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1 ry (EA) populations identified nine loci for serum magnesium.
2 xtend our understanding of the metabolism of serum magnesium.
3 ificantly correlated with increased level of serum magnesium.
5 ession models to assess associations between serum magnesium and radiographic subarachnoid hemorrhage
6 of HF is greater among individuals with low serum magnesium and those with high serum phosphorus and
10 owed a statistically significant increase in serum magnesium concentration at 6 hrs when compared wit
11 ficant trends in fracture risk in men across serum magnesium concentration groups were apparent for s
12 together explained 2.8 % of the variance in serum magnesium concentration in ARIC African-American p
13 loci that explained 2.8% of the variance in serum magnesium concentration in ARIC African-American p
14 se and MUC1 and between insulin and TRPM6 on serum magnesium concentration in ARIC European-American
15 m-glucose cotransporter 2 inhibitors elevate serum magnesium concentration in patients with or withou
19 75-675/> 675 ng/gHb respectively) (99%), low serum magnesium concentrations (< 0.75 mmol/L) (59%), an
21 yte magnesium concentrations correlated with serum magnesium concentrations (r(s) = 0.338, p < 0.05)
22 001) and basal ETKA (p < 0.05) increased and serum magnesium concentrations decreased (p < 0.001).
23 ole blood TDP and basal ETKA increased while serum magnesium concentrations decreased, indicating inc
25 um potassium concentrations of 3.5 mmol/L or serum magnesium concentrations of < 1.8 mg/dL (< 0.74 mm
27 ine magnesium concentrations, the changes in serum magnesium concentrations were not significantly di
29 mmon practice in critical care to supplement serum magnesium for the purpose of preventing episodes o
34 ere to identify genetic loci associated with serum magnesium in an African-American (AA) population u
35 educes kidney magnesium wasting and restores serum magnesium in cisplatin-treated rats, likely throug
37 duals within NIPAL1 families suggested lower serum magnesium in NPC compared to unaffected members.
40 highest frequency rate (72%) and lowest mean serum magnesium level (0.66 +/- 0.17 mmol/L) in patients
41 wed an inverse relationship between baseline serum magnesium level and NODAT (hazard ratio [HR], 1.24
43 r results suggest that lower post-transplant serum magnesium level is an independent risk factor for
45 te to severe shivering (p = 0.04), and lower serum magnesium levels (p = 0.01) were associated with g
46 f rs17251221 was also associated with higher serum magnesium levels (P = 1.2 * 10(-3)), lower serum p
53 iosis and craniofacial anomalies should have serum magnesium levels monitored closely after surgery.
54 ate ratios of ischemic stroke for those with serum magnesium levels of <or=1.5, 1.6, 1.7, and >or=1.8
60 an for 3 months moving at 3-month intervals) serum magnesium levels while adjusting for potential con
63 participants, GDs were associated with lower serum magnesium, lower educational achievement, and high
66 nterval, 6.7-11.9) in the lowest quartile of serum magnesium (</=1.77 mg/dL) compared with 6.3 per 10
69 on (P>0.11 for all biomarkers), whereas both serum magnesium (P<0.001) and uric acid levels (P=0.008)
71 Magnesium doses were targeted to achieve serum magnesium ranges of 1.0-1.85 mmol/L or 1.25-2.5 mm
72 further work is required to identify optimal serum magnesium targets for atrial fibrillation prophyla
73 ntensive care unit (ICU) patients undergoing serum magnesium testing was conducted in 93 ICUs across
77 correlate with hypomagnesemia and to predict serum magnesium values in critically ill pediatric patie
83 heart disease, impaired renal function, and serum magnesium, was identified as an independent risk f
84 odels, individuals in the lowest quartile of serum magnesium were ~50% more likely to develop AF (adj
85 e kidney plays a central role in maintaining serum magnesium within a narrow range (0.70-1.10 mmol/L)