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1 s of peripheral mature B cells and decreased serum immunoglobulin.
2 east or by immobilized protein A/G to remove serum immunoglobulin.
3 me activated in the presence of neutralizing serum immunoglobulin.
4 mbers showed progressive loss of B cells and serum immunoglobulins.
5 nd central memory T cells and an increase in serum immunoglobulins.
6 vestigated the prevalence of abnormally high serum immunoglobulin A (IgA) and IgG anti-gliadin antibo
7 nses; all volunteers who received H10407 had serum immunoglobulin A (IgA) and IgG responses, and all
9 for RV1 and RV5 to correlate anti-rotavirus serum immunoglobulin A (IgA) antibody titers vs efficacy
11 Half of the volunteers mounted a positive serum immunoglobulin A (IgA) response to S. flexneri lip
16 trifluoroacetylated protein adduct-specific serum immunoglobulin and hepatotoxicity were reduced.
17 oved B-cell function with increases in total serum immunoglobulin and increased splenic mitogen respo
18 inflammation and hyperresponsiveness (AHR), serum immunoglobulin and splenic T cells were assessed.
19 erved as lesser allergic phenotypes, reduced serum immunoglobulins and allergic mediators, lower mast
20 methods included flow cytometry, analysis of serum immunoglobulins and autoantibodies, lymphocyte sti
22 Ptpn6(f/f);CD19-cre mice exhibited elevated serum immunoglobulins and impaired antibody responses to
23 tive IgE, which is the least abundant of the serum immunoglobulins and occurs at subnanomolar levels,
27 in early B-cell development, near absence of serum immunoglobulin, and recurrent bacterial infections
28 Eosinophil recruitment, cytokine production, serum immunoglobulins, and airway histology were analyze
29 sponse (ASR), anaphylaxis, body temperature, serum immunoglobulins, and mouse mast cell protease-1 (m
30 ignificant reductions in lymphocyte count or serum immunoglobulin, anticardiolipin antibody, or rubel
32 d activation parameters are normal; however, serum immunoglobulins are increased and kidney function
33 iency associated with partial persistence of serum immunoglobulin arising from a missense mutation in
34 nificantly reduced titers of IgG2a and IgG2b serum immunoglobulins as well as autoantibodies, but mai
35 solated using the device was consistent with serum immunoglobulin assays that are commonly used in MM
36 Ku70-deficient mice lacked mature B cells or serum immunoglobulin but, unexpectedly, reproducibly dev
37 als produced significantly less IgG1 and IgE serum immunoglobulins, but maintained comparable levels
39 d a normal immune cell repertoire, unchanged serum immunoglobulin concentrations and an intact immune
41 ations depended on DCs, in contrast to total serum immunoglobulin concentrations, suggesting an effec
43 sistent with eradication of B-lineage cells, serum immunoglobulins decreased to very low levels after
44 ogeneous and miniscule fraction of the total serum immunoglobulin displaying identical properties oth
45 sponses, characterized by increases in total serum immunoglobulin E (IgE) and specific serum IgG1 lev
48 This study examined the association between serum immunoglobulin E (IgE) levels for a panel of commo
50 al hyperresponsiveness (BHR), elevated total serum immunoglobulin E (IgE) levels, and skin tests posi
51 dies of intermediate phenotypes, one each on serum immunoglobulin E (IgE) levels, blood eosinophil co
53 ent of CD4-CD8- peripheral T cells, elevated serum immunoglobulin E (IgE), and possible pulmonary inf
54 cts on markers of Th2 inflammation, reducing serum immunoglobulin E (IgE), chemokine ligands 13 and 1
55 However, the level of a systemic Th2 marker, serum immunoglobulin E (IgE), correlated significantly w
56 ypersensitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE); a strong T2 cytokine pheno
58 d with asthma phenotypes, such as high total serum immunoglobulin E and bronchial hyperresponsiveness
60 ti-TNF-alpha-treated mice exhibited elevated serum immunoglobulin E and inhibition of the anticryptoc
61 levels at year 1 and repeated assessments of serum immunoglobulin E antibodies (year 1, 4.5, 6), atop
62 the allergen specificity, allergen-specific serum immunoglobulin E concentration, or individual labo
68 eukin-4 production by NKT cells, to increase serum immunoglobulin E levels, and to promote the genera
70 he accumulation of eosinophils and levels of serum immunoglobulin E were increased in NFAT1 -/- mice.
71 ormed with asthma, AHR, lung function, total serum immunoglobulin E, and blood eosinophil levels.
