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1  by liver biopsy, transient elastography, or serum markers).
2 s instituted prospectively on the basis of a serum marker.
3 CT perfusion parameters with tumor grade and serum markers.
4 relation between CT perfusion parameters and serum markers.
5          Parameters were correlated with HCC serum markers.
6 ondiagnostic electrocardiograms and negative serum markers.
7 ssion was documented by biopsy or increasing serum markers.
8 sociations between cardiac SSc and candidate serum markers.
9 sociations between cardiac SSc and candidate serum markers.
10 ns, however, unclear and we lack mechanistic serum markers.
11 with further improvement upon integration of serum markers.
12 lates with the body mass index and metabolic serum markers.
13 d by improvements in noninvasive imaging and serum markers.
14 r cannot explain racial disparities in these serum markers.
15 avior were comparable with that of other IBD serum markers.
16 ere documented in blood eosinophil counts or serum markers after eHF intake.
17              Malnutrition was measured using serum markers (albumin <3.5 g/dL) as well as body mass i
18            However, when the values of known serum markers alpha fetoprotein, des-gamma carboxyprothr
19                                              Serum markers also appear to provide useful prognostic i
20  patients with advanced fibrosis as a single serum marker and in combination with APRI and FIB-4.
21 naire and metabolic risks were determined by serum markers and anthropometric measures at pre- and po
22 sure mitochondrial absorbance, infarct size, serum markers and apoptotic index.
23 c inflammation was assessed by histology and serum markers and fibrosis by collagen proportionate are
24                  Secondary outcomes included serum markers and histologic measures of fibrosis improv
25                      At the current writing, serum markers and imaging methods are available and incr
26                             A combination of serum markers and imaging tools appears useful in reduci
27 ltahepa) or control mice, based on levels of serum markers and microscopic and histologic analysis of
28 nd still enrolling HeadSMART II (Head Injury Serum Markers and Multi-modalities for Assessing Respons
29                     The relationship between serum markers and quantitative hepatic collagen content
30 e of the newer noninvasive methods including serum markers and radiologic tests.
31 tion for treatment as they rely primarily on serum markers and radiologist-defined imaging features.
32 ve adverse vascular effects not reflected in serum markers and that nonlipid macronutrients can modul
33 eviews that assessed the association between serum markers and the presence of and/or predicting the
34 lgorithms combining standard ultrasound with serum markers and transient elastography (TE) for detect
35  A sequential approach or the combination of serum markers and transient elastography is able to sign
36 se, including quantitative imaging features, serum markers, and functional biomarkers, is needed to o
37 ue to frequent late-stage diagnosis, lack of serum markers, and limited information regarding biliary
38 dditional parameters including tumor biopsy, serum markers, and subclassification of current staging
39 s of recovery, such as genetic associations, serum markers, and the impact of medical therapy or vent
40                         Available diagnostic serum markers are not sensitive or specific enough, and
41      Ghr(-/-);Mdr2(-/-) mice showed elevated serum markers associated with liver damage and cholestas
42                                              Serum markers associated with the acute proinflammatory
43        Guidelines recommend that testing for serum markers be repeated every 12-24 weeks during antiv
44                                More-reliable serum markers, better tumour localisation and identifica
45 nflammatory markers, and neurological-damage serum markers between never-ventilated subjects, subject
46   We compared YKL-40 with two ovarian cancer serum markers, CA125 and CA15-3, for the detection of ea
47                                        These serum markers can be used as a non-invasive method in th
48 erated diagnostic protocols with new cardiac serum markers can detect myocardial ischemia or infarcti
49 ine decarboxylase (HDC) gene expression; and serum markers (CCL2, CCL5, CCL11, IL-3, and thymic strom
50 od to assess the prognostic value of CTC and serum marker changes during treatment.
51                                              Serum marker determinations at baseline and 60-min after
52                                     Frequent serum marker determinations to estimate marker decline d
53                 One hour after intervention, serum markers did not differ between interventions.
54 vs non-neutrophilic asthma phenotypes, while serum markers did not differ.
55    The relationships between IL-23 blockade, serum markers downstream of IL-23 signaling, and withdra
56 nd magnetic resonance elastography (MRE) and serum markers e.g. APRI and FIB-4 scores were assessed a
57 hy scans, bronchoalveolar lavage and various serum markers (e.g., surfactant protein D and KL-6) each
58 es emphasized are computer-generated models, serum markers, echocardiography, and nuclear imaging in
59                               Among nonlipid serum markers examined, only C-reactive protein levels a
60 gression including metabolomics, circulating serum markers, exercise physiology, and both structural
61 aimed to reveal a potential new inflammatory serum marker for assessing disease activity and severity
62 ific antigen (PSA) has been widely used as a serum marker for cancer of the prostate.
