ライフサイエンスコーパス: small bowel resection

1 tive response of Math1-null crypts following small bowel resection.

2 nderwent adhesiolysis only and 352 underwent small bowel resection.

3 owel syndrome is a morbid product of massive small bowel resection.

4 by gut ischemia, which necessitated massive small bowel resection.

5 ithelial lymphocytes are decreased following small bowel resection.

6 irect apoptosis and adaptation after massive small-bowel resection.

7 surgery for Crohn disease (CD) to avoid wide small-bowel resections.

8 3%]) were more often performed with ANS than small-bowel resection (527 [6%]) and colectomy (945 [4%]

9 had poor survival and impaired adaptation to small-bowel resection, an effect that was rescued by cro

10 es of active extravasation, and he underwent small bowel resection and subsequent IR embolization due

11 Bax-null mice had no apoptosis response to small-bowel resection and displayed an amplified adaptat

12 ed similar physiologic adaptive responses to small bowel resection as measured by changes in body wei

13 ese 7 procedures included partial colectomy, small-bowel resection, cholecystectomy, operative manage

14 less lean body mass after undergoing massive small bowel resection compared to non-obese rats.

15 s significantly higher in patients requiring small bowel resection compared with those requiring adhe

16 Patients undergoing adhesiolysis only or small bowel resection for SBO from 1991 to 2002 were sel

17 ergoing adhesiolysis only and 47% undergoing small bowel resection had more than 1 complication (P <

18 omy, partial colectomy, inguinal hernia, and small-bowel resection in a procedure with a resident phy

19 cholecystectomy to a median of 3.7% in open small bowel resection, in colorectal from 0.3% in minima

20 randomized to either underwent a 75% distal small bowel resection (massive resection) or small bowel

21 patients, and the predominant procedure was small bowel resection, mostly in young adults.

22 ineered small intestine (TESI) after massive small bowel resection (MSBR).

23 oscopically included ileocecectomy (n = 46), small bowel resection (n = 22), fecal diversion (n = 7),

24 ity of adult male Lewis rats followed by 75% small bowel resection (n=24).

25 ctomy and Crohn disease (CD) with concurrent small bowel resection or colectomy from 2000 to 2013.

26 d-type littermates at baseline and following small bowel resection or sham surgery was performed.

27 Rats underwent either 80% massive small bowel resection or transection and were harvested

28 ctor transgenic mice) conditions after a 50% small-bowel resection or sham operation.

29 Normal intestinal adaptation to massive small-bowel resection requires intact epidermal growth f

30 (group C), implantation alone (n=9); (2) the small bowel resection (SBr) group, after 75% SBr (n=9);

31 Following massive small bowel resection (SBR), the remnant intestine under

32 del to a more primitive state one week after small bowel resection (SBR); therefore, this study focus

33 Intestinal adaptation to small-bowel resection (SBR) after necrotizing enterocoli

34 on included recurrent disease after previous small bowel resection, thickened mesentery, large inflam

35 After massive small bowel resection, tuft cells and Tm were diminished

36 We examined the impact of upfront small bowel resection (USBR) for metastatic SB-NET compa

37 ut prior gastrointestinal resection or whose small bowel resection was limited to < 100 cm of ileum w

38 range, 16.4-66.6 years) undergoing elective small-bowel resection were recruited between July 2006 a