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1 the contrast loss caused by greater temporal smoothing.
2 h time point independently and then performs smoothing.
3 lso generated without any postreconstruction smoothing.
5 tures or rely on either manual inspection or smoothing algorithms that lose the underlying agent-base
6 by Bayesian Smoothing) capable of producing smooth and accurate nowcasts in multiple disease setting
7 lysis offers unique opportunities to achieve smooth and clean functionalization of drugs by unlocking
8 ese motors coordinate their action to ensure smooth and fast transport along the flagellum without st
9 e and scanning electron microscopy suggest a smooth and homogeneous distribution of grains on the fil
10 om healthy subjects were similar between the smooth and rough morphotypes of M. abscessus However, in
11 nsmission electron microscope testing of the smooth and rough nanoboxes show them to yield at 130 +/-
13 introduction system was designed to ensure a smooth and uninterrupted delivery of desolvated particle
14 our approach to scRNA-seq transformed by kNN smoothing and found that our model successfully classifi
15 ber-band baseline correction, Savitzky-Golay smoothing and standard normal variate transformation.
18 00 and 900 cm(-1) followed by Savitzky-Golay smoothing, baseline correction and normalization to Amid
21 sian approach, NobBS (Nowcasting by Bayesian Smoothing) capable of producing smooth and accurate nowc
23 round tissue level implants with a partially smooth collar between patients with thin (<=2 mm) and th
25 and is attributed to an ultra-thin and ultra-smooth copper-doped silver film with low optical loss an
28 g MNAs fulfill the geometric (sharp tips and smooth edges) and mechanical-strength requirements for f
30 als with high anchoring strengths can ensure smooth electrodeposition of lithium metal, thus paving t
31 m channels, and/or internal release from the smooth endoplasmic reticulum) near the postsynaptic dens
33 target compensate for the reduced vigour of smooth eye movements that occurs with the ingestion of l
35 Our purpose was to determine the effect of a smoothing filter and partial-volume correction (PVC) on
37 inger to rub or tap three textured surfaces (smooth flat, medium rough, and rough) with three levels
38 % lower bending rigidity, thus destabilizing smooth [Formula: see text]um curvature radius structures
40 the logarithm of mean gene counts as sums of smooth functions with the smoothing parameters and coeff
41 th tailored chirality distributions, such as smooth gradients in chirality and micropatterns with tai
42 scalloped hammerhead shark (Sphyrna lewini), smooth hammerhead shark (S. zygaena) and shortfin mako s
43 fter 270 days to be more yellow, rubbery and smooth, have a less sour and lingering aftertaste and ar
45 mogeneous channel formation, indicative of a smooth interface between the TMD and underlying electrol
47 latter consisting of multifocal opacity and smooth interlobular septal thickening, possibly with sma
48 illing of a rough clinical isolate and not a smooth isolate, suggesting that unique host-pathogen int
49 assessed using locally-weighted scatterplot smoothing (LOWESS) regression and change-point analyses
50 er detection of lipid A derived from intact (smooth) LPS from host-pathogen MSI studies, proved elusi
51 on vaginal products (KY Jelly, Replens Silky Smooth lubricant, coconut oil, Replens Long-Lasting mois
56 n promotes beta-AR desensitization in airway smooth muscle (ASM) and compromises airway relaxation re
57 odorants on the contractile state of airway smooth muscle (ASM) and uncovered a complex mechanism of
59 2B subunits are expressed in mouse lymphatic smooth muscle (LSM) and form functional K(ATP) channels
61 s widely expressed in many tissues including smooth muscle (SM), although its role in the regulation
62 00A4 is expressed in many tissues, including smooth muscle (SM), but its physiologic function is unkn
64 protein] receptor) is expressed in vascular smooth muscle (VSM) and increased VSM PTH1R signaling mi
67 ss fibers, exhibited greater levels of alpha-smooth muscle actin (alpha-SMA) expression, and exerted
68 itative proteomic comparison of mature alpha-smooth muscle actin (alpha-SMA)+ myofibroblasts (verifie
69 ding collagen Ialpha1 (colIalpha1) and alpha-smooth muscle actin (alpha-SMA), and cell migration/woun
70 lls, associated with overexpression of alpha-smooth muscle actin (alpha-SMA), and differential upregu
73 s I and VI, and the profibrotic factor alpha-smooth muscle actin compared with placebo in subcutaneou
74 own of GalR1 in cholangiocytes reduced alpha-smooth muscle actin expression in LX-2 cells treated wit
75 uces collagen I, fibronectin, elastin, alpha-smooth muscle actin in human adult dermal (HDFs) and neo
77 s >60% of fibroblasts formed alphaSMA (alpha-smooth muscle actin) stress fibers and expressed myofibr
78 markers CNN-1 (calponin 1), alpha-SMA (alpha-smooth muscle actin), and SM22-alpha (smooth muscle prot
79 fibrotic markers f-actin, fibronectin, alpha smooth muscle actin, and collagen type 1 were equally di
80 fibrotic markers f-actin, fibronectin, alpha smooth muscle actin, and collagen type 1 were performed.
