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1 d dental caries (Streptococcus mutans and S. sobrinus).
2 ) for Streptococcus mutans and Streptococcus sobrinus.
3 d by infection with cariogenic Streptococcus sobrinus.
4 e construct to include both S. mutans and S. sobrinus.
5  replace the P1 protein, the spaA gene of S. sobrinus 6715 was isolated from plasmid pX1303 and inser
6 (GBP-2, GBP-3, and GBP-5) from Streptococcus sobrinus 6715 were compared structurally by mass spectro
7 tinomyces viscosus and Streptococcus mutans (sobrinus) 6715, and fed a cariogenic diet.
8 hildren under 18 mos were: S. mutans 70%, S. sobrinus 72%, P. gingivalis 23%, B. forsythus 11%, and A
9  vaginalis, and S. mutans with Streptococcus sobrinus (all p < 0.05) were positively associated with
10      Multivariate modeling employing HOT, S. sobrinus and S. mutans (PCR/qPCR), and sugar snacks sepa
11                                   S. mutans (sobrinus) and A. viscosus established readily in all ani
12 e strain each of S. gordonii, S. sanguis, S. sobrinus, and S. vestibularis.
13 nel of streptococci, including S. mutans, S. sobrinus, and Streptococcus australis, and Alloprevotell
14 tination of Streptococcus mutans 3209 and S. sobrinus B13.
15 hat SpaA plays a role in the virulence of S. sobrinus by specifically interacting with fluid-phase sa
16 educed dental caries after infection with S. sobrinus compared with sham-immunized animals.
17                                 Thus, the S. sobrinus ComRS pathway provides new tools for studying t
18  among the mutans group streptococci, the S. sobrinus ComRS system is most similar to the competence
19        We discovered that most strains of S. sobrinus contain a new class of the ComRS competence sys
20              In conclusion, S. mutans and S. sobrinus correlated with Romanian adolescents with carie
21                                     Thus, S. sobrinus GBP-2 and GBP-3 appear to be distinct proteins
22                                           S. sobrinus GBP-5 may be a proteolytic fragment of GBP-3, o
23                             None of these S. sobrinus GBPs appeared to have a strong antigenic relati
24                                Streptococcus sobrinus glucosyltransferase (GTF), an enzyme involved i
25 ere immunized as above or with Streptococcus sobrinus GTF and then infected with S. sobrinus to explo
26  also inhibited the ability of Streptococcus sobrinus GTF to synthesize insoluble glucan.
27  at 7- to 14-day intervals with peptides, S. sobrinus GTF, or phosphate-buffered saline.
28 ibody from the communized group inhibited S. sobrinus GTF-mediated insoluble glucan synthesis in vitr
29 soluble glucan synthesis by S. mutans and S. sobrinus GTFs (P < 0.0001 and P < 0.05, respectively).
30                 Two mutants of Streptococcus sobrinus incapable of aggregation by high-molecular-weig
31                                Streptococcus sobrinus is an etiologic cause of dental caries (tooth d
32                          Our knowledge of S. sobrinus is scant despite the organism's important role
33                                  Although S. sobrinus is typically placed among the mutans group stre
34                It is widely believed that S. sobrinus lacks the natural competence pathways that are
35 as also prevented genetic manipulation of S. sobrinus, limiting our knowledge of its pathogenicity.
36 ther cariogenic Streptococcus (Streptococcus sobrinus) or with oral nonstreptococci species.
37 eptococcus mutans (p < 0.005), Streptococcus sobrinus (p < 0.005), and Bifidobacteria (p < 0.0001) we
38       Unlike all other ComRS systems, the S. sobrinus pathway contains 2 copies of the transcriptiona
39         The results show that even though S. sobrinus produces several proteins capable of binding al
40 s, failed to reveal cross-reactivity with S. sobrinus proteins.
41 ectrophoretic mobility similar to that of S. sobrinus SpaA protein and a minor 210-kDa protein and a
42                            The Streptococcus sobrinus SpaA protein and the Streptococcus mutans P1 pr
43 was inhibited specifically by antisera to S. sobrinus SpaA protein.
44 iment, rats were infected with Streptococcus sobrinus strain 6715 under an identical protocol.
45 to detect Streptococcus mitis, Streptococcus sobrinus, Streptococcus mutans, Streptococcus oralis, an
46 ted were Streptococcus mutans, Streptococcus sobrinus, Streptococcus sanguinus, Lactobacillus acidoph
47           We also identified 2 strains of S. sobrinus that appear to be "cheaters" by either not resp
48 occus sobrinus GTF and then infected with S. sobrinus to explore the effects of immunization on immun
49  S. pyogenes, S. gordonii, S. mutans, and S. sobrinus, using PCR amplification, and sequenced.
50 associated with S-ECC, and S. mutans with S. sobrinus was associated with lesion recurrence (p < 0.05
51 s from culture supernatants of Streptococcus sobrinus were able to bind avidly to Sephadex G-75.
52 ess to dental care, whereas S. mutans and S. sobrinus were detected infrequently in Swedish adolescen
53   Synthetic XIP enables transformation of S. sobrinus with plasmid or linear DNA, and we leverage thi