ライフサイエンスコーパス: simplex

1 omal recessive form of epidermolysis bullosa simplex.

2 ng the threshold value using the Nelder-Mead simplex algorithm.

3 ndent exercise-induced anaphylaxis, Anisakis simplex allergy and mast cell disorders.

4 rally high-functioning participants with ASD simplex and 66 typically developing control subjects wit

5 Here, we investigate whether simplex and multiplex vitiligo comprise different diseas

6 genes are co-clustered in genomes of Digenea simplex and Palmaria palmata.

7 cells, DNA viruses such as vaccinia, herpes simplex, and adenovirus induced increased IFN production

8 (c.1434_1435insC [p.Glu479Argfs( *)18]) in a simplex case subject.

9 bjects with a family history of tics than in simplex case subjects.

10 in a multi-ethnic cohort of 718 families and simplex cases with OM.

11 to risk in multiplex vitiligo cases than in simplex cases, supporting a special role for adaptive im

12 GWAS in multiplex vitiligo cases relative to simplex cases.

13 ow-effect-size variants that are relevant in simplex cases.

14 Constrained simplex-centroid design was applied in the optimization

15 s of published genotype data from the Simons Simplex Collection (SSC) and the Autism Genetic Resource

16 ified CNVs in two autism populations (Simons Simplex Collection and MSSNG) and two unselected populat

17 reover, individuals with ASD from the Simons Simplex Collection carrying damaging de novo mutations i

18 enome Project data [1590 parents] and Simons Simplex Collection data [1962 parents]).

19 in both the Autism Genome Project and Simons Simplex Collection datasets.

20 or atypical development of 0.83; the Simons Simplex Collection of ASD families shows similar PPVs.

21 lly discordant sibling pairs from the Simons Simplex Collection study for autism spectrum disorders,

22 In the Simons Simplex Collection, there was also significant evidence

23 ere obtained from 2614 trios from the Simons Simplex Collection.

24 and their unaffected siblings in the Simons Simplex Collection.

25 ents affected by dystrophic, junctional, and simplex EB.

26 Fatal herpes simplex encephalitis (HSE) results from immune pathology

27 Herpes simplex encephalitis (HSE), caused by HSV type 1 (HSV-1)

28 umours, usually ovarian teratoma, and herpes simplex encephalitis are known triggers of NMDAR autoimm

29 he class means collapse to the vertices of a simplex equiangular tight frame (ETF).

30 to the class means or in other words, to the simplex ETF (i.e., to a self-dual configuration).

31 In the trio analysis of 30 simplex families (patient and parent trios), we identifi

32 work to 1,790 autism spectrum disorder (ASD) simplex families reveals a role in disease for noncoding

33 ere preferentially found in multiplex versus simplex families.

34 rios of Ashkenazi Jewish descent, selecting "simplex" families with no family history of BD and an ea

35 ion (including cytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV], blood

36 ing the underlying molecular abnormality: EB simplex, junctional EB, dystrophic EB and Kindler EB.

37 ients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent during

38 Ocular herpes simplex keratitis (HSK) is a consequence of viral reacti

39 Herpes simplex keratitis (HSK), caused by herpes simplex virus

40 life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients without

41 ng the statistical tool of mixture planning, simplex-lattice design.

42 The optimization was carried out using a simplex method.

43 Simplex mRNA Sequencing (RNA-Seq) based isoform quantifi

44 estigate models of microbial dynamics in the simplex of relative abundances.

45 er, were determined using the super modified simplex optimization method by means of percentage error

46 ion of the cerebellar vermis (lobule 4/5) or simplex produced reliable and specific effects in hippoc

47 step and refined the two models by downhill simplex runs against experimental data for the force-vel

48 Exact simplex solvers based on rational arithmetic require a n

49 family age-related hearing loss (mARHL) and simplex/sporadic age-related hearing loss (sARHL) cases

50 cal trial of 106 patients with active herpes simplex stromal keratitis who had not received any corti

51 ion and in shortening the duration of herpes simplex stromal keratitis.

52 ctural organization of the virions of Herpes Simplex Type 1 viruses and bacteriophage MS2.

