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1 genes encoding pro-IL-1beta and pro-IL-18 in sinusoidal endothelial cells.
2  vascular ectonucleotidase CD39 increased on sinusoidal endothelial cells.
3 me that both C-type lectins are expressed on sinusoidal endothelial cells.
4 othelial cell populations, including hepatic sinusoidal endothelial cells.
5 take of 125I-HA at 37 degrees C by rat liver sinusoidal endothelial cells.
6 oteinase-9 and matrix metalloproteinase-2 by sinusoidal endothelial cells.
7  largely restricted to lymph nodes and liver sinusoidal endothelial cells.
8 cription factor 3 (STAT3) in hepatocytes and sinusoidal endothelial cells.
9 ow that in the liver, L-SIGN is expressed by sinusoidal endothelial cells.
10 ed in whole-liver extracts were localized to sinusoidal endothelial cells.
11 ) agonist, increased cAMP levels in cultured sinusoidal endothelial cells.
12 ars very early after injury and derives from sinusoidal endothelial cells.
13 dependent iNOS mRNA induction in Kupffer and sinusoidal endothelial cells.
14  only nonparenchymal cells, predominantly in sinusoidal endothelial cells.
15 e week starting with hepatocytes followed by sinusoidal endothelial cells.
16 of circulating leukocytes as well as hepatic sinusoidal endothelial cells.
17 lymphatic endothelial cells (LECs) and liver sinusoidal endothelial cells.
18  adipocytes, and comprised a minor subset of sinusoidal endothelial cells.
19 activity in hepatic stellate cells and liver sinusoidal endothelial cells.
20 actor-1 receptors, CXCR7 and CXCR4, in liver sinusoidal endothelial cells.
21 nteractions among rat HSCs, hepatocytes, and sinusoidal endothelial cells.
22 ing verified the expression of E-selectin on sinusoidal endothelial cells 4 hours after Gal/ET inject
23 ), hepatic B cells (81.5 +/- 9.3%) and liver sinusoidal endothelial cells (64.6 +/- 13.7%) interacted
24 n increase in factor VIII (produced by liver sinusoidal endothelial cells), a decrease of the natural
25 milarly, selective CXCR7 activation in liver sinusoidal endothelial cells abrogated fibrogenesis.
26                                        Liver sinusoidal endothelial cell activation and stellate cell
27 tokine release, Kupffer cell activation, and sinusoidal endothelial cell activation.
28 er acute injury, CXCR7 upregulation in liver sinusoidal endothelial cells acts with CXCR4 to induce t
29 Additionally, GIT1 expression was reduced in sinusoidal endothelial cells after liver injury, consist
30 ival, but paradoxically decreased killing of sinusoidal endothelial cells after storage and reperfusi
31 andin E(2), another agent that preconditions sinusoidal endothelial cells against storage/reperfusion
32 d from LPS-stimulated Kupffer cells protects sinusoidal endothelial cells against storage/reperfusion
33                                     In liver sinusoidal endothelial cells, alcohol-induced TGF-beta s
34 ncreased by 60% +/- 27% over basal values in sinusoidal endothelial cells and 98% +/- 40% in stellate
35 ude the accumulation of gammaDPGA in hepatic sinusoidal endothelial cells and a gammaDPGA clearance r
36 rage involves reperfusion-induced killing of sinusoidal endothelial cells and activation of Kupffer c
37 es exposed to acetaminophen, but not hepatic sinusoidal endothelial cells and biliary epithelial cell
38 ctor-b (TGF-beta) family ligands produced by sinusoidal endothelial cells and endogenous LXR ligands
39 eceptor 3 (TLR3)-activated Kupffer and liver sinusoidal endothelial cells and further controlled the
40 of PltC subunit to sulfated glycans on liver sinusoidal endothelial cells and gallbladder epithelial
41 f Disse and maintain close interactions with sinusoidal endothelial cells and hepatic epithelial cell
42                          Interaction between sinusoidal endothelial cells and hepatocytes is a prereq
43 ersion atomization, to direct genes to liver sinusoidal endothelial cells and hepatocytes, respective
44  with up-regulation of adhesion molecules on sinusoidal endothelial cells and hepatocytes.
