ライフサイエンスコーパス: sinus node dysfunction

1 , atrial standstill, conduction disease, and sinus node dysfunction.

2 r (DDDR) versus ventricular (VVIR) pacing in sinus node dysfunction.

3 of dual-chamber versus ventricular pacing in sinus node dysfunction.

4 ich reduced Na+ channel function might cause sinus node dysfunction.

5 med SAN at birth, the mutant mice manifested sinus node dysfunction.

6 vent diseases such as atrial fibrillation or sinus node dysfunction.

7 closely resemble those observed in clinical sinus node dysfunction.

8 d risk of death during pacemaker therapy for sinus node dysfunction.

9 with subsequent stroke in patients paced for sinus node dysfunction.

10 and exhibited ventricular preexcitation and sinus node dysfunction.

11 nts (92%) to determine the late incidence of sinus node dysfunction.

12 4 years after the Fontan operation, 44% had sinus node dysfunction.

13 high-grade atrioventricular block and 1 for sinus node dysfunction.

14 principally in the subgroup of patients with sinus-node dysfunction.

15 odds ratio [OR], 1.90 [95% CI, 1.36-2.67]), sinus node dysfunction (1 point; OR, 1.84 [95% CI, 1.04-

16 At follow-up, 7 (6%) of 108 patients had sinus node dysfunction, a permanent pacemaker, or both,

17 iminished P-wave amplitude characteristic of sinus node dysfunction, an AF risk factor in human patie

18 of pacemaker activity, reminiscent of human sinus node dysfunction and "tachy-brady" syndrome.

19 All 11 patients presented with sinus node dysfunction and 10 had atrial arrhythmias.

20 parasympathetic influence has been linked to sinus node dysfunction and arrhythmia.

21 Respective subdomain sizes and severity of sinus node dysfunction and atrial arrhythmia susceptibil

22 c variation-driven ectopic PITX2 expression, sinus node dysfunction and atrial arrhythmogenesis, illu

23 or gene (PITX2), identified in patients with sinus node dysfunction and atrial fibrillation and model

24 than the broad categories and indications of sinus node dysfunction and atrioventricular block.

25 cribes an arrhythmia phenotype attributed to sinus node dysfunction and diagnosed by electrocardiogra

26 Following that operation she developed sinus node dysfunction and had a permanent epicardial du

27 We sought to eliminate sinus node dysfunction and postoperative bradyarrhythmia

28 t loss of an RE at the HCN4 locus results in sinus node dysfunction and reduced gene expression.

29 rmal SAN function and the pathophysiology of sinus node dysfunction and suggest new potential targets

30 more likely to occur in patients with early sinus node dysfunction and those with longer follow-up.

31 g the 300 patients enrolled, 190 (63.3%) had sinus-node dysfunction and 100 (33.3%) had atrioventricu

32 -venetoclax (cardiac failure, pneumonia, and sinus node dysfunction) and in one patient receiving chl

33 econd-degree atrioventricular blocks, 4 with sinus node dysfunction, and 5 sudden cardiac deaths.

34 s more major adverse events, major bleeding, sinus node dysfunction, and pacemaker implantation.

35 gous for the RE deletion showed bradycardia, sinus node dysfunction, and selective loss of Hcn4 expre

36 Bradycardia and sinus node dysfunction are common causes of early postop

37 pacing on subsequent stroke in patients with sinus node dysfunction are not known.

38 cardiac conduction disorder associated with sinus node dysfunction, arrhythmia, and right and occasi

39 conduction, and human SCN5A mutations cause sinus node dysfunction, atrial fibrillation, conductiona

40 Indications for HBP were sinus node dysfunction, atrioventricular conduction dise

41 ies with a phenotypic spectrum consisting of sinus node dysfunction, AV conduction defects, and hyper

42 hamber pacing were observed in patients with sinus-node dysfunction, but not in those with atrioventr

43 Patients with sinus-node dysfunction, but not those with atrioventricu

44 ant and persistent atrioventricular block or sinus node dysfunction can occur and indicate a need for

45 nd at 6 months, decreased R wave amplitudes, sinus node dysfunction, cardiac hypertrophy, interstitia

46 r groups, affected individuals mainly showed sinus node dysfunction, conduction defects, and atrial a

47 cing (DDDR) and ventricular pacing (VVIR) in sinus node dysfunction, demonstrated no difference in de

48 In sinus-node dysfunction, dual-chamber pacing does not imp

49 The most common bradyarrhythmia was sinus node dysfunction followed by high-grade atrioventr

50 iciency in mice may cause the stress-induced sinus node dysfunction found in many aged individuals an

51 Sinus node dysfunction has been previously reported to o

52 I (hazard ratio, 4.0; P=0.04), and previous sinus node dysfunction (hazard ratio, 8.0; 95% confidenc

53 ofosbuvir and daclatasvir, he had an extreme sinus node dysfunction (heart rate of 27beats/min).

