ライフサイエンスコーパス: sickness

1 st mortality (=death or delisting because of sickness).

2 sceral leishmaniasis, and gambiense sleeping sickness).

3 stage East African trypanosomiasis (sleeping sickness).

4 risks of hypobaric hypoxia and decompression sickness.

5 igate the social environment during times of sickness.

6 insights into the mechanism of sleep during sickness.

7 rs who might provide support and care during sickness.

8 in other organisms to regulate sleep during sickness.

9 nic trypanosomes that cause African sleeping sickness.

10 ate/inefficient RH alleles exhibit synthetic sickness.

11 extracellular parasite that causes sleeping sickness.

12 ain the weight loss associated with sleeping sickness.

13 ntribute to loss of appetite observed during sickness.

14 called African trypanosomiasis, or sleeping sickness.

15 t causes the deadly disease African sleeping sickness.

16 te gastric lesions related to acute mountain sickness.

17 ncluding malaria, dengue fever, and sleeping sickness.

18 23andMe database who were surveyed about car sickness.

19 share underlying genetic factors with motion sickness.

20 esiense, the cause of acute African sleeping sickness.

21 somiasis (HAT), also called African sleeping sickness.

22 brucei, the causal agent of African sleeping sickness.

23 panosomes, protecting against human sleeping sickness.

24 cei, the causative agent of African sleeping sickness.

25 d neuronal impairment and succumbed to fatal sickness.

26 s to treat Chagas disease and human sleeping sickness.

27 , pregnancy-related ailments such as morning sickness.

28 ock, including Bluetongue, and African Horse Sickness.

29 promising lead to treat late-stage sleeping sickness.

30 ral neuroendocrine mechanism of sleep during sickness.

31 ore to assess the severity of acute mountain sickness.

32 an African trypanosomiasis (HAT) or sleeping sickness.

33 d to suicide rates but others, such as staff sickness (-0.12, 0.37) and patient satisfaction (-0.06,

34 .0 emotional (+8.1; 95% CI, 1.8 to 14.4) and Sickness (-8.2; 95% CI, -14.2 to -2.2) scores.

35 i is the causative agent of African sleeping sickness, a devastating disease endemic to sub-Saharan A

36 thern Africa are foci for Rhodesian sleeping sickness, a fatal zoonotic disease caused by trypanosome

37 rypanosomiases, including Rhodesian sleeping sickness, a zoonosis associated with wilderness areas of

38 ported by an individual and the odds of paid sickness absence (1 adversity: odds ratio (OR) = 1.26, 9

39 The outcome variables were paid sickness absence (yes/no) and number of days of paid sic

40 The higher prevalence of sickness absence after breast cancer was mainly a result

41 The primary outcome measure was change in sickness absence among those supervised by each of the m

42 were associated with higher odds ratios for sickness absence and disability pension (odds ratio rang

43 ted and sociodemographic factors with future sickness absence and disability pension among women with

44 Purpose To explore future diagnosis-specific sickness absence and disability pension among women with

45 Information about relatively low future sickness absence and disability pension levels can be us

46 the annual prevalence of diagnosis-specific sickness absence and disability pension over 5 years.

47 absence hours divided by the sum of hours of sickness absence and hours of attendance.

48 Advanced cancer at diagnosis, > 90 days sickness absence before diagnosis, low education, and be

49 en in the first year, most women were not on sickness absence for a substantial time, and even in hig

50 leave seperately, with rate being defined as sickness absence hours divided by the sum of hours of si

51 ondents reported a greater number of days of sickness absence in response to worsening psychosocial j

52 absence (yes/no) and number of days of paid sickness absence in the past year (2005-2012).

53 Conclusion The level of sickness absence increased substantially in women with b

54 Sickness absence is associated with adverse health, orga

55 d even in high-risk groups, many were not on sickness absence or disability pension in the following

56 e included in the study via their anonymised sickness absence records.