45  found that donor hematopoietic cells act on sinusoidal endothelial cells and induce host blood vesse
46     This function was observed in both liver sinusoidal endothelial cells and Kupffer cells even at v
47 ne alkaloid, with reported toxicity in liver sinusoidal endothelial cells and Kupffer cells.
48 marily by ICAM-1 constitutively expressed on sinusoidal endothelial cells and Kupffer cells.
49 ly abundant endocytic receptors expressed by sinusoidal endothelial cells and parenchymal cells in th
50 tive B7-H1 expression on dendritic cells and sinusoidal endothelial cells and promptly induced B7-H1
51 inusoidal leukocyte aggregates and activated sinusoidal endothelial cells, and (3) sustained producti
52 erived APCs including Kupffer cells or liver sinusoidal endothelial cells, and apparently can occur e
53 nts of isolated primary hepatocytes, hepatic sinusoidal endothelial cells, and biliary epithelial cel
54 taline caused depolymerization of F-actin in sinusoidal endothelial cells, and blocking of F-actin de
55 c macrophages, liver-associated lymphocytes, sinusoidal endothelial cells, and hepatic stellate cells
56  inputs, including signals from hepatocytes, sinusoidal endothelial cells, and hepatic stellate cells
57  liver cell types such as progenitors, liver sinusoidal endothelial cells, and hepatic stellate cells
58 ntributions of liver progenitor cells, liver sinusoidal endothelial cells, and hepatic stellate cells
59 stribution in cultured human cholangiocytes, sinusoidal endothelial cells, and hepatocytes revealed t
60 ng expression of eotaxins in hepatocytes and sinusoidal endothelial cells, and induces IL-5 expressio
61 tivated hepatic stellate cells (HSCs), liver sinusoidal endothelial cells, and Kupffer cells are resp
62 r, CD302 was expressed by hepatocytes, liver sinusoidal endothelial cells, and Kupffer cells.
63                         Kupffer cells, liver sinusoidal endothelial cells, and leukocytes express PD-
64 epatic stellate cells, myofibroblasts, liver sinusoidal endothelial cells, and macrophages to illustr
65 atocytes, recruited macrophage, capillarized sinusoidal endothelial cells, and permeable intestinal e
66 gets of VEGF in liver may not be confined to sinusoidal endothelial cells, and that VEGF responses re
67                                        Liver sinusoidal endothelial cells are a major endogenous sour
68 nisms by which it regulates eNOS activity in sinusoidal endothelial cells are not well understood.
69 tellate cells, and Kupffer cells showed that sinusoidal endothelial cells are the major source of bot
70                                      Hepatic sinusoidal endothelial cells are unique among endothelia
71               AAV5 and AAV8 transduced liver sinusoidal endothelial cells as efficiently as hepatocyt
72                 Apoptosis of hepatocytes and sinusoidal endothelial cells, assessed by in situ TUNEL
73 ) maturation and localization to bone marrow sinusoidal endothelial cells (BMECs), stimulating thromb
74 ebrand factor, which is synthesized by liver sinusoidal endothelial cells but not hepatocytes, were u
75 ve (Thy1(+)) oval cells, stellate cells, and sinusoidal endothelial cells but not to hepatocytes.
76 ependent manner and cross-presented by liver sinusoidal endothelial cells, but not dendritic cells, t
77  expression was present in Kupffer cells and sinusoidal endothelial cells, but not in hepatocytes.
78 as detected in whole liver, hepatocytes, and sinusoidal endothelial cells, but not in Kupffer cells.
