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1 infections of several cell lines and a human skin lesion.
2 g antibiotic treatment and resolution of the skin lesion.
3 xtract important dermoscopic features of the skin lesion.
4 ptors exist in clinically resolved psoriatic skin lesions.
5 in decisions to biopsy a series of pigmented skin lesions.
6 more than five destructively treated actinic skin lesions.
7 ut also dramatically resolved the associated skin lesions.
8 athogens were frequently identified in these skin lesions.
9 cruitment of leukocytes to the EB-associated skin lesions.
10  pruritic skin disease with multiple nodular skin lesions.
11  practiced because it resulted in occasional skin lesions.
12  a dermatotropic poxvirus that causes benign skin lesions.
13 a) and interleukin-6 (IL-6) in MRSA-infected skin lesions.
14 acute care clinic with multiple small tender skin lesions.
15 ative color channel to detect malignancy for skin lesions.
16 phritis, reduced spleen size, and diminished skin lesions.
17       The levels of IL-22 are elevated in AD skin lesions.
18 elevated in AD, and Th22 cells infiltrate AD skin lesions.
19 t with cutaneous FL who showed regression of skin lesions.
20 ents, and primary care providers to diagnose skin lesions.
21  attractant protein-1 (MCP-1) in MRSA USA300 skin lesions.
22 mice are completely resistant to HFD-induced skin lesions.
23 rmoscopy may be applicable for monitoring of skin lesions.
24 atory skin disease characterized by pruritic skin lesions.
25 orted to have frequent epistaxis and similar skin lesions.
26 d through symptoms of pruritus and recurrent skin lesions.
27 presents a morphologic hallmark of psoriatic skin lesions.
28  any skin abnormality/lesion and significant skin lesions.
29 e response amplification, and development of skin lesions.
30 ecurrent fever, arthralgia, and disseminated skin lesions.
31 -LTR promoter develop spontaneous neoplastic skin lesions.
32 r BALB/c or C57BL/6 mice resulted in smaller skin lesions.
33 e evaluation of itch and pain for suspicious skin lesions.
34 le tool for physicians evaluating suspicious skin lesions.
35 piRNAs examined are downregulated in leprosy skin lesions.
36 ccal biofilms have been reported on adult AD skin lesions.
37 /6-EZH2 pathway in human and mouse psoriatic skin lesions.
38  for surgical biopsies of atypical pigmented skin lesions.
39 vant types of benign and malignant pigmented skin lesions.
40 liaceus, which is characterised by exclusive skin lesions.
41 m 400 Bangladeshi individuals with arsenical skin lesions.
42 ropical disease characterized by disfiguring skin lesions.
43 d 77.8% of the qPCR positive individuals had skin lesions.
44 astatic tumours versus normal skin or benign skin lesions.
45 ) is markedly upregulated in human psoriatic skin lesions.
46 essing potential overtreatment of suspicious skin lesions.
47 ouse model of imiquimod-induced psoriasiform skin lesions.
48 ine-grained variability in the appearance of skin lesions.
49 ts with early AD, but T cells predominate in skin lesions.
50 esented 34 species and 4 families; 27.5% had skin lesions, 13.3% were positive for O. ophiodiicola DN
51 but also in patients without a history of BU skin lesions (24.7%).
52 the medical records identified 2891 biopsied skin lesions; 457 of these were nonpigmented neoplasms.
53                   A convenience sample of 40 skin lesions (8 nevi, 8 seborrheic keratoses, 7 basal ce
54         Computational results also show that skin lesion abrupt cutoff is a reliable indicator of mal
55 t in humans is known to exacerbate psoriasis skin lesion activity at distant sites.
56 ho received ustekinumab showed regression of skin lesions after 2 weeks and almost complete resolutio
57 effect, as the same mice did not exhibit any skin lesions after chronic UV irradiation.
58 d to the skin and spontaneous improvement of skin lesions after several years.
59 rtiary referral medical center with purpuric skin lesions after using epidermal growth factor recepto
60 6beta(-/-) mice developed larger zosteriform skin lesions along infected neurons.
