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1 mparable to those of peak expiratory flow or serum cortisol.
2 riod, blood samples were taken for 8:00 A.M. serum cortisol.
4 ients without cholestasis by measuring total serum cortisol, adrenocorticotropic hormone (ACTH), as w
5 iated with a significant decrease in IOP and serum cortisol, along with an improvement in optic nerve
6 letion of a corticotrophin stimulation test, serum cortisol and 11beta-deoxycortisol concentrations w
10 ealed that ozone exposure markedly increased serum cortisol and corticosterone together with increase
12 , metabolic responses (serial blood glucose, serum cortisol and insulin, plasma epinephrine, plasma n
13 xia nervosa subtype, waist-to-hip ratio, and serum cortisol and leptin levels on treatment outcome, o
14 erein, we examined the relationships between serum cortisol and multimodality brain AD biomarkers in
16 Seltorexant's effects on polysomnography, serum cortisol, and cortisol waking response were also m
17 d plasma aldosterone, plasma renin activity, serum cortisol, and estimation of trans tubular potassiu
18 y and levels of adrenocorticotropic hormone, serum cortisol, and saliva cortisol among all patients a
20 eroid hormones, and accurate measurements of serum cortisol are necessary for proper diagnosis of adr
22 vents, coping by means of denial, and higher serum cortisol as well as with lower cumulative average
23 The 91 patients with sepsis began with a serum cortisol at 29.3 +/- 2.5, and it increased to 40.1
28 itical illness is characterized by increased serum cortisol concentrations and bioavailability result
29 macokinetic profiles and a lack of impact on serum cortisol concentrations at predicted therapeutic d
31 in infusion raised plasma growth hormone and serum cortisol concentrations significantly (P < 0.001 f
35 morning (approximately 8 am) measurements of serum cortisol, corticotropin, and dehydroepiandrosteron
39 epinephrine levels, morning plasma ACTH and serum cortisol, fasting glucose and insulin, and lipid p
42 showed a significant decrease (P <= .001) in serum cortisol level and an improved QOL (P = .001).
49 addition, there was a sustained elevation of serum cortisol levels, reduced circulating melatonin lev
52 mg overnight dexamethasone-suppression test (serum cortisol: <50 nmol/L, nonfunctioning adrenal tumor
54 tigraphy assessment, and 24-hour analyses of serum cortisol, melatonin, and peripheral clock gene exp
55 y ill patients is best made by a delta total serum cortisol of < 9 microg/dL after adrenocorticotroph
57 Perceived Stress Scale (PSS) questionnaire, serum cortisol (sCOR), salivary Cortisol Awakening Respo
58 termined time points for plasma cannabinoid, serum cortisol, serum haptoglobin, liver enzymes, serum
59 d "stressed" (i.e., exhibited an increase in serum cortisol), there was no effect on the microbiota a
61 (37%) were steroid responsive; the baseline serum cortisol was 14.1 +/- 5.2 microg/dL in the steroid
64 immune cell proportions, cytokines, CRP and serum cortisol were measured at baseline and during the
66 thalamic-pituitary-thyroid axis, we measured serum cortisol, which also has a circadian secretory pat