ライフサイエンスコーパス: sinus

1 d an left ventricular branch of the coronary sinus.

2 endocardium and epicardium via the coronary sinus.

3 subspecies zooepidemicus from the maxillary sinus.

4 d sampling from coronary artery and coronary sinus.

5 onal epicardial ablation within the coronary sinus.

6 t supplies the lateral wall of the maxillary sinus.

7 valuate 200 patients making up 400 maxillary sinuses.

8 the frontal, anterior ethmoid, and sphenoid sinuses.

9 forming system for localized delivery to the sinuses.

10 (4.7%), anastomotic fistula (0.8%), chronic sinus (0.9%), and anastomotic stricture in 3.6% of cases

11 the intraosseous artery to the floor of the sinus, (2) the average length of the artery, (3) the dia

12 core was -0.89 (-1.07 to -0.71; p<0.0001) in SINUS-24 and -0.87 (-1.03 to -0.71; p<0.0001) in SINUS-5

13 s -2.06 (95% CI -2.43 to -1.69; p<0.0001) in SINUS-24 and -1.80 (-2.10 to -1.51; p<0.0001) in SINUS-5

14 ores was -7.44 (-8.35 to -6.53; p<0.0001) in SINUS-24 and -5.13 (-5.80 to -4.46; p<0.0001) in SINUS-5

15 LIBERTY NP SINUS-24 and LIBERTY NP SINUS-52 were two multinational,

16 -52 up to week 24 and the dupilumab group in SINUS-24 and the placebo groups in both studies until we

17 SINUS-24 was done in 67 centres in 13 countries, and SIN

18 Patients in SINUS-24 were randomly assigned (1:1) to subcutaneous du

19 d Aug 3, 2017, 276 patients were enrolled in SINUS-24, with 143 in the dupilumab group and 133 in the

20 ooled population of both dupilumab groups in SINUS-52 up to week 24 and the dupilumab group in SINUS-

21 was done in 67 centres in 13 countries, and SINUS-52 was done in 117 centres in 14 countries.

22 Patients in SINUS-52 were randomly assigned (1:1:1) to dupilumab 300

23 LIBERTY NP SINUS-24 and LIBERTY NP SINUS-52 were two multinational, multicentre, randomised

24 Aug 28, 2017, 448 patients were enrolled in SINUS-52, with 150 receiving at least one dose of dupilu

25 S-24 and -5.13 (-5.80 to -4.46; p<0.0001) in SINUS-52.

26 S-24 and -0.87 (-1.03 to -0.71; p<0.0001) in SINUS-52; and difference in Lund-Mackay CT scores was -7

27 S-24 and -1.80 (-2.10 to -1.51; p<0.0001) in SINUS-52; difference in nasal congestion or obstruction

28 mus time (>=100 ms) and reversal of coronary sinus activation during pacing from the left atrial appe

29 mus conduction time and reversal in coronary sinus activation to falsely suggest block.

30 ligonucleotides led to a reduction in aortic sinus and en face lesion areas (47.2% or 58.8% decrease

31 sentation of skin folds coincides with blood sinus and folds of the rostrum gyrus.

32 xillary sinuses, ethmoid air cells, sphenoid sinus and frontal sinuses yielded the highest prediction

33 ernans voltage on the body surface, coronary sinus and left ventricle leads, requires a delivered cha

34 169/Siglec-1-mediated capture by subcapsular sinus and marginal zone metallophilic macrophages for tr

35 ng laterally at the proximal-middle coronary sinus and septally at the left atrial ridge.

36 model of CRS, TEMPS was maintained in rabbit sinuses and effectively reduced sinonasal inflammation a

37 ions, but these infections lack the draining sinuses and fungal grains characteristic of eumycetoma.

38 bullosa is connected with the development of sinuses and the incidence of inflammation within them.

39 g from lymphatic vasculature along the dural sinuses and the middle meningeal artery.