53 ents with bacterial and viral (due to herpes simplex, varicella zoster, and enteroviruses) meningitis

54 d monoclonal IgG antibody against the herpes simplex viral protein glycoprotein D (gD) was radiolabel

55 We developed a herpes simplex viral vector to rapidly yet transiently overexpr

56 ridae-positive, which included 9 with herpes simplex virus (8.8%), 5 with varicella-zoster virus (4.9

57 d polymerase chain reaction (PCR) for herpes simplex virus (HSV) and varicella zoster virus was done

58 Herpes simplex virus (HSV) can cause severe infection in neonat

59 Neonatal herpes simplex virus (HSV) disease results in unacceptable morb

60 into the brain stem.IMPORTANCE Latent herpes simplex virus (HSV) DNA has been detected in the central

61 rus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions with a vi

62 Herpes simplex virus (HSV) establishes latency in neurons of th

63 Herpes simplex virus (HSV) establishes lifelong latent infectio

64 Herpes simplex virus (HSV) glycoprotein C (gC) functions in vir

65 We previously reported that herpes simplex virus (HSV) glycoprotein K (gK) binds to signal

66 ein-protein interactions between four herpes simplex virus (HSV) glycoproteins (gD, gH/gL, and gB) dr

67 The herpes simplex virus (HSV) heterodimer gE/gI and another membra

68 n was associated with reduced odds of herpes simplex virus (HSV) infection among MSM overall (0.84, 0

69 erous cases point to the link between herpes simplex virus (HSV) infection and multifocal CNS demyeli

70 Herpes simplex virus (HSV) infection is restricted to epithelia

71 Herpes simplex virus (HSV) infection is widespread in the human

72 equently complicates the treatment of herpes simplex virus (HSV) infections in immunocompromised pati

73 Herpes simplex virus (HSV) is a common and often benign infecti

74 Herpes simplex virus (HSV) is a neuroinvasive virus that has be

75 Herpes simplex virus (HSV) is among the most prevalent viral in

76 The terminase of herpes simplex virus (HSV) is composed of three subunits encode

77 cells expressing the LATs.IMPORTANCE Herpes simplex virus (HSV) is responsible for significant morbi

78 Herpes simplex virus (HSV) is the main cause of viral encephali

79 us or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 7

80 isualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

81 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerve destr

82 particles and a replication-defective herpes simplex virus (HSV) recombinant vector.

83 Herpes simplex virus (HSV) requires fusion between the viral en

84 The herpes simplex virus (HSV) single-strand DNA binding protein (S

85 emic antimicrobial use, imaging data, herpes simplex virus (HSV) testing, and overall hospital charge

86 munotherapy with oncolytic engineered herpes simplex virus (HSV) therapy, are urgently warranted.

87 The herpes simplex virus (HSV) type I alkaline nuclease, UL12, has

88 meningoencephalitis, 6% influenza, 6% herpes simplex virus (HSV), and 6% Mycoplasma pneumoniae; 25% (

89 nal swabs were collected to test HPV, herpes simplex virus (HSV), and 7 STIs.

90 V, respiratory syncytial virus (RSV), herpes simplex virus (HSV), and measles.

91 ertain viruses, including poliovirus, herpes simplex virus (HSV), cytomegalovirus (CMV), and influenz

92 diverse glycan specificities such as Herpes Simplex Virus (HSV), Influenza A Virus (IAV), and Merkel

93 Other alphaherpesviruses, including herpes simplex virus (HSV), utilize low-pH-mediated endocytosis

94 osed with-congenital cytomegalovirus, herpes simplex virus (HSV), varicella zoster virus (VZV), and r

95 The ubiquitous human pathogens, herpes simplex virus (HSV)-1 and HSV-2, are distinct viral spec

96 Herpes simplex virus (HSV)-1 proteins pUL7 and pUL51 form a com

97 n STING are unexpectedly resistant to Herpes Simplex Virus (HSV)-1, despite lacking STING-induced typ

98 ) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

99 The herpes simplex virus (HSV-1) latency-associated transcript (LAT

100 is a viral restriction factor against herpes simplex virus (HSV-1).

101 One compound was also active against herpes simplex virus (HSV1 and HSV2), and another compound was

102 to constitutively express the type I Herpes Simplex Virus (HSV1) HSV-1 receptor, nectin-1, to allow

103 Oncolytic herpes simplex virus (oHSV) selectively replicates in cancer ce

104 (KO) mice were infected ocularly with herpes simplex virus 1 (HSV-1) (strain McKrae).

