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1 ant role in Fc(gamma)R-mediated inflammatory skin reaction.
2 o bind HLA-DR did not elicit an inflammatory skin reaction.
3 lls contribute to the sustained inflammatory skin reaction.
4 out the trial and contemporaneously with the skin reaction.
5 r multikinase inhibitor-associated hand-foot skin reaction.
6 s from a patient who developed an urticarial skin reaction.
7 covered by EORTC systems including radiation skin reaction.
8 3 toxicities were hypertension and hand-foot-skin reaction.
9 stant to IgE-mediated cutaneous inflammatory skin reaction.
10 s and other wound complications, and adverse skin reactions.
11 ulatory properties predicted for SAg-induced skin reactions.
12 n of drug and radiation did not modify acute skin reactions.
13 ediate (IH) or delayed (DH) hypersensitivity skin reactions.
14 history of athlete's foot but also caused IH skin reactions.
15 sitivity, photocarcinogenesis and eczematous skin reactions.
16 ower than what is required for deterministic skin reactions.
17 at 18% of IAVI G002 participants experienced skin reactions.
18 mon adverse event being transient urticarial skin reactions.
19 ; adverse effects were occasional mild local skin reactions.
20 ective, yet with limitations owing to strong skin reactions.
21 ed to sodium lauryl sulfate-induced irritant skin reactions.
22 in treatment were prospectively examined for skin reactions.
23 c anaphylaxis and to participate in allergic skin reactions.
24 y early event in the development of allergic skin reactions.
25 cities were restricted to grade 1 to 2 local skin reactions.
26 patients (95.2%) experienced transient local skin reactions: 1 (4.8%) herpes zoster, 3 (14.3%) transa
27 well tolerated in both groups, except for 6 skin reactions: 1 in the CMD group (1.0%) and 5 in the c
28 atment-related toxicities included hand-foot skin reaction (10%), hypertension (4%), fatigue (2%), an
30 grades) consisted of local DC injection site skin reactions (100%), transient post-DC infusion chills
32 n grade 3 or 4 adverse events were hand-foot skin reaction (154 [28%] of 559 patients in the sorafeni
33 s were common, most frequently hand and foot skin reaction (16 patients, 33%), diarrhoea (five patien
36 ere hypertension (31 of 132, 23%), hand-foot skin reaction (26 of 132, 20%), and diarrhoea (seven of
38 oups, previous DHR was mostly represented by skin reactions (46.4% in CMD and 82.9% in the control gr
39 tients [5%] in the placebo group), hand-foot skin reaction (47 patients [13%] vs one [1%]), fatigue (
42 (any grade) in the combination arm included skin reactions (67.1%) and peripheral neuropathy (60.5%)
43 events in the sorafenib group were hand-foot skin reaction (76.3%), diarrhoea (68.6%), alopecia (67.1
44 er in the sorafenib group included hand-foot skin reaction (8.6% v 0.3%), fatigue (7.3% v 3.6%), rash
45 higher related to regorafenib were hand-foot skin reaction (83 patients, 17%), fatigue (48, 10%), dia
47 ipsilateral forearm resulting in a positive skin reaction, a clear increase in IFN-gamma ELISPOT cou
48 trointestinal symptoms, hypersensitivity and skin reactions, acute kidney injury, and secondary infec
49 lorectal cancer who had developed no or mild skin reactions after 21 days of treatment at the standar
53 ves: To evaluate the association between BCG skin reaction and mycobacteria-specific immune responses
54 atients in stratum two had grade 3 hand-foot skin reaction and/or rash as dose-limiting toxicities (D
56 events, all in the intervention group-mostly skin reactions and exercise-related pain exacerbations.
57 risk for unusually intense radiation-induced skin reactions and late tissue damage from high-dose int
58 th more patients developing chills and local skin reactions and more patients stopping PIXY321 due to
59 owever, p38alpha inhibitors produced adverse skin reactions and other toxic effects that often outwei
62 vents were skin rash/desquamation, hand-foot skin reaction, and fatigue; 9% of patients discontinued
63 f actinic keratoses (primary outcome), local skin reactions, and immune activation parameters were as
64 ccus vaccines, delayed-type hypersensitivity skin reactions, and mucosal defense (secretory immunoglo
66 ded bleeding episodes, thrombocytopenia, and skin reactions, and were assessed in all randomly assign
69 recommended in adults with only generalized skin reactions as it results in significant improvements
70 Individual items on the Radiation-Induced Skin Reaction Assessment Scale also favored the MF for b
71 care provider score using Radiation-Induced Skin Reaction Assessment Scale, the MF arm had significa
73 ntinib were evaluated for the development of skin reactions at each treatment visit from October 2012
74 emas, as well as from localized inflammatory skin reactions at pegylated IFN-alpha injection sites, w
77 tamine-induced sensations, dysesthesias, and skin reactions but not the sensations and dysesthesias e
80 ater incidence of nausea, sleep disturbance, skin reactions, constipation, and depression, with only
84 mmon adverse effects, most notably hand-foot skin reaction, diarrhea, and hypertension, compared with
86 the incidence and causes of heparin-induced skin reactions during pregnancy in a prospective cohort
87 ith LamOVA induced significantly lower local skin reactions during therapy compared to unconjugated O
88 [18%] in the standard-dose group), hand-foot skin reaction (eight [15%] patients vs ten [16%] patient
91 of novel D7 reactions as well as increasing skin reactions from D3 to D7 was analysed separately.
