ライフサイエンスコーパス: serum lipid

1 ysis as the mechanism for niacin's effect on serum lipids.

2 circumference, blood pressure, glucose, and serum lipids.

3 ased relative proportion of omega-6 PUFAs in serum lipids.

4 ples, was now evaluated for the profiling of serum lipids.

5 the end of each diet period and analyzed for serum lipids.

6 nal adjustment was made for levels of BP and serum lipids.

7 may be partly mediated by effects of TFAs on serum lipids.

8 and pectin was attenuated by adjustment for serum lipids.

9 beta-muricholic acid as well as hepatic and serum lipids.

10 There was no difference in serum lipids.

11 ssover design trial to assess the effects on serum lipids.

12 , dietary intake (via 7-d food records), and serum lipids.

13 ement the growth phenotype in the absence of serum lipids.

14 his response independently of its effects on serum lipids.

15 r function, estrogen treatment in women, and serum lipids.

16 exhibit global perturbations of circulating serum lipids.

17 r sex, age, body mass index (BMI), and total serum lipids.

18 r hemoglobin A1c, fasting blood glucose, and serum lipids.

19 resistance and increased body adiposity and serum lipids.

20 ncreased with decreasing eGFR and decreasing serum lipids.

21 change in overall fatty acid composition in serum lipids.

22 Dietary sugars influence blood pressure and serum lipids.

23 Among subjects with serum lipid abnormalities who were not using a statin, h

24 nfection has been clinically associated with serum lipid abnormalities, yet our understanding of the

25 be associated with retinal hard exudate and serum lipid abnormalities.

26 hoblastic leukemia treatment arm, body mass, serum lipids, albumin and cortisol levels, dexamethasone

27 In adults, male sex, serum lipids, alcohol consumption, and age were positive

28 e, obesity, diabetes, hypertension, smoking, serum lipids, alcohol intake, and physical activity.

29 ipid-class fatty acid profile indicated that serum lipid alterations were localized to the cholestero

30 ism, located proximal to the IL28 gene, with serum lipid and apolipoprotein levels in 746 subjects wi

31 ict DXA total and regional body composition, serum lipid and diabetes markers, and functional strengt

32 3DO PCs improve prediction of serum lipid and diabetes markers, and functional strengt

33 ription factor involved in the regulation of serum lipid and glucose levels, has recently been associ

34 Serum lipid and glycemic indexes were evaluated biweekly

35 Mean serum lipid and lipoprotein concentrations and plasma tr

36 w examines the effects of n-3 fatty acids on serum lipid and lipoprotein concentrations in seven spec

37 n apo E(-/-)/LDLR(-/-) mice had no effect on serum lipid and lipoprotein concentrations, although it

38 Serum lipid and lipoprotein levels were quantified using

39 d plaque regression is associated with lower serum lipids and ACEI in patients after TX.

40 Serum lipids and blood pressure measurements were availa

41 icant benefits to participants with elevated serum lipids and blood pressure.

42 ytoin and carbamazepine produce increases in serum lipids and C-reactive protein, as well as decrease

43 Serum lipids and factor VII were measured in both studie

44 icated many genes previously associated with serum lipids and found previously recognized association

45 SO) against ADRP, and effects on hepatic and serum lipids and glucose homeostasis were examined.

46 iants across the genome for association with serum lipids and have generated novel hypotheses about t

47 lude glucose tolerance, appetite regulation, serum lipids and inflammatory markers.

48 ietary factors explain ethnic differences in serum lipids and insulin profiles in children, independe

49 low calorific value also beneficially lower serum lipids and lipid deposition in animals fed on athe

50 Although serum lipids and lipoprotein profiles did not differ bet

51 d plant oils (alpha-linolenic acid) on human serum lipids and lipoproteins are reviewed.

