ライフサイエンスコーパス: simple diffusion

1 mum, owing mainly to directional movement or simple diffusion.

2 xtended further than might be predicted from simple diffusion.

3 ds of times faster than what is expected for simple diffusion.

4 gesting that recycling pool vesicles move by simple diffusion.

5 es in the IS and that its movement occurs by simple diffusion.

6 sion and increases the proportion exhibiting simple diffusion.

7 esting that nuclear entry does not occur via simple diffusion.

8 obility was not directed but was governed by simple diffusion.

9 cur in a non-targeted fashion as a result of simple diffusion.

10 ggest that anandamide uptake is a process of simple diffusion.

11 tes, cross the intact blood-brain barrier by simple diffusion.

12 n, and from the theoretical distribution for simple diffusion.

13 1, suggesting that import does not occur via simple diffusion.

14 the coexistence of facilitated diffusion and simple diffusion.

15 trations, at rates much faster than that via simple diffusion.

16 barriers, and membrane rafts also occurs by simple diffusion and does not require facilitation by me

17 ly unlike the familiar spreading patterns of simple diffusion, as the kinetics of response are surpri

18 exceeded the expected transit times assuming simple diffusion by orders of magnitude.

19 hown to overcome the slow signaling speed of simple diffusion by utilizing diffusive relays, in which

20 ent with IFNgamma spread being governed by a simple diffusion-consumption model and offer insight int

21 l biologically relevant diffusion processes (simple diffusion, continuous-time random walk, caged dif

22 ding of Ca2+ to the regulatory site is not a simple diffusion-controlled reaction.

23 ith a model in which AEA uptake is driven by simple diffusion coupled to FAAH metabolism.

24 rane (<4 microm), the ISMs pose a barrier to simple diffusion, creating an ion trap.

25 These include simple diffusion, "cytonemes", filopodia, "argosomes", a

26 m of the Bicoid profile is consistent with a simple diffusion/degradation model, the observed length

27 A simple diffusion/extraction steady state model is propos

28 l-molecule inhibitors, including delivery by simple diffusion in the growth medium and concentration-

29 Simple diffusion is sufficient to deliver signals from c

30 Many peptides cross the BBB by simple diffusion, mainly explained by their lipophilicit

31 Taken together, our results suggest that simple diffusion may account for the nuclear export obse

32 t of endocrine cells is not always through a simple diffusion mechanism as presently thought, but ins

33 minute in a manner that is consistent with a simple diffusion mechanism.

34 Recently, however, we challenged the simple diffusion model by showing that a Drosophila orga

35 A simple diffusion model can describe mobility of lipid mo

36 and simulation-based inference, we find this simple diffusion model fails to explain the experimental

37 These data are fit well by a simple diffusion model in which the rate and variability

38 A simple diffusion model indicated that such gradients req

39 A simple diffusion model indicates that such [Na](SL) depl

40 luble recombinant Shh (ShhN)) to support the simple diffusion model of long-range Shh signalling, the

41 A simple diffusion model predicts that the time course of

42 Based on a simple diffusion model, we estimate that this MR imaging

43 A simple diffusion model, where the measured diffusion coe

44 , whose frequency could be predicted using a simple diffusion model.

45 h the brain [1], because of the predominant "simple diffusion" model of steroid hormone transport acr

46 3 orders of magnitude less than expected for simple diffusion models, and the binding progress curves

47 subsecond scales that cannot be explained by simple diffusion models.

48 ne transport in vitro, most methods focus on simple diffusion of a single analyte across nonbiomimeti

49 Concerning flux across the plasma membrane, simple diffusion of ascorbic acid plays only a small or

50 s can be shaped by mechanisms other than the simple diffusion of RA from a localized posterior source

51 o account for the long-range effects of Shh: simple diffusion of Shh, a relay mechanism in which Shh

52 probably different mechanisms and not due to simple diffusion of the protein in a lipid bilayer.

53 erns with a mathematical model that combines simple diffusion of the signal with logistic growth of t

54 g chain acyl-CoA synthesis (LACS) occurs via simple diffusion or a more complex mechanism.

55 a localized site, but whether this occurs by simple diffusion or by more elaborate mechanisms is uncl

56 wer in vivo than compared to in vitro, and a simple diffusion process describes the autocorrelation f

57 A simple diffusion resistance model is shown to be suffici

58 entrations and/or REE profiles indicative of simple diffusion, signifying minimal alteration, have be

59 We review how simple diffusion through solution may be unable to accou

60 ic radius of RNP particles (86 nm) precludes simple diffusion through the mesh of cytoskeletal fibers

61 molecules (<10 kDa) permeate the nucleus by simple diffusion through the pore, but molecules larger

62 ween cells, based on mechanisms ranging from simple diffusion to fluid flow.

63 .8 nm) is representative of native vWF after simple diffusion to the hydrophobic surface, followed by

64 hough many membrane proteins pass the NPC by simple diffusion, two yeast proteins, ScSrc1/ScHeh1 and

65 Apart from simple diffusion, various modes of signal transfer have

66 The passive processes include simple diffusion via the aqueous phase and facilitated d

67 , but they are not trapped and can escape by simple diffusion, which may be slow, fast, or directed.

68 uch steeper concentration profiles than does simple diffusion, which may facilitate neutrophil chemot

69 toplasm and nucleoplasm cannot be modeled by simple diffusion with a unique diffusion constant.