyphoidal serovars of Salmonella.
96           Infantis) is one of the ubiquitous serovars of the bacterial pathogen S. enterica and recen
97 ultures) mice challenged with C. trachomatis serovars of the same complex were protected but not thos
98 ca serovar Typhi, whereas 86% (131/152) were serovars other than Typhi (nontyphoidal Salmonella).
99 uals were shown to be infected with a single serovar over a lengthy period.
100 e fatality (27.8%) was higher than for other serovars (P = .0009).
101  a focused minireview on Salmonella enterica serovar Panama, a serovar responsible for invasive salmo
102 human challenge model of Salmonella enterica serovar Paratyphi A infection.
103                          Salmonella enterica serovar Paratyphi A is a human-specific serovar that, to
104 i or Salmonella enterica subspecies enterica serovar Paratyphi A or C were only isolated in 14 (0.03%
105 by infections with drug-resistant S enterica serovar Paratyphi A.
106 and the expression of SPI-1 in the typhoidal serovarS Paratyphi A compared to that of the nontyphoida
107 e chronic shedding of Leptospira interrogans serovar Pomona in California sea lions (Zalophus califor
108  were identified, with three dogs having two serovars present.
109                        Pathogenic Salmonella serovars produce clinical manifestations ranging from sy
110 , whether infection or immunization with one serovar provides protection against other serovars has n
111 abolically with most, if not all, Salmonella serovars, representing a novel approach to control of th
112 iew on Salmonella enterica serovar Panama, a serovar responsible for invasive salmonellosis worldwide
113 nging to 28 serovars, including, S. enterica serovars S.
114 ple independent lineages such as S. enterica serovars S.
115  CVD 1944 protected mice against the group D serovar Salmonella enterica serovar Dublin (85% vaccine
116  CVD 1931 protected mice against the group B serovar Salmonella enterica serovar Stanleyville (91% va
117 sistance against challenge with nontyphoidal serovar Salmonella Enteritidis than with another nontyph
118 ed immunosensor to know the concentration of serovar Salmonella typhimurium.
119 sing infectious bacteria Salmonella enterica serovar (Salmonella typhi) in 10 muL of sample volume.
120 hat immunization of mice with live typhoidal serovar, Salmonella Typhi, generates cross-reactive immu
121 a Enteritidis than with another nontyphoidal serovar, Salmonella Typhimurium.
122                 Exposure to the dominant NTS serovars, Salmonella enterica serovars Typhimurium and E
123 terica serovar Typhi and Salmonella enterica serovar Sendai, causes enteric fever.
124 with Salmonella enterica subspecies enterica serovar Senftenberg are often associated with exposure t
125 al was put forward that they comprised a new serovar, serovar 16.
126                                        These serovars share a high degree of homology at the genome a
127                                   Salmonella serovars sidestep the competition by using their virulen
128 onstruct promoted strong immune responses to serovar-specific epitopes, the conserved LNPTIAG epitope
129 se invasive disease may be linked to certain serovar-specific genetic factors.
130 inst the group B serovar Salmonella enterica serovar Stanleyville (91% vaccine efficacy), and S. Ente
131 rica serovar Paratyphi A is a human-specific serovar that, together with Salmonella enterica serovar
132                                              Serovars that are uncommonly associated with human disea
133                         There are over 2,600 serovars that cause a range of disease manifestations ra
134 found that, in comparison with a noninvasive serovar, the invasive Salmonella strains Ty2 and D23580
135 this new niche support a bloom of Salmonella serovars, thereby ensuring transmission of the pathogen
136  protective Abs elicited with one Salmonella serovar to engage with and consequently provide protecti
137 ion of typhoidal and nontyphoidal Salmonella serovars to invasive disease varies considerably in plac
138 sia, multidrug-resistant Salmonella enterica serovar Typhi (S Typhi) has been the main cause of enter
139 luence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.
140                          Salmonella enterica serovar Typhi (S Typhi) is responsible for an estimated
141 d by Salmonella enterica subspecies enterica serovar Typhi (S. Typhi) and can lead to systemic illnes
142                          Salmonella enterica serovar Typhi (S. Typhi) causes substantial morbidity an
143 d subsequent invasion of Salmonella enterica serovar Typhi (S. Typhi), a human-restricted pathogen.
144        The population of Salmonella enterica serovar Typhi (S. Typhi), the causative agent of typhoid
145 e (AMR) in the bacterium Salmonella enterica serovar Typhi (S. Typhi).
