コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 o standard diagnostic methods (ImmunoCAP and skin prick test).
2 nd ELISA inhibition, basophil activation and skin prick test.
3 t basophils and elicit positive responses in skin prick tests.
4 he patient's allergic triggers or performing skin prick tests.
5 en Asthma Study completed questionnaires and skin prick tests.
6 Atopy was assessed by using skin prick tests.
7 s: all had negative results for aeroallergen skin prick tests.
8 Atopy was assessed using skin prick tests.
9 gg allergy was primarily determined by using skin prick tests.
10 n observed pasteurized raw egg challenge and skin prick tests.
11 85; P = 0.02), and with similar findings for skin prick tests.
12 and 6 years by specific IgE assessments and skin prick tests.
13 inical examinations including serum IgEs and skin prick tests.
14 saline challenge tests, questionnaires, and skin prick tests.
15 ystematic reintroduction analysis but not by skin-prick tests.
16 ldren were examined for eczema and underwent skin prick testing.
17 en patients assigned to immediate or delayed skin prick testing.
18 pic status were determined by spirometry and skin prick testing.
19 ted for more detailed assessments, including skin prick testing.
20 ng a rat basophil leukaemia cell line and by skin prick testing.
21 ed with IL-31 and NaCl (negative control) by skin prick testing.
22 s underwent a clinical examination including skin prick testing.
23 of rhinitis but without positive results on skin prick testing.
24 ent report, clinical examination, and repeat skin prick testing.
25 ly to attend research clinics and consent to skin-prick testing.
26 pecific IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge p
28 with parental face-to-face interviews and/or skin prick testing, 238 (10.4%) were eligible for a DBPC
29 iagnostic testing, 47.3% was assessed with a skin prick test, 39.9% with a serum specific IgE test, a
30 gE/4 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.9
32 nates, and allergic disease was evaluated by skin prick test and clinical examination at 12 months of
34 le NSAID in history were tested first with a skin prick test and if negative challenged with the culp
36 without systemic IgE-sensitisation tested by skin prick test and serum allergen-specific IgE (sIgE) d
38 nical-demographic questionnaire, spirometry, skin prick test and specific IgE were evaluated yearly.
42 pplied for selected cases where the history, skin prick test and/or specific IgE are not definitive f
43 intervention (structured allergy history and skin prick testing and appropriate advice on allergy avo
44 gy intervention (structured allergy history, skin prick testing and appropriate allergy avoidance adv
46 s defined as one or more positive results on skin prick testing and clinically relevant symptoms of r
47 olerant (ST), or food allergic (FA) based on skin prick testing and food challenge at 12 months of ag
48 stic correlates included end point titration skin prick testing and measurement of CM-specific IgE an
49 vited for a standardized physician exam with skin prick testing and parental interview at age 2 years
52 re, taking a structured allergy history with skin prick testing and tailored advice on allergy avoida
53 tic sensitivity to 65% compared with 20% for skin prick tests and 46% ImmunoCAP using kiwi extract.
54 nonallergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergen
57 re, taking a structured allergy history with skin prick tests and giving tailored advice on allergy a
65 reened 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food c
67 ust mites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2),
68 icult, because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts
71 formed consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; bein
73 e markers measured, small wheal diameters on skin-prick testing and increases in egg-specific IgG4 an
75 thma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the
76 ondary outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 meas
78 nical-demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were
80 minth Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siber
81 e an interviewer-administered questionnaire, skin prick testing, and measurement of lung function fro
82 similar in children positive and negative on skin prick testing, and were not appreciably altered by
84 ecific immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled f
86 te use of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the tim
87 food-allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and f
91 e positive ELISA results correlated with the skin prick test areas with the whole body and the setae
93 ldren (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data colle
95 sensitization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age
105 lected at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challen
106 white specific IgE (sIgE) levels in serum or skin prick test has been shown to be a poor predictor of
109 uch as family history (50.2%) and conducting skin prick testing in non-high-risk children (43.9%).
110 hensive set of recommendations on the use of skin prick tests in allergic rhinitis-conjunctivitis and
111 to determine whether C+ assayed by means of skin prick tests influenced AR symptom severity in contr
113 -of-function samples, we performed histamine skin prick tests, investigated the contribution of STAT3
115 in and/or gastrointestinal symptoms only and skin prick test < 8 mm) are considered for home-based mi
117 post-bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity
121 ract, which was determined with the use of a skin-prick test--one consisting of participants with no
122 including detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination
123 ly relevant sensitizations are elucidated by skin prick testing or by the determination of specific I
124 nly 4 predictors of the original model: sex, skin prick test, peanut sIgE, and total IgE minus sIgE.