63 e cancer (PCa) and is the most commonly used serum marker for cancer.
64                            PCT is a reliable serum marker for determining the presence or absence of
65 Chromogranin A appears to be the most useful serum marker for diagnosis, staging, and monitoring.
66               We have identified a potential serum marker for glioblastoma multiforme (GBM) using mic
67                             The conventional serum marker for HB, alpha-fetoprotein (AFP), has its li
68  concluded that GPC3 is inferior to AFP as a serum marker for HB.
69  increasing grade of ACR and may be a useful serum marker for intestinal rejection.
70                            There is no known serum marker for intestinal rejection.
71 d and neck cancer and represents a promising serum marker for monitoring affected patients.
72 alyl-Lewis a), which is used as a prognostic serum marker for pancreatic cancer, and its isomer sialy
73  results merit further investigation of this serum marker for potential diagnostic and prognostic pur
74 tate-specific antigen (PSA) is a widely used serum marker for prostate cancer (PCa) but has limited s
75 tate specific antigen (PSA) is a widely used serum marker for prostate cancer (PCa), but has limited
76 tate-specific antigen (PSA) is a widely used serum marker for prostate cancer (PCa), but in the criti
77 , previously known as CA125, is a well-known serum marker for the diagnosis of ovarian cancer and has
78  the potential of this protein to serve as a serum marker for the early stage diagnosis of PC.
79                  ApoM levels may be a useful serum marker for the identification of MODY3 patients.
80             Methods for the determination of serum markers for bone and cartilage destruction, as wel
81 veloped massive cardiac damage as defined by serum markers for cardiomyocyte cell death, electrocardi
82 y in hematopoietic tissues and are potential serum markers for certain hematopoietic malignancies.
83            This study sought to determine if serum markers for collagen I and III synthesis, the carb
84  CCSA-3 and CCSA-4 show promise as potential serum markers for detection of colorectal cancer and adv
85 the proposed intervention and identify early serum markers for each of those subgroups.The mathematic
86 ents (30%) and 3 of 59 controls (5%) without serum markers for HBV infection.
87 tly, there remains a great need for reliable serum markers for IBD.
88 nd inflammatory responses and serve as early serum markers for monitoring acute allograft rejection.
89 ntibodies against citrullinated proteins and serum markers for osteoclast-mediated bone resorption in
90               Her white blood cell count and serum markers for ovarian cancer were normal (alpha-feto
91                                 Detection of serum markers for pancreatic cancer has been elusive.
92 ometry successfully identified 154 potential serum markers for pancreatic cancer.
93 large-scale proteomics to identify potential serum markers for pancreatic cancer.
94  to controls, Fic(-/-) mice exhibit elevated serum markers for pancreatic dysfunction and display enh
95 ts suggest that both CYT-MAA and HMW-MAA are serum markers for residual melanoma in patients with res
96                                              Serum markers for the diagnosis of early HCC (<2 cm in d
97  and a literature search to select candidate serum markers for the diagnosis of lung cancer.
98           The measurement of infarct size by serum markers has multiple theoretical and practical lim
99  in whom postoperative elevations of cardiac serum markers have been correlated to medium- and long-t
100 ge of targeted therapies and as a diagnostic serum marker in cancer, is confounded by its variable tu
101 o support recommendations for using nonlipid serum markers in decisions regarding statin therapy for
102                  The chosen reviews assessed serum markers in GC, and the quality assessment was cond
103                             The investigated serum markers in the studies included SOD activity, sele
104 imaging data were supported by modulation of serum markers in vitro and ex vivo histopathology.
105                                              Serum markers include chromogranin A for neuroendocrine
106 rol and rmTSG-6-treated animals when various serum markers (including pro- and anti-inflammatory cyto
107                                        Novel serum markers, including C-reactive protein and homocyst
108                                  Analysis of serum markers indicated that 95% (84/88) had evidence of
109 neoplasia could make this protein a possible serum marker indicating the presence of high-risk premal
110        Inflammatory responses (physiological/serum markers, innate-signaling transcripts) are therefo
111 ly history of liver cancer and hepatitis B/C serum markers is associated with an over 70-fold elevate
112         Use of Feno values, bEOS counts, and serum marker levels (eg, CCL26 and CCL17) in combination
113                 Furthermore, proinflammatory serum markers may be used as inclusion criteria or endpo
114 ose a paradigm to improve the sensitivity of serum marker measurement by modifying them with an exter
115                                        Novel serum markers need to be explored.
116 ents were analyzed for cardiac troponin I, a serum marker not detected in the blood of healthy person
117 el candidate for development as a diagnostic serum marker of early stage colon cancer.
