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1 ine release from nerve terminals, and airway smooth muscle contraction.
2 n to both motion and force generation during smooth muscle contraction.
3 protein caldesmon (CaD) reversibly inhibits smooth muscle contraction.
4 ajal (ICC), the pacemaker cells that control smooth muscle contraction.
5 secretion, sensory signal transduction, and smooth muscle contraction.
6 sory transduction, epithelial secretion, and smooth muscle contraction.
7 fluid secretion, neuronal excitability, and smooth muscle contraction.
8 negatively modulate force development during smooth muscle contraction.
9 implicated in Rho-mediated cell adhesion and smooth muscle contraction.
10 a crucial and selective role in ETA-mediated smooth muscle contraction.
11 and play an important role in regulating the smooth muscle contraction.
12 function as signaling platforms to regulate smooth muscle contraction.
13 tase activity in the absence of Ca2+ induces smooth muscle contraction.
14 kinase (MLCK) is essential for initiation of smooth muscle contraction.
15 of myosin (MLC20) phosphorylation and airway smooth muscle contraction.
16 aggregation, lipolysis, glycogenolysis, and smooth muscle contraction.
17 PT1.PP1C.P-CPI-17, leading to an increase in smooth muscle contraction.
18 re preceded by long-duration waves of airway smooth muscle contraction.
19 take part in the thin filament regulation of smooth muscle contraction.
20 depletion of paxillin by antisense inhibits smooth muscle contraction.
21 in filament dynamics and organization during smooth muscle contraction.
22 thought to be involved in the regulation of smooth muscle contraction.
23 +) sensitivity of myosin phosphorylation and smooth muscle contraction.
24 ing pathways, important regulators of airway smooth muscle contraction.
25 in filament dynamics and organization during smooth muscle contraction.
26 that paxillin may be involved in regulating smooth muscle contraction.
27 oconstriction of human airway by stimulating smooth muscle contraction.
28 heads plays a key role in the regulation of smooth muscle contraction.
29 of CPI-17 to produce Ca(2+) sensitization of smooth muscle contraction.
30 on and subsequent inhibition (relaxation) of smooth muscle contraction.
31 which might play a role in the regulation of smooth muscle contraction.
32 hectomy, parasympathetic stimulation elicits smooth muscle contraction.
33 proliferation and airway as well as vascular smooth muscle contraction.
34 oteins known to regulate aortic and tracheal smooth muscle contraction.
35 gests that NO may be closely associated with smooth muscle contraction.
36 ability persisted beyond the duration of the smooth muscle contraction.
37 , which facilitates actin polymerization and smooth muscle contraction.
38 in part from amplified GPCR-mediated airway smooth muscle contraction.
39 light chain, which is essential for vascular smooth muscle contraction.
40 critical physiological processes, including smooth muscle contraction.
41 neuronal cell death, acute inflammation, and smooth muscle contraction.
42 involved in cytoskeletal reorganization and smooth muscle contraction.
43 the regulation of Mlc20 phosphorylation and smooth muscle contraction.
44 e for lysophosphatidic acid-induced vascular smooth muscle contraction.
45 interactor 1) participates in the control of smooth muscle contraction.
46 re involved in blood pressure regulation and smooth muscle contraction.
47 of myosin light chain, which induces airway smooth muscle contraction.
48 the involvement of ZIPK in the regulation of smooth muscle contraction.
49 enin by lentivirus-mediated shRNA attenuated smooth muscle contraction.
50 oA, inactivating RhoA and suppressing airway smooth muscle contraction.
51 latory protein that has a role in modulating smooth muscle contraction.
52 can reduce inflammation and inhibit vascular smooth muscle contraction.
53 ructural alterations of epithelial cells and smooth muscle contraction.
54 n plays a critical role in the regulation of smooth muscle contraction.
55 ssed ovalbumin-mediated guinea pig bronchial smooth muscle contraction.
56 regulation of thick filament length, such as smooth muscle contraction.
57 dying mechanical stress-mediated decrease in smooth muscle contraction.
58 ranging from mucosal secretion to regulating smooth muscle contraction.
59 ural remodelling of the artery, or increased smooth muscle contraction.
60 substrate (CAS) have been shown to regulate smooth muscle contraction.
61 s interaction inhibited IP3-dependent airway smooth muscle contraction.
62 intracellular Ca(++) (via IP(3) release) and smooth muscle contraction.
63 tion process in response to agonist -induced smooth muscle contraction.
64 ch have been implicated commonly in vascular smooth muscle contraction.
65 ing that RGS5 negatively regulates bronchial smooth muscle contraction.
66 tion, cardiac and neuronal excitability, and smooth muscle contraction.
67 ts in the fetal period, requires coordinated smooth muscle contraction.
68 mbryo and is regulated by adrenergic uterine smooth muscle contractions.
69 directly inhibited various tonic and phasic smooth muscle contractions.
70 s system, including cardiac, arteriolar, and smooth muscle contractions.
71 each other and are both necessary for normal smooth muscle contractions.
72 directional beating of epithelial cilia, and smooth muscle contractions.
73 requency of spontaneous colonic longitudinal smooth muscle contractions.
