ライフサイエンスコーパス: serum bicarbonate

1 ster rate of decline of serum hematocrit and serum bicarbonate.

2 ls of serum chloride and decreased levels of serum bicarbonate.

3 trointestinal tract, leads to an increase in serum bicarbonate.

4 L/min per 1.73 m(2)) and metabolic acidosis (serum bicarbonate 12-20 mmol/L), who had completed the 1

5 ce [CrCl] 15 to 30 ml/min per 1.73 m(2)) and serum bicarbonate 16 to 20 mmol/L to either supplementat

6 inary albumin/creatinine (ACR) >=50 mg/g and serum bicarbonate 20-28 meq/L.

7 mbrane depolarization correlated with raised serum bicarbonate and base excess, indicating compensate

8 ublished evidence on the association between serum bicarbonate and clinical outcomes.

9 e B-type natriuretic peptide, uric acid, and serum bicarbonate and decreases in hemoglobin and hemato

10 Serum bicarbonate and pH decreased.

11 Serum bicarbonate and pH levels plateau after 48 hrs of

12 Serum bicarbonate and potassium concentrations were draw

13 We compared changes in serum bicarbonate and urinary citrate-to-creatinine rati

14 Hemoglobin (Hb), serum bicarbonate, and creatinine; use of erythropoiesis

15 Other medications, serum bicarbonate, and renal function were not different

16 albuminuria, serum calcium, serum phosphate, serum bicarbonate, and serum albumin (C statistic, 0.917

17 circulating NT-proBNP levels, a decrease in serum bicarbonate, and significant changes in general me

18 owing CKD progression, but the difference in serum bicarbonate between placebo and drug arms was only

19 2 mm Hg; P = .25), although daily changes of serum bicarbonate (between-group difference, -0.8 mEq/L;

20 With alkali therapy, there was no change in serum bicarbonate, but the urinary citrate-to-creatinine

21 ly associated with normal serum lactate were serum bicarbonate, chloride, and pulmonary disease, whil

22 sessed the long-term effects of veverimer on serum bicarbonate concentration and physical functioning

23 Due to multiple compensating mechanisms, the serum bicarbonate concentration is a relatively insensit

24 dney function in CKD, even if initiated when serum bicarbonate concentration is normal.

25 L/min per 1.73 m(2)) and metabolic acidosis (serum bicarbonate concentration of 12-20 mmol/L).

26 ncrease of 4 mmol/L or more from baseline in serum bicarbonate concentration or serum bicarbonate in

27 urinary ammonium excretion was 25% lower and serum bicarbonate concentration was 1.3 meq/L higher in

28 systemic pH caused by a primary decrease in serum bicarbonate concentration), as seen in clinical di

29 95% CI, 0.90 to 0.99 per 1 mEq/L increase in serum bicarbonate concentration).

30 of the patients initially had acidosis (mean serum bicarbonate concentration, 12.9 mmol per liter).

31 Serum bicarbonate concentrations remained significantly

32 higher prevalence of vasopressor use, lower serum bicarbonate concentrations, and a higher prevalenc

33 tinoculation percent weight change per h and serum bicarbonate concentrations, the virulence of the S

34 y associated with acid accumulation, whereas serum bicarbonate did not.

35 seline in serum bicarbonate concentration or serum bicarbonate in the normal range of 22-29 mmol/L, a

36 .6 mEq/L on day 7 (P < 0.001 for both); mean serum bicarbonate increased from 20 4 to 21 4 mEq/L on P

37 entilation in multivariate analysis included serum bicarbonates less than 20 mM (odds ratio, 4.9 [95%

38 We suggest that: 1) clinicians use a serum bicarbonate level <27 mmol/L to exclude the diagno

39 ted multinomial logistic regression model, a serum bicarbonate level less than 10 mEq/L (compared wit

40 pH was greater than 7.45, and .4 days where serum bicarbonate level was greater than 28 mmol/L, duri

41 ssociated with the number of days with a low serum bicarbonate level, but was not associated with inc

42 ith increased frequency of an elevated pH or serum bicarbonate level.

43 eGFR 15-44 ml/min per 1.73 m 2 ) with normal serum bicarbonate levels (22-27 mEq/L).

44 Both high and low serum bicarbonate levels associate with an increased ris

45 Lower serum bicarbonate levels, even within the normal range,

46 participants with CKD stage 3b-4 with normal serum bicarbonate levels.

47 function in participants with CKD and normal serum bicarbonate levels.

48 red urinary ammonium excretion and increased serum bicarbonate more than the lower dose but was assoc

49 4.5; 95% confidence interval [CI], 2.7-7.6), serum bicarbonate of <20 mmol/L (OR, 2.9; 95% CI, 1.6-5.

50 l/min per 1.73 m 2 ) and metabolic acidosis (serum bicarbonate of 12-20 mEq/L) from 35 countries were

51 Serial osmolalities, lactates, serum bicarbonate, PaCO2, and pH were measured during lo

52 aps earlier acid-base indicator of risk than serum bicarbonate, particularly in patients without acid

53 ger scores of 6 variables: serum creatinine, serum bicarbonate, pulse, systolic blood pressure, diast

54 Serum bicarbonate rose significantly while the patient w

55 imens at any point over the study period for serum bicarbonate, serum potassium, or urine chloride en

56 able, pragmatic, and a superior parameter to serum bicarbonate (the defended entity) to assess acid-b

57 el incorporating plasma IL-8, protein C, and serum bicarbonate to assign phenotypes on each day.

58 th a baseline and at least one post-baseline serum bicarbonate value).

59 GFR was 29.2+/-6.3 ml/min per 1.73 m 2 , and serum bicarbonate was 17.5+/-1.4 mEq/L; this increased t

60 After randomized withdrawal, the mean serum bicarbonate was 22.0+/-3.0 mEq/L and 20.9+/-3.3 mE

61 seline eGFR was 36+/-9 ml/min per 1.73 m(2), serum bicarbonate was 24+/-2 meq/L, and median (IQR) ACR

62 pace, rhythm, and variability domains, lower serum bicarbonate was associated with worse performance

63 While serum bicarbonate was flat and did not fall below the re

64 In sum, lower serum bicarbonate was independently associated with wors

65 asing proportion of days with elevated pH or serum bicarbonate was not associated with increased mort

66 Serum bicarbonate was reduced by 2.3, 4.2, and 4.4 mEq/L

67 Serum bicarbonate was significantly lower in incremental

68 e gait analysis, we examined associations of serum bicarbonate with eight individual gait variables.