57 breast cancer, the proportion of women with sickness absence was high but decreased continuously fro

58 most rapidly increasing causes of long-term sickness absence, despite improved rates of detection an

59 The impact of job satisfaction upon sickness absence, turnover intention, as well as the inf

60 d to a significant reduction in work-related sickness absence, with an associated return on investmen

61 health problems, and its effect on employee sickness absence.

62 ll psychosocial job quality and variation in sickness absence.

63 mprove the quality of work could help reduce sickness absence.

64 ce to measure the impact of longer shifts on sickness absences for mental health workforce.

65 redictors: age, self-rated health, number of sickness absences in previous year, socioeconomic positi

66 Chagas disease, leishmaniasis and sleeping sickness affect 20 million people worldwide and lead to

67 ts association with energy-saving behaviors, sickness allows increased incentive motivation when the

68 Acute mountain sickness (AMS) affects more than 25% of individuals asce

69 to high altitude, the CBF in acute mountain sickness (AMS) subjects was higher (P < 0.05), while the

70 with symptoms consistent with acute mountain sickness (AMS).

71 Detection of subtle cues indicating sickness and avoidance of sick conspecifics would theref

72 d public health measures drastically reduced sickness and death from infections in children and young

73 provocative of and protective against motion sickness and how vestibular disease may sensitize to mot

74 h is the parasite that causes human sleeping sickness and is also partially responsible for animal tr

75 i is the causative agent of African sleeping sickness and is known for its unique RNA processing mech

76 ainst diseases that include African sleeping sickness and malaria.

77 and observing new associations with altitude sickness and many gastrointestinal conditions.

78 rucei is a protist parasite causing sleeping sickness and nagana in sub-Saharan Africa.

79 n mechanisms and loci associated with motion sickness and nausea per se.

80 sea and vomiting (PONV), vertigo and morning sickness and observing new associations with altitude si

81 ould benefit both victims of acute radiation sickness and patients undergoing hematopoietic cell tran

82 nations and loss of crucial staff because of sickness and public health restrictions.

83 he host immune and metabolic response during sickness and recovery in a mouse model of malaria.

84 e importance of the nervous system in motion sickness and suggest a role for glucose levels in motion

85 individuals is highly susceptible to motion sickness and yet the underlying causes of this condition

86 adverse event, including rash, fever, serum sickness, and anaphylaxis.

87 sthenia, sensory neuropathy, anorexia, serum sickness, and hypertensive encephalopathy.

88 uman pathologies as Chagas disease, sleeping sickness, and leishmaniasis.

89 bited alleviated bodyweight loss, behavioral sickness, and myocardial dysfunction.

90 cei, the causative agent of African sleeping sickness; and Plasmodium spp., the causative agents of m

91 This effect was not due to sickness as icv TTR did not cause a conditioned taste av

92 There is new research on acute mountain sickness associated with inflammation with relevance for

93 morphisms (SNPs) were associated with motion sickness at a genome-wide-significant level (P < 5 x 10(

94 ore, which quantifies the overall feeling of sickness at altitude (VAS[O]; various thresholds), Acute

95 sual analog scale for the overall feeling of sickness at altitude, Acute Mountain Sickness-Cerebral,

96 sera have many disadvantages including serum sickness, batch-to-batch variation in quality and the us

97 Sickness behavior and cognitive dysfunction occur freque

98 igated the impact of glucose availability on sickness behavior and delirium in mice and humans.

99 1 was necessary and sufficient for mediating sickness behavior and drove leukocyte recruitment to the

100 Leucine had no effect of its own on sickness behavior and neuroinflammation.

101 eral and central cytokine levels, along with sickness behavior and telemetry data, were analyzed foll

102 that probiotic (VSL#3) treatment attenuates sickness behavior development in mice with liver inflamm

103 Attenuation of sickness behavior development was associated with reduct

104 ication pathway, thereby reducing subsequent sickness behavior development.

105 croglial activation and subsequently driving sickness behavior development.

106 n the initiation of cognitive impairment and sickness behavior during viral infection through inducti

107 tenuates tumor-induced neuroinflammation and sickness behavior following an immune challenge (i.p. li

108 monocytes and platelets, and development of sickness behavior following bile-duct ligation.