118                     FibroTest is an indirect serum marker of hepatic fibrosis.
119 nce of procalcitonin (ProCT), an established serum marker of infection, in saliva.
120                       A reliable noninvasive serum marker of IRA patency is desired to permit early i
121                    TT levels may be a useful serum marker of poor outcomes among Covid-19 patients.
122 e HIP1 autoantibody test may be an important serum marker of prostate cancer.
123                      sTac has been used as a serum marker of T cell activation in immune disorders an
124 eptide of calcitonin, has been shown to be a serum marker of the severity and mortality of several sy
125 se Neuroimaging Initiative-1 (ADNI1) cohort, serum markers of 5-HT, tryptophan, and Kyn were measured
126 gned to assess safety, pharmacokinetics, and serum markers of angiogenesis in patients with solid tum
127                                              Serum markers of angiogenic activity did not provide ins
128 e consistent hyposecretors of acid, most had serum markers of atrophic gastritis.
129 n total serum bile acid (BA) concentrations, serum markers of BA synthesis and steady-state pharmacok
130 ne levels were normal (35 +/- 21 pg/ml), the serum markers of bone formation (osteocalcin and bone-sp
131 on in bone, loss of osteoblasts, and reduced serum markers of bone formation, including osteocalcin a
132 rum markers of bone resorption, but elevated serum markers of bone formation.
133 ography, and bone turnover was quantified by serum markers of bone resorption and formation via enzym
134 eoclasts, Phlpp1 deficiency did not increase serum markers of bone resorption, but elevated serum mar
135 hose discontinuing alendronate had increased serum markers of bone turnover compared with continuing
136                                              Serum markers of bone turnover were also determined.
137 lasts, Shn3-deficient animals show decreased serum markers of bone turnover.
138 d the dynamics of, and relationship between, serum markers of brain injury [neurofilament light (NfL)
139                 In Step 1, we tested whether serum markers of cholesterol catabolism were associated
140  respective gene expression in liver and the serum markers of circadian function.
141 The goal of this study was to identify novel serum markers of colon cancers and precancerous colon ad
142 creatinine level, urinary protein level, and serum markers of disease activity.
143 -defined patient population for the study of serum markers of familial OA with respect to pathogenesi
144 hepatic venous pressure gradient (HVPG), and serum markers of fibrosis in 475 patients with NASH with
145 fness by MRE and transient elastography, and serum markers of fibrosis were measured at baseline and
146 orphometry), NAFLD Activity Score (NAS), and serum markers of fibrosis.
147 re, Braak score, and 11C-PiB retention, with serum markers of glucose homeostasis using grouped and c
148 oad, and a decrease in both GCF IL-1beta and serum markers of IL-6 response.
149 cell function, lymphocyte surface phenotype, serum markers of immunologic activation, and viral burde
150 rt Form 36 health survey questionnaire), and serum markers of inflammation (erythrocyte sedimentation
151                                              Serum markers of inflammation (high-sensitivity C-reacti
152 y, high viral loads, and increased levels of serum markers of inflammation and hepatic/renal injury.
153                                              Serum markers of inflammation and macrophage activation.
154                                              Serum markers of inflammation and organ injuries that pe
155                                              Serum markers of inflammation and oxidation were also me
156  lack of coagulopathy, and reduced or absent serum markers of inflammation and/or hepatic/renal funct
157 atypical chemokine receptor allele increases serum markers of inflammation could lead to novel therap
158     Hormone therapy has divergent effects on serum markers of inflammation in women with CAD.
159                                   Anemia and serum markers of inflammation were present in all cases.
160 roups had similar fecal microbiota profiles, serum markers of inflammation, and levels of neurotrophi
161 s, and HCMV-specific immunoglobulin G (IgG), serum markers of inflammation, and mycobacterial antibod
162 fecal microbiota, urine metabolome profiles, serum markers of inflammation, neurotransmitters, and ne
163  erythematous left index finger and elevated serum markers of inflammation.
164 rains and was accompanied by an elevation of serum markers of insulin resistance, including increases
165                                              Serum markers of kidney injury, BUN, creatinine, and neu
166 tion analysis to address the contribution of serum markers of liver damage, high aspartate (AST, >49.