105 In the case of the herpes simplex virus 1 (HSV-1) 0DeltaNLS vaccine, the correlate

106 ruses and include the human pathogens herpes simplex virus 1 (HSV-1) and HSV-2 and are significant ca

107 Herpes simplex virus 1 (HSV-1) and HSV-2 can efficiently establ

108 mbly of the neurotropic herpesviruses herpes simplex virus 1 (HSV-1) and pseudorabies virus (PRV) in

109 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (PRV), ha

110 that these cells can be infected with herpes simplex virus 1 (HSV-1) at a multiplicity of infection (

111 nfections in HeLa cells infected with herpes simplex virus 1 (HSV-1) at high multiplicity of infectio

112 olated from mice can be infected with herpes simplex virus 1 (HSV-1) but that subsequent replication

113 Herpes simplex virus 1 (HSV-1) can induce damage in brain regio

114 Herpes simplex virus 1 (HSV-1) can infect virtually all cell ty

115 Herpes simplex virus 1 (HSV-1) causes a lifelong infection of n

116 Herpes simplex virus 1 (HSV-1) causes significant morbidity and

117 Herpes simplex virus 1 (HSV-1) cycles between phases of latency

118 ether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 from the

119 as bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) establish and maintain lifelong

120 to reactivate from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes a lifelong infection

121 Herpes simplex virus 1 (HSV-1) establishes a lifelong latent in

122 Herpes simplex virus 1 (HSV-1) establishes latency in both peri

123 Herpes simplex virus 1 (HSV-1) establishes latency within the s

124 Following acute infection, herpes simplex virus 1 (HSV-1) establishes lifelong latency in

125 Herpes simplex virus 1 (HSV-1) establishes lifelong latent infe

126 reactivation from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes lifelong latent infe

127 Chronic viruses such as herpes simplex virus 1 (HSV-1) evade the hosts' immune system b

128 An earlier report showed that herpes simplex virus 1 (HSV-1) expresses two microRNAs (miRNAs)

129 During all stages of infection, herpes simplex virus 1 (HSV-1) expresses viral microRNAs (miRNA

130 of several cellular receptors used by herpes simplex virus 1 (HSV-1) for cell entry.

131 Reactivation of herpes simplex virus 1 (HSV-1) from neurons in sensory ganglia

132 The Us11 protein encoded by herpes simplex virus 1 (HSV-1) functions to impair autophagy; h

133 Herpes simplex virus 1 (HSV-1) genes are transcribed by cellula

134 ed from the long-arm component of the herpes simplex virus 1 (HSV-1) genome, (iv) pUL36 serves no dir

135 Herpes simplex virus 1 (HSV-1) has infected more than 80% of th

136 cted 80 open reading frames (ORFs) of herpes simplex virus 1 (HSV-1) have been intensively studied fo

137 ation of histone H3K9 associated with herpes simplex virus 1 (HSV-1) immediate early (IE) promoters a

138 The herpes simplex virus 1 (HSV-1) immediate early protein ICP22 pl

139 e of CD8(+) T cells in the control of herpes simplex virus 1 (HSV-1) infection and disease is gaining

140 established a potential link between herpes simplex virus 1 (HSV-1) infection and the development of

141 Analysis of Herpes Simplex virus 1 (HSV-1) infection by population-averaged

142 ollowing vaccination in resistance to herpes simplex virus 1 (HSV-1) infection continues to be rigoro

143 We have previously shown that herpes simplex virus 1 (HSV-1) infection results in the drastic

144 We recently reported that herpes simplex virus 1 (HSV-1) infection suppresses CD80 but no

145 ice were highly susceptible to ocular herpes simplex virus 1 (HSV-1) infection, independent of the pr

146 Herpes simplex virus 1 (HSV-1) infections afflict more than 80%

147 Herpes simplex virus 1 (HSV-1) infects mucosal epithelial cells

148 Herpes simplex virus 1 (HSV-1) infects several types of cells,

149 In our murine oro-ocular (OO) model, herpes simplex virus 1 (HSV-1) inoculation in one side of the l

150 Herpes simplex virus 1 (HSV-1) is a contagious neurotropic herp

151 The envelope glycoprotein I (gI) of herpes simplex virus 1 (HSV-1) is a critical mediator of virus-

152 Herpes simplex virus 1 (HSV-1) is a leading cause of infectious

153 oteolipids in this process.IMPORTANCE Herpes simplex virus 1 (HSV-1) is a neurotropic pathogen that c

154 displacement of the portal.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of severa