146      Salmonella enterica subspecies enterica serovar Typhi (Salmonella Typhi) is the cause of typhoid
147 ovar that, together with Salmonella enterica serovar Typhi and Salmonella enterica serovar Sendai, ca
148                          Salmonella enterica serovar Typhi causes the systemic disease typhoid fever.
149                          Salmonella enterica serovar Typhi causes typhoid fever only in humans.
150   Multiyear epidemics of Salmonella enterica serovar Typhi have been reported from countries across e
151 h the causative pathogen Salmonella enterica serovar Typhi implicated in many outbreaks through histo
152                          Salmonella enterica serovar Typhi is a human-restricted Gram-negative bacter
153                          Salmonella enterica serovar Typhi is the etiological agent of typhoid fever.
154 yphoid fever case with a Salmonella enterica serovar Typhi isolate showing extended spectrum beta-lac
155                          Salmonella enterica serovar Typhi isolates from the 2 hospitals with blood c
156      Salmonella enterica subspecies enterica serovar Typhi or Salmonella enterica subspecies enterica
157 tion of Typhoid toxin in Salmonella enterica serovar Typhi relies on a muramidase.
158 e used a live attenuated Salmonella enterica serovar Typhi strain to create a bivalent mucosal plague
159 ed a Salmonella enterica subspecies enterica serovar Typhi strain with resistance against beta-lactam
160 ctamase (ESBL)-producing Salmonella enterica serovar Typhi was identified, whole-genome sequence type
161  Salmonella enterica and Salmonella enterica serovar Typhi, and Yersinia pestis), and 3 protozoa (Lei
162  agent of typhoid fever, Salmonella enterica serovar Typhi, can partially subvert this critical innat
163  threat has increased in Salmonella enterica serovar Typhi, driven in part by the emergence of succes
164 Enteric fever, caused by Salmonella enterica serovar Typhi, is an important public health problem in
165                          Salmonella enterica serovar Typhi, the causative agent of typhoid fever in h
166 is a virulence factor of Salmonella enterica serovar Typhi, the causative agent of typhoid fever, and
167 ellae, 14% (21/152) were Salmonella enterica serovar Typhi, whereas 86% (131/152) were serovars other
168  antimicrobial-resistant Salmonella enterica serovar Typhi.
169 , with a predominance of Salmonella enterica serovar Typhi.
170 mophilus influenzae, and Salmonella enterica serovar Typhi/Typhimurium.
171  enteric fever caused by Salmonella enterica serovars Typhi and Paratyphi is substantial and has high
172  degrees C-42 degrees C) Salmonella enterica serovars Typhi, Paratyphi A, and Sendai significantly at
173 terial pathogens such as Salmonella enterica serovar Typhimurium (7.8%), Listeria monocytogenes (3.88
174                FrmR from Salmonella enterica serovar typhimurium (a CsoR/RcnR-like transcriptional de
175 sinia enterocolitica and Salmonella enterica serovar Typhimurium (all gram-negative bacteria) and Sta
176       The human pathogen Salmonella enterica serovar Typhimurium (S Typhimurium) contains a complex d
177 araginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell resp
178                          Salmonella enterica serovar Typhimurium (S Typhimurium) is a Gram-negative b
179                          Salmonella enterica serovar Typhimurium (S Typhimurium) relies upon the inne
180 istant to infection with Salmonella enterica serovar Typhimurium (S Typhimurium).
181                          Salmonella enterica serovar Typhimurium (S.
182 steria monocytogenes and Salmonella enterica serovar Typhimurium (S.
183 tory system PhoP/PhoQ of Salmonella enterica serovar Typhimurium (S. Typhimurium) in mildly acidic pH
184 f human macrophages with Salmonella enterica serovar Typhimurium (S. Typhimurium) leads to inflammaso
185  model of infection with Salmonella enterica serovar Typhimurium (STM) to identify changes in intesti
186 t the bacterial pathogen Salmonella enterica serovar Typhimurium (STm).
187 tection of low levels of Salmonella enterica serovar typhimurium and enteritidis in blood samples wit
188 urli fibers, produced by Salmonella enterica serovar Typhimurium and Escherichia coli.
189 scherichia coli O157:H7, Salmonella enterica serovar Typhimurium and S.
190  two bacterial pathogens-Salmonella enterica serovar Typhimurium and Shigella flexneri.
191 pathogens that use T3SS, Salmonella enterica serovar Typhimurium and Yersinia pseudotuberculosis.