109 Sickness behavior in humans is characterized by low mood

110 Despite how common sickness behavior is, its molecular basis is not well un

111 t detected in serum and mice did not display sickness behavior or lose weight.

112 ddition, inflammatory cytokines in serum and sickness behavior profiles were measured before and afte

113 These animals developed sickness behavior symptoms, including anorexia, hypoacti

114 Features of the complex behavioral sickness behavior syndrome were characterized by homecag

115 ered peripherally to adult zebra finches and sickness behavior was recorded 2 or 24 hours later.

116 ntake is a widely conserved manifestation of sickness behavior with poorly characterized effects on a

117 lls as natural gatekeepers for virus-induced sickness behavior, demonstrated tissue specific IFNAR en

118 We found that during sickness behavior, single-stranded RNA viruses, double-s

119 Sickness behavior, temperature, and cytokine profiles of

120 eriphery in response to brain injury induced sickness behavior, which could be abrogated by inhibitio

121 and behavioral changes, collectively termed sickness behavior, which while adaptive, can affect mood

122 ate microglial activation and development of sickness behavior.

123 lls to activate microglial cells and promote sickness behavior.

124 of the microglia, as well as development of sickness behavior.

125 SD-sensitized mice associated with prolonged sickness behavior.

126 mmatory responses are necessary for adaptive sickness behaviors (e.g., fatigue, anorexia, and fever)

127 The resulting sickness behaviors (e.g., pain, disturbed sleep), depres

128 Tumors also attenuated sickness behaviors (hypothermia and lethargy) compared t

129 Sickness behaviors and metabolic responses to invading p

130 Although these so-called sickness behaviors are the most common and familiar symp

131 ced physiologic trade-off was independent of sickness behaviors but required hematopoietic sensing of

132 e immune cells accelerated the recovery from sickness behaviors following SE.

133 rious pathophysiologic mechanisms leading to sickness behaviors have been proposed.

134 tures of circulating monocytes and promoting sickness behaviors in mice with cholestatic liver injury

135 microbiome to treat inflammation-associated sickness behaviors in patients with systemic inflammator

136 mation in specific regions may contribute to sickness behaviors that overlap with the symptoms of MDE

137 mals, activation of the immune system causes sickness behaviors that present during a major depressiv

138 important role of TLR2 in the development of sickness behaviors via stimulation of hypothalamic micro

139 Sickness behaviors were quantified based on time spent b

140 ommonly develop debilitating symptoms (i.e., sickness behaviors) that arise from changes in brain fun

141 mmonly develop debilitating symptoms, called sickness behaviors, which arise via changes in brain fun

142 se these factors are likely to influence how sickness behaviour alters pathogen transmission.

143 common types of pain: systemic inflammation (sickness behaviour), post-surgical pain, diabetic neurop

144 Host sickness behaviours vary in pathogen species-specific ma

145 Such "sickness behaviours" include disrupted circadian rhythms

146 nd are sufficient to generate the associated sickness behaviours, and are the first demonstration tha

147 location of metabolic resources during acute sickness but might also account for maladaptive, motivat

148 omide was used by pregnant women for morning sickness but was removed from the market because it caus

149 ferent levels of protection against sleeping sickness, but this comes with an increased risk of devel

150 may undermine efforts to eliminate sleeping sickness by 2020.

