ライフサイエンスコーパス: sleep-disordered breathing

1 le lung disease, pulmonary hypertension, and sleep disordered breathing.

2 CIH-induced neuropathology in patients with sleep disordered breathing.

3 al importance to the patients suffering from sleep-disordered breathing.

4 sitive pressure ventilation in children with sleep-disordered breathing.

5 s is higher among subjects with than without sleep-disordered breathing.

6 eful second-line treatment for children with sleep-disordered breathing.

7 n the predominant abnormality leading to the sleep-disordered breathing.

8 lt patients evaluated by polysomnography for sleep-disordered breathing.

9 c respiratory cycles in adults evaluated for sleep-disordered breathing.

10 al impairments observed in a rodent model of sleep-disordered breathing.

11 uld have a role in preventing or alleviating sleep-disordered breathing.

12 desaturation (DeltaSa(O(2))) associated with sleep-disordered breathing.

13 ic obstructive pulmonary disease, asthma and sleep-disordered breathing.

14 diovascular risk factor levels in those with sleep-disordered breathing.

15 ace, we studied the effect of spaceflight on sleep-disordered breathing.

16 uality during spaceflight is not degraded by sleep-disordered breathing.

17 nography is invaluable for the evaluation of sleep-disordered breathing.

18 y event index was 14 (IQR = 6-25); 62.8% had sleep-disordered breathing.

19 specific sleep-related parameters, including sleep-disordered breathing.

20 is hypothesis in a group of subjects without sleep-disordered breathing.

21 resence or absence of insulin resistance and sleep-disordered breathing.

22 who have clinically significant REM-specific sleep-disordered breathing.

23 s that insulin resistance is associated with sleep-disordered breathing.

24 oscopy at the time of adenotonsillectomy for sleep-disordered breathing.

25 cy, suggesting abnormal cardiac responses to sleep-disordered breathing.

26 may stand to benefit from AT for obstructive sleep-disordered breathing.

27 lay an important role in the pathogenesis of sleep-disordered breathing.

28 study found an association between PTSD and sleep-disordered breathing.

29 otype and cognitive decline in patients with sleep-disordered breathing.

30 me individuals may partially protect against sleep-disordered breathing.

31 When abnormal, these interactions lead to sleep-disordered breathing.

32 effects of oral antihistamines on asthma and sleep-disordered breathing.

33 omnography studies to assess the presence of sleep-disordered breathing.

34 malities in children with SCD are related to sleep-disordered breathing.

35 rdered breathing compared with those without sleep-disordered breathing: 4.8 versus 0.9% (p=0.003) fo

36 Sleep-disordered breathing accounted for 9 to 10% of eth

37 entral apnea, Cheyne-Stokes respiration, and sleep-disordered breathing-age interaction terms were si

38 ongest in older participants in whom overall sleep-disordered breathing also increased atrial fibrill

39 implicated, including apnoea of prematurity, sleep disordered breathing and congestive heart failure.

40 urbations during sleep (e.g. those caused by sleep disordered breathing and periodic leg movements) m

41 ined the cross-sectional association between sleep- disordered breathing and self-reported CVD in 6,4

42 Compared with those without sleep-disordered breathing and adjusting for age, sex, b

43 events, we aimed to assess the prevalence of sleep-disordered breathing and associated clinical featu

44 Although research supports a sleep-disordered breathing and atrial fibrillation assoc

45 Associations between sleep-disordered breathing and cardiovascular disease (C

46 ights the complex interrelationships between sleep-disordered breathing and cardiovascular disease, p

47 Obese females are less predisposed to sleep-disordered breathing and have higher serum leptin

48 e directionality of the relationship between sleep-disordered breathing and heart failure is controve

49 the association between objectively measured sleep-disordered breathing and hypertension (defined as

50 born preterm exhibit increased incidence of sleep-disordered breathing and hypertension, suggesting

51 ontinuous positive airway pressure (CPAP) on sleep-disordered breathing and its consequences in heart

52 els were used to assess associations between sleep-disordered breathing and outcomes, adjusted for so

53 erity, and perioperative risk of obstructive sleep-disordered breathing and persistent sleep apnea af

54 more in-depth discussion of indications for sleep-disordered breathing and recurrent throat infectio

55 gnificant relation was also observed between sleep-disordered breathing and ventricular ectopic beats

56 ormed in young children, 28.9% in those with sleep-disordered breathing, and 2.8% in those with compl

57 Obesity is a strong causal factor for sleep-disordered breathing, and because of the ongoing o

58 , both overall and in relation to age group, sleep-disordered breathing, and complex chronic conditio

59 trategies for management of hypoventilation, sleep-disordered breathing, and cough insufficiency are

60 d glycation endpoints, autonomic neuropathy, sleep-disordered breathing, and genetic susceptibility t

61 icity and alcohol's contribution to obesity, sleep-disordered breathing, and hypertension.