155 Utilizing a mouse model of herpes simplex virus 1 (HSV-1) keratitis, we found that infecti

156 High rates of wild-type (WT) herpes simplex virus 1 (HSV-1) latency reactivation depend on t

157 nsulator protein CTCF plays a role in herpes simplex virus 1 (HSV-1) latency through the establishmen

158 Corneal infection with herpes simplex virus 1 (HSV-1) leads to infection of trigeminal

159 iously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclear local

160 etry approach, we have shown that the herpes simplex virus 1 (HSV-1) neurovirulence- and autophagy-mo

161 ct on cell-intrinsic immunity against herpes simplex virus 1 (HSV-1) or gammaherpesvirus 68 (gammaHV6

162 umber of the world population harbors herpes simplex virus 1 (HSV-1) potentially leading to blinding

163 tes bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) reactivation.

164 de transneuronal tracers derived from herpes simplex virus 1 (HSV-1) strain 129 (H129) are important

165 The features of herpes simplex virus 1 (HSV-1) strain 129 (H129), including nat

166 4.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN signali

167 We used herpes simplex virus 1 (HSV-1) to infect the human DRG-derived

168 ful stimuli.IMPORTANCE The ability of herpes simplex virus 1 (HSV-1) to periodically reactivate from

169 smission to nerve endings, capsids of herpes simplex virus 1 (HSV-1) travel retrogradely within axons

170 Herpes simplex virus 1 (HSV-1) triggers both the cyclic GMP-AMP

171 n, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which has be

172 Here, we show that herpes simplex virus 1 (HSV-1) virions travel in association wi

173 Entry of the human alphaherpesvirus herpes simplex virus 1 (HSV-1) was independent of both host SM

174 g the three groups, the prevalence of herpes simplex virus 1 (HSV-1) were 9% in saliva and 5% in GCF;

175 rtion of the world population harbors herpes simplex virus 1 (HSV-1), a major cause of infectious cor

176 n 0 (ICP0), an E3 ubiquitin ligase of herpes simplex virus 1 (HSV-1), can derepress viral genes by de

177 Oncolytic herpes simplex virus 1 (HSV-1), devoid of the gamma(1)34.5 gene

178 In cells infected with herpes simplex virus 1 (HSV-1), hnRNPA2B1 was quantitatively ex

179 ive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian pseudor

180 ies of the prototypical herpesviruses herpes simplex virus 1 (HSV-1), HSV-2, human cytomegalovirus (H

181 on understanding the biology of human herpes simplex virus 1 (HSV-1), no tool has been developed to s

182 and viruses, including T4, Phi29, and herpes simplex virus 1 (HSV-1), the portal forms a nucleation c

183 terminal endocytic pathway.IMPORTANCE Herpes simplex virus 1 (HSV-1), the prototype alphaherpesvirus,

184 For herpes simplex virus 1 (HSV-1), we and others have previously p

185 In herpes simplex virus 1 (HSV-1)-infected cells, hnRNPA2B1 was qu

186 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed the vi

187 re distantly related alphaherpesvirus herpes simplex virus 1 (HSV-1).

188 are activated early in infection with herpes simplex virus 1 (HSV-1).

189 Epstein-Barr virus (EBV), and vhs in herpes simplex virus 1 (HSV-1).

190 at can enter via the plasma membrane, herpes simplex virus 1 (HSV-1).

191 bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

192 han other alphaherpesviruses, such as herpes simplex virus 1 (HSV1) or pseudorabies virus (PRV).

193 In human cells, herpes simplex virus 1 (HSV1) UL39-encoded ICP6 blocks RIP homo

194 re derived from the DNA polymerase of herpes simplex virus 1 (Pol peptides).

195 viruses (pseudorabies virus [PRV] and herpes simplex virus 1 [HSV-1]).

196 uman herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex virus 1 [HSV-1], HSV-2, JC virus [JCV], and vari

197 l alphaherpesvirus that is related to herpes simplex virus 1 and causes respiratory, reproductive, an

198 his VZV study also complemented prior herpes simplex virus 1 and pseudorabies virus studies investiga

199 ce of brefeldin A, while studies with herpes simplex virus 1 documented an impaired secondary assembl

200 accharyltransferase with NGI-1 causes herpes simplex virus 1 dysfunction.