151 Decompression sickness can involve the musculoskeletal system, skin, i

152 that the risk for trypanosomiasis (sleeping sickness), carried by tsetse flies in bushy environments

153 Patients with sleeping sickness, caused by the parasite Trypanosoma brucei, hav

154 (VAS[O]; various thresholds), Acute Mountain Sickness-Cerebral score (AMS-C; >/=0.7 indicates AMS), a

155 ling of sickness at altitude, Acute Mountain Sickness-Cerebral, and clinical functional score perform

156 We observed patients with chronic mountain sickness (CMS) in our clinic who developed progressive n

157 Chronic mountain sickness (CMS) is a maladaptation syndrome encountered a

158 The diagnosis of chronic mountain sickness (CMS) is based on a score including 7 clinical

159 ome high-altitude dwellers (chronic mountain sickness [CMS] or Monge's disease) but not others living

160 searched for comorbid phenotypes with motion sickness, confirming associations with known comorbiditi

161 rucei, a causative agent of African Sleeping Sickness, constantly changes its dense variant surface g

162 ve effect of olfactory-visual integration of sickness cues was found in the intraparietal sulcus, whi

163 soma brucei, the causative agent of sleeping sickness, cycles between an insect and a mammalian host.

164 Decompression sickness (DCS) was first diagnosed in marine turtles in

165 pathological responses such as decompression sickness (DCS).

166 cei, the etiologic agent of African Sleeping Sickness, deploys an RNA pol II that contains a non-cano

167 targeted to be used as a novel biomarker of sickness/depressive-like behaviors.

168 threats, their contribution to decompression sickness development requires further study.

169 cei, the causative agent of African sleeping sickness, differs from its human host in several fundame

170 uce immune complex diseases, including serum sickness disease (SSD).

171 gly immunogenic and rabbit ATG induces serum sickness disease in almost all patients without addition

172 Sickness during pregnancy is associated with an increase

173 Sickness effects might also depend on how a social inter

174 Finally, sickness effects might vary with social relationship typ

175 We predicted that sickness effects should be smaller for interactions amon

176 g a poor sleeper) that correlate with motion sickness, findings that could help identify risk factors

177 Trypanosoma brucei causes African sleeping sickness for which no vaccine exists and available treat

178 In 1993, the freedom to choose one's sickness fund was formally introduced, and reforms that

179 extended in 1913 to cover relations between sickness funds and doctors, which in turn led to the rig

180 initially applied mainly to the payers (the sickness funds) but was extended in 1913 to cover relati

181 w vestibular disease may sensitize to motion sickness has increased.

182 rucei, the causative agent of human sleeping sickness, has an intrinsic circadian clock that regulate

183 owever, the polysymptomatic nature of motion sickness, high interindividual variability, and the exte

184 me variable is defined as the ratio of total sickness hours over the total scheduled working hours (f

185 e associated with an increased percentage of sickness hours per week of between 0.73% and 0.98%, the

186 soma brucei, the causative agent of sleeping sickness (Human African Trypanosomiasis, HAT), contains

187 elephone Interview for Cognitive Status, and Sickness Impact Profile.

188 irst genome-wide association study on motion sickness in 80 494 individuals from the 23andMe database

189 humans perceive and integrate early cues of sickness in conspecifics sampled just hours after the in

190 ed by biting midges and causes African horse sickness in equids, with mortality reaching up to 95% in

191 s African trypanosomiasis, known as sleeping sickness in humans and nagana in domestic animals.

192 African trypanosomiasis, sleeping sickness in humans or nagana in animals, is a potentiall

193 infect mammals, they cause African sleeping sickness in humans.

194 has traditionally been used to treat motion sickness in humans.

195 tozoan parasite that causes African sleeping sickness in humans.

196 re is a hallmark feature of chronic mountain sickness in maladapted populations living at high altitu

197 Sickness in mammals can lead to cognition deficits, alth

198 llular stress is similar to sleep induced by sickness in other animals.

199 alaria parasite, Plasmodium chabaudi, shapes sickness in terms of parasite genotype-specific effects

200 indirect centrality is also correlated with sickness in the Agta, suggesting a trade-off.

201 his study, we used a mouse model of sleeping sickness in which the acute infection was treated but br

202 mor flare") (in 11%), anemia (in 11%), serum sickness (in 8%), and fatigue (in 8%).

203 s, distention, bloating, abdominal pain, and sickness) in CD participants compared with HVs (P <= 0.0

204 The prevalence of acute mountain sickness increases with higher altitudes.

205 hat the PSTN complex reacts to neophobia and sickness induced by lipopolysaccharide or cisplatin.