62 re, specific exercise, opioids, treatment of sleep-disordered breathing, and interventions to address

63 sure of physiological stress associated with sleep-disordered breathing, and this measure predicts in

64 The probability of moderate-to-severe sleep-disordered breathing (apnea-hypopnea index >/=15%)

65 The prevalence of sleep-disordered breathing (apnea-hypopnea index of 15 o

66 isk factors for excessive sleepiness: severe sleep-disordered breathing (apnea-hypopnea index, >30 ep

67 t prevalence estimates of moderate to severe sleep-disordered breathing (apnea-hypopnea index, measur

68 Clinical signs of insomnia and sleep-disordered breathing are common in mid-to-late pre

69 overnight by 18-channel polysomnography for sleep-disordered breathing, as defined by the apnea-hypo

70 The primary outcome was prevalence of sleep-disordered breathing, assessed by the apnoea-hypop

71 We found a dose-response association between sleep-disordered breathing at base line and the presence

72 ible predisposition of the pregnant woman to sleep-disordered breathing because of these changes, and

73 We analyzed data on sleep-disordered breathing, blood pressure, habitus, and

74 ent in HFpEF patients, but renal disease and sleep-disordered breathing burdens are similar.

75 opause is considered to be a risk factor for sleep-disordered breathing, but this hypothesis has not

76 Obstructive sleep disordered breathing can cause death and significa

77 Central sleep-disordered breathing can occur with associated car

78 on, a standard-of-care management option for sleep-disordered breathing, can itself trigger specific

79 Sleep disordered breathing causes repetitive episodes of

80 s, dyslipidemia, obstructive sleep apnea and sleep-disordered breathing, certain cancers, and major c

81 Sleep-disordered breathing (characterized by recurrent a

82 drigeminy) were more common in subjects with sleep-disordered breathing compared with those without s

83 Among older women, those with sleep-disordered breathing compared with those without s

84 bstantial impact of this increasingly common sleep-disordered breathing condition.

85 Sleep-disordered breathing contributes to cardiac chambe

86 The prevalence of sleep-disordered breathing, depending on the apnea-hypop

87 articipants who had objective assessments of sleep-disordered breathing during pregnancy were asked t

88 Prospective data suggest that sleep-disordered breathing enhances risk for incident an

89 The frequency of sleep-disordered breathing episodes remained high at 0 a

90 ds of complex arrhythmias than those without sleep-disordered breathing even after adjustment for pot

91 dered breathing, can itself trigger specific sleep-disordered breathing events including air leaks, p

92 airway resistance can increase the risk for sleep-disordered breathing events.

93 relationship between insulin resistance and sleep-disordered breathing for potential confounding var

94 ry artery disease, congestive heart failure, sleep-disordered breathing, gastro-oesophageal reflux di

95 The prevalence of moderate-to-severe sleep-disordered breathing (>/=15 events per h) was 23.4

96 rdered breathing compared with those without sleep-disordered breathing had an increased risk of deve

97 ent coronary heart disease, individuals with sleep-disordered breathing had four times the odds of at

98 Sleep-disordered breathing has also been linked to these

99 thway whereby incident CVD causes or worsens sleep-disordered breathing has not been studied.

100 The prevalence of sleep-disordered breathing has not been well studied in

101 Common polysomnographic measures of sleep-disordered breathing have shown a disappointing ab

102 Individuals with severe sleep-disordered breathing have two- to fourfold higher

103 ly affects the respiratory system leading to sleep-disordered breathing, hypoventilation, and weaknes

104 have also highlighted the manifestations of sleep disordered breathing in children with sickle cell

105 breathing and HVR, which may protect against sleep disordered breathing in obesity.

106 ment V(E) and HVR, which may protect against sleep disordered breathing in obesity.