125 shed, using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE)
126 aluation, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 mo
127 Two hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic
129 wanted to measure geographical variation in skin prick test positivity and assess whether it was exp
130 Geographical variation in the prevalence of skin prick test positivity in Europe is unlikely to be e
131 re fitted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.3
133 for food allergy by standardized interviews, skin prick tests, prick-to-prick tests and ImmunoCAP.
136 Associations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using
138 ion between a chronic helminth infection and skin prick test reactivity even in a developed country.
139 e reactions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitizatio
141 studies demonstrated that exercise increases skin prick test reactivity to and bioavailability of the
142 Total IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all redu
143 o 2.45 +/- 0.24 (P = .001) and a decrease in skin prick test reactivity to house dust mite from 7.0 +
145 children with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-
146 SAFS (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumig
148 ible mold at age 1 year and child's positive skin prick test response to aeroallergens and molds at a
149 ID-independent anaphylaxis to LTPs, positive skin prick test response to LTPs, and serum LTP IgE.
151 PPOIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels
155 tifiable by using routinely available peanut skin prick test responses or specific IgE levels, but th
157 serum total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and I
158 ves with allergic disease) but with negative skin prick test responses to common allergens at randomi
160 th a history of ragweed allergy and positive skin prick test responses to ragweed were randomized and
162 exposure and sensitization (as determined by skin prick test responses) was analyzed in more than 100
163 ith peanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2
164 eanut but have peanut-specific IgE, positive skin prick test responses, or both represents a signific
167 random sample of participants with negative skin prick test results attended a hospital-based food c
168 asing their judgment on clinical history and skin prick test results before and after obtaining the I
170 th antivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-g
173 higher IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower
175 among M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+
176 dence of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitiz
177 atients were classified by clinical history, skin prick test/serum specific IgE (sIgE), and nasal all
182 co-factor enhanced food allergy, assessed by skin-prick tests, specific IgE and oral challenges.
183 al interview combined with blood collection, skin prick tests, spirometry with bronchodilation, and e
186 wheeze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen
189 g the basophil activation test (BAT) and the skin prick test (SPT) and measuring the levels of peanut
190 in had the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and
192 sought to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg pro
193 ized nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut
194 or urticaria upon CHX exposure and positive skin prick test (SPT) and/or positive CHX ImmunoCAP test
195 ercise, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE
196 lergic sensitization was determined based on skin prick test (SPT) of five mites, three molds, and ni
198 ng cows' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, place
199 pecific IgE against aeroallergens (sIgE) and skin prick test (SPT) reactivity for the most common loc
203 action to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/o
204 lergy, milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/o
205 nd 4 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) le
206 ping peanut allergy, and the implications of skin prick test (SPT) screening before peanut introducti
208 protein levels in household dust and peanut skin prick test (SPT) sensitization and likely allergy.
209 nfantile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong a
210 six dog allergens (Can f 1-6) in commercial skin prick test (SPT) solutions and to determine individ
212 of 5276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with
215 eczema, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group I
217 fe (EQ-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smel
224 tern blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to d
225 Commercial allergen extracts for allergy skin prick testing (SPT) are widely used for diagnosing
229 orm beta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra
231 ng hay fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific
239 y against common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE)
240 CFC; <=500 mg of peanut protein), a positive skin-prick test (SPT) result (>=5 mm wheal diameter abov
241 ed risk factors for atopy (allergen-specific skin prick test [SPT] reactivity and IgE [asIgE] sensiti
242 measures of allergic sensitisation (such as skin-prick test [SPT] and serum specific IgE [sIgE]) whe
244 ich they answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and pr
245 ants completed a questionnaire and underwent skin prick tests (SPTs) to egg, peanut, cow's milk, fish
246 were invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2
247 ous reactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut sol
248 erial 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial mil
250 logical work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activati
251 e following outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE
252 7 with mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-P
253 on who initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60
254 e in peanut-specific basophil activation and skin prick test titration compared with nonresponders.
255 iral culture for varicella-zoster virus, and skin prick test to common food and animal allergens were
256 icipants aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were expose
257 r anaphylaxis related to NSAID, (3) positive skin prick test to foods and/or specific IgE to food all
258 easonal allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate
259 ty reaction after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind
260 ample of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's mil
262 ohort were also more likely to have positive skin prick tests to cabbage, lettuce and mustard and sen
266 ciation between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensit
270 ths for scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1
280 easurements of eczema, asthma, rhinitis, and skin prick tests were available for all follow-ups.
283 Food-specific serum IgE measurements and skin prick tests were performed before initiating the di
284 n, IgE inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selec
290 E 33.3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experience
293 grass pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Be
294 subjected to topical cowhage provocation and skin prick testing with histamine and assessed for diffe
296 meat, we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha
299 llenges with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measure