61 ythema migrans (EM) or an annular, expanding skin lesion and uninfected individuals from areas of end
62 enetic ablation of Nrf2 worsened spontaneous skin lesions and accelerated PPK development in footpad
63 hysicians were more likely to care for viral skin lesions and acne (3405 of 7287 visits [46.7%]), whe
64 stigate IL-17 expression in acute eczematous skin lesions and correlate it with markers of remodeling
65 L/6 mice abolished the expression of Arg1 in skin lesions and dLNs but did not affect development and
66 in immunity, which facilitates initiation of skin lesions and establishment of tumors by promoting im
67  measured only with difficulty compared with skin lesions and growth faltering that are apparent for
68 al lepromatous patients, both locally in the skin lesions and in circulating mononuclear cells.
69 GFBR2 mutations in human vemurafenib-induced skin lesions and in sporadic cSCC.
70  molecule, Pak1, in sun-induced premalignant skin lesions and indicates that increased Pak1 activatio
71          In addition, topical PG ameliorated skin lesions and inflammation in a mouse model of psoria
72 a disfiguring disease characterized by large skin lesions and is transmitted by a sand fly vector.
73 ll activation that is primarily localized to skin lesions and lacks the systemic inflammation of mode
74   Reports about the manifestation of various skin lesions and lesions of the vascular system in some
75  Finally, IFI16 was upregulated in psoriasis skin lesions and localized to the cytoplasm in a subpopu
76 ech Republic for the presence of macroscopic skin lesions and O. ophiodiicola.
77  that initiate remodeling in AD prior to any skin lesions and reveal the significance of LPS in OVA u
78 rus to adjacent skin, thus leading to larger skin lesions and satellite lesions in IL-1R1(-/-) mice.
79 e epithelial cytokine IL-33 are increased in skin lesions and serum of patients with AD.
80  human experts in the diagnosis of pigmented skin lesions and should have a more important role in cl
81 maging is important to improve monitoring of skin lesions and skin conditions, ensure the availabilit
82 lyzed the oxygen levels found in leishmanial skin lesions and their effect on the NOS2-dependent leis
83  (interquartile range) experience evaluating skin lesions and using dermoscopy of participants was 6
84                   In vivo, Arg1 was found in skin lesions and, to a much lower extent, also in dLNs o
85 ions, recurrent/persistent thrombocytopenia, skin lesions) and bleedings.
86 th Hymenoptera venom-induced anaphylaxis, no skin lesions, and baseline serum tryptase </=20 ng/mL.
87  healing of the ulcers, stabilization of the skin lesions, and control of the patient's intraocular p
88 erapeutic response, decrease in inflammatory skin lesions, and decrease of circulating NKp44+ ILC3.
89 xA expression, reduced immune infiltrates in skin lesions, and decreased CLASI-A score.
90 including systemic inflammation, destructive skin lesions, and interstitial lung disease.
91 ancer, clearance and prevention of keratotic skin lesions, and intervention-specific adverse events.
92 oporosis, recurrent anaphylaxis, presence of skin lesions, and long-term antihistamine treatment.
93 pathy, interstitial lung disease, ulcerative skin lesions, and premature death.
94 e ability to detect, categorize, or identify skin lesions, and the intervention was evaluated through
95 o 2017, from 83 male patients with ulcers or skin lesions, and were examined using multilocus sequenc
96 menoptera sting-induced anaphylaxis, without skin lesions, and with tryptase levels of less than 11.4
97                     In other cases, however, skin lesions are absent, which represents a diagnostic c
98                                           AD skin lesions are often exacerbated by Staphylococcus aur
99                          Linear nonblanching skin lesions are thought to occur very rarely in patient
100 atric AD, but alterations in early pediatric skin lesions are unknown, limiting advancement of target
101 arning algorithms can diagnose all pigmented skin lesions as accurately as human experts is unclear.
102 with anti-COL7 IgG led to the same extent of skin lesions as in wild-type mice.
103 egulated in the epidermis of human psoriatic skin lesions as well as in a mouse model of psoriasis in
104 We detected high levels of EBER1 RNA in such skin lesions, as well as EBV-microRNAs, but no intact EB
105 cal samples of sunlight-induced premalignant skin lesions assessed by immunohistochemistry.
106 nterquartile range [IQR], 0.5 to 7.4 years), skin lesions associated with the use of anti-TNF therapy
107 KBDM showed a partial reduction in secondary skin lesions at day 8 p.i. compared to the level for HSV
108 dard diagnostic test for leprosy is based on skin lesion biopsy, which is invasive and presents low s
109  a history of spontaneous pneumothoraces and skin lesion biopsy.