52 ents (85 type 1 and 139 type 2) and assessed serum lipids and lipoproteins from fasting blood, skin r

53 Bilberries caused beneficial changes in serum lipids and lipoproteins in group B, whereas the op

54 We measured serum lipids and lipoproteins, fasting plasma glucose an

55 Serum lipids and lipoproteins, glycosylated hemoglobin,

56 used to examine the effects of the diets on serum lipids and lipoproteins: AAD [34% fat; 16% saturat

57 ciated with health effects such as increased serum lipids and liver enzymes, decreased vaccine respon

58 Serum lipids and markers of inflammation (C-reactive pro

59 After each 4-wk diet, serum lipids and oral glucose tolerance were measured.

60 The association between serum lipids and periodontal disease has been studied pr

61 Fasting blood sugar, serum insulin, fasting serum lipids and serum alanine aminotransferase (ALT) le

62 Neither drug altered serum lipids and the marked increase in vascular express

63 associated with incident CVD, independent of serum lipids and traditional CVD risk factors, in ethnic

64 Sustained elevations of serum lipids and/or pro-inflammatory cytokines may have

65 After adjustment for age, gender, race, serum lipids, and baseline of the dependent variable, th

66 subcutaneous adipose tissue, blood pressure, serum lipids, and fasting plasma glucose) were collected

67 (mostly mild or moderate), higher levels of serum lipids, and greater decreases from baseline in bon

68 by euglycemic clamp, blood pressure, fasting serum lipids, and insulin at mean 15 and 22 years of age

69 fects on arterial stiffness, blood pressure, serum lipids, and plasma PS concentrations were investig

70 and end of study as well as change in HbA1c, serum lipids, and renal and hepatic function.

71 In adults, high blood pressure (BP), adverse serum lipids, and smoking associate with cognitive defic

72 e greater with greater age, body mass index, serum lipids, and the use of supplements containing vita

73 Higher serum lipids are associated with increased risk of CSME

74 Serum lipids are associated with SVR, although these par

75 rum lipids during therapy, determine whether serum lipids are associated with virological response, a

76 In serum, lipids are the largest class of periodic metaboli

77 cause R,R-19a produced only a weak effect on serum lipids as compared with niacin, we conclude that t

78 ost severe steatosis and highest hepatic and serum lipids as well as insulin resistance among the eig

79 e conducted a lipidomic analysis to identify serum lipids associated with AAA presence.

80 Outcome data were serum lipids at baseline and ASVD deaths over 10 y (13,6

81 However, the beneficial effects on serum lipid biomarkers, blood pressure, and E-selectin o

82 m any effect of vitamin D supplementation on serum lipids, blood pressure, or CRP in healthy 5- to 7-

83 BP, serum lipids, body mass index, and smoking were assessed

84 Serum lipids, body weight, and glucose metabolism were t

85 increases in hyperglycemia and decreases in serum lipids, body weight, and SAT, with preserved VAT a

86 showed small effects on lipid intakes and on serum lipids, but no adverse effects.

87 rculation longer in participants with higher serum lipids, but the (2)H-alpha-tocopherol absorbed was

88 f RCTs, coffee was associated with a rise in serum lipids, but this result was affected by significan

89 tion likely contributes toward the uptake of serum lipids by infected hepatocytes.

90 Serum lipid, cholesterol, and lipoprotein contents are i

91 prevalence of adolescents with one abnormal serum lipid component was 43.2% and was significantly di

92 ND PARTICIPANTS: Cross-sectional analysis of serum lipid concentrations among 16,116 youths aged 6 to

93 onal data suggest there were improvements in serum lipid concentrations among US children and adolesc

94 luded strategies intended to improve overall serum lipid concentrations among youths.

95 Fasting serum lipid concentrations and body composition [total,

96 The relation between birth weight and serum lipid concentrations at age 7 through 11 years was

97 endpoints were the mean percentage change in serum lipid concentrations from baseline to week 12; the

98 Improving serum lipid concentrations in adults over 65 y of age wi

99 the influence of esterified plant sterols on serum lipid concentrations in adults with mild-to-modera

100 in use and whole-grain intake in relation to serum lipid concentrations in adults.

101 ace-, sex-, and age-specific upper decile of serum lipid concentrations in children born with low bir

102 effect of dietary behenic acid (behenate) on serum lipid concentrations in humans is assumed to be ne

103 Adjustment for serum lipid concentrations partly attenuated these assoc

104 ir was associated with greater reductions in serum lipid concentrations than was continuation of lopi

105 988-1994 and 2007-2010, a favorable trend in serum lipid concentrations was observed among youths in

106 Mean serum lipid concentrations were extremely low: total cho

107 s included weight at 3, 6, and 12 months and serum lipid concentrations, blood pressure, urinary keto

108 t anterior descending coronary artery (LAD), serum lipid concentrations, serum thiocyanate (for smoki

109 Key safety endpoints were change in fasting serum lipid concentrations, the incidence of adverse eve

110 With additional adjustment for serum lipid concentrations, vitamin E concentrations wer

111 on of the effects of alpha-linolenic acid on serum lipid concentrations.