81 in, alpha(1) -antitrypsin, glypican-3, alpha-smooth muscle actin, and collagen type 1A2 markers were
82 n, hepatic expressions of collagen-1a, alpha-smooth muscle actin, and mononuclear cell infiltration (
84 and PECAM1 and increases in collagen, alpha-smooth muscle actin, TGFbeta receptor 1, and the transcr
85 stress to promote fibrosis in the absence of smooth muscle actin-expressing myofibroblasts, a key pro
86 ted hepatic stellate cell; aHSCs) expressing smooth muscle alpha-actin (alphaSMA) and platelet-derive
87 ), a cytoplasmic protein expressed in airway smooth muscle and bronchial epithelium that regulates th
88 he variant were differentiated into vascular smooth muscle and endothelial cells that demonstrated ha
89 tion, a mechanism by which those channels in smooth muscle are thought to be targets of endothelium-d
91 sected advanced atherosclerotic lesions with smooth muscle cell (SMC) and endothelial lineage tracing
93 sm development and identified a key role for smooth muscle cell (SMC) reprogramming into a mesenchyma
95 a-1 (TGFbeta1) is a major driver of vascular smooth muscle cell (VSMC) phenotypic switching, an impor
98 ic inflammation and remodeling via decreased smooth muscle cell activation and neutrophil transendoth
99 elastic fiber disruption, and an increase in smooth muscle cell alpha-actin expression compared to un
100 es and evaluates the changes in rat vascular smooth muscle cell biomechanics following statin-mediate
101 holesterol depletion remodels total vascular smooth muscle cell cytoskeletal orientation that may add
102 gene encoding NOTCH3 and results in vascular smooth muscle cell degeneration, stroke, and dementia.
105 caspase-1 secretion and attenuated leukocyte-smooth muscle cell interactions under high glucose or li
107 Abi1 knockdown by shRNA reduced human airway smooth muscle cell migration, which was restored by Abi1
108 tics allow the investigation of pericyte and smooth muscle cell physiology and their role in regulati
109 g is associated with an increase in vascular smooth muscle cell proliferation and changes in vessel m
110 nhibition of TRPV4 channels mitigates aortic smooth muscle cell-dependent inflammatory cytokine produ
112 f blood outgrowth endothelial cells (BOECs), smooth muscle cells (BO-SMCs), and leukocytes were obtai
116 involved ongoing proliferation of intestinal smooth muscle cells (ISMC) with expression of platelet-d
117 duced vascular endothelial cells (iVECs) and smooth muscle cells (iSMCs) by direct reprogramming of h
120 rly stage of atherosclerosis and on vascular smooth muscle cells (SMC) remain to be fully elucidated.
122 g extra domain A (Fn-EDA) is associated with smooth muscle cells (SMCs) following vascular injury.
