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1 osal tissues (the main HIV-1 entry portal on sexual transmission).
2 st immune cells that HIV-1 encounters during sexual transmission.
3 t immune cells to interact with HIV-1 during sexual transmission.
4 anogenital stratified squamous mucosa during sexual transmission.
5 ils that are exploited by HIV to promote its sexual transmission.
6 the first cell types to encounter HIV during sexual transmission.
7 evention strategies to protect against HIV-1 sexual transmission.
8 immunodeficiency virus (HIV) at the point of sexual transmission.
9 1 entry to prevent early dissemination after sexual transmission.
10 IV-1 infection may be a determinant of HIV-1 sexual transmission.
11 e ingredient of a microbicide to prevent HIV sexual transmission.
12 eports regarding the evidence for or against sexual transmission.
13 preciated role in decreasing the rate of HIV sexual transmission.
14 irus propagation and dissemination following sexual transmission.
15 of microbicides as a method to prevent HIV-1 sexual transmission.
16 s HIV trans-infection and may play a role in sexual transmission.
17 nt as a vaginal microbicide to prevent HIV-1 sexual transmission.
18 er human immunodeficiency virus (HIV) during sexual transmission.
19 bility to human immunodeficiency virus (HIV) sexual transmission.
20 nea pigs received approximately 10(2) IFU by sexual transmission.
21 ts of a topical microbicide to prevent HIV-1 sexual transmission.
22 mains essential to understanding the risk of sexual transmission.
23  both rapid disease progression and enhanced sexual transmission.
24 ce that HIV-1 selection indeed occurs during sexual transmission.
25 iruses in its ability also to spread through sexual transmission.
26              The epidemiology of BV supports sexual transmission.
27 he female lower genital tract barrier during sexual transmission.
28 avoid adaptive immunity - to allow efficient sexual transmission.
29 in may contribute to the prevention of HIV-1 sexual transmission.
30 ecent studies have shown an increase through sexual transmission.
31 the male reproductive system poses a risk of sexual transmission.
32 ert selective pressure on the viruses during sexual transmission.
33 es should focus on pathogenesis and modes of sexual transmission.
34 d to investigate potential mechanisms of HIV sexual transmission.
35 on from recovered patients have been through sexual transmission.
36 h injecting drugs in 2018, with male-to-male sexual transmission accounting for a continuously increa
37 onhospital healthcare (clinic) settings; and sexual transmission among HIV-infected persons.
38 t a reported travel history, consistent with sexual transmission among men who have sex with men.
39 infection in the male reproductive tract and sexual transmission, an ability to cross the placenta du
40 ons may help explain the high rates of HIV-1 sexual transmission and accelerated HIV-1 disease progre
41  very different in the anatomical context of sexual transmission and begin to explain the lack of sti
42 ptimize treatment, and the relative roles of sexual transmission and endogenous infection in BV epide
43 ion in cerebrospinal fluid (CSF) and between sexual transmission and human immunodeficiency virus typ
44 in HIV-negative patients are consistent with sexual transmission and local immunity, whereas in HIV-p
45  public health issue due to their widespread sexual transmission and oncogenic potential.
46                            The likelihood of sexual transmission and persistence of DRM was assessed
47                                              Sexual transmission and persistence of Zika virus (ZIKV)
48  are the first cells to encounter HIV during sexual transmission and their interaction with HIV may d
49  to expanded prevention programs targeted at sexual transmission and transmission among injecting dru
50  after the onset of illness and evidence for sexual transmission and viability of the viruses in seme
51 ates due to injection drug use and secondary sexual transmission and with an apparent West Coast focu
52 g either the need for a threshold of HCV for sexual transmission and/or variability in sexual practic
53 ation with abnormal fetal brain development, sexual transmission, and lack of a preventive vaccine ha
54 ave not as yet resulted in either disease or sexual transmission, and may represent benign endpoint i
55 us (EBOV) RNA persistence in semen, reported sexual transmission, and sporadic clusters at the end of
56                 Since viruses isolated after sexual transmission are mainly R5 viruses, this suggests
57 d mosquito-borne flavivirus, have identified sexual transmission as a new route of disease spread, wh
58 extracted and summarised with a focus on HIV sexual transmission at varying HIV viral loads.