112 cluded age, body mass index, blood pressure, serum lipids, diabetes, family history of myocardial inf

113 nhibitor studies and the transplant studies, serum lipids did not differ significantly between groups

114 ment for energy intake, body mass index, and serum lipids did not impact these relations.

115 Changes in serum lipids differed significantly between the groups.

116 viral therapy was associated with changes in serum lipids during and after antiviral therapy, with th

117 udy were to evaluate baseline and changes in serum lipids during therapy, determine whether serum lip

118 ion, adjusting for age, reference date year, serum lipids, education, race/ethnicity, smoking, and al

119 ng for age, body mass index, serum cotinine, serum lipids (except for PFCs), and study site (Michigan

120 Ethnic differences in serum lipids exist and appear to be associated with diff

121 e diets had no significant adverse effect on serum lipid, fasting serum glucose, and fasting serum in

122 fore and 6 weeks after the switch to measure serum lipid fractions, lipoprotein(a), C-reactive protei

123 analysis of 188 patients, changes in fasting serum lipids from baseline to month 12 were compared bet

124 of the cholesterol measures, statin use, or serum lipid genes and any of the AMD outcomes in the met

125 cumference, body mass index, blood pressure, serum lipids, glucose, and insulin levels.

126 waist circumference, blood pressure, fasting serum lipids, glucose, and insulin were measured, and ov

127 Serum lipids, glucose, insulin, bile acids, and endothel

128 The correlation of HbA1C and serum lipid (HDL, LDL, total cholesterol, and triglyceri

129 regularly assessed for socioeconomic status, serum lipids, high-sensitivity C-reactive protein (hs-CR

130 DeltaEC mice had reduced adiposity, impaired serum lipid homeostasis, and a higher respiratory exchan

131 We measured serum lipid, homocysteine, folate, and vitamin B-12 conc

132 Flow-mediated dilatation (FMD), FGF-23, serum lipid, hsCRP levels, BMI and HOMA were assessed.

133 ct in bile approximately 2-fold and elevated serum lipid hydroperoxides approximately 4-fold.

134 ic exposure did not significantly affect the serum lipids in AB-treated mice.

135 OBJECTIVE To examine trends in serum lipids in adults between 1988 and 2010.

136 e effect of reasonable intakes of cashews on serum lipids in adults with or at risk of high LDL chole

137 ptor may have a role in regulating levels of serum lipids in animal models of obesity and insulin res

138 Almonds had beneficial effects on serum lipids in healthy adults and produced changes simi

139 tion regarding the importance of the role of serum lipids in patients with elevated serum lipid level

140 utic role for PPARbeta in the improvement of serum lipids in the setting of metabolic syndrome.

141 temporal profiles of fasting blood glucose, serum lipids including triglycerides, cholesterol profil

142 Pre- and post fasting blood was drawn for serum lipid, insulin, 8-isoprostaglandin F(2alpha) (8-is

143 rols Hfe(-/-) mice exhibit no differences in serum lipid, insulin, glucagon, or thyroid hormone level

144 Cardiovascular disease risk factors (serum lipids, insulin, glucose, and cortisol) were asses

145 the relative proportion of omega-6 PUFAs in serum lipids is inversely related to the incidence of me

146 Serum lipid, leptin, adiponectin, insulin, total and dir

147 nce, skinfold thickness, blood pressure, and serum lipid, leptin, and C-reactive protein levels at ag

148 ilar approach involving standardization plus serum lipid level adjustment generally performed well.