79               Monocrotaline decreased GSH in sinusoidal endothelial cells, but not in liver homogenat
80                           Synthesis of NO in sinusoidal endothelial cells by endothelial nitric-oxide
81 binding was evaluated on primary human liver sinusoidal endothelial cells by flow cytometry and confi
82 e potent vaccines engaged both KCs and liver sinusoidal endothelial cells by inducing higher titers o
83  prevented the protective preconditioning of sinusoidal endothelial cells by LPS, whereas pretreatmen
84 f the hepatocytes is separated from adjacent sinusoidal endothelial cells by the space of Disse, wher
85 egulation of fibronectin splicing in primary sinusoidal endothelial cells by transfecting a minigene
86 cation of C4d staining in proximity to liver sinusoidal endothelial cell capillarization and stellate
87 tiguous expression of junctional proteins at sinusoidal endothelial cell-cell contacts, switching cap
88 pecific interaction whereby WNT2 secreted by sinusoidal endothelial cells controls cholesterol uptake
89 tion, constitutive FGFR1 signalling in liver sinusoidal endothelial cells counterbalanced CXCR7-depen
90               Inducible deletion of Cxcr7 in sinusoidal endothelial cells (Cxcr7(iDeltaEC/iDeltaEC))
91 ivation of KCs, inhibited APAP-induced liver sinusoidal endothelial cell damage and improved hepatic
92 lar niche predominantly represented by liver sinusoidal endothelial cells deploys paracrine trophogen
93        In this study, we observed that liver sinusoidal endothelial cells derived from ethanol-fed ra
94                                        Liver sinusoidal endothelial cell-derived bone morphogenetic p
95                     Decreased HGF release by sinusoidal endothelial cells, despite high levels of VEG
96 ransferase, aspartate aminotransferase), and sinusoidal endothelial cell dysfunction (hyaluronic acid
97  significantly attenuated APAP-induced liver sinusoidal endothelial cell dysfunction and ameliorated
98 istology revealed that monocytes reside near sinusoidal endothelial cells (ECs) and leptin receptor (
99 icular, molecular and physical adaptation of sinusoidal endothelial cells (ECs) promote HSPC BM occup
100 l cells, we demonstrate that ET-1 binding to sinusoidal endothelial cell ETB receptors led to increas
101 nisms underlying death of cultured rat liver sinusoidal endothelial cells exposed to chemical hypoxia
102                                  Bone marrow sinusoidal endothelial cells express high levels of Angp
103 ls, Kupffer cells, stellate cells, and liver sinusoidal endothelial cells express key molecules that
104 ning whether hepatocytes, Kupffer cells, and sinusoidal endothelial cells express mRNA and enzyme act
105                            Like human spleen sinusoidal endothelial cells, Flp-In 293 cell lines tran
106 activates eNOS, was substantially reduced in sinusoidal endothelial cells from injured livers.
107                                        Using sinusoidal endothelial cells from normal or injured live
108    Furthermore, GRK2 expression increased in sinusoidal endothelial cells from portal hypertensive ra
109 tion syndrome are initiated by dehiscence of sinusoidal endothelial cells from the space of Disse.
110 ic acid uptake are reliable markers of liver sinusoidal endothelial cell function and that normal or
111 ar group, suggesting that hepatocyte and not sinusoidal endothelial cell function is adversely affect
112 interleukin-1beta levels as well as impaired sinusoidal endothelial cell function were detected in ac
113 in LPS/RAN-cotreated rats, suggested altered sinusoidal endothelial cell function.
114                                 Depletion of sinusoidal endothelial cell glutathione (GSH) has been p
115 onine sulfoximine starting day - 2 decreased sinusoidal endothelial cell GSH and attenuated the prote
116  starting day - 1, prevented the decrease in sinusoidal endothelial cell GSH and protected against hi
117  toxicity, at least partially by maintaining sinusoidal endothelial cell GSH levels.
118 nclusion, monocrotaline selectively depletes sinusoidal endothelial cell GSH.
119  cell adhesion and rolling along bone marrow sinusoidal endothelial cells has been defined, and mecha
120 2a-Vegf axis as a prime node in coordinating sinusoidal endothelial cell-hepatocyte crosstalk during
121 ed sulphur colloid to assess the function of sinusoidal endothelial cells, hepatocytes, and Kupffer c
122                          In vitro studies of sinusoidal endothelial cells, hepatocytes, stellate cell
123 hylnitrosamine (DMN), in vitro human hepatic sinusoidal endothelial cells (HHSEC) and Human Umbilical
124  ICAM-1 and VCAM-1 expression in human liver sinusoidal endothelial cells (HLSECs) and the adhesion o
125 eptor class F, member 1 (SCARF-1) on hepatic sinusoidal endothelial cells (HSEC).