59             The cluster likely resulted from sexual transmission between men having sex with men livi
60 k factors account for many cases of apparent sexual transmission between sexual partners; the prevale
61 njecting heroin users (NIUs) are a potential sexual transmission bridge to "lower risk" partners, 180
62 possibly, HCV-infected NIUs, are a potential sexual transmission bridge.
63  type 1 (HIV-1) load and reduces the risk of sexual transmission, but little is known about the effic
64 t with HHV-8 being primarily associated with sexual transmission, but the HHV-8 seropositivity rate i
65 al data strongly support the hypothesis that sexual transmission by acutely infected individuals has
66 ine the approximate infection dose in actual sexual transmission by comparing the kinetics of infecti
67 -1 is suggested to be a major contributor to sexual transmission by mucosal routes.
68 ated research during disease outbreaks where sexual transmission can have a key role.
69                                    Following sexual transmission, Chlamydia trachomatis specifically
70                  These findings suggest that sexual transmission constrains viral diversity at the po
71 ance mutations (DRM) may be a consequence of sexual transmission, de novo mutations, or technical err
72                         These data show that sexual transmission does not select for enhanced macroph
73 if infection of these organs is important in sexual transmission during long-term (chronic) infection
74                      Here we examine whether sexual transmission during multiple matings of queens is
75                          A suspected case of sexual transmission from a male survivor of Ebola virus
76 ter transmission were the consequence of (i) sexual transmission from the source, (ii) de novo emerge
77                                     Although sexual transmission has a relatively low contribution in
78 sions, bodily fluids, respiratory droplets), sexual transmission has also been theorized in the curre
79 ion to humans is through mosquito bites, but sexual transmission has also been well documented in hum
80                                        While sexual transmission has been documented in humans, and m
81 ut can persist in human semen and sperm, and sexual transmission has been documented.
82 alent in sub-Saharan Africa, but the role of sexual transmission has not been well characterized.
83                           Anecdotal cases of sexual transmission have been reported, and HCV nucleoti
84 complications such as ocular involvement and sexual transmission have been reported.
85           Among the women with a low risk of sexual transmission, HHV-8 seroprevalence was 0 percent
86         In addition to blood transfusion and sexual transmission, HTLV-1 is transmitted mainly throug
87 approach to decrease the efficiency of HIV-1 sexual transmission.IMPORTANCE In the absence of an effe
88                These data support a role for sexual transmission in HTLV-II infection.
89 f Chlamydia closely parallels C. trachomatis sexual transmission in humans and may be a good model sy
90  epididymal epithelial cells are critical to sexual transmission in immunocompromised mice.
91  strains of Campylobacter are circulating by sexual transmission in MSM populations across diverse ge
92  strains of Campylobacter are circulating by sexual transmission in MSM populations across diverse ge
93 raditionally at low risk for shigellosis via sexual transmission in MSM.
94 tted among injection drug users, the role of sexual transmission in the spread of HCV remains controv
95  which certain selfish genetic elements skew sexual transmission in their own favor.
96 results suggest an approach to prevent HIV-1 sexual transmission in which a virus-sensitizing microbi
97 majority of new HIV infections occur through sexual transmission, in which HIV is transferred from th
98  rate and mortality rate of mosquitoes while sexual transmission increases the risk of infection and
99 00), in which the percentage contribution of sexual transmission is 3.044% (95% CI: 0.123-45.73).
