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1 uate nucleus (ARC) to maintain low levels of serum prolactin.
2 nships between changes in CSF monoamines and serum prolactin and clinical outcome were examined, and
5 and spike duration of TIDA cells, increased serum prolactin can promote dopamine release to limit it
6 s were assessed by measuring the increase in serum prolactin concentration during 5 hours following s
7 osa, the fenfluramine-stimulated increase in serum prolactin concentration was significantly less tha
8 verse events, extrapyramidal symptom scores, serum prolactin concentration, body weight, and metaboli
10 SLE patients display hyperprolactinemia, and serum prolactin correlates with disease activity in some
12 nd cyclosporine are reported to increase the serum prolactin level, producing gynecomastia in men.
13 in, antiparkinson medication use, akathisia, serum prolactin level, QTc prolongation, and sedation.
19 at persistent mild-to-moderate elevations in serum prolactin levels induce a break in self tolerance
21 ential effects of sustained "high" and "low" serum prolactin levels on bone mineral density and perip
24 rolactin levels and a decrease in endogenous serum prolactin levels, indicating that ovine prolactin
25 e alpha-2 receptor subtype within the PVN on serum prolactin levels; however, the functional signific
26 significantly higher maximum mean levels of serum prolactin (men, 34.56 microg/L [95% CI, 29.75-39.3
28 pment was analyzed by selectively increasing serum prolactin (PRL) concentration in thymus-grafted co
29 udy was to evaluate the relationship between serum prolactin (PRL) concentrations and prolactinoma si
33 d 3-methoxy-4-hydroxyphenylglycol (MHPG) and serum prolactin were measured during drug-free and antip
34 th more weight gain and greater increases in serum prolactin, whereas haloperidol decanoate was assoc