126 cyte differentiation and the role of hepatic sinusoidal endothelial cells (HSECs) in this process are
127 ed extracellular matrices, and human hepatic sinusoidal endothelial cells (HSECs) were quantified in
128 id is a unique vascular bed lined by hepatic sinusoidal endothelial cells (HSECs), a functionally and
129                                              Sinusoidal endothelial cells in bone marrow have been re
130 study human B-cell migration through hepatic sinusoidal endothelial cells in flow-based adhesion assa
131 IGNR, a human DC-SIGN homologue expressed on sinusoidal endothelial cells in liver and lymph node, al
132 VE-1 is also present in normal hepatic blood sinusoidal endothelial cells in mice and humans.
133 ue inhibitor of metalloproteases confined to sinusoidal endothelial cells in response to myeloid cell
134 iserum showed that DC-SIGNR was expressed on sinusoidal endothelial cells in the liver and on endothe
135 ves lymphocyte transmigration across hepatic sinusoidal endothelial cells in vitro.
136           Because graft failure is linked to sinusoidal endothelial cell injury after storage/reperfu
137 sure to LPS on graft survival in relation to sinusoidal endothelial cell injury after storage/reperfu
138  containing trypan blue for determination of sinusoidal endothelial cell injury by counting trypan bl
139 contrast, LPS-induced hepatocyte and hepatic sinusoidal endothelial cell iNOS expression was signific
140 er-selective MMP-9 inhibition restored liver sinusoidal endothelial cell integrity, enhanced liver re
141 pecific Notch1 decoys increased sprouting of sinusoidal endothelial cells into micrometastases, there
142 sly showed that apoptosis of hepatocytes and sinusoidal endothelial cells is a critical mechanism of
143     The study suggests that the apoptosis of sinusoidal endothelial cells is a pivotal mechanism of p
144 erlying impaired activity of eNOS in injured sinusoidal endothelial cells is defective phosphorylatio
145 thelial cell nitric oxide synthase (eNOS) in sinusoidal endothelial cells is reduced in the injured l
146 o-regenerative angiocrine signals from liver sinusoidal endothelial cells is subverted to promote fib
147 ajor FcgammaRIIB-expressing cell type, liver sinusoidal endothelial cells, is required for both proce
148 es expression of eotaxins in hepatocytes and sinusoidal endothelial cells isolated from wild-type mic
149                              Because hepatic sinusoidal endothelial cells kill tumor cells in vitro b
150   A brief period of liver ischemia decreases sinusoidal endothelial cell killing after cold liver sto
151           Ischemic preconditioning decreased sinusoidal endothelial cell killing after storage/reperf
152 or agonist, CGS-21680, and DB-cAMP decreased sinusoidal endothelial cell killing to the same extent a
153 alpha-gal A in several cell types, including sinusoidal endothelial cells, Kupffer cells, and hepatoc
154                                    Rat liver sinusoidal endothelial cells (LECs) express two hyaluron
155  endocytic hyaluronan (HA) receptor of liver sinusoidal endothelial cells (LECs) is responsible for t
156 and PL liver localization as expected; liver sinusoidal endothelial cell (LSEC) depletion reduced PL
157               Capillarization, lack of liver sinusoidal endothelial cell (LSEC) fenestration, and for
158                                        Liver sinusoidal endothelial cell (LSEC)-selective Cre deleter
159 endothelial cell (EC) populations into liver sinusoidal endothelial cells (LSEC) and continuous endot
160 lial cell targeting, we isolated mouse liver sinusoidal endothelial cells (LSEC) and examined cell bi
161 ed LPS in liver became associated with liver sinusoidal endothelial cells (LSEC) and only approximate
162 or (FcgammaRIIb, RIIb) is expressed on liver sinusoidal endothelial cells (LSEC) and that the liver i
163                                        Liver sinusoidal endothelial cells (LSEC) are identified as an
164        In this study, we observed that liver sinusoidal endothelial cells (LSEC) derived from ethanol
165                                        Liver sinusoidal endothelial cells (LSEC) have been reported t
166  and fenestra-forming role of PLVAP in liver sinusoidal endothelial cells (LSEC) have remained contro
167 (HC), hepatic stellate cells (HSC) and liver sinusoidal endothelial cells (LSEC) in accelerated liver
168 that the coculture of hepatocytes with liver sinusoidal endothelial cells (LSEC) significantly increa
169           Priming of CD4(+) T cells by liver sinusoidal endothelial cells (LSEC) supported migration
170 he liver-resident macrophages) and the liver sinusoidal endothelial cells (LSEC) which line the sinus
171  multiple cell types, including hepatocytes, sinusoidal endothelial cells (LSEC), Kupffer cells, and
172 g z-axis resolution, we identified the liver sinusoidal endothelial cells (LSEC), marked by FcgammaRI
173                      Interestingly, in liver sinusoidal endothelial cells (LSEC), the cells that form
174 scopy (QPM) to obtain 3D morphology of liver sinusoidal endothelial cells (LSEC).