73 es on swIg(+) cells until they disappear and serum immunoglobulin falls to a low level, in which case
77 one marrow plasmacytosis, extremely abnormal serum immunoglobulin free light chain ratio, and multipl
78 C-reactive protein, human interleukin-6, and serum immunoglobulin free light chains decreased with th
80 n ovarian cancer phage display library using serum immunoglobulins from an ovarian cancer patient as
81 s resulted in the induction of FimA-specific serum (immunoglobulin G [IgG] and IgA) and salivary (IgA
85 ity to S. typhimurium severely depressed the serum immunoglobulin G (IgG) and IgA anti-FrgC response
86 -CAT construct induced significant levels of serum immunoglobulin G (IgG) and IgA antibody to both pa
88 with Invaplex was found to enhance anti-OVA serum immunoglobulin G (IgG) and IgA responses and induc
89 oximately 2-week intervals and evaluated for serum immunoglobulin G (IgG) and IgG subclass and for sa
91 throughout the experiment and evaluated for serum immunoglobulin G (IgG) and IgM and salivary IgA an
93 or no bacteria in the liver and lungs, high serum immunoglobulin G (IgG) and IgM antibody titers, an
94 lpha stimulated strong antigen (Ag)-specific serum immunoglobulin G (IgG) and IgM responses, while MI
95 ion of mice with CVLPs induced gp41-specific serum immunoglobulin G (IgG) and intestinal secretory Ig
96 luenza virus showed significant increases in serum immunoglobulin G (IgG) and mucosal IgA antibodies
97 lones were found to stimulate high levels of serum immunoglobulin G (IgG) and mucosal IgA antibodies
98 oxin (CT) A2 and B subunits (CTA2/B) develop serum immunoglobulin G (IgG) and mucosal IgA antibody re
99 hesizing either PspA fusion protein elicited serum immunoglobulin G (IgG) and mucosal IgA responses a
102 hK63 or LThR72 exhibited high titers of both serum immunoglobulin G (IgG) and mucosal secretory IgA a
103 ed animals demonstrated significantly higher serum immunoglobulin G (IgG) and salivary IgA antibody l
107 enuation elicited higher Salmonella-specific serum immunoglobulin G (IgG) and/or mucosal secretory-Ig
108 later, significant reductions were seen for serum immunoglobulin G (IgG) antibodies at week 14 and f
110 recombinant proteins were reacted with human serum immunoglobulin G (IgG) antibodies in Western immun
113 ta provide evidence that a critical level of serum immunoglobulin G (IgG) antibodies to the surface p
114 were found to be immunogenic since elevated serum immunoglobulin G (IgG) antibodies without a specif
115 Oral and i.p. vaccines elicited O11-specific serum immunoglobulin G (IgG) antibodies, but IgA was obs
116 isms are found and the induction of specific serum immunoglobulin G (IgG) antibodies, we examined the
117 ay was used to measure the seroprevalence of serum immunoglobulin G (IgG) antibody among NHANES parti
119 ria during the study interval, and increased serum immunoglobulin G (IgG) antibody levels substantiat
120 nce of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibody levels to P. gingi
122 oPn produced greater titers of MoPn-specific serum immunoglobulin G (IgG) antibody than mice that rec
123 zed by enzyme-linked immunosorbent assay for serum immunoglobulin G (IgG) antibody to Aggregatibacter
126 rticular) demonstrated significantly reduced serum immunoglobulin G (IgG) antibody, salivary IgA anti
127 Ribi had the highest levels of PorB-specific serum immunoglobulin G (IgG) by enzyme-linked immunosorb
130 mean titer (GMTs) and 4-fold rise of anti-Vi serum immunoglobulin G (IgG) enzyme-linked immunosorbent
131 ntigenic MDI-albumin products, as defined by serum immunoglobulin G (IgG) from MDI-exposed workers, w
132 ale C57BL/6 mice treated daily with purified serum immunoglobulin G (IgG) from patients with longstan
134 immunosorbent assay to measure beta-specific serum immunoglobulin G (IgG) levels and used it to compa
136 haride (PS) conjugate vaccines may prime for serum immunoglobulin G (IgG) memory responses to meningo
137 transplantations indicate that low levels of serum immunoglobulin G (IgG) negatively impact outcomes.
138 Overall, 23% of patients developed either a serum immunoglobulin G (IgG) response (9%) or sputum IgA
139 . denticola induced a significant (400-fold) serum immunoglobulin G (IgG) response compared to that i
142 ized with replication-defective HSV, durable serum immunoglobulin G (IgG) responses were elicited.