149 This study was conducted to determine serum lipid level changes in patients who received cloza

150 Serum lipid level testing and influenza vaccine administ

151 ence creatinine (e.g., age and hydration) or serum lipid levels (e.g., body mass index and recent fat

152 possibly represent a return to preinfection serum lipid levels after accounting for expected age-rel

153 ination Survey has shown favorable trends in serum lipid levels among children and adolescents aged 6

154 The mean peak serum lipid levels among patients in the sirolimus group

155 is also associated with favorable changes in serum lipid levels and an improvement in glycemic contro

156 TCDD) alters glucose transport and increases serum lipid levels and blood pressure.

157 idence that genetic variants associated with serum lipid levels and body mass index influence CAC lev

158 ide Y gene has been associated with elevated serum lipid levels and cardiovascular disease.

159 We evaluated the relationships between serum lipid levels and clinically significant macular ed

160 ntify novel loci predisposing to unfavorable serum lipid levels and coronary heart disease (CHD).

161 The relationship of serum lipid levels and diabetic retinopathy has interest

162 le of serum lipids in patients with elevated serum lipid levels and diabetic retinopathy.

163 l gene transfer resulted in normalization of serum lipid levels and in the clearance of apo E-contain

164 Serum lipid levels and lipoprotein distributions did not

165 onsumption has been linked to a reduction in serum lipid levels and oxidative stress.

166 the effects of experimentally induced IH on serum lipid levels and pathways of lipid metabolism in t

167 o be more than a casual relationship between serum lipid levels and systemic health (particularly car

168 and haplotypes are associated with elevated serum lipid levels and the metabolic syndrome (P = 0.008

169 mmon HNF4A variants are associated with high serum lipid levels and the metabolic syndrome.

170 sults of this study suggest that ectopic and serum lipid levels are positively associated with bone m

171 oci have been reported to be associated with serum lipid levels at genome-wide statistical significan

172 4, fasting sample) for direct measurement of serum lipid levels before study treatments were started.

173 Serum lipid levels correlated with FEV1 only in the subj

174 Body weight and serum lipid levels did not change.

175 er adjustment for family medical history and serum lipid levels did not substantially change these re

176 lambda gene variant was also associated with serum lipid levels in chronic hepatitis C patients.

177 tive estrogen-receptor modulator, on fasting serum lipid levels in men receiving ADT for prostate can

178 ant correlations were found between MPOD and serum lipid levels or age.

179 steine was not significantly associated with serum lipid levels or family history of cardiovascular d

180 They had lower serum lipid levels than noncarriers (P < 0.05), had more

181 ly identification of novel regions linked to serum lipid levels that would have otherwise been missed

182 These findings link elevated serum lipid levels to a proinflammatory signaling cascad

183 The mice were killed, serum lipid levels were determined, and the eyes were ul

184 ceived that glucocorticoids adversely affect serum lipid levels, although results of prospective stud

185 Blood glucose, serum lipid levels, and arterial blood pressure were wit

186 Body mass index, serum lipid levels, and hematocrit were strong phenotypi

187 Acute treatments with 25HC3S decreased serum lipid levels, and long-term treatments decreased h

188 Body weight, body composition, fasting serum lipid levels, and tolerability.

189 ms previously reported to be associated with serum lipid levels, CAD, and blood pressure.

190 r outcomes, including bone density and other serum lipid levels, did not change.

191 Serum lipid levels, especially triglyceride (TG) levels,

192 nonblack race, higher body mass index, lower serum lipid levels, HIV replication, low nadir CD4(+) ce

193 gle-nucleotide polymorphisms associated with serum lipid levels, hypertension, and CAD and tested the

194 trated no beneficial changes in body weight, serum lipid levels, hypothyroid symptoms as measured by

195 c loci for coronary artery calcification and serum lipid levels, one of the strongest risk factors fo

196 erences in plasma post-heparin LPL activity, serum lipid levels, or lipoprotein distribution between

197 typic association with type 2 diabetes, BMI, serum lipid levels, serum adiponectin levels, and measur

198 fter adjusting for age, sex, smoking status, serum lipid levels, systemic and dietary factors, and CF

199 iple regression model adjusted for age, sex, serum lipid levels, years of smoking, and pack-years, ye

200 f atherosclerosis in mice, without affecting serum lipid levels.

201 ene regions responsible for the variation in serum lipid levels.

202 istance, and other proatherogenic changes in serum lipid levels.

203 y macular edema, is associated with elevated serum lipid levels.