206 Now, Wang et al. identify that sickness-induced anorexia differentially shapes the meta

207 Sickness-induced anorexia is a conserved behavior induce

208 microbes have evolved mechanisms to modulate sickness-induced behaviors to promote health of their ho

209 ine article, Garre et al. (2017) report that sickness-induced cortical dendritic spine loss and impai

210 Study 2 examined the performance of the Sickness Insight in Coping Questionnaire among 100 ICU p

211 The Sickness Insight in Coping Questionnaire had good intern

212 Overall, the Sickness Insight in Coping Questionnaire has good psycho

213 This study showed that the Sickness Insight in Coping Questionnaire has good struct

214 ICU clinicians can use the Sickness Insight in Coping Questionnaire to gain insight

215 n = 103 hospitalized patients) addressed the Sickness Insight in Coping Questionnaire's performance r

216 res in testing the construct validity of the Sickness Insight in Coping Questionnaire-subscales (figh

217 t-proxy agreement of a novel instrument, the Sickness Insight in Coping Questionnaire.

218 Loss of appetite during sickness is a common and often debilitating phenomenon.

219 Sleeping sickness is a fatal disease caused by the protozoan para

220 ne signaling.SIGNIFICANCE STATEMENT Sleeping sickness is a fatal disease that disrupts the circadian

221 Motion sickness is an emerging hazard in information technologi

222 Inflammation-induced sickness is associated with a large set of behavioral al

223 Sickness is triggered by exposure to environments such a

224 man African trypanosomiasis ([gHAT] sleeping sickness) is a vector-borne disease that is typically fa

225 n trypanosomiasis (HAT), or African sleeping sickness, is a fatal disease found throughout sub-Sahara

226 Human African trypanosomiasis, or sleeping sickness, is caused by infection with parasites of the g

227 n African trypanosomiasis (HAT), or sleeping sickness, is caused by the protozoan parasite Trypanosom

228 cei, the causative agent of African sleeping sickness, is transmitted to its mammalian host by the ts

229 Two hours post LPS, sickness-like behaviors increased, the transcription of

230 mmatory drug-associated angioedema and serum sickness-like reactions, are more frequent among young p

231 ma brucei, the etiological agent of sleeping sickness, localized its replication origins to the bound

232 fect of extended working hours on short term sickness (< 7 days) on staff is examined.

233 tic trypanosomes that cause African sleeping sickness, mating occurs during transmission by the tsets

234 ndings suggest that the symptoms of sleeping sickness may be because of alterations in homeostatic ad

235 ogy and brain regions associated with motion sickness may provide for more effective medication in th

236 ity due to heart failure in chronic mountain sickness most likely reduces fitness.

237 s the subcellular localization and synthetic sickness of Heh2DeltaNLS mutants.

238 ed to explicitly consider how the effects of sickness on social network structure can differ dependin

239 from feeding when the animal is subjected to sickness or exposed to a previously unknown source of fo

240 es, including those associated with satiety, sickness or unpalatable food.

241 r human African trypanosomiasis (or sleeping sickness), orally dosed fexinidazole stands poised to re

242 sleep need; because these conditions include sickness, our findings provide a link between sleep and

243 ort (P = .03), fatigue (P = .03), and motion sickness (P = .01).

244 In the sleeping sickness parasite Trypanosoma brucei, the kinetoplast is

245 el, we evaluated the effects of the sleeping sickness parasite, Trypanosoma brucei, on sleep patterns

246 The sleeping sickness parasite, Trypanosoma brucei, uses quorum sensi

247 The strategy that sleeping sickness parasites use to evade the mammalian immune sys

248 rare cell types in blood (including sleeping sickness parasites), and has the potential to enable sin

249 hlighting the need for better assessments of sickness, particularly in women.

250 ion in both murine models and human sleeping sickness patients.

251 eric Core Scales (PedsQL4.0), and an overall Sickness question.