40 in implants inserted in augmented maxillary sinuses and to analyze possible risk factors.

41 metabolomics on blood from artery, coronary sinus, and femoral vein in 110 patients with or without

42 ncement of the vein of Labbe, sphenoparietal sinus, and superficial middle cerebral vein was graded b

43 The nose, paranasal sinuses, and associated lymphoid tissues play important

44 on was shown in lungs, upper airway, cranial sinuses, and intestines because of improved field homoge

45 one graft consolidation within the maxillary sinus are rare.

46 cement of bioglass and/or allograft into the sinus area using an osteotome technique in 37 patients w

47 nction (SND) clinically include bradycardia, sinus arrest, and chronotropic incompetence and may serv

48 we assessed the influence of the Respiratory Sinus Arrhythmia (RSA) while estimating the resting f(H)

49 changes in respiration-corrected respiratory sinus arrhythmia (RSAc)-an established metric of HRV tha

50 he symbiotic pacemaker successfully corrects sinus arrhythmia and prevents deterioration.

51 ere increased in rats paced with respiratory sinus arrhythmia compared to monotonic pacing, via impro

52 Respiratory sinus arrhythmia is physiological pacing of the heart th

53 ed daily for 2 weeks with either respiratory sinus arrhythmia or paced monotonically at a matched hea

54 tor to heart rate variability is respiratory sinus arrhythmia or RSA - an intrinsic respiratory modul

55 We propose that respiratory sinus arrhythmia pacing reverse-remodels the heart in he

56 e proposed that reinstatement of respiratory sinus arrhythmia would improve cardiac function in rats

57 t rhythm abnormalities (i.e. sinus pause and sinus arrhythmias) when compared to control mice.

58 enome of mice resulted in SAN hypoplasia and sinus arrhythmias.

59 ate T follicular helper cells) and lymphatic sinus-associated SIGNR1(+) macrophages (which can activa

60 regeneration, guided bone regeneration, and sinus augmentation (P < 0.0001).

61 tients who underwent lateral or transcrestal sinus augmentation and received dental implants.

62 ecords of 551 patients who underwent lateral sinus augmentation at Tufts University School of Dental

63 rafting such as guided bone regeneration and sinus augmentation compared with socket preservation (P

64 Sinus augmentation with synthetic HA/TCP and DBBM exhibi

65 edback reflex mediated by aortic and carotid sinus baroreceptors when systemic arterial pressure is l

66 Eighty-five human sinus biopsies were harvested 6 +/- 1 months after MSFA.

67 lications with untapped opportunities (e.g., sinus, bladder, and colon).

68 An electrocardiogram showed sinus bradycardia and nonspecific T wave changes.

69 ation, general supraventricular tachycardia, sinus bradycardia and sinus rhythm including sinus irreg

70 e characterized by sinus dysrhythmia, severe sinus bradycardia, sinus pauses and chronotropic incompe

71 use model of heart failure in which there is sinus bradycardia, there is upregulation of a microRNA (

72 in and I(f) in the sinus node and blunts the sinus bradycardia.

73 TCP) particles were inserted into one of the sinus cavities using the extra-oral approach, where depr

74 ng presurgical nasal lavage of patients at a sinus clinic.

75 -target binding regions included the ethmoid sinus, clivus, meninges, substantia nigra, but not the b

76 graphic examination, the mean thicknesses of sinus cortexes for DBBM and HA/TCP groups were similar (

77 ly ablated from within the proximal coronary sinus (CS) guided by recorded potentials at the roof of

78 ue to an inability or impediment to coronary sinus (CS) lead implantation.