204 wing differential DNA methylation related to serum lipid levels.

205 trols were 32 healthy men and women for whom serum lipid, lipoprotein, and cytokine concentrations we

206 th a variety of critical illnesses, for whom serum lipid, lipoprotein, and cytokine concentrations we

207 ical records indicated that the CR group had serum lipid-lipoprotein and BP levels in the usual range

208 Subsequently, we monitored serum lipids, liver VLDL secretion, and hepatic expressi

209 nclude smoking cessation, exercise, lowering serum lipids, lowering high blood pressure, and daily an

210 in the diabetic and provide a rationale for serum lipid-lowering strategies in the treatment of diab

211 We show that serum and the serum lipid, lysophosphatidic acid (LPA), increased Cdc4

212 biological actions of the cell membrane and serum lipid lysophosphatidylcholine (LPC) in atheroscler

213 rine n-3 fatty acid) have similar effects on serum lipid markers at intakes recommended for primary p

214 ned to an intervention or control group, and serum lipids, markers of insulin sensitivity, and serum

215 Serum lipids may act as a mediator between dietary fiber

216 Recent studies suggest that serum lipids may be associated with treatment response.

217 l infarction; treatment-related increases in serum lipids may contribute to greater cardiovascular di

218 al periodontal parameters were measured, and serum lipids, MDA, and 8-OHdG levels were assessed in bl

219 nd 35 years were estimated by using repeated serum lipid measurements over 20 years in the CARDIA stu

220 y-low-density and high-density subfractions, serum lipid measures, glycolysis-related metabolites, ke

221 This study was designed to evaluate serum lipid metabolite changes that are associated with

222 imosis, skeletal malformation, and increased serum lipid metabolites.

223 secondary outcome measures included fasting serum lipids, microvascular reactivity, arterial stiffne

224 t understanding the mechanisms through which serum lipids modulate Abeta could offer new approaches t

225 of this study was to evaluate the effect on serum lipids of a yeast-derived beta-glucan fiber in 15

226 ecent observations concerning the effects of serum lipids on immune cell phenotype/function, and a he

227 asurement error bias caused by variations in serum lipids or by urinary diluteness, we recommend impr

228 in the absence of significant differences in serum lipids or insulin sensitivity.

229 care, there appeared to be little effect of serum lipids or statins on the incidence of proliferativ

230 Exposure of cells to serum, lipids, or the tumor promoter PMA suppressed form

231 rmal FA (and/or RD) in BD, and also examined serum lipid peroxidation (lipid hydroperoxides, LPH and

232 ned after 2 to 3 hours' postischemia and the serum lipid peroxide levels followed the same pattern.

233 B(1)) in liver plays a key role in increased serum lipid production, fatty liver, and possibly diet-i

234 dies have addressed the relationship between serum lipid profile and HOA.

235 designed to assess prospectively changes in serum lipid profile and myocardial perfusion with serial

236 stinct phenolic and fibre concentrations, on serum lipid profile and oxLDL of male Wistar rats fed a

237 The association between serum lipid profile and RHOA was modeled using multivari

238 aim was to determine the association between serum lipid profile and the incidence of radiographic ha

239 m oxidative stress and improved (p<0.05) the serum lipid profile in the high-fat dietary groups; mean

240 , transferrin and transferrin saturation and serum lipid profile on a routine follow-up examination.

241 Policosanol does not alter the serum lipid profile over an 8-wk period in adults with m

242 Serum lipid profile variables were analysed as continuou

243 psyllium seed husk (PSH)/d for 24 wk on the serum lipid profile were assessed in this randomized, do

244 ose tolerance (oral glucose tolerance test), serum lipid profile, and dietary intake.

245 0.10-1.45) but no improvement in the overall serum lipid profile, blood pressure, and glycemic contro

246 Rabbits serum lipid profile, hematology and histology were inves

247 to IH for five days to determine changes in serum lipid profile, liver lipid content, and expression

248 osing and are accompanied by improvements in serum lipid profile.

249 es investigating the association of NPs with serum lipid profile.

250 estigated the association of SFA intake with serum lipid profiles and ASVD mortality in a population-

251 Little is known about their serum lipid profiles and corresponding risk of cardiovas

252 n on host metabolism, we examined changes in serum lipid profiles and intrahepatic expression of lipi

253 a triglycerides, and implications of altered serum lipid profiles for atherogenic risk.

