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1 -OIT-FDEIA groups, except for level of total serum immunoglobulin E.
3 d with asthma phenotypes, such as high total serum immunoglobulin E and bronchial hyperresponsiveness
5 ti-TNF-alpha-treated mice exhibited elevated serum immunoglobulin E and inhibition of the anticryptoc
7 levels at year 1 and repeated assessments of serum immunoglobulin E antibodies (year 1, 4.5, 6), atop
8 the allergen specificity, allergen-specific serum immunoglobulin E concentration, or individual labo
10 and had higher blood eosinophil numbers and serum immunoglobulin E concentrations that were specific
13 e of type 2 (T2)-low asthma (i.e., low total serum immunoglobulin E, fractional exhaled nitric oxide,
14 sponses, characterized by increases in total serum immunoglobulin E (IgE) and specific serum IgG1 lev
17 This study examined the association between serum immunoglobulin E (IgE) levels for a panel of commo
19 al hyperresponsiveness (BHR), elevated total serum immunoglobulin E (IgE) levels, and skin tests posi
20 dies of intermediate phenotypes, one each on serum immunoglobulin E (IgE) levels, blood eosinophil co
22 ent of CD4-CD8- peripheral T cells, elevated serum immunoglobulin E (IgE), and possible pulmonary inf
23 cts on markers of Th2 inflammation, reducing serum immunoglobulin E (IgE), chemokine ligands 13 and 1
24 However, the level of a systemic Th2 marker, serum immunoglobulin E (IgE), correlated significantly w
25 ypersensitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE); a strong T2 cytokine pheno
27 ility of severe AD was associated with total serum immunoglobulin E levels greater than 1708 IU/mL an
29 eukin-4 production by NKT cells, to increase serum immunoglobulin E levels, and to promote the genera
33 measures included biological markers (total serum immunoglobulin E (tIgE), number of positive skin p
34 he accumulation of eosinophils and levels of serum immunoglobulin E were increased in NFAT1 -/- mice.