123 erations in extracellular matrix and loss of smooth muscle cells (SMCs) in the medial layer of the ao
125 phages (in the absence of serum or HDL) onto smooth muscle cells (SMCs) that had been metabolically l
126 se vasodilatory factors that act directly on smooth muscle cells (SMCs) to induce arterial dilation a
127 ediated NO dioxygenation process in vascular smooth muscle cells (SMCs), and the requisite reducing s
128 ontractility and differentiation in vascular smooth muscle cells (SMCs), but the specific function of
129 uman atherosclerotic plaques associated with smooth muscle cells (SMCs), inflammation, extracellular
130 c motor neurons and SIP syncytium, including smooth muscle cells (SMCs), interstitial cells of Cajal
131 tional cells of the SIP syncytium, including smooth muscle cells (SMCs), interstitial cells of Cajal
134 mechanical fluctuations applied to vascular smooth muscle cells (VSMCs) regulates mitochondrial netw
135 a hybrid phenotype of striated and vascular smooth muscle cells (VSMCs), we performed lineage tracin
138 tin, MCP-1) in endothelial cells or vascular smooth muscle cells and decreased monocytes adhesion to
139 on PIP(2) as a regulator of ion channels in smooth muscle cells and endothelial cells-the two major
142 ese results identify P2Y(2) receptors in RTN smooth muscle cells as requisite determinants of respira
143 We also identify P2Y(2) receptors in RTN smooth muscle cells as the substrate responsible for thi
144 blockade or genetic deletion of P2Y(2) from smooth muscle cells blunted the ventilatory response to
145 gregated within the mitochondria of vascular smooth muscle cells can drive an hour-long disruption.
146 e, mutant transgenic PDE3A overexpression in smooth muscle cells confirmed that mutant PDE3A causes h
147 sed a strategy whereby human endothelial and smooth muscle cells derived from blood progenitors from
149 differentiated human macrophages, and aortic smooth muscle cells from humans with diabetes), MCC950 s
150 e-expression of P2Y(2) receptors only in RTN smooth muscle cells fully rescued the CO(2)/H(+) chemore
152 rimentally by treating human coronary artery smooth muscle cells in an in vitro calcification assay.
154 nd to be expressed predominantly on vascular smooth muscle cells in lesions of athero-prone Apoe(-/-)
156 cepted role of the protein Kv2.1 in arterial smooth muscle cells is to form K(+) channels in the sarc
158 intercellular Ca(2+) waves are generated in smooth muscle cells of colonic longitudinal muscles (LSM
159 gene signatures of mesangial cells, vascular smooth muscle cells of the afferent and efferent arterio
161 ed in mice harboring specific endothelial or smooth muscle cells or cardiomyocyte or myeloid cell def
162 ssion of Akt1E17K to endothelial, cardiac or smooth muscle cells resulted in viable offspring and rem
163 crine prostaglandin E(2) signaling in airway smooth muscle cells that eventually triggered cAMP/PKA-d
164 e Kv7.4 and Kv7.5 alpha-subunits in vascular smooth muscle cells to determine which components are es
165 ogy, adhesion, and migration of human airway smooth muscle cells transfected with PKAc variants conta
166 endothelial cells and alpha-SMA(+) vascular smooth muscle cells were detected within all cellular zo
167 neurons(1), cardiomyocytes(2-4) and vascular smooth muscle cells(5), where they are involved in the r
169 r cells, like endothelial cells and vascular smooth muscle cells, cardiac myocytes and inflammatory c
170 ised intracellular Ca(2+) levels in arterial smooth muscle cells, constricted arterioles ex vivo and
171 ro-inflammatory actions of TWEAK on vascular smooth muscle cells, decreasing NF-kB activation, cytoki
173 ing through activation of nuclear ERalpha in smooth muscle cells, inviting to revisit the mechanisms
185 unter to the expectation that the absence of smooth muscle constriction would lead to a more relaxed
186 regression and niche relocation through the smooth muscle contractile machinery that generates centr
187 the first time, that effects of compromised smooth muscle contractility are more important in terms
188 st that directly inhibits calcium influx and smooth muscle contractility, leading to voiding dysfunct
190 a(1A)-adrenoreceptor, a GPCR that stimulates smooth muscle contraction in response to binding noradre
191 expression along with reduced expression of smooth muscle differentiation markers in the carotids.
194 ated with the life-threatening multisystemic smooth muscle dysfunction syndrome (MSMDS) due to mutati
195 irway hyperresponsiveness as an indicator of smooth muscle dysfunction, and treating them appropriate
199 myosin heavy chain (smMHC) and several other smooth muscle gene transcripts, indicating these cells a
202 We show that Poldip2 deficiency in vascular smooth muscle in vitro and in vivo induces the expressio
203 channel localizes to vascular endothelium or smooth muscle is controversial and the distribution and
204 strate that during development, while airway smooth muscle is dispensable for epithelial branching, i
206 xcitability and contractility of the uterine smooth muscle layer, the myometrium, increase drasticall
208 t and cardiovascular system, blood, kidneys, smooth muscle lineage and limb skeleton in the developin
209 or DR3 restricted increases in peribronchial smooth muscle mass and accumulation of lung collagen, pr
211 id cells type 2 (ILCs), and increased airway smooth muscle mass via recruitment of mesenchymal progen
213 polymerization in vitro of nonphosphorylated smooth muscle myosin filaments by the addition of MgATP
214 PDGFRalpha(+) cells in the PKJ co-expressed smooth muscle myosin heavy chain (smMHC) and several oth
215 airway smooth muscle (ASM) against a loss of smooth muscle myosin heavy chain (SMMHC) expression.