175                                        Liver sinusoidal endothelial cells (LSECs) act as a filter bet
176                       The fenestrae of liver sinusoidal endothelial cells (LSECs) allow passive trans
177  lining the hepatic sinusoids, such as liver sinusoidal endothelial cells (LSECs) and hepatic stellat
178 mRNA in purified populations of murine liver sinusoidal endothelial cells (LSECs) and hepatocytes, bu
179 We evaluated the kinetics by which rat liver sinusoidal endothelial cells (LSECs) are repopulated in
180 s in hepatocyte growth factor (HGF) in liver sinusoidal endothelial cells (LSECs) are thought to driv
181                                        Liver sinusoidal endothelial cells (LSECs) are unique organ-re
182                                        Liver sinusoidal endothelial cells (LSECs) are uniquely differ
183 Proteomic and mRNA analyses of primary liver sinusoidal endothelial cells (LSECs) both revealed that
184 he association of human platelets with liver sinusoidal endothelial cells (LSECs) by immunohistochemi
185               Here we demonstrate that liver sinusoidal endothelial cells (LSECs) constitute a unique
186                                        Liver sinusoidal endothelial cells (LSECs) defenestrate and ca
187  antimetastatic niche characterized by liver sinusoidal endothelial cells (LSECs) defenestration extr
188                                        Liver sinusoidal endothelial cells (LSECs) differ, both struct
189                                        Liver sinusoidal endothelial cells (LSECs) have long been note
190                       To explore the role of sinusoidal endothelial cells (LSECs) in the adult liver,
191 isms by which forces are transduced by liver sinusoidal endothelial cells (LSECs) into pressure and m
192                                        Liver sinusoidal endothelial cells (LSECs) make up a large pro
193                                 Normal liver sinusoidal endothelial cells (LSECs) promote quiescence
194 ifference, the roles of Kupffer cells, liver sinusoidal endothelial cells (LSECs), hepatocytes, scave
195 n of the two receptors was detected in liver sinusoidal endothelial cells (LSECs), monocytes, and Kup
196 cterized by loss of differentiation of liver sinusoidal endothelial cells (LSECs), precedes the onset
197 iators of hepatic immune tolerance are liver sinusoidal endothelial cells (LSECs).
198 ver, but nonspecific uptake of ADCs by liver sinusoidal endothelial cells (LSECs).
199 ls, hepatic stellate cells (HSCs), and liver sinusoidal endothelial cells (LSECs).
200 bone morphogenetic protein 6 (BMP6) in liver sinusoidal endothelial cells (LSECs).
201 s (BM SPCs) repopulate the sinusoid as liver sinusoidal endothelial cells (LSECs).
202 igated the role of Notch1 signaling in liver sinusoidal endothelial cells (LSECs).
203 ent of bone marrow (BM) progenitors of liver sinusoidal endothelial cells (LSECs, also called sinusoi
204      Morphological and functional changes to sinusoidal endothelial cells mediated by soluble factors
205  Surprisingly, Kupffer cells, but also liver sinusoidal endothelial cells, mounted responses to rAAV,
206  of steatotic liver isografts via preventing sinusoidal endothelial cell necrapoptosis and consequent
207 on after transplantation induces significant sinusoidal endothelial cell necrapoptosis in steatotic Z
208 cal abnormalities have been described in the sinusoidal endothelial cells of cirrhotic livers, but th
209                                              Sinusoidal endothelial cells of human liver, lymph node,
210 E, 315- and 190-kDa, are highly expressed in sinusoidal endothelial cells of liver, lymph node, and s
211 RE isoforms (190 and 315 kDa) are present in sinusoidal endothelial cells of liver, spleen, and lymph
212                 We conclude that HARE in the sinusoidal endothelial cells of lymph nodes and liver li
213 s membrane proteins, are highly expressed in sinusoidal endothelial cells of lymph nodes, liver, and
214  found as two isoforms (315- and 190-kDa) in sinusoidal endothelial cells of the liver, lymph node, a
215 5- and approximately 300-kDa HARE species in sinusoidal endothelial cells of the liver, spleen, and l
216 plenic macrophages and the Kupffer cells and sinusoidal endothelial cells of the liver.