143 e the long-term course of H. pylori-specific serum immunoglobulin G (IgG) responses with respect to s
144 were effective in eliciting antigen-specific serum immunoglobulin G (IgG) responses, and these respon
146 fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1
147 were consistent with significantly declined serum immunoglobulin G (IgG) titers for HHV-6 of MS pati
148 ckerboard analyses of eight plaque bacteria, serum immunoglobulin G (IgG) titers to 17 bacteria, and
151 colonization, 9 of 40 patients developed new serum immunoglobulin G (IgG) to OMP CD, as measured by e
152 ollowing infection and who had developed new serum immunoglobulin G (IgG) to their infecting strain w
154 eleased from bovine fetuin, polyclonal human serum immunoglobulin G (IgG), and human alpha1-acid glyc
155 he most effective means of immunization: the serum immunoglobulin G (IgG), IgA, and IgM anti-fragment
158 adermal comparative tuberculin skin test and serum immunoglobulin G [IgG] responses) against a range
159 vated during primary infections (acute-phase serum immunoglobulin G [IgG] titers, <25), compared with
160 nses (HI, antibody-secreting cell [ASC], and serum immunoglobulin G [IgG]) in 15 LRs and 25 control H
161 minent specific anti-CtxB responses in vivo (serum immunoglobulin G [IgG], P </= 0.05; serum IgA, P <
162 ly correlated with the level of prechallenge serum immunoglobulin G against the O127 lipopolysacchari
163 l aplasia, several vaccine/pathogen-specific serum immunoglobulin G and A (IgG and IgA) titers remain
166 Only conjugates with O acetyls elicited serum immunoglobulin G anti-LPS with bactericidal activi
167 ith the level of protection correlating with serum immunoglobulin G anti-protective antigen titers.
168 VRP resulted in high levels of DENV-specific serum immunoglobulin G antibodies and significant titers
170 ll-length block 2 antigens showed that human serum immunoglobulin G antibodies induced by infection c
171 orbent assays (ELISAs) for quantification of serum immunoglobulin G antibodies specific for N. mening
172 concentrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (
173 tion, as well as the specificity, of anti-P1 serum immunoglobulin G antibodies were demonstrated in g
175 d formation, an impaired ability to generate serum immunoglobulin G antibodies, and significant inhib
176 treated patients with LA have high levels of serum immunoglobulin G antibodies, and sometimes low lev
177 cator of inflammation), or edentulism and 2) serum immunoglobulin G antibody response to Aggregatibac
178 e also developed sustained anti-H. hepaticus serum immunoglobulin G antibody responses and elevated a
179 Less marked, but significant, anti-SREHP serum immunoglobulin G antibody responses were also indu
181 PI) owing to the presence of highly elevated serum immunoglobulin G antibody titers with a high avidi
182 cant levels of salivary immunoglobulin A and serum immunoglobulin G antibody to the respective inject
185 ortal vein diameter, splenomegaly, increased serum immunoglobulin G level, and increasing number of a
190 , with a 44% reduction in intracardiomyocyte serum immunoglobulin G localization in het-treated-8 mic
191 cination geometric mean titre of LT-specific serum immunoglobulin G of 3400.29, compared with 315.41
194 the naturally infected SCID/NCr mice had no serum immunoglobulin G response against H. hepaticus.
195 vaccine doses elicited a strong PA-specific serum immunoglobulin G response with a geometric mean ti
197 ufficient to decrease P. gingivalis-specific serum immunoglobulin G responses, but lower antibody tit
198 osted 4 weeks later induced higher levels of serum immunoglobulin G specific for PspA and for outer m
201 ked immunosorbent assay (ELISA) to determine serum immunoglobulin G titers against Shigella LPS.
202 he vhs(-)/ICP8(-) mutant showed prechallenge serum immunoglobulin G titers comparable to those immuni
203 a clinical oral-health examination, and had serum immunoglobulin G titers measured against 19 period
204 of variants with ChoP correlates with higher serum immunoglobulin G titers to LPS containing this str
209 e deficiency, characterized by low levels of serum immunoglobulin G, A, and/or M with loss of antibod
211 ut adjuvant stimulated induction of specific serum immunoglobulin G, whereas antigen alone or admixed
213 asma could not be duplicated by pooled human serum, immunoglobulin G, or fibrinogen, whether used sep
214 s well as the production of antigen-specific serum immunoglobulin G1 (IgG1) and IgG2a antibodies.