167                                 Knowledge of serum markers of liver decompensation would facilitate c
168 d is sufficient for preventing elevations in serum markers of liver dysfunction in this population un
169                                              Serum markers of liver function were used to evaluate LR
170 id not modulate baseline characteristics and serum markers of liver injury in CHC patients.
171 ung age, male sex, and advanced disease, and serum markers of liver injury, particularly bilirubin an
172  high-resolution CT (HRCT) abnormalities and serum markers of lung fibrosis.
173 rmined that DPD in mice and humans increases serum markers of metabolic health.
174 e displayed increased inflammation, enhanced serum markers of myocardial damage, and an increased inf
175 st groups with a similar trends observed for serum markers of myocardial injury and apoptotic index.
176                                              Serum markers of myocardial injury and inflammation (tro
177  aim of this study was to assess the role of serum markers of myocardial necrosis after cardiac surge
178 growth and should be considered as potential serum markers of neoplastic prostate diseases.
179 osition, apoptosis, histologic features, and serum markers of oxidative stress (OS) and cell death in
180  with stable plaques, and may correlate with serum markers of plaque instability and inflammation.
181                                              Serum markers of renal and cardiac function, oxidative s
182  to examine associations between SBI and two serum markers of renal function: Serum creatinine (SCr)
183                                              Serum markers of renal injury were significantly decreas
184   Currently, quantification of pretransplant serum markers of the HBV antigen load does not predict t
185 chemokine 10, in parallel with depression of serum markers of the myeloid cell activation, such as CC
186 ve developed a process for identification of serum markers of this disease based upon standardized fr
187 xtures of urinary phthalate metabolites with serum markers of thyroid function and autoimmunity.
188                                  Established serum markers of tumor spread were measured serially and
189 cardiolipin IgG is associated with increased serum markers of vascular inflammation, and IgG purified
190 weeks of gestation, did not reduce systemic (serum) markers of inflammation.
191                  Additionally, the origin of serum markers often cannot be localized to a specific ce
192 umor cells along with reduction in malignant serum markers (osteopontin, Cxcl12) were noted.
193       To date, a number of methods including serum marker panels and ultrasound-based transient elast
194                                              Serum marker panels, initially developed for determining
195 iew is broad and includes topics such as the serum marker procalcitonin, gene expression profiling, m
196   A standard amphotropic vector expressing a serum marker protein, human alpha 1-antitrypsin, was inf
197                                              Serum markers provide an alternative way to monitor the
198                  A disparate array of plasma/serum markers provides evidence for chronic inflammation
199                               Two additional serum markers rapidly renormalized after polypectomy: gr
200                                              Serum markers related to bone turnover, calcium, phospho
201                                              Serum markers such as myoglobin and creatine kinase, MB
202  Previous genetic studies of blood group and serum markers suggested that Jewish groups had Middle Ea
203 regarding the measurement of infarct size by serum markers, technetium-99m sestamibi single-photon em
204 mes, it is desirable to identify a sensitive serum marker that is closely related to the degree of my
205 proteomic analysis in an attempt to discover serum markers that can assist in the early detection of
206               Finally, we sought to identify serum markers that differentiate SIRS with and without i
207                 Longitudinal physiologic and serum markers, time of death.
208         Although PSA is the most widely used serum marker to detect and follow patients with prostati
209       We assessed the added value of CTCs or serum markers to prognostic clinicopathological models i
210 n characterized in mastocytosis, such as the serum marker tryptase and the immune checkpoint molecule
211   Assessment of liver fibrosis with multiple serum markers used in combination is sensitive, specific
212 sent, specificity and sensitivity of current serum markers used to diagnose PSC are limited and often
213 iagnosis of GC, this umbrella focuses on the serum markers valuable in GC, for whether detecting or p
214           A panel of 10 diabetes and obesity serum markers was determined using MAGPIX.
215 d the slope of increase over 60 min for each serum marker were significantly higher in patients with
216 of detection of Down's syndrome for the five serum markers were as follows: 17 percent for alpha-feto
217          CCL2 levels (but not those of other serum markers) were significantly higher during anaphyla
218 ated significantly and better than any other serum marker with apoptosis and liver damage, such as ba
219 ent of cardiac troponin I (cTnI), which is a serum marker with high sensitivity and specificity for c
220                     The integration of these serum markers with clinical parameters into a new multi-
221              The stronger correlation of the serum markers with Ishak scores suggests that serum fibr
222 in saturation (SaO(2)) during sleep] and all serum markers with pain thresholds and tolerances at bas
223 ular epithelial cells, and lower cholestasis serum markers within 2 weeks after DDC treatment.
224 assess whether the immediate availability of serum markers would increase the appropriate use of thro

 
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