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1 Four children died, of whom only one had a skull fracture.
2 racranial injury can occur in the absence of skull fracture.
3 was done on 156 children, of whom 107 had a skull fracture.
4 acranial lesions, even when they have linear skull fractures.
5 whom had severe head injuries with multiple skull fractures.
6 njuries concerning for maltreatment, such as skull fractures.
8 iocervical junction, and tend also to have a skull fracture, a thin film of subdural haemorrhage, but
9 geminal nerve anesthesia-one following basal skull fracture and another following large posterior fos
10 tabase of all patients with TBI (findings of skull fracture and/or intracranial hemorrhage on an init
11 uding (a) healed rib fractures, (b) hairline skull fractures and a compression deformity of the thora
12 s, non-severe injury mechanism, no palpable skull fracture, and acting normally according to the par
13 Lower GCS score, midline shift, depressed skull fracture, and epidural hematoma are key risk facto
14 tracranial hemorrhage (other than epidural), skull fracture, and higher head/neck injury severity.
15 severe injury mechanism, no signs of basilar skull fracture, and no severe headache) had a negative p
17 DVST was seen in only those patients with skull fractures extending to a dural sinus or jugular bu
18 bulbs in 57 (40.7%) of the 140 patients with skull fractures extending to a dural sinus or jugular bu
21 anatomically exquisite portrayals of growing skull fractures for neurosurgical planning, but its rout
23 mon procedures were elevation of a depressed skull fracture in the low HDI tier (69 [45%]), evacuatio
24 the acutely ill patient or identification of skull fractures in the assessment of a patient with head
26 of consciousness, post-traumatic amnesia, or skull fracture) in Olmsted County, Minnesota, during the
27 was diagnosed (eg, cervical spine fracture, skull fracture, intracranial bleeding, liver and spleen
28 venograms were reviewed for the presence of skull fractures, intracranial hemorrhage, and traumatic
29 ospective database on all patients with TBI (skull fracture/intracranial hemorrhage on head computed
30 physical examination findings suggestive of skull fracture (likelihood ratio [LR], 16; 95% CI, 3.1-5
31 were brain contusion with subdural hematoma, skull fracture, loss of consciousness or amnesia for mor
32 ldren who were admitted to hospital or had a skull fracture (n = 883) were included in the study.
36 ed tomography (CT) was done in children with skull fractures on radiography and in those without frac
37 that mTBI model did not produce brain edema, skull fracture or sensorimotor coordination dysfunctions
38 shift (OR, 6.8; 95% CI, 3.4-13.8), depressed skull fracture (OR, 6.5; 95% CI, 3.7-11.4), and epidural
39 ury (OR, 4.16; 95% CI, 2.13-8.10; P < .001), skull fractures (OR, 3.20; 95% CI, 1.76-5.82; P < .001),
40 onsciousness for 30 minutes to 24 hours or a skull fracture), or severe (loss of consciousness or amn
41 , ejected from vehicle, fall >1 m, suspected skull fracture, or GCS score <15 at 2 hours) had an LR o
42 for atraumatic brain abnormalities, isolated skull fractures, or chronic intracranial hemorrhage.
43 er age, traumatic brain injury severity, and skull fractures predict anterior pituitary disorders, wh
44 traumatic brain injury; seven studies), and skull fractures (risk ratio, 1.73; 95% CI, 1.03-2.91; si
46 2 through 1995, 87 consecutive children with skull fractures visible at plain radiography were referr