ライフサイエンスコーパス: sialic acid receptor

1 193T substitution is involved in binding the sialic acid receptor.

2 nin-neuraminidase (HN) glycoprotein with its sialic acid receptor.

3 nin-neuraminidase (HN) glycoprotein with its sialic acid receptor.

4 a preference for avian-like alpha2,3-linked sialic acid receptors.

5 ges to recognize host-specific variations in sialic acid receptors.

6 h of these mutations increased HA binding to sialic acid receptors.

7 e [HN]) protein, which binds to cell surface sialic acid receptors.

8 agglutinin (HA) that often affect binding to sialic acid receptors.

9 residues and failed to bind alpha2,3-linked sialic acid receptors.

10 lates also displayed binding to "human-like" sialic acid receptors.

11 without a shift to a preference for alpha2,6 sialic acid receptors.

12 SNA) and virus (H1N1) adhesion to endogenous sialic acid receptors.

13 te, which is critical for binding human type sialic acid receptors.

14 ch binding from "avian-type" alpha2-3-linked sialic acid receptors (2-3Sia) to "human-type" Siaalpha2

15 (2-3Sia) to "human-type" Siaalpha2-6-linked sialic acid receptors (2-6Sia).

16 viral entry and egress, including binding to sialic acid receptors, activating the fusion (F) protein

17 toward human (alpha2,6) and avian (alpha2,3) sialic acid receptor analogues, and the potential to tra

18 cid, which enhances overall virus binding to sialic acid receptor analogues.

19 relatively high affinity for alpha2,6-linked sialic acid receptor and acid and temperature stability.

20 Thus, virus binding to a sialic acid receptor and to immunoglobulin-like receptor

21 H7N9 viruses are able to bind to human sialic acid receptors and are also able to develop resis

22 a key role in the recognition process of the sialic acid receptors and catalytic efficiency of NA.

23 Additionally, these cells expressed sialic acid receptors and supported the active replicati

24 and duck myotubes expressed avian and human sialic acid receptors and were readily susceptible to lo

25 virulence by its ability to bind avian-type sialic acid receptors, and that pdm/09 RNP conferred the

26 wcastle disease virus mediates attachment to sialic acid receptors, as well as cleavage of the same m

27 ion of complexes with the NA protein and the sialic acid receptors, as well as provide HA activity to

28 of this Viet04 HA reveals an avian alpha2-3 sialic acid receptor binding preference.

29 d with antibody combining site A or B or the sialic acid receptor binding site.

30 HA) gene, at sites in close proximity to the sialic acid receptor-binding pocket.

31 Depletion of sialic acid receptors by DAS181 was evaluated by lectin-

32 -specific IgG protected neonates through the sialic acid receptor CD22(6,7), which suppressed IL-10 p

33 ricate balance between host cell binding and sialic acid receptor destruction is carefully maintained

34 microscopy techniques, we characterized the sialic acid receptor distribution and the cellular compo

35 and C-22, that exhibit increased avidity for sialic acid receptors due to single amino acid changes i

36 Sialic acid receptors for both human and avian viruses,

37 parasite strain that is heavily dependent on sialic-acid receptors for invasion, and show that the Pf

38 in interactions and HN protein attachment to sialic acid receptors, HN and F protein-containing compl

39 nhance binding to human-like alpha2,6-linked sialic acid receptors.IMPORTANCE The interaction of infl

40 ity from the preference of avian viruses for sialic acid receptors in alpha2,3 linkage to the prefere

41 rcine Kupffer cells, suggesting a role for a sialic-acid receptor in innate cellular recognition of x

42 enza H17N10 virus neither bind to nor cleave sialic acid receptors, indicating that this virus employ

43 The sialic acid receptor induces a cascade of conformational

44 an avian virus-like preference for alpha2,3 sialic acid receptors, it is unable to replicate in male

45 -adapted influenza virus hemagglutinins bind sialic acid receptors linked via alpha2-3 glycosidic bon

46 hy to visualize influenza virions bound to a sialic acid receptor mimic.

47 Binding of HN to its sialic acid receptor on a target cell triggers its activ

48 (ii) the interaction between the HA and its sialic acid receptor on the effector cell.

49 lutinin ligands on influenza viruses and the sialic acid receptors on biosensors or on host cells, ou

50 utation dramatically decreases HA binding to sialic acid receptors on cell surfaces.

51 inin that regulates hemagglutinin binding to sialic acid receptors on host cells.

52 avian alpha-2,3- and human alpha-2,6-linked sialic acid receptors on the apical surface and supports

53 but are not a natural host and have distinct sialic acid receptor profiles compared to humans.

54 Enzyme-mediated removal of sialic acid receptors represents a novel antiviral strat

55 es attach to cells via a sialic acid moiety (sialic acid receptor) that is alpha2-3 linked or alpha2-

56 ping reagents with an increased affinity for sialic acid receptors through linking CBM40 modules toge

57 that human influenza A virus uses canonical sialic acid receptors to infect bat cells, even though b

58 Progenitor epithelial cells expressed sialic acid receptors utilized by avian and mammalian in

59 es generally mediate binding to cell surface sialic acid receptors via the hemagglutinin (HA) glycopr

60 The ability of ZM1 to bind sialic acid receptors was inhibited 10-fold less than fo

61 se (HN) attachment protein of NDV recognizes sialic acid receptors, whereas the NiV G attachment prot

62 ng binding to both human-type and avian-type sialic acid receptors, which may influence respiratory t

63 in showed stronger binding to the human-type sialic acid receptor, with preferential binding to alpha