107 compensation suggesting that exacerbation of sleep disordered breathing in REM (compared to NREM) sle

108 between the use of replacement hormones and sleep-disordered breathing in a sample of 2,852 noninsti

109 ves: Here, we took advantage of our model of sleep-disordered breathing in diet-induced obese mice, r

110 ld underlie the etiology of certain forms of sleep-disordered breathing in humans.SIGNIFICANCE STATEM

111 lic consequences and community prevalence of sleep-disordered breathing in mildly obese, but otherwis

112 Sleep-disordered breathing in neuromuscular diseases is

113 e in addressing pitfalls in the diagnosis of sleep-disordered breathing in neuromuscular diseases, id

114 ptin will bypass leptin resistance and treat sleep-disordered breathing in obesity.

115 in the carotid bodies could be a therapy for sleep-disordered breathing in obesity.

116 changes, and results of published studies of sleep-disordered breathing in pregnancy are discussed.

117 We estimated the prevalence of sleep-disordered breathing in the United States for the

118 for sleep duration, sleep fragmentation, and sleep-disordered breathing) in the development of cognit

119 m the brain, and hypoxemia characteristic of sleep-disordered breathing increases Abeta production.

120 iated with an increased likelihood of having sleep-disordered breathing, independent of known confoun

121 ptin resistance and significantly attenuated sleep-disordered breathing independently of body weight.

122 In addition, sleep disordered breathing, inflammation, left ventricul

123 Sleep disordered breathing is common in people with, or

124 Sleep-disordered breathing is a common disorder with a r

125 Sleep-disordered breathing is a prevalent condition asso

126 Thus, sleep-disordered breathing is a prevalent condition in m

127 Sleep-disordered breathing is also more common in patien

128 Sleep-disordered breathing is associated with an increas

129 A significant portion of sleep-disordered breathing is associated with ApoE epsil

130 Sleep-disordered breathing is associated with major morb

131 Sleep-disordered breathing is associated with worse func

132 Sleep-disordered breathing is correlated with RMS and th

133 Sleep-disordered breathing is highly prevalent in elderl

134 These results suggest that sleep-disordered breathing is highly prevalent, with imp

135 The findings suggest that sleep-disordered breathing is likely to be a risk factor

136 Nevertheless, sleep-disordered breathing is often unrecognized in chil

137 Sleep-disordered breathing is prevalent in the general p

138 If sleep-disordered breathing is shown in future follow-up

139 intermittent hypoxia (IH), such as occurs in sleep-disordered breathing, is associated with neurobeha

140 control instability contributes to apnea and sleep-disordered breathing later in childhood.

141 reathing, the current evidence suggests that sleep-disordered breathing may function as a risk factor

142 sis was limited to those 27 patients who had sleep-disordered breathing (more than 5 apneas or hypopn

143 Mild sleep-disordered breathing (mSDB) in children is associa

144 Sleep-disordered breathing occurs after surgery even in

145 Sleep-disordered breathing occurs in Alzheimer disease p

146 ompatible with modest to moderate effects of sleep-disordered breathing on heterogeneous manifestatio

147 logical mechanisms underlying the effects of sleep-disordered breathing on the brain.

148 lectively studied in populations at risk for sleep-disordered breathing or cardiovascular diseases.

149 ng young children (aged <3 y) and those with sleep-disordered breathing or complex chronic conditions

150 None had symptoms of sleep-disordered breathing or conditions that impacted o

151 lass II to IV heart failure and suspicion of sleep-disordered breathing or excessive daytime sleepine

152 ent studies show either absence of change in sleep-disordered breathing or improved sleep cardiovascu

153 relationship between symptoms of obstructive sleep disordered breathing (oSDB) and childhood behavior

154 tonsillectomy to treat pediatric obstructive sleep-disordered breathing (OSDB) often falls on individ

155 ssure (BP) in children with mild obstructive sleep-disordered breathing (oSDB) remains unclear.

156 , incident CVD was associated with worsening sleep-disordered breathing over 5 years.

157 Sleep-disordered breathing, particularly the obstructive

158 gnition; however, it remains unclear whether sleep-disordered breathing precedes cognitive impairment

159 tion association, prospective data examining sleep-disordered breathing predicting incident atrial fi

160 oing obesity epidemic, previous estimates of sleep-disordered breathing prevalence require updating.

