ライフサイエンスコーパス: skin blood flow

1 kin temperature, sweating, and laser-Doppler skin blood flow.

2 and ButOH increase erythema as a function of skin blood flow.

3 ser-Doppler flowmetry to provide an index of skin blood flow.

4 ugh a neurogenic mechanism that also affects skin blood flow.

5 sity, but neither affected histamine-induced skin blood flow.

6 circadian and ultradian (12 h) variations in skin blood flow.

7 function, keratinocyte differentiation, and skin blood flow.

8 Baseline skin blood flow (31 [17-113] vs 16 [9-32] arbitrary perf

9 These measurements were compared with skin blood flow after various stimuli: heating the skin,

10 ema and PG increased erythema with decreased skin blood flow, all as a function of ADHIB haplotype.

11 pler flowmetry (LDF) was used as an index of skin blood flow and cutaneous vascular conductance (CVC)

12 Previous studies show that the rise in skin blood flow and cutaneous vascular conductance durin

13 At inclusion, skin blood flow and DeltaSBF/DeltaT were lower in patien

14 attenuated in aged skin resulting in greater skin blood flow and heat loss during cold exposure.

15 t of thermal stimuli or distal scratching on skin blood flow and histamine-induced itch in healthy vo

16 ds histamine iontophoresis was performed and skin blood flow and itch intensity were measured immedia

17 d flow but reduced histamine-induced maximal skin blood flow and itch intensity.

18 Scratching reduced mean histamine-induced skin blood flow and itch intensity.

19 application in healthy and irritated skin on skin blood flow and its relationship to barrier function

20 ormation process, informed by data regarding skin blood flow and reactive hyperemia in response to pr

21 Paw skin blood flow, angiographic score, and capillary densi

22 tions of skin sympathetic nerve activity and skin blood flow, arterial pressure, and R-R intervals, o

23 Enhancements in basal skin blood flow at 4 hours and 7 days post-administratio

24 osed workers had significantly lower resting skin blood flow at both 21 degrees C and 4 degrees C tha

25 Neuropathy and skin blood flow at rest were assessed in response to ace

26 ing of a limb causes not only an increase in skin blood flow but also in muscle blood flow.

27 ation was found between skin temperature and skin blood flow but not with TEWL.

28 Noxious heat pain increased basal skin blood flow but reduced histamine-induced maximal sk

29 that baroreflexes are capable of modulating skin blood flow, but the effects of baroreceptor loading

30 Alterations in fingertip skin blood flow can be evaluated using a laser Doppler t

31 P did not produce a significant increase in skin blood flow compared to the initial baseline or the

32 ause of enhanced thermoregulatory demand for skin blood flow coupled with dehydration and hyperthermi

33 o and metabolic activity in vivo via altered skin blood flow (Doppler velocimeter) and erythema (refl

34 ing rate (evaporative heat loss) and reduced skin blood flow (dry heat loss) for a given core tempera

35 We evaluated whether changes in skin blood flow during circulatory shock were related to

36 hetic nerve activity (SSNA) that control the skin blood flow during heat stress in CHF patients.

37 These data suggest the rise in skin blood flow during whole body heating contains an H1

38 The reduction in skin blood flow during whole-body cooling is impaired in

39 onal VEGF-A protein expression and transient skin blood flow enhancement in men with T2DM.

40 ation as there was no observable increase in skin blood flow following a second administration of sub

41 icroneurography) from the peroneal nerve and skin blood flow (forearm laser Doppler) in 9 patients wi

42 Skin blood flow has been imaged during stimulation of fi

43 for matricellular proteins in the regulation skin blood flow has never been proposed.

44 Laser-Doppler flowmetry was used to measure skin blood flow in a total of 18 subjects.

45 and, correspondingly, greater reductions in skin blood flow in HTN.

46 ng the role of substance P in the control of skin blood flow in humans.

47 e more important in regulating microvascular skin blood flow in regions rich in arteriovenous anastom

48 The initial, rapid increase in skin blood flow in response to direct application of hea

49 increase in SNA was accompanied by decreased skin blood flow, increased skin vascular resistance, and

50 prostaglandin production to the increase in skin blood flow induced following the iontophoresis of A

51 Skin blood flow is rapidly altered during circulatory sh

52 we measured skin SNA (microneurography) and skin blood flow (laser Doppler velocimetry) as well as h

53 es, heart rate, beat-by-beat blood pressure, skin blood flow (laser-Doppler flowmetry), local sweat r

54 arm from heart level such that the sites of skin blood flow measurement were 34 +/- 1 cm below the h

55 seline ; P < 0.01) and greater reductions in skin blood flow (NTN: -16 +/- 2%baseline vs. HTN: -28 +/

56 -noxious warming the skin did not affect the skin blood flow or itch intensity.

57 Nicotine decreased resting skin blood flow (P < 0.05); this response was inhibited

58 cated on sensory nerves, would attenuate the skin blood flow response to local heating in humans.

59 The initial skin blood flow response to rapid local heating is an ax

60 Skin blood flow responses to graded intradermal microdia

61 The mechanisms involved in the human skin blood flow responses to iontophoretic application o

62 l fluid that would be related with a rise in skin blood flow (SkBF) and temperature sensation.

63 ed with intradermal microdialysis to measure skin blood flow (SkBF) during graded ET-A (BQ-123) and E

64 72-week-old male mice underwent analysis of skin blood flow (SkBF) via laser Doppler in response to

65 Skin blood flow (SkBF) was continuously monitored by las

66 Skin blood flow (SkBF) was monitored by laser-Doppler fl

67 Nicotine affects the regulation of skin blood flow (SkBF), but the mechanisms involved are

68 Esophageal temperature, skin blood flow, sweat rate, and perceived thermal sensa

69 -Doppler flowmetry, and CVC was the ratio of skin blood flow to mean arterial pressure.

70 We assessed skin blood flow using skin laser Doppler on the fingerti

71 Mean arterial pressure, skin blood flow via laser-Doppler flowmetry and core tem

72 es C for 20-30 min until a stable plateau in skin blood flow was achieved.

73 in at 4 microl min(-1) in sites 3 and 4, and skin blood flow was allowed to return to baseline (appro

74 The subsequent increase in skin blood flow was allowed to return to baseline (appro

75 Skin blood flow was also monitored over adjacent sites t

76 Forearm skin blood flow was measured by laser-Doppler flowmetry,

77 An index of skin blood flow was measured directly over each microdia

78 Skin blood flow was measured while local skin heating (4

79 al heating to 42 degrees C was performed and skin blood flow was measured with laser Doppler flowmetr

80 1 older (61-77 years) men and women, forearm skin blood flow was monitored at three sites using laser

81 s) and 11 older (62-76 years) men and women, skin blood flow was monitored at two forearm sites with

82 Skin blood flow was monitored by laser-Doppler flowmetry

83 Skin blood flow was monitored during antidromic stimulat

84 bstance P was then delivered to the skin and skin blood flow was monitored for 45-60 min.

85 In addition, skin blood flow was monitored over an area of forearm sk

86 In supine individuals, forearm skin blood flow was monitored via laser-Doppler flowmetr

87 The techniques used to monitor skin blood flow were laser Doppler perfusion imaging and

88 Baseline measurements of skin blood flow were obtained on the flexor aspect of th

89 ance P produced a dose-dependent increase in skin blood flow with the concentrations of substance P t

90 IPA increases skin blood flow without erythema and PG increased erythe