192 es, Escherichia coli and Salmonella enterica serovar Typhimurium as model microbes, a common redox ac
193 d the vT3SS and fT3SS of Salmonella enterica serovar Typhimurium at ~5 and ~4 nm resolution using ele
194  The food-borne pathogen Salmonella enterica serovar Typhimurium benefits from acute inflammation in
195                          Salmonella enterica serovar Typhimurium can inject effector proteins into ho
196              Conversely, Salmonella enterica serovar Typhimurium causes gastroenteritis in humans and
197  intracellular bacterium Salmonella enterica serovar Typhimurium causes persistent systemic inflammat
198  reduced ability to kill Salmonella enterica serovar Typhimurium compared to that of macrophages isol
199 loodstream infections by Salmonella enterica serovar Typhimurium constitute a major health burden in
200    The ST313 pathovar of Salmonella enterica serovar Typhimurium contributes to a high burden of inva
201 lso demonstrate that the Salmonella enterica serovar Typhimurium core promoter is more active than pr
202  inflammatory response induced by Salmonella serovar Typhimurium creates a favorable niche for this g
203 e intracellular pathogen Salmonella enterica serovar Typhimurium decreases H-NS amounts 16-fold when
204 ane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the variable N terminus o
205 t colonization niche for Salmonella enterica serovar Typhimurium during gastrointestinal infections.
206 that Salmonella enterica subspecies enterica serovar Typhimurium employs a dedicated mechanism, drive
207 recent study showed that Salmonella enterica serovar Typhimurium exhibits sliding motility under magn
208                          Salmonella enterica serovar Typhimurium exploits the host's type I interfero
209 IS) to screen mutants of Salmonella enterica serovar Typhimurium for their ability to infect and grow
210 sis of core genes within Salmonella enterica serovar Typhimurium genomes reveals a high degree of all
211 bilities to infection by Salmonella enterica serovar Typhimurium has just been published in Nature Mi
212 ase and a chitinase from Salmonella enterica serovar Typhimurium hydrolyze LacNAc from Galbeta1-4GlcN
213 ed to restrict growth of Salmonella enterica serovar Typhimurium in host tissues by causing magnesium
214 in cells in vitro and increased virulence of serovar Typhimurium in mice.
215 pathway and promote virulence of S. enterica serovar Typhimurium in mice.
216 srupt biofilms formed by Salmonella enterica serovar Typhimurium in vitro and in vivo.
217 found that intracellular Salmonella enterica serovar Typhimurium induced the binucleation of a large
218 pathogen and host during Salmonella enterica serovar Typhimurium infection and reveal the molecular i
219 ells more susceptible to Salmonella enterica serovar Typhimurium infection in a NOD1-dependent manner
220                          Salmonella enterica serovar Typhimurium infection of immunocompetent mice re
221 RD9 is suppressed during Salmonella enterica serovar Typhimurium infection, facilitating increased IL
222                   During Salmonella enterica serovar Typhimurium infection, host inflammation alters
223 e intracellular pathogen Salmonella enterica serovar Typhimurium initiates an anti-inflammatory trans
224                          Salmonella enterica serovar Typhimurium is a common cause of food-borne gast
225                          Salmonella enterica serovar Typhimurium is a facultative intracellular patho
226                          Salmonella enterica serovar Typhimurium is an intracellular bacterial pathog
227 ate immune resistance of Salmonella enterica serovar Typhimurium is attributed to the high-molecular-
228 c mechanism of FrmR from Salmonella enterica serovar Typhimurium is triggered by metals in vitro, and
229 vention for inactivating Salmonella enterica serovar Typhimurium LT2 (ST2) in tender coconut water (T
230 re absent in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of S Montevide
231 al microcompartment from Salmonella enterica serovar Typhimurium LT2, one of the most widely studied
232 li K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococcus aureus, and Stre
233 s initially found on the Salmonella enetrica serovar Typhimurium multi-resistance plasmid pMG101 from
234 ompetent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its ability to compete wi
235 escued the virulence defect of a S. enterica serovar Typhimurium mutant specifically defective in its
236 ing turtles, but S. enterica subsp. enterica serovar Typhimurium or lesions associated with Salmonell
237 ture of the prototypical Salmonella enterica serovar Typhimurium pathogenicity island 1 basal body, d
238 viA gene in nontyphoidal Salmonella enterica serovar Typhimurium reduced flagellin-induced pyroptosis
239 teropathogenic bacterium Salmonella enterica serovar Typhimurium requires a T6SS encoded within Salmo
240 ins PrgH and PrgK in the Salmonella enterica serovar Typhimurium Salmonella pathogenicity island 1 (S
241 lpha and Salmonella enterica subsp. enterica serovar Typhimurium secretome (STS)-induced outcomes in
242 se to the catalytic triad, is variable, with serovar Typhimurium SpvD having an arginine and serovar
243 t the genomes of S. enterica subsp. enterica serovar Typhimurium strain LT2 and Salmonella bongori st
244 genetically engineered a Salmonella enterica serovar Typhimurium strain of multilocus sequence type 3
245 cterium tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.