79 that the presence of dual muscular coronary sinus (CS) to left atrial (LA) connections, coupled with

80 We collected questionnaires and sinus CT scans from 646 participants selected from a sou

81 ted with hemorrhage in patients with lateral sinus DAVFs than does CVR, and thus may offer guidance i

82 The presence of paranasal sinus disease in association with loss of vision even in

83 s significantly correlated with radiographic sinus disease severity (r = 0.56; P < .001) and were ass

84 tibiotic allergy, lower FEV(1), radiographic sinus disease severity, nasal polyposis, and systemic co

85 mical variations and their relation to known sinus drainage pathways.

86 int prostheses, the presence of preoperative sinus drainage was significantly associated with reinfec

87 to look for mucosal disease of the paranasal sinuses, drainage pathways, and presence of anatomical v

88 a complex cardiac phenotype characterized by sinus dysrhythmia, severe sinus bradycardia, sinus pause

89 ltilevel analysis, history of periodontitis, sinus elevation with lateral approach, and one-stage sin

90 model including the nasal cavity, maxillary sinuses, ethmoid air cells, sphenoid sinus and frontal s

91 tivity was assessed in the superior sagittal sinus, evaluating the breath-hold index.

92 Renal sinus fat (RSF) is a perivascular fat compartment locate

93 compression by visceral, perirenal and renal sinus fat; increased renal sympathetic nerve activity (R

94 definitions, without assessment of objective sinus findings.

95 of 100 mum proceeding from the bottom of the sinus floor (SF) up to the apical top of the biopsy.

96 Maxillary sinus floor augmentation (MSFA) is a well-established an

97 This study investigates influence of the sinus floor configuration on dimensional stability of gr

98 rgical removal of impacted teeth and lateral sinus floor elevation are more prone to more severe comp

99 rgical removal of impacted teeth and lateral sinus floor elevation had the highest incidence and seve

100 both lateral window technique and one-stage sinus floor elevation seemed to represent significant ri

101 evation with lateral approach, and one-stage sinus floor elevation significantly correlated with the

102 al procedures including, but not limited to, sinus floor elevation, guided tissue regeneration, crown

103 remolar area is <21% during a lateral window sinus floor elevation.

104 T cells subsequently migrate randomly on the sinus floor independent of both chemokines and integrins

105 ivering localized treatment to the paranasal sinuses for diseases such as chronic rhinosinusitis (CRS

106 tissue remodeling of the nose and paranasal sinuses, frequently occurring with nasal polyps and alle

107 Mean initial gain of sinus grafted bone height was 7.0 +/- 1.9 mm, and later

108 y of grafted bone height after the osteotome sinus grafting procedure.

109 Peri-sinus IgA plasma cells increased with age and following

110 e expression in the arterial wall and aortic sinus induced by severe periodontitis.

111 is not a well-documented cause of paranasal sinus infection.

112 (74%) and most commonly consisted of ear or sinus infections (43 of 120, 36%) and cerebrospinal flui

113 ngitis occurs mainly in patients with ear or sinus infections and cerebrospinal fluid leakage.

114 s in the wall of the aortic arch and carotid sinus initiates autonomic reflexes to change heart rate

115 The method employs transverse sinus injections of 2-4 muL of AAV9 at P0.

116 sinus bradycardia and sinus rhythm including sinus irregularity rhythm.

117 total fusion in which the superior sagittal sinus is shared.

118 Lead placement via the coronary sinus is the mainstay approach of cardiac resynchronizat

119 i because placing a stent in stenosed venous sinuses is a novel treatment option in patients who are

120 r7 (-/-) mice showed reduced aortic arch and sinus lesion areas.

121 Here, we identify a subset of lymphoid organ sinus lining macrophage (SMs) that provide a cell-cell c

122 (NPS), nasal congestion or obstruction, and sinus Lund-Mackay CT scores (a coprimary endpoint in Jap

123 decreased the network of SSMs and medullary sinus macrophages (MSMs).