217 was observed, without evident effects on the sinusoidal endothelial cell or on the hepatocyte.
218 loid cell recruitment more than either liver sinusoidal endothelial cells or Kupffer cells.
219 that targeting a heterologous NOS isoform to sinusoidal endothelial cells or other perisinusoidal cel
220  quiescent HSCs, hepatocytes, Kupffer cells, sinusoidal endothelial cells, or biliary cells showed mi
221  porcine aortic, femoral arterial, and liver sinusoidal endothelial cells (PAEC/PFAEC/PLSEC).
222 e liver injury, where it has been shown that sinusoidal endothelial cells produce EIIIA-fibronectin.
223                       HSCs, hepatocytes, and sinusoidal endothelial cells produced and secreted fibro
224 fter liver injury, bone marrow-derived liver sinusoidal endothelial cell progenitor cells (BM SPCs) r
225                         Recruitment of liver sinusoidal endothelial cell progenitor cells (sprocs) fr
226  was presented that bone marrow (BM)-derived Sinusoidal endothelial cell PROgenitor Cells (sprocs) pl
227 soidal endothelial cells (LSECs, also called sinusoidal endothelial cell progenitor cells [sprocs]) w
228 s well as the kinetics of hepatocellular and sinusoidal endothelial cell proliferation, were assessed
229 e to the coordination between hepatocyte and sinusoidal endothelial cell proliferation.
230 this study, we investigated the mechanism of sinusoidal endothelial cell protection after ischemic pr
231 r pathway coupled to increased cAMP mediates sinusoidal endothelial cell protection by ischemic preco
232 Sleeping Beauty transposon targeted to liver sinusoidal endothelial cells provided long-term expressi
233 rses of the recovery suggest that successful sinusoidal endothelial cell recovery may depend upon pri
234                         To determine whether sinusoidal endothelial cells released MMPs when placed i
235  stellate cells and CD34 expression by liver sinusoidal endothelial cells remained stable, consistent
236 re expressed in the liver by parenchymal and sinusoidal endothelial cells, respectively.
237 cts of the vascular ectonucleotidase CD39 on sinusoidal endothelial cell responses following partial
238 roduction of cytokines and free radicals and sinusoidal endothelial cell (SEC) activation, may contri
239                                              Sinusoidal endothelial cell (SEC) apoptosis is a central
240 the liver followed by reperfusion results in sinusoidal endothelial cell (SEC) apoptosis.
241 gnificant vascular remodeling with increased sinusoidal endothelial cell (SEC) capillarization, vascu
242                                              Sinusoidal endothelial cell (SEC) function and permeabil
243 milar effects of GdCl3 on one of the hepatic sinusoidal endothelial cell (SEC) functions, i.e., hyalu
244         The detrimental role of platelets in sinusoidal endothelial cell (SEC) injury during liver tr
245 ertension, a disorder characterized by liver sinusoidal endothelial cell (SEC) injury with resultant
246 donor prodrug, restored NO levels, preserved sinusoidal endothelial cell (SEC) integrity and sinusoid
247                                              Sinusoidal endothelial cell (SEC) porosities were compar
248 patocyte division has mostly subsided, while sinusoidal endothelial cell (SEC) proliferation is initi
249                                              Sinusoidal endothelial cells (SEC) are a target of CI/WR
250 gested that more than 50% of hepatocytes and sinusoidal endothelial cells (SEC) are undergoing apopto
251 bazine showed selective in vitro toxicity to sinusoidal endothelial cells (SEC) compared with hepatoc
252 O is examined in further detail by isolating sinusoidal endothelial cells (SEC) from the rat liver.
253 ity on the cellular events that occur in rat sinusoidal endothelial cells (SEC) in the cold.
254                 Platelet adhesion to hepatic sinusoidal endothelial cells (SEC) is a major mechanism
255       We hypothesize that capillarization of sinusoidal endothelial cells (SEC) is permissive for hep
256 hat cause HVOD initially causing HVOD target sinusoidal endothelial cells (SEC) perhaps via profound
257                                              Sinusoidal endothelial cells (SEC) showed evidence of ap
258             HIR-induced apoptosis of hepatic sinusoidal endothelial cells (SEC) within 6 hours of HIR
259 ticles contain Hh ligands that alter hepatic sinusoidal endothelial cells (SEC).
