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1 ession, somatic complaints, and motor/mental slowness.
2  beneath the same region also have anomalous slowness.
3 l hallucinations (15 [46.9%] vs 42 [11.6%]), slowness (23 [71.9%] vs 95 [26.2%]), apathy (23 [71.9%]
4                                         This slowness allows infection to become well established; th
5 inally, we investigate the trade-off between slowness and sparseness when combined in one objective f
6 In contrast to this claim, we here show that slowness and sparsity drive the representations towards
7 43), dissociative phenomena (34), cognitive 'slowness' and excessive effort (26), communication diffi
8  movement difficulty (eg, tremor, stiffness, slowness), and psychological or cognitive problems (eg,
9 ness), cognitive symptoms (mental fogginess, slowness), and symptoms of hyperacusis (sensitivity to l
10 partial inactivation might produce weakness, slowness, and loss of independence of one or two adjacen
11 tion might produce dissociation of weakness, slowness, and relative independence of movement, and whi
12 exhaustion, low physical activity, weakness, slowness, and shrinking.
13 ase-like movement problems such as rigidity, slowness, and tremor.
14 le mass, exhaustion, low energy expenditure, slowness, and weakness) was obtained for participants.
15 teria: shrinking, exhaustion, sedentariness, slowness, and weakness.
16                                          The slowness anomalies are not accompanied by significant tr
17 eria (weight loss, exhaustion, low activity, slowness) at >/=2 visits, or at 1 visit with >/=1 criter
18 aDBS algorithm intended to mitigate movement slowness by delivering targeted stimulation increases du
19 device tracked frailty markers like cadence (slowness), daily steps (inactivity), postural transition
20 indings; other possible explanations include slowness due to comorbidities misinterpreted as bradykin
21 uptions in this network lead to rigidity and slowness during movement execution.
22 like phenotypes with dystonia, vertical gaze slowness, dysexecutive syndrome, predominant axial rigid
23       Parkinson's disease is associated with slowness, especially of sequential movements, and is cha
24 5]) of five frailty components (weight loss, slowness, exhaustion, weakness, and low physical activit
25 by resting tremor, rigidity, bradykinesia or slowness, gait disturbance, and postural instability.
26                        The building sector's slowness in adopting decarbonization strategies can be e
27                    Heritability was high for slowness in eating (84%; 95% CI: 83%, 86%) and satiety r
28 ite [enjoyment of food, food responsiveness, slowness in eating (SE), satiety responsiveness (SR), an
29 "enjoyment of food," "food responsiveness," "slowness in eating," and "satiety responsiveness." Herit
30 mmarize behavioural observations of movement slowness in Parkinson's disease and discuss these findin
31 ivity has been proposed to underlie movement slowness in Parkinson's disease.
32 ing of lysozyme arises not from any inherent slowness in the formation of the native structure but ra
33 here, exhibiting extremes of "fastness" and "slowness" in their life histories.
34  with the only difference that SSA maximizes slowness instead of sparsity.
35                                         This slowness is even more severe than cubic-time free energy
36                   This unsupervised temporal slowness learning (UTL) was substantial, increased with
37 cale cubically with the RNA length, and this slowness limits their use in genome-wide applications.
38 la, and the Mid-Atlantic Ridge have observed slownesses more than 64 percent greater than predicted b
39            Only six logs-bulk density, shear slowness, neutron porosity, toolface azimuth, spontaneou
40 otection is also poor because of the extreme slowness of an intramolecular Galpha refolding step (iso
41 ontribute significantly to the rate-limiting slowness of cellulose hydrolysis.
42                                          The slowness of concerted transfer of H* between Os(IV)NHPh
43                The mechanism of this dynamic slowness of contracted airways probably involves intrins
44                              It started with slowness of gait and mood dysfunction.
45 cle is about the neural conundrum behind the slowness of human behavior.
46                   Bradykinesia as defined by slowness of initiation of movement and a progressive red
47 Parkinson's disease defines bradykinesia as 'slowness of initiation with progressive reduction in spe
48 lymph node infiltration assay to address the slowness of metastasis of tumor xenografts.
49 se: an age-dependent and levodopa-responsive slowness of movement associated with diminished dopamine
50 n understanding the exact mechanisms driving slowness of movement has been impeded due to the heterog
51                           The characteristic slowness of movement in Parkinson's disease relates to a
52 cardinal motor symptoms of tremor, rigidity, slowness of movement, and impairments of posture, gait,
53 disease causes symptoms including stiffness, slowness of movement, and tremor.
54 afflicts patients later in life with tremor, slowness of movement, gait instability, and rigidity.
55                             Bradykinesia, or slowness of movement, is a defining feature of Parkinson
56  Parkinson's disease, including the hallmark slowness of movement, termed bradykinesia, were describe
57  of progressive mild left-sided weakness and slowness of movements.
58 ting a mechanism for the cannabinoid-related slowness of movements.
59 bined effect of the conduction delay and the slowness of muscle contraction.
60                                          The slowness of nucleation can be reconciled with the simila
61 mechanistic insights on the counterintuitive slowness of PT at water-hydrophobe boundaries and its re
62 ral experiments in which cannabinoid-related slowness of purposeful movements was reverted by cannabi
63  selectivity filter are at the origin of the slowness of recovery in K(+) channels.
64 ncertainties and latency associated with the slowness of seismic waves.
65 ver, appear too insignificant to explain the slowness of the beta-octabromination protocols, which se
66                                 Finally, the slowness of the lockdown process is dictated by the need
67 n intersystem barrier and is responsible for slowness of the spin-forbidden deprotonation of (1)HNO b
68  signal-to-noise phase, the arrival time and slowness of which agree with theoretical predictions for
69 PAs are an emergent phenomenon in which the "slowness" of the network is due to interactions between
70 ic latencies that obviate the impact of limb slowness on executing response decisions.
71 , known PD motor deficits characterized by a slowness or inability to switch between motor programs.
72 ve the conflict it emerges from (despite its slowness), or does it operate on future conflicts that i
73 ame properties may be equally derived from a slowness principle.
74 zophrenia is a consequence of depression and slowness, rather than a primary feature of the disease.
75 eurologic condition characterized by tremor, slowness, stiffness, and unstable posture.
76 's disease, bradykinesia is characterized by slowness, the reduced amplitude of movement, and sequenc
77 quently showed signs of movement poverty and slowness, tremor and subtle cognitive deficits.
78  also could produce dissociated weakness and slowness versus loss of independence in a given finger m
79           Physical frailty, often defined as slowness, weakness, low physical activity, shrinking, an
80 teria-defined bradykinesia, characterized by slowness with progressive reduction in amplitude and spe
81                               Because of the slowness with which dementia can develop, unbiased asses