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1 ined for koayu fish and related allergens by skin prick and allergen-specific immunoglobulin E (IgE)
5 tization in an unsupervised manner, based on skin prick and sIgE tests taken throughout childhood and
8 ) LEN) survey, we measured the prevalence of skin prick positivity to a panel of allergens, and geome
13 lected at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challen
14 in and/or gastrointestinal symptoms only and skin prick test < 8 mm) are considered for home-based mi
21 wheeze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen
24 g the basophil activation test (BAT) and the skin prick test (SPT) and measuring the levels of peanut
25 in had the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and
27 sought to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg pro
28 ized nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut
29 or urticaria upon CHX exposure and positive skin prick test (SPT) and/or positive CHX ImmunoCAP test
30 ercise, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE
31 lergic sensitization was determined based on skin prick test (SPT) of five mites, three molds, and ni
33 ng cows' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, place
37 action to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/o
38 lergy, milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/o
39 nd 4 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) le
40 ping peanut allergy, and the implications of skin prick test (SPT) screening before peanut introducti
42 protein levels in household dust and peanut skin prick test (SPT) sensitization and likely allergy.
43 nfantile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong a
44 six dog allergens (Can f 1-6) in commercial skin prick test (SPT) solutions and to determine individ
46 of 5276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with
49 eczema, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group I
51 fe (EQ-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smel
57 pecific IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge p
58 ed risk factors for atopy (allergen-specific skin prick test [SPT] reactivity and IgE [asIgE] sensiti
60 le NSAID in history were tested first with a skin prick test and if negative challenged with the culp
62 without systemic IgE-sensitisation tested by skin prick test and serum allergen-specific IgE (sIgE) d
64 nical-demographic questionnaire, spirometry, skin prick test and specific IgE were evaluated yearly.
68 pplied for selected cases where the history, skin prick test and/or specific IgE are not definitive f
69 e positive ELISA results correlated with the skin prick test areas with the whole body and the setae
75 white specific IgE (sIgE) levels in serum or skin prick test has been shown to be a poor predictor of
77 including detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination
78 aluation, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 mo
80 wanted to measure geographical variation in skin prick test positivity and assess whether it was exp
81 Geographical variation in the prevalence of skin prick test positivity in Europe is unlikely to be e
82 re fitted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.3
85 Associations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using
87 ion between a chronic helminth infection and skin prick test reactivity even in a developed country.
88 e reactions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitizatio
90 studies demonstrated that exercise increases skin prick test reactivity to and bioavailability of the
91 Total IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all redu
92 o 2.45 +/- 0.24 (P = .001) and a decrease in skin prick test reactivity to house dust mite from 7.0 +
93 children with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-
94 SAFS (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumig
96 ID-independent anaphylaxis to LTPs, positive skin prick test response to LTPs, and serum LTP IgE.
98 PPOIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels
102 tifiable by using routinely available peanut skin prick test responses or specific IgE levels, but th
104 serum total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and I
105 ves with allergic disease) but with negative skin prick test responses to common allergens at randomi
106 th a history of ragweed allergy and positive skin prick test responses to ragweed were randomized and
108 exposure and sensitization (as determined by skin prick test responses) was analyzed in more than 100
109 ith peanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2
110 eanut but have peanut-specific IgE, positive skin prick test responses, or both represents a signific
113 random sample of participants with negative skin prick test results attended a hospital-based food c
115 th antivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-g
117 higher IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower
118 among M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+
119 e in peanut-specific basophil activation and skin prick test titration compared with nonresponders.
120 iral culture for varicella-zoster virus, and skin prick test to common food and animal allergens were
121 icipants aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were expose
122 r anaphylaxis related to NSAID, (3) positive skin prick test to foods and/or specific IgE to food all
123 easonal allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate
124 ty reaction after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind
130 E 33.3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experience
133 thma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the
136 iagnostic testing, 47.3% was assessed with a skin prick test, 39.9% with a serum specific IgE test, a
137 ondary outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 meas
139 nical-demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were
142 nly 4 predictors of the original model: sex, skin prick test, peanut sIgE, and total IgE minus sIgE.
143 shed, using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE)
147 gE/4 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.9
148 atients were classified by clinical history, skin prick test/serum specific IgE (sIgE), and nasal all
149 CFC; <=500 mg of peanut protein), a positive skin-prick test (SPT) result (>=5 mm wheal diameter abov
150 measures of allergic sensitisation (such as skin-prick test [SPT] and serum specific IgE [sIgE]) whe
153 on who initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60
154 ract, which was determined with the use of a skin-prick test--one consisting of participants with no
155 dence of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitiz
159 tern blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to d
160 Commercial allergen extracts for allergy skin prick testing (SPT) are widely used for diagnosing
163 orm beta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra
165 ng hay fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific
169 intervention (structured allergy history and skin prick testing and appropriate advice on allergy avo
170 gy intervention (structured allergy history, skin prick testing and appropriate allergy avoidance adv
172 s defined as one or more positive results on skin prick testing and clinically relevant symptoms of r
173 olerant (ST), or food allergic (FA) based on skin prick testing and food challenge at 12 months of ag
176 re, taking a structured allergy history with skin prick testing and tailored advice on allergy avoida
180 uch as family history (50.2%) and conducting skin prick testing in non-high-risk children (43.9%).
182 ly relevant sensitizations are elucidated by skin prick testing or by the determination of specific I
183 ample of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's mil
185 grass pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Be
186 subjected to topical cowhage provocation and skin prick testing with histamine and assessed for diffe
188 with parental face-to-face interviews and/or skin prick testing, 238 (10.4%) were eligible for a DBPC
189 minth Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siber
190 e an interviewer-administered questionnaire, skin prick testing, and measurement of lung function fro
191 similar in children positive and negative on skin prick testing, and were not appreciably altered by
194 post-bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity
195 ths for scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1
211 y against common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE)
213 ich they answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and pr
214 ants completed a questionnaire and underwent skin prick tests (SPTs) to egg, peanut, cow's milk, fish
215 were invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2
216 ous reactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut sol
217 erial 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial mil
219 logical work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activati
220 e following outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE
221 7 with mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-P
222 tic sensitivity to 65% compared with 20% for skin prick tests and 46% ImmunoCAP using kiwi extract.
223 nonallergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergen
226 re, taking a structured allergy history with skin prick tests and giving tailored advice on allergy a
234 reened 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food c
236 ust mites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2),
237 icult, because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts
239 ergenicity of Ory c 3 was confirmed by using skin prick tests and the basophil activation assay.
240 formed consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; bein
241 ldren (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data colle
242 sensitization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age
245 to determine whether C+ assayed by means of skin prick tests influenced AR symptom severity in contr
246 Two hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic
249 ohort were also more likely to have positive skin prick tests to cabbage, lettuce and mustard and sen
253 ciation between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensit
257 easurements of eczema, asthma, rhinitis, and skin prick tests were available for all follow-ups.
259 Food-specific serum IgE measurements and skin prick tests were performed before initiating the di
260 n, IgE inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selec
266 meat, we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha
269 llenges with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measure
271 ecific immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled f
273 te use of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the tim
274 food-allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and f
277 -of-function samples, we performed histamine skin prick tests, investigated the contribution of STAT3
279 for food allergy by standardized interviews, skin prick tests, prick-to-prick tests and ImmunoCAP.
282 al interview combined with blood collection, skin prick tests, spirometry with bronchodilation, and e
299 At Day 85, 6 weeks after the last dose, skin prick wheal responses to allergen were suppressed b