215 wever, there was an increase in the level of serum immunoglobulin G1 (IgG1) and IgG2b as well as a mi
216 fected with wild-type H. hepaticus developed serum immunoglobulin G1 (IgG1) and IgG2c responses again
218 i antibody levels in serum showed a dominant serum immunoglobulin G1 (IgG1) response in immunized C57
220 sponse, including the levels of RSV-specific serum immunoglobulin G1 (IgG1), IgG2a, IgA, and total Ig
221 onstrated an increased level of CVB3-binding serum immunoglobulin G1 in mice inoculated with CVB3-PL2
222 le, VirB9, VirB10, and CTP are recognized by serum immunoglobulin G2 (IgG2) and stimulate memory T-ly
223 Patients with LJP have elevated levels of serum immunoglobulin G2 (IgG2), and this is most strikin
224 e demonstrated between the levels of anti-P1 serum immunoglobulin G2a (IgG2a) and IgG2b, but not of I
225 e vector and passenger antigen, resulting in serum immunoglobulin G2a (IgG2a) and modest mucosal IgA
226 mRNAs; high levels of antibody, particularly serum immunoglobulin G2a (IgG2a) and respiratory IgA; an
227 hat oral reovirus infection elicits specific serum immunoglobulin G2a (IgG2a), while parenteral reovi
228 by the production of high titers of specific serum immunoglobulin G2a antibody and the production of
229 n the dermis and subcutaneous fat, increased serum immunoglobulin G2a levels, and lymphadenopathy ass
230 al immunization with LT-R72 induced a potent serum immunoglobulin G2a response, indicating that this
231 he cecum, as well as elevated Th1-associated serum immunoglobulin G2a(b) compared to infection of B6
233 ry sclerosing cholangitis but with increased serum immunoglobulin G4 and infiltrating immunoglobulin
235 o rotavirus infection, we analyzed levels of serum immunoglobulin (Ig) A and IgG antibodies to variou
237 ek 6 (predicted peak of pollen) to determine serum immunoglobulin (Ig) E concentrations and Treg perc
239 red orally without adjuvant, and they elicit serum immunoglobulin (Ig) G and intestinal IgA responses
240 pithelial antigen that reacted strongly with serum immunoglobulin (Ig) G from the mouse model of BA w
242 ever, in the absence of constant reexposure, serum immunoglobulin (Ig) levels rapidly decline and ful
243 samples were collected for quantification of serum immunoglobulin (Ig) levels, specific antibodies ag
244 Consistent with B-lymphocyte increases, serum immunoglobulin (Ig) M, IgG, and IgE were significa
245 fective B-cell function characterized by low serum immunoglobulin (Ig) M, low IgM antibody production
247 ) cell cytotoxic activity and were devoid of serum immunoglobulin (Ig) throughout a 37-week lifespan.
249 ymic stromal lymphopoietin, IL-5 and IL-13), serum immunoglobulin (Ig)E and airway hyper-responsivene
251 re examined pulmonary pathophysiological and serum immunoglobulin (Ig)E responses in mice of 12 inbre
254 ing checkerboard DNA-DNA hybridizations, and serum immunoglobulin (Ig)G levels were measured against
255 ity to carriage did not correlate with total serum immunoglobulin (Ig)G to the homotypic capsular pol
256 pression, mutant mice had elevated levels of serum immunoglobulin (Ig)G1, IgG2b, and IgE and produced
259 long-lived plasma cells, which produce most serum immunoglobulin, is central to humoral immunity.
260 re was widespread variation in the levels of serum immunoglobulin isotypes as well as in the percenta
261 o, and baseline levels of the Th2-controlled serum immunoglobulin isotypes, IgE and IgG1, were also s
262 ith baseline levels, we noted an increase in serum immunoglobulin levels across all classes, and a re
267 Absolute numbers of lymphocyte subsets and serum immunoglobulin levels were all within normal range
273 ) is characterized by variable reductions in serum immunoglobulin levels which cause most ICF patient
274 rently normal lymphocyte development, normal serum immunoglobulin levels, and the capacity to respond
279 cell numbers; immune complex, complement, or serum immunoglobulin levels; delayed type hypersensitivi
280 was associated with higher brain CFU, lower serum immunoglobulin M (IgM) and IgG responses to glucur
281 cryptocococcal pulmonary infection elicited serum immunoglobulin M (IgM) and IgG to the capsular pol
284 (+)CD4(-) T cells, and decreases in elevated serum immunoglobulin M and inflammatory markers includin
287 se with nonsense CXCR4 mutations have higher serum immunoglobulin M levels and incidence of symptomat
288 88 show lower bone marrow disease burden and serum immunoglobulin M levels but show an increased risk
289 vement declined from 55% to 10% (P = .0004), serum immunoglobulin M levels declined from 4,830 to 1,1
290 ysts had no detectable mature T and B cells, serum immunoglobulin M, or even Thy-1(+) and B220(+) pre
292 higher bone marrow (BM) disease involvement, serum immunoglobulin-M levels, and symptomatic disease r
293 CXCR4(WILDTYPE (WT)) had intermediate BM and serum immunoglobulin-M levels; those with MYD88(WT)CXCR4
294 luble and membrane-bound TGF-beta as well as serum immunoglobulin production are high in these mice.
295 ural killer cells, lymphocyte phenotype, and serum immunoglobulin subclass levels were evaluated.
296 scue of mature B cells with normal levels of serum immunoglobulins, together with complete rescue of
297 enzyme-linked immunosorbent assay, specific serum immunoglobulin was detected as early as 134 days p
300 ically by inadequate quantity and quality of serum immunoglobulins with increased susceptibility to i