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1 hereas the lack of Kindlin-1 causes profound skin defects.
2 recursors in skin and corrected the hair and skin defects.
3              K14-CXCR4KO pups had no obvious skin defects.
4                               Full-thickness skin defects (225 mm2) were created on the dorsum of rat
5 xin-linked diseases ranging from deafness to skin defects and blindness.
6 enotypic defects of the EEC syndrome include skin defects and limbal stem-cell deficiency.
7 rome (SLS), a rare disorder characterized by skin defects and mental retardation.
8 stational metabolic defect causes the severe skin defects and neonatal lethality remain important una
9 including failure of eyelid fusion, hair and skin defects, and abnormalities of lung development.
10 clude micro- phthalmia, sclerocornea, linear skin defects, and agenesis of the corpus callosum.
11 ssion led to rescue of the neonatally lethal skin defects, and the resulting mice were viable and fer
12  definitively that the Dicer- and Dgcr8-null skin defects are both striking and indistinguishable.
13                                Surprisingly, skin defects are not apparent in mice lacking 25-hydroxy
14 lly for over a year, 9V;C* pups had a lethal skin defect as did 0S;C* mice resembled that of 0S mice.
15 e also showed similar, although less severe, skin defects as dCKO mice.
16       Aplasia cutis manifests with localized skin defects at birth and is a feature in various syndro
17 ful as a dermal substitute in full-thickness skin defects, but healing with xenogenic ADM was poor.
18 ardi syndrome and microphthalmia with linear skin defects, but in these sporadic conditions, evidence
19 eratinocytes induced a range of inflammatory skin defects characteristic of psoriatic and actinic ker
20  Prmt5 conditional knockouts exhibited gross skin defects, compromised skin barrier function, and red
21 e by side on 15 patients with full-thickness skin defects for 1 week before autografting.
22                       CDPX2 patients display skin defects including linear or whorled atrophic and pi
23           Aquaporin 3-deficient mice exhibit skin defects, including decreased glycerol content and i
24 ouse ectoderm triggers both craniofacial and skin defects, including hyperproliferation, loss of spin
25                           Impaired repair of skin defects is a major complication of diabetes; yet, t
26 e, although grossly normal at birth, exhibit skin defects, lung hypoplasia, severe runting, poor body
27                   Microphthalmia with linear skin defects (MLS) is an X-linked dominant male-lethal s
28                   Microphthalmia with linear skin defects (MLS) is an X-linked dominant, male-lethal
29                   Microphthalmia with linear skin defects (MLS) syndrome is an X-linked male-lethal d
30                These findings correlate with skin defects observed in mice and alterations found in h
31 MLS syndrome have microphthalmia with linear skin defects of face and neck, sclerocornea, corpus call
32 l mice for Klf4 die shortly after birth from skin defects, precluding their analysis at later stages.
33 l(nkt) double-mutant mice indicates that the skin defect remains under the absence of T and B cells.
34 us skin grafting is a standard treatment for skin defects such as burns.
35 xpanded to treat less severe burns and other skin defects, such as chronic diabetic wounds.
36                   Microphthalmia with linear skin defects syndrome (MLS) is an X-linked dominant, mal
37                   Microphthalmia with linear skin defects syndrome (MLS) is an X-linked male-lethal d