コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 adult heterozygous Dyrk1a mice also rescued social recognition.
2 ry bulb (GnRH(OB)) of adult mice can mediate social recognition.
3 forward inhibition of CA3 and CA2 to promote social recognition.
4 PV IN-mediated inhibition of CA3 and CA2 and social recognition.
5 campus to the posterior hippocampus to guide social recognition.
6 on or avoidance behavior is thought to guide social recognition.
7 gan, an olfactory substructure essential for social recognition.
8 s Spheniscus vocalisations are important for social recognition.
9 /- mice showed decreased social interest and social recognition.
10 (V1aR) or oxytocin receptor (OTR) related to social recognition.
11 801), cognitive impairments, and deficits in social recognition.
12 veral days before behavioral testing reduced social recognition.
13 iral vector resulted in a complete rescue of social recognition.
14 but not the medial amygdala, is critical for social recognition.
15 sensitive PFC to BLA circuit is required for social recognition.
16 easured by prepulse inhibition, and impaired social recognition.
17 insights about neuroendocrine regulation of social recognition.
18 n explored concerning its modulatory role in social recognition.
19 e estrogen control over the OT regulation of social recognition.
20 experimental social experience on subsequent social recognition.
21 OT treatment fully restores social recognition.
22 la during the initial exposure to facilitate social recognition.
23 asopressin in the olfactory bulb impairs the social recognition abilities of rats and that vasopressi
24 alian social behaviors such as pair bonding, social recognition and aggression causally increases hum
25 ptor (V1aR) exhibit a profound impairment in social recognition and changes in anxiety-like behavior.
26 core autism-like traits, including defective social recognition and communication, increased stereoty
28 er that corresponded with reduced short-term social recognition and enhanced background (pre-investig
29 ories in rodents, focusing on two paradigms: social recognition and fear conditioning, representing a
31 Oxytocin (OT) is a critical molecule for social recognition and memory that mediates social and e
33 ove useful in studies of disorders affecting social recognition and social engagement, and the treatm
35 n play a particularly prominent role both in social recognition and the expression of appropriate soc
36 physiological responses including alertness, social recognition, and hunger, yet, their mechanism of
37 es abnormalities in respiratory function and social recognition, and improves motor coordination in y
38 ts were tested in a short-term memory model, social recognition, and in a separate group cortical and
39 g the monkey delayed matching-to-sample, rat social recognition, and mouse inhibitory avoidance model
41 d violence, perceived respect from patients, social recognition as well as physician-nurse coordinati
42 reated mice exhibit impaired sociability and social recognition at 72 h post-exposure, which correlat
43 type (wt) mice resulted in a potentiation of social recognition behavior and a mild increase in anxie
44 and vertebrates share similarities in their social recognition behavior, indicating that these behav
45 olved in species-typical behavior, including social recognition behavior, maternal behavior, social b
46 atal novelty exposure-induced enhancement in social recognition broadens the impact of early life sti
47 region-selective receptor deletion impaired social recognition but left odor discrimination and reco
55 t a critical role for the oxytocin system in social recognition has been conserved across perceptual
60 odel that binge alcohol consumption disrupts social recognition in females and potentiates sensorimot
67 al hormones play a role in the modulation of social recognition including estrogen, oxytocin and argi
68 could provide a basis for different types of social recognition, including kin and species recognitio
74 es typical behavioral functions that include social recognition, maternal-infant attachment, and modu
82 k3-deficient rats exhibit impaired long-term social recognition memory and attention, and reduced syn
83 signaling in modulating short- and long-term social recognition memory and found that it is necessary
84 found that RAG1-deficient mice show impaired social recognition memory compared to mice wildtype for
86 R2B overexpression in the forebrain enhances social recognition memory for different strains and anim
87 in regulating short- and long-term forms of social recognition memory has not been fully investigate
88 e data trace the origin of the impairment in social recognition memory in RAG1-deficient mice to the
89 isition model in rat pups at 0.1 mg/kg and a social recognition memory model in adult rats at 0.01 mg
90 found that RAG1-deficient mice show impaired social recognition memory relative to heterozygous or RA
92 terone concentration, turning asymmetry, and social recognition memory suggest that stress hormones a
93 RAG1 plays a role in brain function using a social recognition memory task, an assessment of the acq
94 y undescribed role for the SuM in regulating social recognition memory via oxytocin signaling and rei
95 alamic-pituitary-adrenal axis, we found that social recognition memory was prolonged to at least 24 h
96 ion of food preference learning and impaired social recognition memory without affecting sociality.
97 ort that young adult AC1+ mice have enhanced social recognition memory, and normal fear and spatial m
98 ay an important role in the consolidation of social recognition memory, at least in part through enha
99 dependent learning and memory, as assayed by social recognition memory, novel object recognition, and
100 investigate the role of the NMDA receptor in social recognition memory, specifically the consequences
108 The data strongly suggest the involvement in social recognition of the four genes coding for ER-alpha
109 social memory capabilities, the majority of social recognition papers explore short-term memories an
110 ng produced by a 120-min delay that impaired social recognition performance to 29% of 30-min delay co
111 ng specific roles for amygdala OTR in female social recognition potentially mediated by anxiety in a
113 ing the functional genomics of OT and OTR in social recognition should help elucidate the neurobiolog
114 dors serve as important safety-promoting and social recognition signals, but their role in human brai
115 re critically involved in the development of social recognition skills within rodent species, primate
120 ed with vehicle-treated Cplx2-/- mice in the social recognition test (34 compared with 13%, P<0.01).
122 rference of MLIs during distinct phases of a social recognition test revealed the cerebellar engageme
124 chrogaster) to novel females in a multitrial social recognition test to investigate whether individua
125 nificantly improved short-term memory in rat social recognition that was not likely due to alteration
127 roducts in a micronet required for mammalian social recognition, through which an individual learns t
128 ning, spontaneous alternation in the Y maze, social recognition, trace and contextual fear conditioni
129 avior enables the evolution and stability of social recognition, whereas conditional helping leads to
130 ated mice, iE-DAP-exposed mice show impaired social recognition while maintaining normal motor activi
131 ecent studies exploring neural substrates of social recognition with a focus on the potential role of