54 To determine the early incidence of sinus node dysfunction, hospital records and perioperati

55 stroke in a population of patients paced for sinus node dysfunction in a large prospective clinical t

56 CPVT, such as the pathophysiological role of sinus node dysfunction in CPVT, and whether the arrhythm

57 Unlike sinus node dysfunction in nontransplanted patients, whic

58 o determine the early and late incidences of sinus node dysfunction in patients systematically and un

59 node function between the 2 stages, 23% had sinus node dysfunction in the early postoperative period

60 r its blood supply is a significant cause of sinus node dysfunction in the orthotopic heart transplan

61 Because sinus node dysfunction in the transplanted heart does no

62 patients, which typically worsens with time, sinus node dysfunction in the transplanted heart usually

63 uld be a therapeutic target for pathological sinus node dysfunction in veteran athletes.

64 Perioperative sinus node dysfunction is common after both the hemi-Fon

65 nus node function between the 2 stages, late sinus node dysfunction is common and more likely to occu

66 Sinus node dysfunction is increasingly common with longe

67 Sinus node dysfunction is the most common indication for

68 tained atrial tachyarrhythmia, implying that sinus node dysfunction is unlikely to be the dominant me

69 ficant clinical manifestation of progressive sinus node dysfunction, is the most frequent indication

70 ity, spontaneous type I ECG, and presence of sinus node dysfunction might be considered as risk facto

71 Sinus node dysfunction occurred in 12 patients in the GP

72 Sinus node dysfunction occurs commonly after orthotopic

73 Bradycardia, due to sinus node dysfunction or atrioventricular (AV) block, w

74 nical trials in patients with pacemakers for sinus node dysfunction or atrioventricular block (AVB) a

75 eatment for patients with bradycardia due to sinus node dysfunction or atrioventricular block.

76 and 7 of these patients also had evidence of sinus node dysfunction (P < .005).

77 ulmonary connection may increase the risk of sinus node dysfunction, previous studies have not report

78 Pacing-induced chronic AF induces sinus node dysfunction, prolongs intra-atrial conduction

79 of atrial fibrillation and in patients with sinus node dysfunction, reduces heart failure symptoms w

80 Ten patients with sinus node dysfunction scheduled for dual-chamber pacema

81 Although sinus node dysfunction (SND) and atrial arrhythmias freq

82 ration family (n=25) with autosomal dominant sinus node dysfunction (SND) and atrioventricular block

83 ut genetic overlap has not been reported for sinus node dysfunction (SND) and noncompaction cardiomyo

84 Inherited forms of sinus node dysfunction (SND) clinically include bradycar

85 ildren experienced more frequent episodes of sinus node dysfunction (SND) compared with older subject

86 Sinus node dysfunction (SND) is a major clinically relev

87 Sinus node dysfunction (SND) is a major public health pr

88 Sinus node dysfunction (SND) is often associated with at

89 nt burden testing in 460,000 individuals for sinus node dysfunction (SND), distal conduction disease

90 ) compared with ventricular (VVIR) pacing in sinus node dysfunction (SND).

91 ndomized trial of DDDR versus VVIR pacing in sinus node dysfunction (SND).

92 we studied a family with DCM associated with sinus node dysfunction, supraventricular tachyarrhythmia

93 cardiac arrhythmia syndrome associated with sinus node dysfunction that is distinct from long QT syn

94 ing are alternative treatment approaches for sinus-node dysfunction that causes clinically significan

95 Even after appropriate pacing for sinus node dysfunction, the sinus node may recover and p

96 domly assigned a total of 2010 patients with sinus-node dysfunction to dual-chamber pacing (1014 pati

97 tment of pacemaker syndrome in patients with sinus node dysfunction treated with ventricular-based (V

98 ain containing 1 (Popdc1) or Popdc2 leads to sinus node dysfunction under stressed conditions in aged

99 , whereas observed survival of patients with sinus node dysfunction was not significantly different f

100 Sinus node dysfunction was present in 7% of the patients

101 A total of 2,010 patients with sinus node dysfunction were randomized to ventricular or

102 ECG analysis revealed severe sinus node dysfunction when freely roaming mutant animal

103 stro-esophageal reflux, retinal disease, and sinus-node dysfunction, whereas related heterozygotes ha

104 art failure hospitalization in patients with sinus node dysfunction who require pacemaker therapy is