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1 jective sleep quality, sleep efficiency, and sleep latency.
2 mplicated CG44153, Piezo, Proc-R and Rbp6 in sleep latency.
3 genetic factors that influence variation in sleep latency.
4 in 248 genes contributing to variability in sleep latency.
5 culate sleep quantity, sleep efficiency, and sleep latency.
6 eep across nights and a shorter baseline REM sleep latency.
7 receptor, Rdl(A302S), specifically decreases sleep latency.
8 netics of GABA(A) receptor signaling dictate sleep latency.
9 leep apnea was demonstrated only for reduced sleep latency.
10 tonin synthesis, pupillary light reflex, and sleep latency.
11 ion was seen between Epworth scores and mean sleep latency.
12 uration and intensity of NREMS and prolonged sleep latency.
13 e conduct a genome-wide association study of sleep latency.
14 and social media were associated with longer sleep latency.
15 egions, increased sleep times, and shortened sleep latencies.
16 normal sleep latency (>10 minutes), moderate sleep latency (5 to 10 minutes), or severe sleep latency
17 mokers, current smokers had a longer initial sleep latency (5.4 minutes, 95% confidence interval (CI)
19 ell an individual is sleeping) and increased sleep latency (amount of time to fall asleep), suggestin
22 y endpoints (Maintenance of Wakefulness Test sleep latency and Epworth Sleepiness Scale score) were m
24 In the phase II study, tasimelteon reduced sleep latency and increased sleep efficiency compared wi
26 with placebo, modafinil decreased nighttime sleep latency and increased slow-wave sleep time in coca
28 s disrupted by methamphetamine by decreasing sleep latency and increasing sleep efficiency compared w
29 flies are resistant to the effects of CBZ on sleep latency and that mutant RDL(A302S) channels are re
30 zed by decreased total sleep time, increased sleep latency, and decreased sleep efficiency, without d
31 ariables-male sex, sleepiness, nocturnal REM sleep latency, and extent of oxygen desaturation-could r
32 period significantly reduced REM, shortened sleep latency, and increased EEG delta power in rats.
33 gat(33-1)) increased sleep amount, decreased sleep latency, and increased sleep consolidation at nigh
34 duction in plasma tryptophan concentrations, sleep latency, and REM latency, as well as increased REM
35 ates that dual use is associated with longer sleep latency, and suggests that the shared component of
36 thanol microinjections significantly reduced sleep latency, and tended (P<0.06) to increase total sle
37 imination, delayed gastric emptying, altered sleep latency, anxiety-like behavior, and age-dependent
38 piness Scale), objective sleepiness (reduced sleep latency as determined by the Multiple Sleep Latenc
39 groups of patients who were defined by mean sleep latency as having normal sleep latency (>10 minute
41 afinil was associated with increased daytime sleep latency, as measured by the Multiple Sleep Latency
42 l biases were observed for total sleep time, sleep latency, awakening after falling asleep, sleep eff
44 ystemic zolpidem administration also reduced sleep latency, but less so than for histamine neurons.
45 ld-derived population of flies, we calculate sleep latency, confirming significant, heritable genetic
46 phically assessed (upper or lower quartiles) sleep latency, continuity, and duration (RRs = 2.2-4.7;
48 stress-induced insomnia in humans: increased sleep latency, decreased non-REM (nREM) and REM sleep, i
50 These experiments uncouple the regulation of sleep latency from that of sleep duration and suggest th
51 fined by mean sleep latency as having normal sleep latency (>10 minutes), moderate sleep latency (5 t
52 slow-wave sleep time, total sleep time, and sleep latency in cocaine-dependent and healthy participa
58 ore and after sleep onset, such as prolonged sleep latency, loss of stage 3-4 sleep, reduced rapid ey
61 e sleep latency (5 to 10 minutes), or severe sleep latency (<5 minutes) (analysis of variance, P = 0.
63 cy, sleep onset and rapid eye movement [REM] sleep latencies, non-REM and REM sleep stages, and wakef
64 hted effect sizes for subjective measures of sleep latency, number of awakenings, wake time after sle
66 ), a 5-min decrease in the MSLT-derived mean sleep latency (OR = 1.9, 95% CI = 1.3 to 2.8), a 90-min
67 s with Parkinson disease exhibited increased sleep latency (P = .04), reduced sleep efficiency (P = .
69 ion was seen between Epworth scores and mean sleep latency (Pearson correlation, -0.17 [95% CI, -0.35
70 takes the view on insomnia, i.e., prolonged sleep latency, problems to maintain sleep, and early mor
71 tional tobacco was associated with increased sleep latency relative to non-smokers/non-vapers by mult
72 In many patients, a short rapid eye movement sleep latency (REML) during the NPSG is also observed bu
73 n ICU patients is characterized by prolonged sleep latencies, sleep fragmentation, decreased sleep ef
74 bal sleep quality, subjective sleep quality, sleep latency, sleep duration, sleep efficacy, sleep med
75 In the phase III study, tasimelteon improved sleep latency, sleep efficiency, and wake after sleep on
76 ality, sleep architecture (total sleep time, sleep latency, sleep efficiency, nonrapid eye movement,
77 rsomnolence was quantified with the multiple sleep latency test (MSLT) and survival analysis was used
78 deficiency, is diagnosed using the Multiple Sleep Latency Test (MSLT) following nocturnal polysomnog
79 ent (REM) periods (2omSOREMPs) on a Multiple Sleep Latency Test (MSLT) raise the possibility of narco
81 ts with cataplexy but with a normal multiple sleep latency test (MSLT) result, or if MSLT is not inte
83 tive sleepiness was assessed by the Multiple Sleep Latency Test in all subjects, problem sleepiness b
84 Sleepiness Scale and the objective multiple sleep latency test may evaluate different, complementary
86 sleep latency as determined by the Multiple Sleep Latency Test), and neurobehavioral functioning (la
87 rements included polysomnography, a multiple sleep latency test, an oral glucose tolerance test, dete
88 e sleep latency, as measured by the Multiple Sleep Latency Test, and a nearly significant decrease in
89 tory tests such as polysomnography, multiple sleep latency test, and actigraphy, along with referral
91 ormed with the use of the nighttime Multiple Sleep Latency Test, the Clinical Global Impression of Ch
98 me, 2 nights of polysomnography and multiple sleep latency tests in the laboratory, and body composit
99 age change group showed significantly longer sleep latencies than the gentle handling group, indicati
101 ent from baseline in mean (+/-SEM) nighttime sleep latency (the interval between the time a person at
103 oring and sleep logs to measure time in bed, sleep latency (time required to fall asleep), sleep dura
105 sleep, and with increases in latency to REM sleep, latency to persistent sleep, and percent Stage 3/
109 5 (standard deviation (SD), 1.2) hours, mean sleep latency was 21.9 (SD, 29.0) minutes, mean sleep du
114 e the onset of melatonin secretion and short sleep latencies were observed close to the temperature n
115 eye movement (REM) sleep and shortened NREM sleep latency with a simultaneous decrease in core tempe