74 oded by TMEM16A control neuronal signalling, smooth muscle contraction, airway and exocrine gland sec
75 bronchoconstriction is largely due to airway smooth muscle contraction, airway structural changes kno
77 elieved to be important in the regulation of smooth muscle contraction, although the precise mechanis
78 inflammatory mechanisms that lead to airway smooth muscle contraction and airway hyperresponsiveness
80 There is emerging evidence indicating that smooth muscle contraction and Ca2+ influx through voltag
81 oth muscle cells (VSMCs) which could enhance smooth muscle contraction and cell growth activated by c
83 kinase that is involved in the regulation of smooth muscle contraction and cytoskeletal reorganizatio
84 al lipopolysaccharide (LPS, 20 mg/kg, IP) on smooth muscle contraction and endothelial relaxation in
85 ing genes with potential regulatory roles in smooth muscle contraction and extracellular matrix-recep
86 ion of Vh peptide also inhibited ACh-induced smooth muscle contraction and inhibited ACh-induced acti
87 , red wine, and green tea inhibited arterial smooth muscle contraction and intestinal Cl(-) secretion
89 ential for many biological processes such as smooth muscle contraction and neurotransmitter release.
90 The major mechanism of Ca2+ sensitization of smooth muscle contraction and non-muscle cell motility i
91 ain kinase is important in the initiation of smooth muscle contraction and other contractile processe
92 ial permeability; it also triggers bronchial smooth muscle contraction and participates in autoregula
95 6 inhibitor NAV2729 inhibits human prostate smooth muscle contraction and proliferation of stromal c
96 ys an important role in cardiac development, smooth muscle contraction and psychiatric disorders.
97 y myosin light chain kinase (MLCK) initiates smooth muscle contraction and regulates actomyosin-based
98 noid signaling impedes coordinated oviductal smooth muscle contraction and relaxation crucial to norm
100 ses such as excitation-contraction coupling, smooth muscle contraction and relaxation, and various fo
103 involvement of ARF6 in promotion of prostate smooth muscle contraction and stromal growth, and define
104 lays a significant role in the regulation of smooth muscle contraction and suggest that CaMKII activa
105 ermine how Tyr(1065) phosphorylation affects smooth muscle contraction and the conformation and cellu
106 a vital mechanism for the control of airway smooth muscle contraction and thus are a critical factor
107 a vital mechanism for the control of airway smooth muscle contraction and thus are a critical factor
108 ce receptors and cause leukocyte activation, smooth muscle contraction and vascular permeability.
109 uences endothelin (ET) ETA receptor-mediated smooth muscle contraction and, if so, to define the unde
110 ) participates in the regulation of vascular smooth muscle contraction and, if so, to investigate the
111 vestigation may identify agents that inhibit smooth muscle contraction and/or restrain or reverse obs
112 sure decreases the interval between systemic smooth muscle contractions and increases the rate of mor
114 egulation of breathing, gas exchange, airway smooth-muscle contraction and relaxation, blood flow thr
116 a pivotal role in regulating actin dynamics, smooth muscle contraction, and airway hyperresponsivenes
119 ) participates in the regulation of vascular smooth muscle contraction, and if so, to investigate the
122 e pharmacomechanical coupling that activates smooth muscle contraction, and possibly regulation of di
126 n, transforming growth factor-beta, vascular smooth muscle contraction, and the hedgehog and Wnt sign
127 ching morphogenesis, the frequency of airway smooth muscle contraction, and the rate of developmental
128 le cells may be dynamically regulated during smooth muscle contraction, and this dynamic regulation m
129 ing pathways, induce inhibition of the ileal smooth muscle contractions, and affect distinct physiolo
131 affect Ca2+-sensitive pathways implicated in smooth muscle contraction, as tested with alpha-toxin pe
132 ted K(+) channel that underlies skeletal and smooth muscle contraction, audition, hormone secretion a
133 ha1 adrenergic receptors not only stimulates smooth muscle contraction but also modifies gene express
134 ator release, and allergen-induced bronchial smooth muscle contraction by CRACM-channel blockers supp
135 m by which RhoA regulates actin dynamics and smooth muscle contraction by expressing the dominant neg
136 , these findings suggest that Plk1 regulates smooth muscle contraction by modulating vimentin phospho
137 9beta1 appears to serve as a brake on airway smooth muscle contraction by recruiting SSAT, which faci
138 n phosphatase (MLCP) plays a pivotal role in smooth muscle contraction by regulating Ca(2+) sensitivi
141 regulates biological functions as diverse as smooth muscle contraction, cardiac function, and axon gu
142 in network have been implicated in mediating smooth muscle contraction, cell migration, and mitosis.