201 sease can occur after reactivation of herpes simplex virus 1 in the trigeminal ganglia, leading to di

202 Furthermore, time-lapse imaging of herpes simplex virus 1 infected epithelial cells enabled by our

203 pendently of this activity.IMPORTANCE Herpes simplex virus 1 invades the nervous system by entering n

204 thin tumor cells.IMPORTANCE Oncolytic herpes simplex virus 1 is a promising agent for cancer immunoth

205 ctional antibody responses.IMPORTANCE Herpes simplex virus 1 is the leading cause of infectious corne

206 ively kill senescent cells expressing herpes simplex virus 1 thymidine kinase (HSV-TK).

207 ct association between infection with herpes simplex virus 1, a ubiquitous human pathogen generally a

208 influenza virus, dengue virus type 2, herpes simplex virus 1, and nonenveloped human adenovirus 5.

209 itis virus, Semliki Forest virus, and herpes simplex virus 1, elicit the neuronal expression of a hos

210 eas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a strong ant

211 gher incidence of cytomegalovirus and herpes simplex virus 1, possibly influenced by demographic cova

212 worldwide are latently infected with herpes simplex virus 1.

213 an immunodeficiency virus (HIV-1) and herpes simplex virus 2 (HSV-2) affect hundreds of millions of p

214 Herpes simplex virus 2 (HSV-2) can be transmitted in the presen

215 Reactivation of herpes simplex virus 2 (HSV-2) from latency causes viral sheddi

216 the last virus inoculation.IMPORTANCE Herpes simplex virus 2 (HSV-2) infects nearly 500 million perso

217 Herpes simplex virus 2 (HSV-2) is a common sexually transmitted

218 Reactivation of herpes simplex virus 2 (HSV-2) results in infection of epitheli

219 double-stranded RNA or infection with herpes simplex virus 2 (HSV-2).

220 Herpes simplex virus 2 (HSV2) causes genital herpes in >400 mil

221 ition to one of three TAP inhibitors: herpes simplex virus 2 ICP47, bovine herpes virus 1 UL49.5, or

222 certain microbial stimuli, including herpes simplex virus 2, and blocked by antibodies against the i

223 nstead, upon secondary challenge with herpes simplex virus 2, circulating memory B cells that enter t

224 Thirteen patients (62%) had herpes simplex virus and 8 patients (38%) had varicella zoster

225 ecimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 months apar

226 , presence of ovarian teratoma, prior herpes simplex virus encephalitis, and isolated psychiatric pre

227 reports linking TLR3 deficiency with herpes simplex virus encephalitis.

228 Like all the herpesviruses, herpes simplex virus encodes machinery that enables it to move

229 tromal fibroblasts restores oncolytic herpes simplex virus function.

230 binding protein 4)(27); MICA(41); the herpes simplex virus glycoprotein HSVgI(33); ATPase-apolipoprot

231 of tests are available for detecting herpes simplex virus infection, but only a subset are useful an

232 ound bacterial pigment in controlling herpes simplex virus infection, for which diverse and multimoda

233 During Herpes Simplex Virus infection, viral replication compartments

234 ion of neonatal infections.IMPORTANCE Herpes simplex virus is among the most serious infections of ne

235 creased the recurrence of any type of herpes simplex virus keratitis by approximately half.

236 cally diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated be

237 ty during HSV-1 infections.IMPORTANCE Herpes simplex virus persists lifelong in neurons and can react

238 respiratory syncytial virus (RSV) and herpes simplex virus type 1 (HSV-1) accumulate a rich and disti

239 Here we report that GA inhibits Herpes simplex virus type 1 (HSV-1) by inhibition of both fusio

240 This study aimed at characterizing herpes simplex virus type 1 (HSV-1) epidemiology in the Middle

241 Herpes simplex virus type 1 (HSV-1) has the ability to delay it

242 Orolabial herpes simplex virus type 1 (HSV-1) infection has a wide spectr

243 es simplex keratitis (HSK), caused by herpes simplex virus type 1 (HSV-1) infection, is the commonest

244 In herpes simplex virus type 1 (HSV-1) infection, the coupling of

245 ciated with protective and pathogenic herpes simplex virus type 1 (HSV-1) infections remains unclear.