100           However, acute HIV-1 infection via sexual transmission is a low-probability event in which
101 ealth Service has estimated that the risk of sexual transmission is approximately 5%, well below the
102       Moreover, the fraction of cases due to sexual transmission is estimated below 4% of the cumulat
103 tment might relate to evidence that suggests sexual transmission is integral to the pathogenesis of b
104 xual men, but determining specific routes of sexual transmission is methodologically challenging, and
105                                              Sexual transmission is negligible in sex-partner studies
106                                  The risk of sexual transmission is probably lower than for Chlamydia
107                                              Sexual transmission is the main transmission route in AG
108                                              Sexual transmission is the most frequent (86%) route of
109 ly, a growing body of evidence suggests that sexual transmission is the predominant mode of transmiss
110            Increasing the rate of horizontal sexual transmission means that vector control rate or in
111                    To implement a horizontal sexual transmission model, male mice were inoculated in
112                              This provides a sexual-transmission model of human AIDS that can be used
113                                           If sexual transmission normalized by July 2021 and HIV care
114  conceptual framework for the male to female sexual transmission observed during ZIKV infection.
115          From January 2003 to December 2013, sexual transmission of 2 clades of Campylobacter jejuni
116 V(+)) youth, and the attendant potential for sexual transmission of antiretroviral (ARV) drug-resista
117  reviewed trials of interventions to prevent sexual transmission of any STI, employing a multilevel p
118 s new mouse model of horizontal and vertical sexual transmission of Chlamydia closely parallels C. tr
119                                 Fortunately, sexual transmission of chlamydiae has been described for
120 based combination microbicide to prevent the sexual transmission of diverse HIV-1 variants.
121 ve response to the threat of persistence and sexual transmission of Ebola is required and should buil
122                                              Sexual transmission of Ebola virus in Liberia has now be
123 a, the genomic analysis provides evidence of sexual transmission of EBOV and evidence of the persiste
124              Furthermore, no animal model of sexual transmission of EBOV exists.
125 his study, we describe a roadmap to modeling sexual transmission of EBOV using a mouse-adapted EBOV i
126  who have sex with men (MSM) are at risk for sexual transmission of enteric pathogens.
127 ne transfer (HGT) in bdelloids precludes the sexual transmission of entire haplotypes, for which we h
128 se (EVD) in Western Africa may contribute to sexual transmission of EVD and generate new clusters of
129  study did not evaluate directly the risk of sexual transmission of EVD.
130 r model suggests that BV is initiated by the sexual transmission of Gardnerella vaginalis, which has
131  model hypothesizing that BV is initiated by sexual transmission of Gardnerella vaginalis.
132      Epidemiologic studies suggest that most sexual transmission of genital herpes occurs when person
133 idence is that there is no increased risk of sexual transmission of HCV among heterosexual couples in
134 e providers need to pay special attention to sexual transmission of HCV among HIV-infected individual
135 sed injection drugs; however, more recently, sexual transmission of HCV has been recognized among HIV
136   Other observations, however, weigh against sexual transmission of HCV infection.
137 voir of HCV-infected individuals is sizable, sexual transmission of HCV likely contributes to the tot
138 d seminal HCV RNA levels could contribute to sexual transmission of HCV, but other factors, including
139 te HCV infection given its potential role in sexual transmission of HCV.
140  is approximately 5%, well below the risk of sexual transmission of hepatitis B or human immunodefici
141                                              Sexual transmission of hepatitis C virus (HCV) is uncomm
142 ations have been cited to support a role for sexual transmission of hepatitis C.
143 ve therapy with valacyclovir reduces risk of sexual transmission of herpes simplex virus type 2 (HSV-
144                                              Sexual transmission of herpes simplex virus type 2 (HSV-
145 be more important than semen or stool in the sexual transmission of HHV-8.
146   There is strong epidemiologic evidence for sexual transmission of high-risk genital human papilloma
147 tential candidate microbicide to prevent the sexual transmission of HIV and AIDS.
148 of the data on the use of ART to prevent the sexual transmission of HIV and identify challenges to im
149 ignificance, for example, as a ligand in the sexual transmission of HIV and stimulator of natural kil
150 on (VMMC) is capable of reducing the risk of sexual transmission of HIV from females to males by appr
151 tes in HIV-infected semen, may contribute to sexual transmission of HIV from men to their partners.
152 s could be effective tools in preventing the sexual transmission of HIV in humans.
