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1 ynaptic contact between motor axons and each skeletal muscle fibre.
2 myocyte preparation or a skinned slow-twitch skeletal muscle fibre.
3 normal expression of laminin alpha2 on their skeletal muscle fibres.
4 ensitivities of contraction in slow and fast skeletal muscle fibres.
5 ontribute to mitochondrial ROS generation in skeletal muscle fibres.
6 lasma membrane of many cell types, including skeletal muscle fibres.
7  of DOX-induced myopathy in both cardiac and skeletal muscle fibres.
8 aintaining the resting membrane potential of skeletal muscle fibres.
9 t a physiological PO2 in intact single mouse skeletal muscle fibres.
10 ochrome c oxidase (COX)-deficient regions in skeletal muscle fibres.
11 molecular remodelling of the triads in adult skeletal muscle fibres.
12 iac myocytes and fast-twitch and slow-twitch skeletal muscle fibres.
13 low and similar between cardiac myocytes and skeletal muscle fibres.
14  forms insoluble aggregates in the nuclei of skeletal muscle fibres.
15 hancement of force production in fast twitch skeletal muscle fibres.
16 r physiological functions in adult mammalian skeletal muscle fibres.
17 genomic actions in isolated intact mammalian skeletal muscle fibres.
18  regarding the role of potassium released by skeletal muscle fibres.
19 PABPN1 forms aggregates within the nuclei of skeletal muscle fibres.
20 m)) following osmotic challenge in amphibian skeletal muscle fibres.
21 h enhancing SR Ca(2+) pumping rate in single skeletal muscle fibres.
22 CICR to operate even in such fully polarized skeletal muscle fibres.
23 s, as well as in fast-twitch and slow-twitch skeletal muscle fibres.
24 f sarcomeres in series (sarcomere number) in skeletal muscle fibres.
25 been measured in single, frog (Rana pipiens) skeletal muscle fibres.
26 anism was investigated in permeabilized frog skeletal muscle fibres.
27 y motor neurons regulates gene expression in skeletal muscle fibres.
28 and spinal cord form elaborate synapses with skeletal muscle fibres(1).
29 ned in single, intact frog (Rana temporaria) skeletal muscle fibres (3.0 C).
30 enes implicated in structure and function of skeletal muscle fibres (ACTG1), neuronal maintenance and
31 h), but remote dilatations to contraction of skeletal muscle fibres also occur.
32 generated from AMP that is released from the skeletal muscle fibres and dephosphorylated by ecto 5'nu
33 -mediated ROS production in both cardiac and skeletal muscle fibres and the prevention of DOX-induced
34 mechanism is present in all cells, including skeletal muscle fibres, and disruption of the muscle clo
35 ane repair is therefore an active process in skeletal muscle fibres, and dysferlin has an essential r
36 st cells in the body are mononuclear whereas skeletal muscle fibres are uniquely multinuclear.
37 gical functions of glucocorticoids, in adult skeletal muscle fibres, are mediated by a glucocorticoid
38 dy weight and composition, leg lean mass and skeletal muscle fibre area all remained unchanged follow
39 cyte preparations, and fast- and slow-twitch skeletal muscle fibres at 12 degrees C.
40 ration of NO in isolated single mature mouse skeletal muscle fibres at rest and following a period of
41                       We also noted enlarged skeletal muscle fibres, brown fat necrosis and calcifica
42 cellular metabolism in isolated, intact long skeletal muscle fibre bundles from adult mice.
43 assessment of cellular respiration in intact skeletal muscle fibre bundles obtained from the extensor
44                               Treating small skeletal muscle fibre bundles with the synthetic glucoco
45 ment of mitochondrial parameters in isolated skeletal muscle fibre bundles.
46 ndrial function and fuel utilisation in live skeletal muscle fibre bundles.
47  force production in isolated, intact, mouse skeletal muscle fibre bundles.
48 these actions in mouse fast- and slow-twitch skeletal muscle fibre bundles.
49 siological role in isolated intact mammalian skeletal muscle fibre bundles.
50  fully polarized, fluo-3-loaded, intact frog skeletal muscle fibres by exposure to hypertonic Ringer
51                  They consist of specialized skeletal muscle fibres, called intrafusal fibres, which
52                                COX-deficient skeletal muscle fibres contained supra-threshold levels
53                    K(+) released from active skeletal muscle fibres could facilitate vasodilatation i
54 Ia histone deacetylases (HDACs) move between skeletal muscle fibre cytoplasm and nuclei in response t
55                    Growth and maintenance of skeletal muscle fibres depend on coordinated activation
56 cle fibres, but not for the earlier steps of skeletal muscle fibre differentiation, elongation, fusio
57 )(on)) and cessation (V(o)(2)off)) in single skeletal muscle fibres differing in oxdidative capacity,
58 ctions onset ( ) and cessation ( ) in single skeletal muscle fibres differing in oxidative capacity,
59  the Ca(2+)-fura-2 reaction) from single rat skeletal muscle fibres, either fully dissociated from th
60 2+) ) release is enhanced in isolated single skeletal muscle fibres following NO(3) (-) supplementati
61  Nav1.4 currents from intact murine (C67BL6) skeletal muscle fibres for the first time.
62         The sliding filament theory explains skeletal muscle fibre force change as a function of leng
63                                       Intact skeletal muscle fibres from adult mammals exhibit neithe
64 may help to explain the weakness observed in skeletal muscle fibres from mdx mice and, possibly, Duch
65 ron Radiation Facility from small bundles of skeletal muscle fibres from Rana esculenta at sarcomere
66 gulation was studied in mechanically skinned skeletal muscle fibres from rat extensor digitorium long
67 been shown to be involved in turning on slow skeletal muscle fibre gene expression.