161 The high prevalence of sleep-disordered breathing recorded in our population-ba

162 owever, its indication for all patients with sleep-disordered breathing, regardless of daytime sympto

163 , and body mass index: (1) 228 subjects with sleep-disordered breathing (respiratory disturbance inde

164 ce index>or=30) and (2) 338 subjects without sleep-disordered breathing (respiratory disturbance inde

165 Sleep-disordered breathing (respiratory event index >=10

166 Although most children with sleep-disordered breathing respond to surgical treatment

167 We propose that sleep-disordered breathing results from loss of preBotC

168 Sleep disordered breathing (SDB) is a complex, sex speci

169 Sleep disordered breathing (SDB) is a well-established c

170 tent hypoxia during sleep (IH), as occurs in sleep disordered breathing (SDB), induces spatial learni

171 However, its impact on children with mild sleep-disordered breathing (SDB) (i.e., habitual snoring

172 Sleep-disordered breathing (SDB) affects over 50% of obe

173 ing evidence suggests an association between sleep-disordered breathing (SDB) and cognitive decline i

174 of the insertion/deletion polymorphism with sleep-disordered breathing (SDB) and hypertension in 1,1

175 investigate the prospective associations of sleep-disordered breathing (SDB) and insomnia with incid

176 tional association has been reported between sleep-disordered breathing (SDB) and insulin resistance,

177 Increasing recognition of sleep-disordered breathing (SDB) and its morbidity have

178 Sleep-disordered breathing (SDB) and sleep apnea have be

179 The influence of sleep-disordered breathing (SDB) and sleep-related hypox

180 Sleep-disordered breathing (SDB) during pregnancy has be

181 Sleep-disordered breathing (SDB) has been associated wit

182 Sleep-disordered breathing (SDB) has been noted commonly

183 gaps exist regarding health implications of sleep-disordered breathing (SDB) identified in pregnancy

184 Trazodone with L-tryptophan can treat sleep-disordered breathing (SDB) in an animal model of O

185 This study examined risk factors for sleep-disordered breathing (SDB) in children and adolesc

186 Sleep-disordered breathing (SDB) in children is associat

187 Obstructive sleep-disordered breathing (SDB) in children is characte

188 haracterization of anatomic risk factors for sleep-disordered breathing (SDB) in children.

189 Sleep-disordered breathing (SDB) is a common disorder in

190 Sleep-disordered breathing (SDB) is a prevalent disorder

191 Whether sleep-disordered breathing (SDB) is a risk factor for le

192 There is increasing evidence that sleep-disordered breathing (SDB) is an independent risk

193 Sleep-disordered breathing (SDB) is associated with dayt

194 Clinic-based studies suggest that sleep-disordered breathing (SDB) is associated with gluc

195 Sleep-disordered breathing (SDB) is associated with hype

196 Rationale: Sleep-disordered breathing (SDB) is associated with incr

197 Sleep-disordered breathing (SDB) is associated with path

198 Sleep-disordered breathing (SDB) is both prevalent and a

199 Prior studies have found that sleep-disordered breathing (SDB) is common among those w

200 Sleep-disordered breathing (SDB) is common in patients w

201 ng of intrinsic information in children with sleep-disordered breathing (SDB) is different from healt

202 Sleep-disordered breathing (SDB) is frequently associate

203 Yet, sleep-disordered breathing (SDB) is highly prevalent in

204 xcessive daytime sleepiness in patients with sleep-disordered breathing (SDB) is not well defined.

205 Prevalence of sleep-disordered breathing (SDB) is reported to increase

206 Most polysomnograms are performed because sleep-disordered breathing (SDB) is suspected, but perio

207 We assessed the extent to which sleep-disordered breathing (SDB) may explain association

208 The effect of sleep-disordered breathing (SDB) on right heart structur

209 ol abnormalities in predisposing to familial sleep-disordered breathing (SDB) was assessed in 31 subj

210 Genetic determinants of sleep-disordered breathing (SDB), a common set of disord

211 ss body weight is positively associated with sleep-disordered breathing (SDB), a prevalent condition

212 iciency), sleep duration, sleep consistency, sleep-disordered breathing (SDB), and sleep architecture

213 sibility (UAC) is increased in children with sleep-disordered breathing (SDB), but during wakefulness