124 In lymph nodes, subcapsular sinus macrophages (SSMs) form an immunological barrier t

125 as strains, potentially originating from the sinuses, may seed the allograft leading to infections an

126 Of these types, only ring-sinus Merkel endings exhibited slowly adapting propertie

127 anoreceptors in the vibrissal follicle: ring-sinus Merkel; lanceolate; clublike; and rete-ridge colla

128 vivo study, PrSPCs mixed with rat urogenital sinus mesenchyme were grafted under the renal capsule of

129 Although the sinus microbial ecology is highly variable between indiv

130 Furthermore, the variability of the sinus microbiome across geographical divides remains une

131 ization and translational application of the sinus microbiota.

132 tractants, whereas LECs lining the medullary sinus (MS) expressed a C-type lectin CD209.

133 n = 12), Hinman syndrome (n = 6), urogenital sinus (n = 4), and other pathologies (n = 4) were includ

134 sing carotid body signalling through carotid sinus nerve (CSN) modulation may offer a therapeutic app

135 We found that leptin enhanced carotid sinus nerve activity at baseline and in response to 10%

136 Leptin increased carotid sinus nerve activity at baseline and in response to hypo

137 e is most often due to damage of the carotid sinus nerve because of neck surgery or radiation.

138 Sinus node (SAN) dysfunction (SND) manifests as low hear

139 The sinus node (SAN) is the primary pacemaker of the human h

140 In right atrial preparations, sinus node (SN) was dominant and AVRs displayed 1:1 impu

141 emature cessation of exercise before maximal sinus node activation.

142 stores HCN4 mRNA and protein and I(f) in the sinus node and blunts the sinus bradycardia.

143 metry, we here quantify >7,000 proteins from sinus node and neighbouring atrial muscle.

144 commonly occurring in these patients affect sinus node beating rate and could be responsible for sev

145 y, peak HR remained low, suggesting impaired sinus node beta-receptor function may not fully account

146 performing single-nucleus RNA sequencing of sinus node biopsies, we attribute measured protein abund

147 computational model of human sinus node cells to account for the dynamic intracellula

148 er implantation (atrio-ventricular blocks-5; sinus node disease-2), 3 patients developed atrial fibri

149 Inherited forms of sinus node dysfunction (SND) clinically include bradycar

150 Sinus node dysfunction (SND) is often associated with at

151 t loss of an RE at the HCN4 locus results in sinus node dysfunction and reduced gene expression.

152 CPVT, such as the pathophysiological role of sinus node dysfunction in CPVT, and whether the arrhythm

153 eatment for patients with bradycardia due to sinus node dysfunction or atrioventricular block.

154 gous for the RE deletion showed bradycardia, sinus node dysfunction, and selective loss of Hcn4 expre

155 Indications for HBP were sinus node dysfunction, atrioventricular conduction dise

156 med SAN at birth, the mutant mice manifested sinus node dysfunction.

157 eous depolarization of cardiomyocytes in the sinus node forming the primary natural pacemaker.

158 The sinus node is a collection of highly specialised cells c

159 ng, we estimate ion channel copy numbers for sinus node myocytes.

160 nd may serve as disease models to understand sinus node physiology and impulse generation.

161 n potential model result in pacemaking and a sinus node-like action potential.

162 he corresponding ionic current, I(f), in the sinus node.

163 ion of I(f), and bradycardia in the isolated sinus node.

164 mphatic endothelial cells in the subcapsular sinus of the LN.

165 Computed tomography simulation predicted sinus of Valsalva sequestration and resultant coronary o

166 Macrophage numbers in aortic sinuses of CD11d(-/-) mice were reduced without affectin

167 unding large veins draining toward the dural sinuses on fluid-attenuated inversion recovery in subjec

168 ral population-based sample in Pennsylvania, sinus opacification was more common among men than in wo

169 severe CRSwNP, dupilumab reduced polyp size, sinus opacification, and severity of symptoms and was we

170 pelvic abscess, anastomotic fistula, chronic sinus, or anastomotic stricture.