92 (n = 44) was associated with an increase in skin reactions >/= grade 2 compared with standard (n = 4
93 city, hyperkeratotic folliculitis, hand-foot skin reaction, hair changes, verrucous papillomas, kerat
94 4%), hypertension (18% v 12%), and hand-foot skin reaction/hand- foot syndrome (HFSR/HFS; 90% v 66%);
95 h a cutaneous adverse event called hand-foot skin reaction (HFSR), in which sites of pressure or fric
96 Nine patients (1%) had concomitant severe skin reactions; implicated agents were lamotrigine, azit
99 irizine) on type I hypersensitivity itch and skin reaction in AD using a patient and examiner-blinded
102 high-dose group; only one (an acute allergic skin reaction in the low-dose group) was assessed to be
104 red in 1.98% (95% CI, 1.50%-2.57%), allergic skin reactions in 1.80% (95% CI, 1.34%-2.37%), heparin-i
108 portedly a difference in the diameter of the skin reaction induced by different types of skin prick t
109 re associated with a reduction in late-phase skin reactions induced by whole allergens and bronchial
110 rently available with regard to fluoroscopic skin reactions is based on a table published in 1994.
111 luded diarrhea, nausea, vomiting, mucositis, skin reactions, liver test abnormalities, and infusion-r
112 r clinical phenotypes of NSAID-induced acute skin reactions manifesting with angioedema, urticaria, o
113 nificantly higher maximum physician-assessed skin reaction (moist desquamation, 28.5% vs 6.6%, P < .0
115 atment-related adverse events were hand-foot skin reaction (n = 10); neutropenia (n = 7); fatigue (n
116 l interest adverse events, treatment-related skin reactions occurred in 47.3% of patients receiving e
117 patients (16.9%); drug hypersensitivity and skin reactions occurred in the experimental arm in 3 pat
119 ttoos and the frequent occurrence of adverse skin reactions, particularly from colored inks, highligh
121 among arms were found in incidence of acute skin reaction, pneumonitis, dyspnea, cough, dysphagia, o
122 r (VEGF) are features of late-phase allergic skin reactions, previously proposed as a model of CSU.
124 ons, constipation, and depression, with only skin reactions reaching statistical significance (14.4%
126 e patients developed hematuria and one had a skin reaction resembling grade 3 hand-foot syndrome.
128 and resolved within a mean time of 32 d; G3 skin reactions (severe skin changes without pain) were p
130 were PPIX photobleaching and clinical local skin reactions, supported by noninvasive reflectance mea
131 ed serious adverse events included hand-foot skin reaction (ten [2%]), abnormal hepatic function (fou
132 safety profile with mostly mild-to-moderate skin reactions that were observed to decrease over time.
133 fined protein associated with both DH and IH skin reactions; these reactions are characterized by dis
134 e reaction was headache; aseptic meningitis, skin reactions, thromboembolic events, and renal tubular
135 trongly associated with early-onset BCC were skin reaction to first summer sun for 1 hour (severe sun
137 25 pulmonary TB patients who had a positive skin reaction to mumps and/or candida antigens showed pe
138 ble-adjusted RR = 0.98, 95% CI: 0.92, 1.05), skin reaction to prolonged sun exposure (for painful bur
139 using hair color, skin tanning ability, and skin reaction to prolonged sun exposure as surrogate mea
143 viduals that can range in severity from mild skin reactions to severe and life-threatening anaphylaxi
144 mal necrolysis (TEN) is a fatal drug-induced skin reaction triggered by common medications and is an
148 ecorded, and the incidence of CHG-associated skin reactions was 1.2 per 1000 days (95% CI 0.60-2.02).
150 patients with 1 or more TEAEs (largely local skin reactions) was similar across all groups in year 1:
156 ) plus irinotecan, patients with </= grade 1 skin reactions were randomly assigned to standard-dose (
161 tients, 62.5% developed transient urticarial skin reactions, with onset during the first infusion, di
162 l wheal formation and complete resolution of skin reactions within 7days, and generated no systemic a