214 Sleep-disordered breathing (SDB), characterized by speci

215 Sleep-disordered breathing (SDB), including obstructive

216 Intermittent hypoxia, a consequence of sleep-disordered breathing (SDB), may contribute to an i

217 Primary sleep disorders such as sleep-disordered breathing (SDB), sleep-related movement

218 In this case-control family study of sleep-disordered breathing (SDB), we describe the distri

219 however, the relation of sleepiness to mild sleep-disordered breathing (SDB), which affects as much

220 Snoring is central to sleep-disordered breathing (SDB), which arises from noct

221 Obstructive sleep-disordered breathing (SDB), which includes primary

222 Children with mild sleep-disordered breathing (SDB), who may not be recomme

223 children are at greatest risk for developing sleep-disordered breathing (SDB)-associated behavioral m

224 ial triggers for arrhythmia in patients with sleep-disordered breathing (SDB).

225 ronic sleepwalkers frequently presented with sleep-disordered breathing (SDB).

226 d to discrepant estimates of the severity of sleep-disordered breathing (SDB).

227 and functional status in subjects with mild sleep-disordered breathing (SDB).

228 o guide treatment decisions in children with sleep-disordered breathing (SDB).

229 Rationale: One in 10 children experiences sleep-disordered breathing (SDB).

230 en linked to disrupted sleep associated with sleep-disordered breathing (SDB).

231 frequent obstructive breathing events (mild sleep-disordered breathing [SDB]) is unknown.

232 Evaluation for sleep-disordered breathing should be a priority for meno

233 pioids on sleep quality, sleep architecture, sleep-disordered breathing, sleep apnea endotypes, venti

234 The relationship of sleep-disordered breathing (SOB) to neuropsychological d

235 (2010-2015), were offered participation in a sleep-disordered breathing study including a home sleep

236 Compared with the 193 women without sleep-disordered breathing, the 105 women (35.2%) with s

237 though stroke can lead to the development of sleep-disordered breathing, the current evidence suggest

238 y cross-sectional area in an animal model of sleep-disordered breathing, the English bulldog.

239 mic dysregulation, systemic inflammation and sleep-disordered breathing; these morbidities are exacer

240 Cross-sectional studies have linked sleep-disordered breathing to poor cognition; however, i

241 re reasonable endpoints for future trials of sleep-disordered breathing treatment in stroke.

242 If effective, sleep-disordered breathing treatment may somewhat lessen

243 thing in neuromuscular diseases, identifying sleep-disordered breathing triggered by noninvasive vent

244 OSA, and 16 healthy control subjects in whom sleep disordered breathing was excluded by complete over

245 Sleep-disordered breathing was ascertained by apnea-hypo

246 Sleep-disordered breathing was associated more strongly

247 Sleep-disordered breathing was associated with worse fun

248 Sleep-disordered breathing was defined as an apnea-hypop

249 Sleep-disordered breathing was defined by an AHI of at l

250 inverse association between hormone use and sleep-disordered breathing was evident in various subgro

251 Sleep-disordered breathing was indicated by the frequenc

252 pause, perimenopause, and postmenopause with sleep-disordered breathing was investigated with a popul

253 The prevalence of sleep-disordered breathing was modeled as a function of

254 Sleep-disordered breathing was not associated with globa

255 Determination of sleep-disordered breathing was performed through clinica

256 Sleep-disordered breathing was quantified by the apnea-h

257 Sleep-disordered breathing was quantified by the respira

258 models, neither age younger than 3 years nor sleep-disordered breathing was significantly associated

259 time of the sleep study, moderate levels of sleep-disordered breathing were common, with a median Re

260 rdered breathing, the 105 women (35.2%) with sleep-disordered breathing were more likely to develop m

261 the diagnosis of OHS in obese patients with sleep-disordered breathing when suspicion for OHS is not

262 To reduce the confounding influence of sleep-disordered breathing, which is related to both inc

263 tric patients from 3 to 16 years of age with sleep-disordered breathing who underwent a polysomnogram

264 nography-diagnosed (AHI <3) mild obstructive sleep-disordered breathing who were considered candidate

265 it to study the associations of measures of sleep disordered breathing with RNA-seq in peripheral bl

266 ted with cardiorespiratory diseases, such as sleep-disordered breathing with apnoea, congestive heart

267 to determine the independent association of sleep-disordered breathing with risk of mild cognitive i

268 been previously reported that some patient's sleep-disordered breathing worsened following surgery.