218 Although it has long been recognized that smooth muscle Na/K ATPase modulates vascular tone and bl
219 ation, revealing extensive expression in the smooth muscle of resistance arterioles supplying skeleta
220 g revealed specific localization of TRPV1 to smooth muscle of terminal arterioles in the heart, adipo
221 ver, in plasma and lung endothelium, but not smooth muscle or adventitia, miR-210 was observed in kno
222 erstanding of the roles of these channels in smooth muscle physiology and disease, particularly in co
223 (alpha-smooth muscle actin), and SM22-alpha (smooth muscle protein 22alpha) and an increase in synthe
224 C processes and indicating the importance of smooth muscle response to vascular stress and phenotypic
226 cellular electrode measurements in lymphatic smooth muscle revealed only subtle, but not significant,
228 s, our study identifies the dermal sheath as smooth muscle that drives follicle regression for reunit
231 l mesenchymal specification to cartilage and smooth muscle, coupling epithelial identity with mesench
234 -type VGCCs are expressed in mouse lymphatic smooth muscle, they do not play a significant role in mo
240 rlap of these QTLs and their relationship to smooth muscle-specific genes and transcription factors.
241 ation genes (proteins) to the human vascular smooth muscle-specific protein-protein interactome (218
242 and lung inflammation in germline and airway smooth muscle-specific Rgs4(-/-) mice and in mice treate
243 To examine this possibility, we developed a smooth muscle-specific TRPC3 knockout (TRPC3smcKO) mouse
250 lpha5-containing GABA(A) receptors in airway smooth muscles are considered as an emerging target for
253 ion, such that firing probability grows as a smooth nonlinear function of membrane potential; and (2)
254 a smaller task related to factoring: finding smooth numbers, which is an essential step of the Number
256 the changes are attributed to atomic-scale "smoothing" of the surface by preferential sublimation of
257 arious factors such as hydrophobic material, smooth optic surface, and sharp posterior optic edge pla
258 the presence of signs favoring PLC on HRCT (smooth or nodular septal lines, subpleural nodularity, p
259 te that the formation of a relatively thick, smooth oxide sheath can improve the catalytic stability
260 counts as sums of smooth functions with the smoothing parameters and coefficients estimated simultan
268 ysaccharides in IDFs had lower proportion of smooth region and higher proportion of hairy region that
272 raded impact crater in Elysium Planitia on a smooth sandy, granule- and pebble-rich surface with few
273 branes via structural optimization: first, a smooth selective layer should be maintained to ensure un
277 n empirical recurrence rates ratio (ERRR), a smoothing statistic that is gradually gaining currency i
279 shear zones and fault planes have spatially smooth stress with predominantly time-dependent stress i
280 m laminar to turbulent flow occurring over a smooth surface is a particularly important route to chao
284 ubSP and the previously identified bumpy and smooth-surfaced spherical particles, may help flavivirus
286 m) have been observed to generate remarkably smooth surfaces not predicted by low-pressure deposition
289 dingly, protraction-tuned units encode rough-smooth texture better than retraction-tuned units and en
290 of our analyzed phenotypes are sufficiently smooth that some evolutionary paths remain accessible fo
291 g-rate selection assumption proposed by CBGB smooths the transition between limiting rates and always
292 ight propagates inside a thin soap membrane, smooth thickness variations in the film act as a correla
293 benzene-containing materials and showed that smooth topographical cues with proper period and orienta
297 the host-pathogen interface: OMVs with long (smooth-type) lipopolysaccharide lipids retain their sphe
298 e, combining chipping of outer enameloid and smooth wear of inner enameloid to preserve an efficient
299 the films reveals that these films are very smooth with low roughness, the thin films synthesized at