171 th larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

172 axillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

173 rt rate and heart rhythm abnormalities (i.e. sinus pause and sinus arrhythmias) when compared to cont

174 sinus dysrhythmia, severe sinus bradycardia, sinus pauses and chronotropic incompetence.

175 Preablation ECVS induced sinus pauses, asystole, and transient atrioventricular b

176 anatomy-anomalous left CA from the opposite sinus, presence of intramurality, abnormal ostium-and sy

177 and cardiac output (CO) while causing reflex sinus rate (heart rate [HR]) increase.

178 ange: 9 to 41) beats/min faster than daytime sinus rates.

179 hese vessels, running alongside dural venous sinuses, recapitulates the meningeal lymphatic system of

180 ted in reduced fungal entrapment in the peri-sinus region and increased spread into the brain followi

181 ells are positioned adjacent to dural venous sinuses: regions of slow blood flow with fenestrations t

182 eeks right ventricle and then 2 weeks normal sinus (resynchronization).

183 lanceolate endings at the level of the ring sinus revealed unique anatomical features that may promo

184 LGE-CMR and electroanatomic mapping (EAM) in sinus rhythm (2960 electroanatomic mapping points analyz

185 < 0.0001) and in all subgroups, particularly sinus rhythm (adjusted HR: 1.25 [95% CI: 1.21 to 1.28])

186 n episodes alternating with short periods of sinus rhythm (odds ratio, 0.18; 95% CI, 0.06-0.52; p = 0

187 superimposed on an AFL substrate (AF+AFLs); sinus rhythm (SR) with an AFL substrate (SR+AFLs; contro

188 +/- 10%), 1,646 presented at diagnosis with sinus rhythm (SR), 317 with paroxysmal AD, and 462 with

189 n characterized by activation slowing during sinus rhythm (SR).

190 brillation alternating with short periods of sinus rhythm and 33 (40%) had refractory ventricular tac

191 n episodes alternating with short periods of sinus rhythm and age less than 50 years were independent

192 ntify the functional substrate for VT during sinus rhythm and guide targeted ablation, obviating the

193 ity in conduction are already present during sinus rhythm and may explain the higher vulnerability to

194 The presence of sinus rhythm at 4 weeks occurred in 193 of 212 patients

195 The primary end point was the presence of sinus rhythm at 4 weeks.

196 early cardioversion in achieving a return to sinus rhythm at 4 weeks.

197 ophageal echocardiography (TEE) performed in sinus rhythm at 6 months to assess left atrial appendage

198 oxysmal or persistent atrial fibrillation in sinus rhythm at baseline were randomly assigned in a 1:1

199 llation episodes alternating with periods of sinus rhythm at the time of implantation had a better su

200 on commonly undergo immediate restoration of sinus rhythm by pharmacologic or electrical cardioversio

201 uration in atrial cardiomyocytes compared to sinus rhythm controls, similar to previous findings in h

202 rified atrial fibrillation before the normal sinus rhythm ECG tested by the model.

203 ncluded 180 922 patients with 649 931 normal sinus rhythm ECGs for analysis: 454 789 ECGs recorded fr

204 All 19 patients underwent 2 detailed sinus rhythm electroanatomic endocardial voltage maps (a

205 orithm applied to electrocardiography during sinus rhythm has recently been shown to detect concurren

206 DCCV restored sinus rhythm in all patients.

207 Catheter ablation is effective in restoring sinus rhythm in atrial fibrillation (AF), but its effect

208 e most frequently used agent for maintaining sinus rhythm in patients with AF, but it impairs the sin

209 ore effective than drug therapy in restoring sinus rhythm in patients with atrial fibrillation (AF),

210 ntricular tachycardia, sinus bradycardia and sinus rhythm including sinus irregularity rhythm.