110                                          Our skin lesion border detection algorithm outperforms other
111                                     Accurate skin lesion border detection is the key to extract impor
112 e address this problem by developing a novel skin lesion border detection method with a robust edge i
113                                    Automated skin lesion border examination and analysis techniques h
114         In the proposed approach, first, the skin lesion border is detected by the density based lesi
115 f texture homogeneity along the periphery of skin lesion borders based on LSP is an effective way of
116    Based on dermatologist drawn ground truth skin lesion borders, the results indicate that our metho
117 d important in the dermoscopic evaluation of skin lesions but their relative significance is unknown.
118 mor necrosis factor (TNF) antibodies develop skin lesions, but the lesions and their clinical course
119 ortively infected cultured cells and a human skin lesion by next-generation sequencing.
120 ion-making process to biopsy or not biopsy a skin lesion by providing the most probable diagnosis via
121                          Analysis of leprosy skin lesions by gene expression profiling and immunohist
122 s studied in chemically induced inflammatory skin lesions by monitoring DsRed expression using confoc
123 cobacterium ulcerans, the causative agent of skin lesions called Buruli ulcers.
124 l genes of the IL-2 pathway were abundant in skin lesions caused by Leishmania braziliensis.
125                  Horses suffer from pruritic skin lesions, caused by type-I/type-IV allergic reaction
126 TIVE-LTC and PEL cells, and Kaposi's sarcoma skin lesion cells.
127 lar and histopathologic profile of psoriatic skin lesions compared with ustekinumab at week 4.
128 fection and increased parasite burden in the skin lesions compared with wild-type controls.
129   The presence of rVSV in synovial fluid and skin lesions confirmed causality.
130                                Biopsy of the skin lesions confirmed the same disease, and [18F]fluoro
131 e, 786 free-ranging snakes were examined for skin lesions consistent with ophidiomycosis and swabbed
132          Lack of LC3 expression in psoriatic skin lesions correlates with parakeratosis and deregulat
133 ned, we provide a systematic overview of the skin lesions described in COVID-19 patients, discuss pot
134 onfidence interval [95% CI] 6.73-10.91), >50 skin lesions destroyed (odds ratio 3.37, 95% CI 2.85-3.9
135 current clinical setting, abrupt cutoff of a skin lesion determined by an examination of a dermatolog
136 frequently been speculated that pruritus and skin lesions develop after topical exposure to aeroaller
137 rized by a strong Th1 response that leads to skin lesion development.
138 hat may variably present with characteristic skin lesions, episodes of mast cell mediator release, an
139 been optimized for measurements on pigmented skin lesions (excitation wavelength 976 nm and a wavelen
140 lts in the development of a zosteriform-like skin lesion followed by a time-dependent increase in pai
141 n sensitization on an atopic dermatitis-like skin lesion, followed by intragastric allergen challenge
142       Specifically, G-1 reduced Hla-mediated skin lesion formation and pro-inflammatory cytokine prod
143 ation of FIT-039 ameliorated the severity of skin lesion formation in mice infected with an acyclovir
144 tral play fighting network position on later skin lesions from aggression was assessed in domestic pi
145                                              Skin lesions from aggression were counted after a dyadic
146 xceeding dermatologists for the diagnosis of skin lesions from clinical and dermoscopic images.
147 ontrol samples (n = 10), as well as fibrotic skin lesions from localized scleroderma and uninvolved s
148 e of one dermatologist of biopsy data of all skin lesions from one clinic during a 10-year period.
149                                              Skin lesions from patients infected with Leishmania braz
150 in molecular and histopathologic profiles in skin lesions from patients with psoriasis receiving risa
151 iameter; n = 19), large lesions (MPCM-large, skin lesions &gt;/= 1 cm in diameter; n = 89), or atypical
152 ion suggests that women and controls with no skin lesion had a higher capacity to methylate the inges
153                              Acute canine AD skin lesions had a significant up-regulation of genes en
154 a in a quasi-therapeutic setting (i.e., when skin lesions had already developed) improved experimenta
155               Two weeks later, the number of skin lesions had increased, and the patient had begun to
156  for severe disease, but its mechanism in AD skin lesions has not been studied.