153                                       During sexual transmission of HIV in women, the first cells lik
154                    Strategies to prevent the sexual transmission of HIV include vaccines that elicit
155 lopment of topical microbicides that prevent sexual transmission of HIV is an active area of investig
156 ne responses required for protection against sexual transmission of HIV is essential for the developm
157   These results suggest that protection from sexual transmission of HIV may be possible by effectivel
158                                              Sexual transmission of HIV selects for highly fit drug-r
159                     We also investigated the sexual transmission of HIV to and from these participant
160 nistered antiretroviral drugs to prevent the sexual transmission of HIV to receptive partners.
161  Only 1 intervention showed efficacy against sexual transmission of HIV, but 22 (53.7%) showed effect
162 nfectivity and may play an important role in sexual transmission of HIV, making them a potential micr
163                                       During sexual transmission of HIV, virus crosses mucosal epithe
164 n of antiretroviral therapy reduced rates of sexual transmission of HIV-1 and clinical events, indica
165 ve important implications for the biology of sexual transmission of HIV-1 and its potential reduction
166        Initial biologic events that underlie sexual transmission of HIV-1 are poorly understood.
167 4 cell count may be associated with enhanced sexual transmission of HIV-1 because of increased number
168 esent a novel strategy to reduce the rate of sexual transmission of HIV-1 in humans.
169                                              Sexual transmission of HIV-1 is an inefficient process,
170 tiviral therapy on HIV-1 in semen and on the sexual transmission of HIV-1 require further study.
171                                              Sexual transmission of HIV-1 requires that small amounts
172  viral entry inhibitors can help prevent the sexual transmission of HIV-1 to humans.
173 onsidered independent methods to prevent the sexual transmission of HIV-1 to women.
174 es in the earliest phases of infection after sexual transmission of HIV-1, a process that is ineffici
175 related morbidity and mortality and prevents sexual transmission of HIV-1.
176 e efficacy of such inhibitors to prevent the sexual transmission of HIV-1.
177 icrobicide that could be used to prevent the sexual transmission of HIV-1.
178 ecretions have important roles in preventing sexual transmission of HIV-1.
179 logic association of trichomoniasis with the sexual transmission of HIV-1.
180 rent genetic bottlenecks associated with the sexual transmission of HIV-1.
181 ecretions will improve studies that evaluate sexual transmission of HIV-1.
182 ical microbicides for their ability to block sexual transmission of HIV-1.
183 vated immune cells and have implications for sexual transmission of HIV-1.
184 ons for the selection of resistant virus and sexual transmission of HIV-1.
185 odel for testing of agents designed to block sexual transmission of HIV-1.
186 ogesterone affects mechanisms underlying the sexual transmission of HIV-1.
187 ction likely increases the efficiency of the sexual transmission of HIV-1.
188 e that such migration may be involved in the sexual transmission of HIV-1.
189 pears to protect homozygous individuals from sexual transmission of HIV-1.
190  women and may play an important role in the sexual transmission of HIV.
191 h less advanced immunodeficiency and reduces sexual transmission of HIV.
192  with a 10- to 20-fold reduction in risk for sexual transmission of HIV.
193 or HIV target cell during the early stage of sexual transmission of HIV.
194 tinually present, has the potential to block sexual transmission of HIV.
195 nital tissues potentially play a key role in sexual transmission of HIV.
196  be useful as a topical microbicide to block sexual transmission of HIV.
197 ls may be a novel strategy to interfere with sexual transmission of HIV.
198 an immunodeficiency virus (HIV) may decrease sexual transmission of HIV.
199 g, protecting, and transferring virus during sexual transmission of HIV.
200 alis may be valuable in reducing the risk of sexual transmission of HIV.
201 vaginal flora could lead to a higher rate of sexual transmission of HIV.
202 this protein as a microbicide to prevent the sexual transmission of HIV.
203 ention or treatment of AGWs may decrease the sexual transmission of HIV.
204 ory role in the efficiency of male-to-female sexual transmission of HIV.