143 n which the magnitude of an agonist-mediated smooth muscle contraction depends on a rapid but limited
144 ht chain phosphorylation (RLC) necessary for smooth muscle contraction depends on the respective acti
147 geneses include connective tissue disorders, smooth muscle contraction disorders, and congenital hear
149 l roles in regulating neuronal excitability, smooth muscle contraction, fluid secretion and gut motil
150 aortic event risk were identified among the smooth muscle contraction genes (ACTA2, MYLK, and PRKG1;
151 ivities, along with a positive enrichment of smooth muscle contraction genes in 75%KD cardiomyocytes/
153 of a beta(2)-AR/cAMP microdomain central to smooth muscle contraction, immune cell activation, and e
155 d in alpha(1)-adrenoceptor-mediated vascular smooth muscle contraction in distinctive time-dependent
157 le for M3-mAChR phosphorylation in bronchial smooth muscle contraction in health and in a disease sta
159 creased in vitro airway narrowing and airway smooth muscle contraction in murine and human airways.
160 sphatase plays a critical role in modulating smooth muscle contraction in response to a variety of ph
161 a(1A)-adrenoreceptor, a GPCR that stimulates smooth muscle contraction in response to binding noradre
162 y is now well recognized to mediate vascular smooth muscle contraction in response to vasoconstrictor
163 The inhibition of myosin phosphatase evokes smooth muscle contraction in the absence of Ca(2+), yet
164 is a multifunctional hormone that regulates smooth muscle contraction in the airways, acid secretion
165 congenital disorder characterized by loss of smooth muscle contraction in the bladder and intestine.
166 ther homologues in inducing well-coordinated smooth muscle contractions in an in vitro rabbit duodena
167 2+) channel expression may suppress circular smooth muscle contractions in the inflamed colon and con
168 its, inhibited adenylyl cyclase, and induced smooth muscle contraction, in similar fashion to the sel
169 hree well-studied pharmacological functions, smooth muscle contraction, increased vascular permeabili
170 vels of activity in myenteric neurons during smooth muscle contractions induced by application of cap
171 , and identified distinct pathways linked to smooth muscle contraction, inflammatory cytokines, immun
174 itiation of force generation during vascular smooth muscle contraction involves a rise in intracellul
176 ption, protein transport, protein synthesis, smooth muscle contraction, ion transport, chemotaxis, an
184 t are unresponsive to capsaicin, bradykinin, smooth muscle contraction, longitudinal or transverse st
185 II) from mast cell renin elicited bronchial smooth muscle contraction mediated by ANG II type 1 rece
186 , many features of bronchial asthma, such as smooth muscle contraction, mucus secretion and recruitme
187 ion transport, olfaction, phototransduction, smooth muscle contraction, nociception, cell proliferati
190 ding mucus secretion, neuronal excitability, smooth muscle contraction, olfactory signal transduction
191 ry effects on inflammation, anaphylaxis, and smooth muscle contraction, PDE-IV inhibitors also produc
192 or a variety of functions including vascular smooth muscle contraction, platelet aggregation, and the
193 ysiological processes as diverse as vascular smooth muscle contraction, platelet aggregation, percept
196 nderstanding of the regulation of myometrial smooth muscle contraction-relaxation is incomplete.
198 valuation of selected proteins implicated in smooth muscle contraction revealed no significant differ
199 l responsiveness was not associated with the smooth muscle contraction since leukotriene C4, histamin
200 ck of PAR-2-mediated platelet aggregation or smooth muscle contraction suggested it might not share t
201 se (approximately total tissue content), and smooth muscle contraction that was rapid in onset and sh
202 cemakers set the origin and frequency of the smooth muscle contractions that propel wastes from the k
204 e ZIPK is involved in regulation of vascular smooth muscle contraction through direct phosphorylation
205 tial cells of Cajal (ICC) control intestinal smooth muscle contraction to regulate gut motility.
206 Excitatory agonists can induce significant smooth muscle contraction under constant free Ca(2+) thr
207 plays acute, allergen-specific, IgE-mediated smooth muscle contractions using precision cut intestina
209 y essential biological activities, including smooth muscle contraction, vesicle secretion, gene expre
210 stimulate secretion, cell proliferation, and smooth muscle contraction via a family of G protein-coup
211 xPANs regulate myenteric neuron activity and smooth muscle contraction via a parasympathetic spinal c
212 se data indicate that IL-17A promotes airway smooth muscle contraction via direct recruitment of Rab3
214 sent study, the role of alpha-actinin during smooth muscle contraction was evaluated in tracheal smoo
216 at CPI-17, a key player in the regulation of smooth muscle contraction, was downregulated by lipopoly
217 to myosin by caldesmon in the regulation of smooth muscle contraction, we investigated the effects o
218 functions as a second messenger in tracheal smooth muscle contraction, we used the criteria set out
220 ene deletion in mice enhances AHR and airway smooth muscle contraction, whereas RGS4 KO mice unexpect
221 ting Ins(1,4,5)P3-dependent Ca2+ release and smooth muscle contraction, whereas the alpha subunits in
222 zation plays an important role in regulating smooth muscle contraction, which is essential for the mo
223 tly reduced by functionally antagonizing the smooth muscle contraction with isoproterenol, or by bloc
224 latory light chain (RLC) phosphorylation for smooth muscle contraction with subsequent dephosphorylat
225 K416Q Abi1 promoted actin polymerization and smooth muscle contraction without affecting myosin light
226 peptide) attenuated actin polymerization and smooth muscle contraction without affecting myosin light