260                      In vitro, human hepatic sinusoidal endothelial cells secreted IFN-inducible prot
261  on ET-1-mediated eNOS activation in hepatic sinusoidal endothelial cells (SECs) and to investigate t
262      To test the hypothesis that fenestrated sinusoidal endothelial cells (SECs) are crucial for this
263                                        Liver sinusoidal endothelial cells (SECs) are generally refrac
264                         Because CRCs contact sinusoidal endothelial cells (SECs) during implantation,
265                            Damage to hepatic sinusoidal endothelial cells (SECs) initiates sinusoidal
266                                     HSCs and sinusoidal endothelial cells (SECs) reside in close prox
267  eNOS interactor, regulates eNOS activity in sinusoidal endothelial cells (SECs) via its interaction
268 ured in isolated hepatocytes, Kupffer cells, sinusoidal endothelial cells (SECs), and hepatic stellat
269 ted in marked pathologic remodeling in liver sinusoidal endothelial cells (SECs), including SEC defen
270 nd activated stellate and Kupffer cells, and sinusoidal endothelial cells (SECs).
271  matrix metalloproteinases (MMPs) by hepatic sinusoidal endothelial cells (SECs).
272 NOS), this isoform has not been described in sinusoidal endothelial cells (SECs).
273          These experiments showed that liver sinusoidal endothelial cells selectively suppress the ex
274       Together, these data reveal that liver sinusoidal endothelial cells sense the microbiome, activ
275 s of nonhematopoietic liver cells, including sinusoidal endothelial cells, stellate cells located in
276  the neuronal NOS gene (nNOS) targeted liver sinusoidal endothelial cells, stellate cells, and hepato
277 that divergent angiocrine signals from liver sinusoidal endothelial cells stimulate regeneration afte
278 ression shifted angiocrine response of liver sinusoidal endothelial cells, stimulating proliferation
279                 Supernatants of isolated rat sinusoidal endothelial cells stored in the cold containe
280 umulated in hepatic stellate cells and liver sinusoidal endothelial cells, suggesting that sPACE NPs
281 eir targeted antibodies to Kupffer and liver sinusoidal endothelial cells that are known to have tole
282 veral microenvironmental regulators of liver sinusoidal endothelial cells that prolong their phenotyp
283                                        Liver sinusoidal endothelial cells, the origin of liver tumor
284 ished close and extensive contact with liver sinusoidal endothelial cells, thereby enhancing adenylyl
285 fter BM suppression supports the assembly of sinusoidal endothelial cells, thereby promoting reconsti
286 id not affect the susceptibility of cultured sinusoidal endothelial cells to Jo2-induced apoptosis.
287 as pretreatment with dimethyl PGE2 protected sinusoidal endothelial cells to the same extent as LPS.
288 timulates cytokine production in neighboring sinusoidal endothelial cells via Tlr9 and the Nalp3 infl
289 um could be replaced with transplanted liver sinusoidal endothelial cells, we developed an animal mod
290 Unlike capsular polysaccharides, the hepatic sinusoidal endothelial cells were also sites for gammaDP
291 eukin-1beta on GMP-140 expression in primary sinusoidal endothelial cells were analyzed.
292          In vivo function of hepatocytes and sinusoidal endothelial cells were evaluated by indocyani
293 al changes that permit the dehiscence of the sinusoidal endothelial cells were investigated.
294                   In spleen, macrophages and sinusoidal endothelial cells were positive, whereas in l
295 lling induced by commensal bacteria in liver sinusoidal endothelial cells, which in turn regulated th
296 taline causes depolymerization of F-actin in sinusoidal endothelial cells, which leads to increased e
297  that TIPS pseudointima are lined by hepatic sinusoidal endothelial cells, which stimulate pseudointi
298 y affected viability of hepatic stellate and sinusoidal endothelial cells, which was reversed by CPA
299 d preservation/reperfusion primarily affects sinusoidal endothelial cells, while hepatocytes are thou
300                  Coculture of hepatocytes or sinusoidal endothelial cells with HSCs increased the lev

 
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