205  they could present a natural barrier to the sexual transmission of HPV and could serve as the basis
206 accine's target disease cervical cancer, the sexual transmission of HPV was minimized, the threat of
207 esigned to control HSV-2 shedding.IMPORTANCE Sexual transmission of HSV-2 results from viral shedding
208                     A microbicide to prevent sexual transmission of HSV-2 would contribute substantia
209 d partners and condom use reduce the risk of sexual transmission of HSV-2.
210  on genital mucosal surfaces and may prevent sexual transmission of HSV.
211                             The incidence of sexual transmission of HTLV-II may be similar to that of
212                        Both intrauterine and sexual transmission of human herpesvirus (HHV)-6 and HHV
213 iretroviral resistance and the prevention of sexual transmission of human immunodeficiency virus (HIV
214    In the acute stage of infection following sexual transmission of human immunodeficiency virus (HIV
215                                              Sexual transmission of human immunodeficiency virus (HIV
216 a (SP) may reduce virus burden and influence sexual transmission of human immunodeficiency virus (HIV
217 as been associated with an increased rate of sexual transmission of human immunodeficiency virus (HIV
218 ntified recently as a cofactor that promotes sexual transmission of human immunodeficiency virus (HIV
219 (BV) is associated with an increased rate of sexual transmission of human immunodeficiency virus (HIV
220 ion and immune activation are key factors in sexual transmission of human immunodeficiency virus (HIV
221                          We report a case of sexual transmission of human immunodeficiency virus (HIV
222                                              Sexual transmission of human immunodeficiency virus type
223 ve role against invading pathogens; however, sexual transmission of human immunodeficiency virus type
224                                              Sexual transmission of human immunodeficiency virus type
225                 Recent studies indicate that sexual transmission of human immunodeficiency virus type
226            Recent findings suggest that most sexual transmission of human immunodeficiency virus type
227 the body fluid most commonly associated with sexual transmission of human immunodeficiency virus type
228 ovaginal mucosa is a relative barrier to the sexual transmission of human immunodeficiency virus type
229                  To explore the mechanism of sexual transmission of human immunodeficiency virus type
230  macaque model to study events that underlie sexual transmission of human immunodeficiency virus type
231                      In the primate model of sexual transmission of human immunodeficiency virus, tre
232 inhibitor of an amyloid fibril that enhances sexual transmission of human immunodeficiency virus.
233 V) 1% vaginal gel has been found to decrease sexual transmission of human immunodeficiency virus.
234 -alpha response is not sufficient to prevent sexual transmission of human immunodeficiency virus.
235                                              Sexual transmission of human papilloma virus is a leadin
236              Cross-sectional studies support sexual transmission of human T lymphotropic virus (HTLV)
237  tissue, supporting a possible mechanism for sexual transmission of KSHV.
238 nd semen, suggesting that the possibility of sexual transmission of Lassa virus should be considered.
239 we experimentally assessed the potential for sexual transmission of LASV in Mastomys.
240 nalysis revealed no substantial evidence for sexual transmission of minority DRM (BF = 0.02).
241    We found no clear evidence to support the sexual transmission of minority resistant variants, and
242  infection in rhesus macaques is a model for sexual transmission of primate retroviruses.
243                               In TB, lack of sexual transmission of rearranged chromosomes associates
244                                           In sexual transmission of simian immunodeficiency virus, an
245 ntial prophylactic for the prevention of the sexual transmission of the human immunodeficiency virus
246 nder development as a microbicide to prevent sexual transmission of the human immunodeficiency virus
247 fter the acute infection, and male-to-female sexual transmission of the virus is well documented.
248 affect fertility, reproductive capacity, and sexual transmission of the virus.
249 xposure to RNA viruses is required to combat sexual transmission of this class of pathogens.
250 l that incorporates both insect-vectored and sexual transmission of this pathogen.
251 ay increase cervical HTLV-I shedding and the sexual transmission of this virus.
252 , we tested the hypothesis of male-to-female sexual transmission of toxoplasmosis.