211 However, whether immediate restoration of sinus rhythm is necessary is not known, since atrial fib

212 For a similar sinus rhythm maintenance at 12 months (90% versus 88%; P

213 the early-cardioversion group, conversion to sinus rhythm occurred spontaneously before the initiatio

214 am mapping was performed pre-ablation during sinus rhythm or LA pacing, and electrogram locations wer

215 ation of tachycardia with the restoration of sinus rhythm or suppression of the tachycardia to <100 b

216 have persistent forms of AF and had a lower sinus rhythm P-wave amplitude.

217 In sinus rhythm patients undergoing cardiac surgery, histop

218 An AI-enabled ECG acquired during normal sinus rhythm permits identification at point of care of

219 le with atrial fibrillation (AF), periods of sinus rhythm present an opportunity to detect prothrombo

220 r it can convert atrial fibrillation (AF) to sinus rhythm remains unclear.

221 Stable sinus rhythm restoration was immediate in 61.5% of patie

222 left ventricular ejection fraction <50% and sinus rhythm should receive beta-blocker therapy even wi

223 shortening to maintain values observed among sinus rhythm subjects.

224 ts with no AF (mean age, 54 years +/- 16) in sinus rhythm to establish control values and convert the

225 s significantly increased from patients with sinus rhythm to paroxysmal AF and persistent AF, respect

226 of atrial fibrillation present during normal sinus rhythm using standard 10-second, 12-lead ECGs.

227 ential durations in atrial cardiomyocytes to sinus rhythm values.

228 s during VT and substrate abnormality during sinus rhythm was also investigated.

229 Although the success rate of restoring sinus rhythm was high, tachyarrhythmias and bradyarrhyth

230 Acute conversion to sinus rhythm was observed in 2 patients after ablation o

231 Sinus rhythm was recorded on 12-lead electrocardiograms

232 Conversion of atrial arrhythmia to sinus rhythm was the primary efficacy end point.

233 disease-related AF) and from 39 patients in sinus rhythm with mitral valve regurgitation (group 2; 3

234 e delayed-cardioversion group, conversion to sinus rhythm within 48 hours occurred spontaneously in 1

235 fibrillation (defined as no plan to restore sinus rhythm) and dyspnea classified as New York Heart A

236 In 13,861 patients in sinus rhythm, beta-blockers reduced mortality versus pla

237 antiarrhythmic drug therapy for maintaining sinus rhythm, but its success varies depending on multip

238 o 6.2+/-0.5 Hz (P<0.01) before converting to sinus rhythm, decreased singularity point density from 0

239 us 3.7% (2.9%-4.5%) in patients with pAF and sinus rhythm, respectively.

240 Electrocardiography showed sinus rhythm, right bundle branch block, T-wave inversio

241 s or older with at least one digital, normal sinus rhythm, standard 10-second, 12-lead ECG acquired i

242 , and superior for successful restoration of sinus rhythm.

243 ) s(-)(1); P=ns) and failed to convert AF to sinus rhythm.

244 emonstrated ventricular preexcitation during sinus rhythm.

245 tilide (1 mumol/L) failed to convert PsAF to sinus rhythm.

246 oversion was superior for the restoration of sinus rhythm.

247 al analysis of ventricular activation during sinus rhythm.

248 with better prognosis, but only for those in sinus rhythm.

249 blation may be more effective in maintaining sinus rhythm.

250 ium, Bachmann bundle, and left atrium during sinus rhythm.

251 antiarrhythmic drug therapy for maintaining sinus rhythm.

252 oped pAF with the remaining 2289 maintaining sinus rhythm.

253 Artefact-free 60-second strips of normal sinus-rhythm ECGs were converted to binary strings using

254 llows identification of horses with PAF from sinus-rhythm ECGs with high accuracy.

255 by complexity analysis of apparently normal sinus-rhythm ECGs.

