ライフサイエンスコーパス: silent period

1 acterized by deep refractoriness to seizure (silent period).

2 terval intracortical inhibition and cortical silent period.

3 did not affect the duration of the cortical silent period.

4 de with voluntary facilitation and a shorter silent period.

5 ds, or firing several bursts then entering a silent period.

6 physiology occurring during the seizure-free silent period.

7 status) and 4.3+/-0.7 late (14 days) in the silent period.

8 t bursts of RNA synthesis followed by longer silent periods.

9 ning and closing, that are separated by long silent periods.

10 xplained by a decrease in the density of the silent periods.

11 nerve response (16 x baseline) followed by a silent period (1-2 s) during which another stimulus evok

12 = 0.01) and the post-motor evoked potential silent period (101 ms; SEM +/- 10) was significantly sho

13 fore conversion, P<.01); (2) an electrically silent period (267+/-45 ms); (3) "organized atrial fibri

14 lices when depolarized during their normally silent period and (2) bursting when depolarized in nonrh

15 t zolpidem reduced hyperexcitability in both silent period and chronically epileptic cells, but was m

16 rtical inhibition was measured with cortical silent period and intracortical inhibition paradigms.

17 rovements correlated with increased cortical silent period and short-interval intracortical inhibitio

18 al intracortical inhibition and the cortical silent period) and GABAA (short-interval intracortical i

19 the input-output recruitment curve, cortical silent period, and amplitude of the motor evoked potenti

20 thresholds, input/output curves or cortical silent period between patients with secondary and primar

21 between respiratory and syringeal control of silent periods between sound units and wing movement cyc

22 eurons that alternate bursting activity with silent periods, but the mechanism underlying this vital

23 ed by gamma-aminobutyric acid-A receptors in silent period cells differed markedly from controls.

24 or-evoked potentials (MEPs) and the cortical silent period (CSP) evoked by a single-pulse TMS, short-

25 in Inventory (BPI), and reduced the cortical silent period (CSP) in a BDNF-dependent manner.

26 al intracortical inhibition (SICI); cortical silent period (CSP)) and excitatory circuitries (short i

27 cortical facilitation, and the contralateral silent period (CSP).

28 tracortical facilitation (ICF), and cortical silent period (CSP).

29 te) and depth-positive deflection during the silent period (Down state).

30 sting motor threshold (P < .05) and cortical silent period duration (P < .001).

31 shold, central motor conduction time (CMCT), silent period duration and the amplitude of compound mus

32 Silent period duration was shorter than normal early in

33 s (APs) by 21 mV without affecting burst- or silent-period durations.

34 d of two bursts per respiratory cycle with a silent period during inspiration.

35 racortical inhibition and prolonged cortical silent period during voluntary activity of an intrinsic

36 onal coinactivation: the occurrence of brief silent periods during which all neurons in the local net

37 igms, such as trace conditioning, in which a silent period elapses between the offset of the conditio

38 The early shortening of silent period, however, probably represents a shift in t

39 rformed a duration discrimination task for a silent period in a rhythmic auditory sequence.

40 netic stimulation to examine the ipsilateral silent period in an arm and hand muscle.

41 -evaluation of the concept of post-traumatic silent period in both animals and humans.

42 The burst rate and the pre-burst silent period in nDF firing of organic dystonia were con

43 ements of their display with atypically long silent periods in their song, potentially avoiding adver

44 .g., synchronization and phase) during these silent periods in vivo (male mice), in vitro (ferrets, e

45 An additional analysis of ipsilateral silent periods indicated that interhemispheric inhibitio

46 ion of the reflex occurred within a cortical silent period induced by transcranial magnetic stimulati

47 spheric inhibition (IHI) and the ipsilateral silent period (iSP), whilst excitability of CTS pathways

48 s seizures develop following an asymptomatic silent period lasting several weeks.

49 ffective at unmasking the underlying IPSP in silent period neurons.

50 VPd neurons only fire during the silent period of DP-PCN neurons, suggesting that they pr

51 of up to 30 s, a time that spans the average silent period of the chorus.

52 multimodal calls are still preferred after a silent period of up to 30 s, a time that spans the avera

53 vements of the display are synchronized with silent periods of song, but it is unknown how this coord

54 en falling asleep, stimulus-induced neuronal silent periods (OFF periods), characteristic of nonrapid

55 ing switching between firing rates, entering silent periods, or firing several bursts then entering a

56 iated IPSPs diminished more gradually in the silent period, reaching a minimum at day 14.

57 tracortical facilitation (ICF), the cortical silent period (SP) and spinal reciprocal inhibition (RI)

58 eter muscles of 12 subjects and the cortical silent period (SP) in nine subjects.

59 rhythmic CS discharges were interleaved with silent periods, suggesting that apamin- and CTX-sensitiv

60 d spontaneous transitions between active and silent periods termed cell cycling.

61 tials and decreased duration of the cortical silent period (the latter only in the conditioned group)

62 The cortical silent period, the startle response and the second and t

63 The distribution of the pre-burst silent period was bimodal with a longer mode of approxim

64 Silent period was estimated during a small background co

65 Cortical silent period was significantly prolonged in writer's cr

66 Cortical silent periods were shortened in leg muscles.

67 asynchronous state) and (2) "filling-in" of silent periods with low-frequency (2-4 Hz) activity (beg