253 sterone, a sex steroid hormone, enhances the sexual transmission of various pathogens, including SIV.
254 cells to come into contact with HIV-1 during sexual transmission of virus.
255 epresent an attractive strategy for blocking sexual transmission of virus.
256       We report on 9 cases of male-to-female sexual transmission of Zika virus in the United States o
257  primary vector for the spread of the virus, sexual transmission of Zika virus is also a significant
258   Also unknown is the relative importance of sexual transmission of ZIKV and asymptomatic ZIKV infect
259 in the FRT and suggest a path for minimizing sexual transmission of ZIKV in women.
260 of mosquito are the primary vector for ZIKV, sexual transmission of ZIKV is a significant route of in
261                            Understanding the sexual transmission of ZIKV through vaginal and rectal r
262  the type I interferon receptor, we examined sexual transmission of ZIKV.
263 he highest viral loads, which is relevant to sexual transmission of ZIKV.
264 investigate the impact of mosquito-borne and sexual transmission on the spread and control of ZIKV an
265                             Yet, the role of sexual transmission on the spread and control of ZIKV in
266                       However, the role that sexual transmission played in the transmission is not ve
267 -4.8% to 14.5%-19.5% in 2025 with increasing sexual transmission, rising to 0%-10.8% and 22.6%-26.0%
268       We used data from the Antiretrovirals, Sexual Transmission Risk and Attitudes (ASTRA) questionn
269 rease genital tract HIV viral load (gVL) and sexual transmission risk to male partners.
270                        Our results suggest a sexual transmission route for beta-HPVs and support the
271 KV should target both the mosquito-borne and sexual transmission routes.
272 observation that the guinea pigs infected by sexual transmission shed organisms for a significantly s
273 e responsible for efficacy against liver and sexual transmission stages of Plasmodium falciparum.
274                             Gametocytes, the sexual transmission stages, form normally and respond in
275        In the current study, we studied both sexual transmission (STx) and vertical transmission (VTx
276      Compared with other risk groups for HIV sexual transmission, such as men who have sex with men,
277                                       During sexual transmission, the high genetic diversity of HIV-1
278                                   Apart from sexual transmission, the virus can also be transmitted f
279 y establishment of HIV-1 infection following sexual transmission, these results may be important in t
280 se of needle-exchange programmes, as well as sexual transmission to injection drug users (IDUs) and s
281            We calculated the contribution of sexual transmission to the basic reproduction number R(0
282  transmission, postexposure prophylaxis, and sexual transmission (topical microbicide).
283 a brucei evansi, causing 'surra') or through sexual transmission (Trypanosoma brucei equiperdum, caus
284 d individual and household factors, examined sexual transmission using a sexual-networks approach, an
285                            The likelihood of sexual transmission was based on viral load, knowledge o
286        Among "pessimistic" simulations where sexual transmission was largely unchanged but viral supp
287 A) loads were stable during observation, and sexual transmission was not attributable to an upward tr
288                                  Limitation: Sexual transmission was not explicitly accounted for in
289                                              Sexual transmission was not observed following mating of
290      Among "optimistic" simulations in which sexual transmission was sharply reduced and viral suppre
291                                              Sexual transmission was the sole risk factor for infecti
292 lective pressures on Nef functions following sexual transmission, we analyzed genetic and functional
293 ne how SEVI might function in the context of sexual transmission, we applied HIV-1 and SEVI to intact
294 have specific biological properties favoring sexual transmission, we inoculated human cervical tissue
295 ority of HIV infections are acquired through sexual transmission, we investigated whether antiviral C
296 rus-cell interactions in this barrier during sexual transmission, we studied the uptake and infection
297 he 12 models, HIV infections attributable to sexual transmission were estimated to surpass those attr
298 d the genito-urinary system, compatible with sexual transmission, whereas children mostly manifested
299 EFV, averting 4900 women's deaths and 20 500 sexual transmissions while adding 300 pediatric deaths.
300  used a mathematical model of HPV 6/11/16/18 sexual transmission within an MSM population in England,

 
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