157                                     Purpuric skin lesions have only rarely been reported in patients
158 osed with KID syndrome due to characteristic skin lesions, hearing deficiency and keratitis.
159 logical features, such as severe emaciation, skin lesions, hemorrhaging, and more zebrafish death.
160 haracterized by higher numbers of T cells in skin lesions, higher IL-17A expression, and stronger exp
161 enters the peripheral nervous system through skin lesions; however, interferon can protect neurons fr
162 ated polyomavirus could be isolated from the skin lesions; however, the spicules were positive for Me
163 f unsupervised segmentation of the ISIC 2018 skin lesion images.
164       Low oxygen levels found at leishmanial skin lesions impaired the NOS2-dependent leishmanicidal
165 nd suppress inflammation to persist in human skin lesions.IMPORTANCE Poxviruses adapt to specific hos
166          RNA-sequencing analysis of an HVLPD skin lesion in a white patient compared with his normal
167 effective method to detect malignancy of the skin lesion in dermoscopy images.
168 c anti-inflammatory mediators on mucosal and skin lesions in autoantibody-mediated diseases.
169 et (HFD) greatly increased the occurrence of skin lesions in C57BL/6 mice.
170 e and characteristics of linear nonblanching skin lesions in children with Henoch-Schonlein syndrome.
171                          After accruing more skin lesions in contest 1, individuals were less likely
172 e been found to be an important part of VACV skin lesions in mice in controlling the infection as wel
173  attenuated imiquimod-induced psoriasis-like skin lesions in mice, and improved imiquimod-induced dis
174 ermis-specific SIRT1 ablation causes AD-like skin lesions in mice, and mice with epidermal SIRT1 dele
175  experimental model to induce psoriasis-like skin lesions in mice.
176 ic mutant strain caused significantly larger skin lesions in mice.
177  associated with the appearance of recurrent skin lesions in millions of people.
178  However, 8 caused the identical microscopic skin lesions in NHPs found with 7, albeit with lower sev
179 howed that ZPL-3893787 improved inflammatory skin lesions in patients with AD, confirming H(4) recept
180 kin to posttreatment lesional skin to active skin lesions in patients with AD.
181 the prevalence and characteristics of linear skin lesions in patients with Henoch-Schonlein syndrome.
182 nalysing two case studies: the assessment of skin lesions in primary care, and the surveillance of pa
183 ake has been associated with arsenic-related skin lesions in South Asia, but its association with ski
184 uscum contagiosum virus, which causes benign skin lesions in young children and more extensive infect
185                                  Sun-induced skin lesions, in particular actinic keratosis, are gener
186 of patients having no apparent history of BU skin lesions, including during the current course of ill
187 nd/or natural killer (NK) cells in blood and skin lesions induced by sun exposure that are infiltrate
188 23, a cytokine highly expressed in psoriatic skin lesions, induces differentiation and promotes proli
189 rn showing diffuse cutaneous maculopapillary skin lesions involving the head, trunk and extremities,
190 pigment pattern cutoff at the periphery of a skin lesion is one of the most important dermoscopic fea
191 ss of pigment patterns at the periphery of a skin lesion is one of the most important dermoscopic fea
192 se to this innate stimulus that causes these skin lesions is poorly understood.
193 g profile of DFU isolates to SAg profiles of skin lesion isolates and vaginal mucosa isolates reveale
194  of DFU isolates was more similar to that of skin lesion isolates.
195 per 17 cytokine, is upregulated in psoriatic skin lesions, its precise role is unclear.
196 nting either with small lesions (MPCM-small, skin lesions &lt;1 cm in diameter; n = 19), large lesions (
197 stive of systemic sarcoidosis, and biopsy of skin lesions may detect patients otherwise missed by rou
198 ficantly more mast cells accumulation in the skin lesions, more severe Th2-mediated inflammation, inc
199 acy of primary care physicians to categorize skin lesion morphology in the same test bank of images w
200 lucagonomas), the most conspicuous signs are skin lesions (necrolytic migratory erythema), while in s
201                                              Skin lesions occur frequently in association with anti-T
202                               show that each skin lesion of disseminated superficial actinic porokera
203 e induction of hBD2 in the barrier-defective skin lesions of AD and indicates therapeutic potential.