256 as connected with larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

257 RR 1.30; 95% CI 0.92-1.82), chronic perineal sinus (RR 1.08; 95% CI 0.53-2.20), and pelviperineal com

258 By leveraging coronary sinus samples and transcriptomic tools, we describe like

259 Subcapsular sinus (SCS) macrophages are strategically positioned at

260 LECs lining the subcapsular sinus (SCS) of LNs abundantly expressed neutrophil chemo

261 nical 3D-sieve barrier of the LN subcapsular sinus (SCS).

262 olvement, imaging of the orbit and paranasal sinuses should be considered early.

263 activity-tracing studies in the mouse aortic sinus showed that the Ahr pathway is active in modulated

264 a at 3-5 years after a functional endoscopic sinus surgery (FESS) and correlate these data to symptom

265 Emergency endoscopic sinus surgery and antibiotic treatment resulted in compl

266 Sinus surgery improves patient-reported outcomes, but no

267 Male sex, migraine headache, and prior sinus surgery were associated with higher odds of CRS(S+

268 had a history of FESS (functional endoscopic sinus surgery) and reported lower symptom severity compa

269 ing systemic corticosteroid use and repeated sinus surgery.

270 h an antibiotic was prescribed for worsening sinus symptoms, and infrequent AECRS was defined as 0 to

271 ties in its structure or function cause sick sinus syndrome, the most common reason for electronic pa

272 on release from arterioles into the red pulp sinuses, T cells latched onto perivascular stromal cells

273 Yet, in inappropriate sinus tachycardia (IST), postural tachycardia syndrome (

274 Cardiac arrhythmias (i.e. inappropriate sinus tachycardia and bradycardia, asystole, and atriove

275 over the last decade, severe and refractory sinus tachycardia, atrial fibrillation, and ventricular

276 man presented to the cardiology service with sinus tachycardia.

277 Microsphere-based-delivery to the Paranasal Sinuses (TEMPS) is developed with the corticosteroid mom

278 the small natural openings leading from the sinuses that can be further obstructed by presence of in

279 of coinciding cerebral infarction and venous sinus thrombosis unveiling the diagnosis of celiac disea

280 usion and left transverse and sigmoid venous sinus thrombosis, along with left jugular vein thrombosi

281 tis, ischemic and hemorrhagic stroke, venous sinus thrombosis, and endothelialitis.

282 cytosis through the floor of the subcapsular sinus thus represents what we believe to be a new physio

283 elet-leukocyte aggregates are present in the sinus tissue and blood of patients with AERD compared wi

284 Sinus tissue was obtained from subjects with AERD, chron

285 n, especially in the nasal cavity, paranasal sinuses, tonsillar fossa, and oral cavity.

286 y disorder characterized by painful nodules, sinus tracts, and scars occurring predominantly in inter

287 and activated Notch1 receptor expression in sinus venosus (SV) endocardium.

288 ificantly increased the distance between the sinus venosus and bulbus arteriosis (SV-BA) at 72 h post

289 cardium is activated in multiple cases where sinus venosus angiogenesis is stunted.

290 The superior sinus venosus atrial septal defect (SVASD) is characteri

291 rogation identified 60% of the patients with sinus venosus defects to be eligible for catheter closur

292 rlapping covered stents were used to exclude sinus venosus defects.

293 terest in nonsurgical correction of superior sinus venosus defects.

294 SOXF/RBPJ and BMP-SMAD pathways are seen in sinus venosus-derived arterial and venous coronaries, re

295 after grafting into Lanyu Taiwanese mini-pig sinuses via split-mouth design.

296 igher than the results in vitro in maxillary sinus volumes with a ratio of 1.05 +/- 0.01 (mean +/- SD

297 Routine multi-slice CT of the paranasal sinuses was performed to look for mucosal disease of the

298 5 implants inserted into augmented maxillary sinuses with a follow-up ranging from 1 to 18 years were

299 th a synthetic biphasic calcium phosphate in sinuses with minimal bone height, the alloplastic and xe

300 thmoid air cells, sphenoid sinus and frontal sinuses yielded the highest prediction accuracy, with Mu