68  on the macrostructure and ultrastructure of skeletal muscle fibres, highlighting the current underst
69  cell membrane of many cell types, including skeletal muscle fibres; however, the exact localisation
70 lock of D2 or disruption of the Dio2 gene in skeletal muscle fibres impaired acute exercise-induced P
71 an release dilator concentrations of K+ from skeletal muscle fibres in CH rats as proposed for N rats
72 fibre type-specific gene expression in adult skeletal muscle fibres in culture.
73 e NO generation in quiescent and contracting skeletal muscle fibres in real time, although peroxynitr
74    These mutations produce depolarization of skeletal muscle fibres in response to reduced extracellu
75 scle contraction, adenosine is released from skeletal muscle fibres independently of NO and acts dire
76  is known of how the microvascular supply to skeletal muscle fibres is affected.
77 genomic actions of DHT in isolated mammalian skeletal muscle fibres is to stimulate amino acid uptake
78 pact of ageing on structure and functions of skeletal muscle fibres, likely to be due to a complex in
79 ct cells revealed that mitochondria in adult skeletal muscle fibres maintain a more activated electro
80 ocal nature of contraction-induced injury to skeletal muscle fibres may arise from heterogeneities in
81 mple dataset of OXPHOS protein abundances in skeletal muscle fibres (myofibres).
82 vestigate mitochondrial parameters of intact skeletal muscle fibres obtained from adult mice.
83 sults suggest that cooperative activation of skeletal muscle fibres occurs primarily through spread o
84  vivo OKSM-mediated partial reprogramming in skeletal muscle fibres of mice to the effects of late-li
85  impaired excitation-contraction coupling in skeletal muscle fibres of the mdx mouse, a model of the
86                                              Skeletal muscle fibres once innervated by neurons that l
87  with Mavacamten for 4 weeks, remodelled the skeletal muscle fibre proteome without any noticeable ef
88 lar vesicles (sEVs) that effectively recover skeletal muscle fibre size and extracellular matrix remo
89                                              Skeletal muscle fibre size is highly variable, and while
90 hese changes occurred without alterations in skeletal muscle fibre size or type.
91 fication in intact voltage-clamped amphibian skeletal muscle fibres studied in the gluconate-containi
92 ivation in intact loose-patch clamped murine skeletal muscle fibres subject to a double pulse procedu
93  in both the ventricular myocardium and slow skeletal muscle fibres such as the masseter and is an im
94 f activation, unloaded shortening of skinned skeletal muscle fibres takes place in two phases: an ini
95 ed to a greater extent in rabbit fast-twitch skeletal muscle fibres than in slow-twitch fibres from b
96 ce that adenosine can stimulate receptors on skeletal muscle fibres that are coupled to ATP-sensitive
97 itochondrial disease and shown in individual skeletal muscle fibres that there exist different patter
98 se data demonstrate in these isolated single skeletal muscle fibres that unchanged peak [Ca(2+)](c) i
99 tosolic ROS balance is compromised in intact skeletal muscle fibres that underwent osmotic shock and
100  both rat slow-twitch and rabbit fast-twitch skeletal muscle fibres the rate of tension redevelopment
101 imation of most of the properties of IKir in skeletal muscle fibres, the model demonstrates that a su
102  the RYR1 ex36 mutation and suggests that in skeletal muscle fibres there is a functional reserve of
103  analysis for the first time in single human skeletal muscle fibres to measure muscle mechanics, incl
104 bular (t) system of mechanically skinned rat skeletal muscle fibres to measure SOCE during intracellu
105 y a product of peripheral changes related to skeletal muscle fibre type and mitochondrial density.
106 tional, 12 week aerobic training protocol on skeletal muscle fibre type distribution and satellite ce
107 ssion in a cell culture model and influences skeletal muscle fibre type proportions in horses.
108 hich altered myostatin expression influences skeletal muscle fibre type remains to be determined.
109 ipheral circulation, and peripheral factors (skeletal muscle fibre type, capillarization and concentr
110 fects mechanical and energetic properties of skeletal muscle fibre types.
111 at such charging phenomena should persist in skeletal muscle fibres unable to release stored Ca(2+).
112 ients in mouse flexor digitorum brevis (FDB) skeletal muscle fibres under voltage clamp, using confoc
113 elease flux were determined optically in cut skeletal muscle fibres under voltage clamp.
114  of tension to [Ca2+] that occurs in skinned skeletal muscle fibres upon stretch also occurs in intac
115                               Contraction of skeletal muscle fibres via electrical field stimulation
116 lcium concentration ([Ca2+]i) in intact frog skeletal muscle fibres was determined at two fibre lengt
117 nt protein (HDAC4-GFP) expressed in isolated skeletal muscle fibres, we now show that activation of P
118                               In single frog skeletal muscle fibres, we utilized supercharging voltag
119                                  Fast-twitch skeletal muscle fibres were enzymatically dissociated fr
120 ndividual sarcomeres of voltage-clamped frog skeletal muscle fibres were examined by laser scanning c
121                                     However, skeletal muscle fibres were hypotrophic and their nuclei
122 tension and sarcomere length of relaxed frog skeletal muscle fibres were measured in response to impo
123 ar-neighbour RU interactions, in contrast to skeletal muscle fibres where the effect was enhanced.
124 man disease Duchenne muscular dystrophy, has skeletal muscle fibres which display incompletely unders

 
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