204                                   Eczematous skin lesions of atopic dermatitis (AD) as well as allerg
205 and L. tropica specific IgG/ADCP/ADNP in the skin lesions of CL patients.
206 d immune cell infiltration were prominent in skin lesions of human IL-26 transgenic mice.
207 t epidermal RABGEF1 expression is reduced in skin lesions of humans diagnosed with either atopic derm
208 eased in PBMCs and exclusively detectable in skin lesions of IBH-affected horses.
209 d FGF7 were significantly upregulated in the skin lesions of imiquimod-treated human IL-26 transgenic
210 preening behaviour and resulted in increased skin lesions of infested birds.
211 ic CD8(+)CD28(-) T cells are resident in the skin lesions of patients in the early stage of the disea
212 ges of genes involved in NK-cell function in skin lesions of patients with atopic dermatitis (AD) wer
213 isease, IL-27 was more strongly expressed in skin lesions of patients with progressive lepromatous le
214 atory cytokine that is elevated in serum and skin lesions of patients with psoriasis.
215 kingly, DUOX1 was drastically induced in the skin lesions of psoriasis and lichen planus patients.
216  of RIP3, was not significantly different in skin lesions of TEN.
217  2013, a novel orthopoxvirus was detected in skin lesions of two cattle herders from the Kakheti regi
218 signatures of CL caused by L. tropica in the skin lesions of ulcerative CL (UCL) and non-ulcerative C
219                                     Multiple skin lesions of varying sizes were identified on the sca
220  he underwent excision of a raised pigmented skin lesion on his left calf that proved to be melanoma
221     We describe a burn patient who developed skin lesions on her skin-graft harvest and skin-graft re
222 o 1 (indicating clearing or near-clearing of skin lesions) on the investigator's global assessment (P
223 ities can be approached by either a study of skin lesions or activated CLA(+) T-cell subsets in perip
224 atent yaws was defined as lack of suspicious skin lesions or presence of ulcers negative for Treponem
225     There were no differences in significant skin lesions or resistance after 6 weeks.
226 equent among those with a history of treated skin lesions or sun-sensitive skin types.
227 el to quantitatively measure abruptness of a skin lesion over our previous method.
228 n (P = .001), and largest diameter length of skin lesion (P < .003).
229 , 2.4-9.1]; P < .001), whereas the number of skin lesions (P = .06) and location on the leg (P = .07)
230                                              Skin lesions (P=0.002), orthopedic illness (P<0.001), ca
231 did not affect development and resolution of skin lesions, parasite burden, NO production, or host ce
232                                              Skin lesions, patient demographic characteristics, treat
233 multisystem disorder characterized by patchy skin lesions, polysyndactyly, diverse cerebral malformat
234 apy and increasing the dosage of prednisone, skin lesions promptly resolved.
235  clinically and/or dermoscopically equivocal skin lesion referred to RCM imaging, from January 2012 t
236  Patients typically present with distinctive skin lesions reflective of vascular drop out.
237 ells to blistering skin and to more advanced skin lesions remains a challenge.
238 lelic desmoglein 1 (DSG1) mutations, exhibit skin lesions reminiscent of erythrokeratodermia variabil
239 an IL-17-mediated inflammatory skin disease, skin lesions resolve with therapy, but often recur in th
240  of the variation in risk for depression and skin lesions resulting from sun exposure (actinic kerato
241  of thousands of non-AS3MT SNPs) and reduced skin lesion risk in an independent sample (p < 0.05).
242 standalone AI tool to correctly categorize a skin lesion's morphology from a test bank of images.
243 AI on identifying and categorizing a primary skin lesion's morphology.
244                                  The primary skin lesion scores were identical in severity and kineti
245 R7 or TRPA1 displayed reduced scratching and skin lesion severity.
246                                              Skin lesions showed a trend for increased CD8, CD25, and
247 e affected individual; in another, biopsy of skin lesions showed features of trichoblastoma.
248              Immunohistochemical staining of skin lesions showed increased C1q in both RR and ENL.
249 of the right hand (main-en-griffe) and a few skin lesions since eighteen months.
250               Diabetic mice showed increased skin lesion size and bacterial load and decreased PGE2 s
251  sagA in MGAS315 synergistically reduced the skin lesion size and GAS burden in the liver and spleen.