254 ive confounders is thus necessary to compare serum lipid profiles in clinical studies.

255 f target concentration, resulted in improved serum lipid profiles in kidney transplant recipients at

256 promote sustained weight loss and favorable serum lipid profiles in obese patients.

257 Our results demonstrate that serum lipid profiles need to be interpreted with caution

258 To further examine this possibility, serum lipid profiles of mice lacking a functional PPARbe

259 uced hypercholestrolemic rats for 28days and serum lipid profiles were estimated.

260 ession or knockdown of Tm6sf2 in mice alters serum lipid profiles, consistent with the association ob

261 ed with oral glucose tolerance tests (OGTT), serum lipid profiles, magnetic resonance imaging (MRI) f

262 0 single-nucleotide polymorphisms (SNPs), of serum lipid profiles, we identified a major linkage sign

263 relationship between glucocorticoid use and serum lipid profiles.

264 regional adiposity, thereby improving their serum lipid profiles.

265 dent from cardiometabolic factors, including serum lipid profiles.

266 ps of mice exhibited similar body weight and serum lipid profiles; however, both MKP-1(+/-) and MKP-1

267 Adverse effects include those on the liver, serum lipids, psyche/behavior and reproductive system.

268 4.2 mumol/L; P = 0.0019) and correlated with serum lipids (r = 0.4938, P = 0.0012).

269 hatidylcholine 18:0/18:1 (PC(18:0/18:1) as a serum lipid regulated by diurnal hepatic PPARdelta activ

270 Serum lipid-regulating therapies may reduce cardiovascul

271 CD4/CD8 ratio, smoking, comorbid conditions, serum lipids, renal function (estimated glomerular filtr

272 asis model assessment of insulin resistance, serum lipids, reproductive hormones, leptin, acylated gh

273 enetic variants that modify the variation in serum lipid response to dietary fat quality.

274 The 3 breakfasts resulted in similar serum lipid responses to the white-bread challenge.These

275 d patients had significant reductions in all serum lipids, SAT, VAT, and muscle.

276 The effects on BP, heart rate (HR), serum lipids, serum and urinary K(+), and tolerability w

277 Furthermore, serum lipids share a genetic predisposition with both ca

278 Although serum lipids (SLPs) and phenol red (PR) are ubiquitous c

279 ociations between smoking, fish consumption, serum lipids, systemic and dietary factors, the CFH sing

280 We studied data from serum lipids that were measured annually among the 1,441

281 l history, physical activity, adiposity, and serum lipids, the hazard ratio in the highest quartile w

282 mia due to its capacity to positively affect serum lipids to a greater extent than other currently ma

283 This study accounted for variability in serum lipid (total cholesterol and triacylglycerol) and

284 Serum lipids (total cholesterol, low-density lipoprotein

285 leotide polymorphisms associate with fasting serum lipid traits and whether the effects are modulated

286 on changes in metabolic parameters (glucose, serum lipids, triglycerides; total, low-density, and hig

287 f HCV-mediated upregulation of LDLR inhibits serum lipid uptake and thereby perturbs HCV replication.

288 ll, our findings highlight the importance of serum lipid uptake by infected hepatocytes in HCV life c

289 Alterations in serum lipid values have been widely reported among perso

290 nd the catalogue of variants responsible for serum lipid variation and, with a full catalogue of vari

291 nerated novel hypotheses about the causes of serum lipid variation.

292 Mediation of the relations by serum lipids was also investigated.

293 Data on blood pressure, serum lipids, weight, diabetes mellitus, smoking, and in

294 Fasting serum lipids were analyzed at baseline and during and af

295 ntal area under the curve (AUCi) method, and serum lipids were determined with the use of standard as

296 Serum lipids were measured.

297 Serum lipids were profiled via liquid chromatography tan

298 Similarly, it is standard to adjust for serum lipids when measuring lipophilic chemicals in seru

299 gen agonist-like actions on bone tissues and serum lipids while displaying potent estrogen antagonist

300 ght-loss regimens have a favorable impact on serum lipids without the accompanying ketosis and have t