246 ostatic activity against Salmonella enterica serovar Typhimurium that is not shared by the related pu
247 tence of Salmonella enterica subsp. enterica serovar Typhimurium through liver-resident immunoregulat
248 rdinates the response of Salmonella enterica serovar Typhimurium to diverse environmental challenges
249  examined the ability of Salmonella enterica serovar Typhimurium to infect the central nervous system
250 e CspA family members of Salmonella enterica serovar Typhimurium to link the constitutively expressed
251  the intestinal pathogen Salmonella enterica serovar Typhimurium uses specialized metal transporters
252 rimentally infected with Salmonella enterica serovar Typhimurium was investigated.
253 , N, O, and Q); however, Salmonella enterica serovar Typhimurium was the most predominant serovar, ac
254 ria monocytogenes V7 and Salmonella enterica serovar Typhimurium were used as model pathogens to eval
255 tection of low levels of Salmonella enterica serovar Typhimurium without culture enrichment.
256              Salmonella (Salmonella enterica serovar Typhimurium) secrete numerous effector proteins,
257 ia (Escherichia coli and Salmonella enterica serovar Typhimurium).
258  isolates, 40% (41) were Salmonella enterica serovar Typhimurium, 10% (10) were Salmonella enterica s
259                          Salmonella enterica serovar Typhimurium, a Gram-negative bacterium, can caus
260                       In Salmonella enterica serovar Typhimurium, DSFs repress the activity of HilD,
261 Klebsiella pneumoniae or Salmonella enterica serovar Typhimurium, enhanced translocation.
262                       In Salmonella enterica serovar Typhimurium, flagella-mediated motility is repre
263  that both flagellins of Salmonella enterica serovar Typhimurium, FliC and FljB, are methylated at su
264                       In Salmonella enterica serovar Typhimurium, Mg(2+) limitation induces transcrip
265 herichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogate murine infection m
266 aps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aeruginosa, Bacillus su
267 itiation of phage P22 in Salmonella enterica serovar Typhimurium, revealing how a channel forms to al
268                       In Salmonella enterica serovar Typhimurium, siroheme is produced by a trifuncti
269  Unlike the nontyphoidal Salmonella enterica serovar Typhimurium, the genomes of S. Typhi and S. Para
270 al functions, strains of Salmonella enterica serovar Typhimurium, the murine model of S Typhi, in whi
271 III protein secretion in Salmonella enterica serovar Typhimurium, we discovered that several TCMs can
272 in their ability to kill Salmonella enterica serovar Typhimurium, which was rescuable after experimen
273 an efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptosis and proinflammato
274 sting the methodology on Salmonella enterica serovar Typhimurium-infected murine bone-marrow-derived
275  significantly less invasive infections than serovar Typhimurium.
276 nd the invasive pathogen Salmonella enterica serovar Typhimurium.
277 es of the model pathogen Salmonella enterica serovar Typhimurium.
278 and aerobic expansion of Salmonella enterica serovar Typhimurium.
279 e intracellular pathogen Salmonella enterica serovar Typhimurium.
280 or upon coinfection with Salmonella enterica serovar Typhimurium.
281 rial infections, such as Salmonella enterica serovar Typhimurium.
282 teric pathogens, such as Salmonella enterica serovar Typhimurium.
283 ypically challenged with Salmonella enterica serovar Typhimurium.
284 stridium perfringens and Salmonella enterica serovar Typhimurium.
285 otypic comparison with the model S. enterica serovar Typhimurium.
286 ella (NTS), particularly Salmonella enterica serovars Typhimurium and Enteritidis, is responsible for
287 e dominant NTS serovars, Salmonella enterica serovars Typhimurium and Enteritidis, were assessed usin
288          Salmonella enterica subsp. enterica serovars Typhimurium and its four closest relatives, Sai
289 typhi A compared to that of the nontyphoidal serovarS Typhimurium.
290 raction was done from Salmonella typhimurium serovars, under the optimized growth conditions for its
291 th the winning metabolic strategy Salmonella serovars use to edge out competing microbes in the infla
292 nvasive NTS from whom 1 of the 4 predominant serovars was isolated in pure culture, 448 (81.0%) were
293                A capsule locus and in silico serovar were identified for all but two nontypeable isol
294 istinct from the previously characterized 15 serovars were described, and a proposal was put forward
295 ic techniques speciated isolates; Salmonella serovars were determined.
296                In the 67 isolates, 27 unique serovars were identified, with three dogs having two ser
297 ons (1.2%) and nontyphoidal Salmonella (NTS) serovars were isolated 10,139 times (6.1%), of which 801
298 ontrolling virulence phenotypes in typhoidal serovars, which is likely to play a role in the distinct
299 ould efficiently concentrate both Salmonella serovars with a capturing efficiency >95%.
300 ant genetic variation was identified between serovars, with the exception of serovars 5 and 12.
301 f the large and diverse range of species and serovars within the genus Leptospira circulating within

 
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