252 tion sickness was probed using the Simulator Sickness Questionnaire modified for pediatric use (Peds

253 nce-in-Difference) are used to compare staff sickness rates before and after the implementation, wher

254 Both depressive and sickness-related behaviors were significantly abundant (

255 Motion sickness remains bothersome in conventional transport an

256 This fundamental aspect of the sickness response reduces the quality of life for people

257 ex, ethnicity, body mass index, and physical sickness response revealed that post-endotoxin depressed

258 blocks the protracted neuroinflammatory and sickness response to peripheral Escherichia coli (E. col

259 These sickness responses are characterized by an acute reorien

260 nged infection-induced neuroinflammatory and sickness responses in aged rats.

261 ne stimulation, and that are associated with sickness responses such as fever, anorexia, and stress h

262 ificantly affect average MSAS, PedsQL4.0, or Sickness score trends.

263 AS, PedsQL4.0 total and subscale scores, and Sickness scores during 20 weeks of follow-up, along with

264 pite greater morbidity (e.g. weight loss and sickness scores) during the acute infection in the 18-mo

265 ad evidence of association between increased sickness sensitivity and reduced glucocorticoid sensitiv

266 P < .03), with significant interaction with sickness severity status.

267 more, in some patients with higher levels of sickness severity, reduced mortality from an optimized a

268 Effects of sickness should be lower for social behaviours that best

269 Importantly, mechanisms regulating sickness sleep are partially distinct from those regulat

270 aptive mechanisms employed in the control of sickness sleep may play a role in correcting cellular ho

271 During sickness, sleep is regulated by cytokines released from

272 Sickness status presented by odor and facial photograph

273 of some anti-mAChR drugs in treating motion sickness suggest that we may, in fact, already be target

274 al protective equipment, (2) staff fever and sickness surveillance, and (3) enhanced medical surveill

275 staff were placed on hospitalwide fever and sickness surveillance, with 1524 frontline staff working

276 that may be related to inflammation-induced sickness symptom as well as other functions, such as blo

277 Cytokines and sickness symptoms were assessed hourly till 8 h after LP

278 Only sickness symptoms were evident in immunodeficient mice i

279 terleukin-6 and tumor necrosis factor-alpha, sickness symptoms, body temperature and self-reported fa

280 lammatory cytokines, vital sign changes, and sickness symptoms, well-established consequences of LPS

281 nosoma brucei, which causes African sleeping sickness, TbISWI down-regulates RNA polymerase I (Pol I)

282 To decrease the possibility of serum sickness, the equine antisera was digested with pepsin t

283 orted discomfort consistent with mild motion sickness; the boy said he was bored and the headset was

284 Other SNPs may affect motion sickness through nearby genes with roles in the nervous

285 ia occurs in disorders ranging from altitude sickness to airway obstruction, apnea, and atelectasis.

286 d by the causative agent of African sleeping sickness, Trypanosoma brucei In mitochondria of this pat

287 etabolism in the causative agent of sleeping sickness, Trypanosoma brucei, with that of human erythro

288 sative agents of Chagas disease and sleeping sickness, two neglected tropical diseases where there is

289 nosomes, parasites that cause human sleeping sickness, undergo a density-dependent differentiation in

290 changes accompanying visually induced motion sickness, using a motion video, hypothesizing that diffe

291 All were exposed to a 10-min motion-sickness video during fMRI.

292 African horse sickness virus (AHSV) is a lethal arbovirus of equids th

293 African horse sickness virus (AHSV), an orbivirus in the Reoviridae fa

294 African horse sickness virus is transmitted by biting midges and cause

295 Visually induced motion sickness was probed using the Simulator Sickness Questio

296 t Barcroft was suffering from acute mountain sickness when he made it!

297 highlanders suffering from chronic mountain sickness, which is characterized by an excessive product

298 ase, high altitude involves chronic mountain sickness with new knowledge of associated cardiovascular

299 We predicted that sickness would impact both the number and duration of so

300 t of Human African Trypanosomiasis (sleeping sickness), yet little is known about which PKs are essen