252  globally, and the correct classification of skin lesions (SLs) into benign and malignant tissue rema
253  improve participants' abilities to diagnose skin lesions; some are more effective than others.
254                                Granulomatous skin lesions sparing warm regions (eg, flexural surfaces
255 ophage cell lineages, primary monocytes, and skin lesion specimens from patients with a disseminated
256 hich HSV-1 genome and mRNA levels in primary skin lesions started to decline in both wild type and IL
257 ene expression profiles derived from leprosy skin lesions suggested a link between IL-27 and the IFN-
258 oking, body mass index and the prevalence of skin lesion suggests that women and controls with no ski
259  improve the clinical diagnostic accuracy of skin lesions suspected of melanoma.
260 s that gene-expression profiles of psoriasis skin lesions, taken in the first 4 weeks of treatment, c
261 reater reduction in pruritus and severity of skin lesions than placebo in patients with prurigo nodul
262 ly Lyme disease is the erythema migrans (EM) skin lesion that develops at the tick bite site typicall
263 tivated antiviral immunity, in particular in skin lesions that are infiltrated with pDCs.
264 l is needed to assist clinical assessment of skin lesions that are suspected of (early) melanoma.
265 is, are generally considered as premalignant skin lesions that can progress into squamous cell carcin
266                    Eleven patients (34%) had skin lesions that covered places other than the lower ex
267 s a worldwide distribution and causes benign skin lesions that may persist for months in young childr
268 treated with oral therapy alone, while 2 had skin lesions that resolved spontaneously.
269 s a long-duration infection characterized by skin lesions that typically display an absence of inflam
270        Within group 3 (no history of treated skin lesions), the strongest factor associated with clin
271            Dermatofibromas are common benign skin lesions, the etiology of which is poorly understood
272 the inability to precisely assess the age of skin lesions, thus affecting the analysis of time-depend
273 ry to look for a portal of entry and classic skin lesions to help diagnose and manage the condition.
274 mote the progression of certain premalignant skin lesions to malignant lesions will permit us to prev
275       Clinical features (age, sex, number of skin lesions, tumor stage, and location as leg vs elsewh
276     Age, sex, period of diagnosis, number of skin lesions, tumor stage, tumor location (leg vs nonleg
277        Here we demonstrate classification of skin lesions using a single CNN, trained end-to-end from
278                  Automated classification of skin lesions using images is a challenging task owing to
279            In situ identification of PTX3 in skin lesion was confirmed by quantitative reverse-transc
280           A signature of the L. braziliensis skin lesion was defined, which includes over 2,000 diffe
281            The median time to development of skin lesions was 3.5 months.
282 The development of ovalbumin-induced AD-like skin lesions was analysed in H4R(-/-) mice and in H4R an
283 s of volunteers (n = 18), and development of skin lesions was monitored over a period of 4 weeks.
284 r and lower limbs without pain, swelling, or skin lesions was noted at physical examination.
285              In the case of the first sister skin lesions were accompanied by lung abnormalities.
286                              Biopsies of the skin lesions were collected from both patients for histo
287                                              Skin lesions were located on the leg in 76% of the cases
288                                     Although skin lesions were mild in most cases, in some snakes the
289 omes and Measures: A total of 1279 equivocal skin lesions were sent for RCM imaging.
290 represent a subset of RNAs detected in human skin lesions which mapped to homologs of numerous postre
291 ous leishmaniasis (CL) presents as 1 or more skin lesions, which makes local therapy inherently attra
292 no identifiable present or past Buruli ulcer skin lesions, which was recorded in at least 14 patients
293 ool to assist physicians in the diagnosis of skin lesions while still requiring the user to criticall
294                            In adults without skin lesions who suffer from mediator-related or other t
295 cialists because the morphologies of primary skin lesions widely vary from patient to patient.
296  IL-25, and IL-33 in human atopic dermatitis skin lesions with high house dust mite sensitization.
297                                              Skin lesions with pruritic vesicles on an erythematous b
298 a common non-contagious chronic inflammatory skin lesion, with frequent recurrence.
299 -/-) mice showed a clear amelioration of the skin lesions, with a diminished influx of inflammatory c
300    In contrast, the scratching persisted and skin lesions worsened over time in sham-treated mice.

 
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