246 Each herpes simplex virus type 1 (HSV-1) replication compartment (RC

247 l stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies upon nu

248 we use direct RNA-seq to profile the herpes simplex virus type 1 (HSV-1) transcriptome during produc

249 d spread of vaccinia virus (VACV) and herpes simplex virus type 1 (HSV-1) were enhanced in HDAC4(-/-)

250 genome packaging and organization in herpes simplex virus type 1 (HSV-1), we developed sequential lo

251 nal approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large DNA geno

252 Herpes simplex virus type 1 (HSV-1)-infected corneas can develo

253 ctive functions during infection with herpes simplex virus type 1 (HSV-1).

254 are not permissive for infection with herpes simplex virus type 1 (HSV-1).

255 Herpes simplex virus type 1 (HSV1) infection has been associate

256 the complexity of the interactome of herpes simplex virus type 1 (HSV1), the prototypical herpesviru

257 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the brains

258 Herpes simplex virus type 1 and Alzheimer's disease: possible m

259 are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

260 t infection with, or reactivation of, herpes simplex virus type 1 or type 1/2 unspecified, cytomegalo

261 The herpes simplex virus type 1 thymidine kinase (HSV1-tk) gene has

262 ponses to chronic infections, such as herpes simplex virus type 2 (HSV-2) in HIV/HSV-coinfected perso

263 tion were seen in smokers, those with herpes simplex virus type 2 (HSV-2) infection, men who had sex

264 at least tripled in individuals with herpes simplex virus type 2 (HSV-2) infection.

265 Genital infection with herpes simplex virus type 2 (HSV-2) is common and increases ris

266 This study investigated herpes simplex virus type 2 (HSV-2) seroprevalence utility as a

267 cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

268 Interestingly, cells infected with herpes simplex virus type-1 (HSV-1) incorporated EdC and EdU at

269 Herpes simplex virus type-1 (HSV-1), one of the most widely spr

270 trols for sociodemographic variables, herpes simplex virus type-2 status, and recreational drug use.

271 he development of acyclovir-resistant herpes simplex virus viremia, primary graft failure, and sinuso

272 richomonas vaginalis, adenovirus, and herpes simplex virus were absent.

273 s, often an adeno-associated virus or herpes simplex virus, among many other types.

274 investigated for Epstein-Barr virus, herpes simplex virus, and HCMV-specific immunoglobulin G (IgG),

275 oma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus, and Eps

276 For herpes simplex virus, this process requires the products of sev

277 onse to HCMV, but not Epstein-Barr or herpes simplex virus, was associated with increased risk of act

278 Herpes simplex virus-1 (HSV-1) encephalitis (HSE) is typically

279 Herpes simplex virus-1 (HSV-1) establishes a latent infection i

280 immunity in mouse and human models of herpes simplex virus-1 (HSV-1) infections.

281 powerful host defense that restricts herpes simplex virus-1 (HSV-1) pathogenesis in neurons.

282 Herpes simplex virus-1 (HSV-1) replicates within the nucleus co

283 Neurotropic viruses, such as herpes simplex virus-1 (HSV-1), also rely on cellular AKT-mTORC

284 Infection by viruses, including herpes simplex virus-1 (HSV-1), and cellular stresses cause wid

285 ncordantly, GPX4 deficiency inhibited herpes simplex virus-1 (HSV-1)-induced innate antiviral immune

286 al other pathogenic viruses including Herpes Simplex Virus-1 and-2, Vaccinia virus, and Zika virus th

287 alovirus (MCMV) or the human pathogen herpes simplex virus-1 compared with littermate control RIPK3-d

288 class B infections (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis E viru

289 nisms underlying rapid elimination of herpes simplex virus-2 (HSV-2) in the human genital tract despi

290 PORTANCE We study the pathogenesis of herpes simplex virus-mediated corneal disease.

291 irus (EBV), cytomegalovirus (CMV) and Herpes simplex virus.

292 clude the significant human pathogens herpes simplex viruses (HSV) and varicella zoster virus (VZV).

293 he same genetic variations.IMPORTANCE Herpes simplex viruses (HSV) infect a majority of adults.

294 Finland are seropositive carriers of herpes simplex viruses (HSV).

295 ort of enveloped particles.IMPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster virus cause

296 erpesvirus related to human pathogens herpes simplex viruses 1 and 2 and varicella-zoster virus.

297 Herpes simplex viruses bud into the nuclear membrane of infecte

298 ses such as varicella-zoster virus or herpes simplex viruses.

299 Most vitiligo cases are "simplex," where there is no family history of vitiligo,

300 itional data refer to the data that lie on a simplex, which are common in many scientific domains suc