ライフサイエンスコーパス: soda

1 y loss of Mn-dependent superoxide dismutase (SodA).

2 of manganese-dependent superoxide dismutase sodA).

3 ter/operator region of superoxide dismutase (sodA).

4 detoxify ROS, such as superoxide dismutase (SodA).

5 (2+) speciation, despite the paucity of holo-SodA.

6 D. radiodurans cells cannot be attributed to SodA.

7 tested whether the same is true for Borrelia SodA.

8 e, with negligible EPR signal from Mn(2+) of SodA.

9 comparison with men who did not consume diet soda.

10 loenzyme to degrade ROS in B. burgdorferi is SodA.

11 ed by substituting alternative beverages for soda.

12 esting that manganese regulates the level of SodA.

13 ated or decaffeinated coffee for low-calorie soda.

14 te dehydrogenase subunits PdhB and PdhD, and SodA.

15 levels of its manganese superoxide dismutase SodA.

16 eef, pork, or lamb sandwich to 0.99 for diet soda.

17 Therefore, this gene was designated sodA.

18 odM, SodA, and a hybrid composed of SodM and SodA.

19 ion of SOD activity by cloned M. catarrhalis sodA.

20 ponential phase of growth, SodM more so than SodA.

21 ssense mutations in the normal stop codon of sodA.

22 ects of the long-term consumption of sugared soda.

23 eas we observed no such association for diet soda.

24 ng/d (daily consumers) of either SSB or diet soda.

25 ceived intolerance to spicy foods, coffee or soda.

26 calories in quickly ingested drinks such as sodas.

27 dolescents and with higher intake of regular soda (73.77 g and 88.28 g for children and 163.67 g and

28 pared with women who consumed less sweetened soda (86.5 pg/mL compared with 74.4 pg/mL, P = 0.01) aft

29 umarase C, Mn-dependent superoxide dismutase SodA, a ferredoxin and ferredoxin reductase and several

30 This manganese-dependent SodA activity allows the bacteria to evade neutrophil ki

31 sed to visualize both native and recombinant SodA activity in bacterial lysates.

32 SodA activity was reduced, but not eliminated in the ssa

33 sion only), and consumption of fast food and soda (adolescent gender expression only) in the past wee

34 We tested SoDA against a set of 120 artificial immunoglobulin sequ

35 onensis metacyclic promastigotes lacking one SODA allele failed to replicate in macrophages and were

36 Reduced expression of SODA also resulted in mitochondrial oxidative damage and

37 s between the superoxide-detoxifying enzyme (SodA), an integral membrane protein (DoxX), and a predic

38 per 100,000 person-years for sugar-sweetened soda and 14 and 47 cases per 100,000 person-years for or

39 Conversely, molecular methods, such as sodA and 16S rRNA gene sequencing, are clinically practi

40 Soda and energy and sports drinks were the largest food

41 tions among white women, whereas caffeinated soda and green tea intakes were associated with increase

42 The association between soda and hip fractures did not differ by body mass index

43 Mycobacterium were identified by rpoB, sodA and hsp65 gene sequencing and strain typed using va

44 drates, less protein, and more caffeine-free soda and juice than whites did during the study (Ps < 0.

45 quire manganese in turn promotes function of SodA and KatN, enzymes that use the metal as a cofactor

46 Optimized method was applied to ice tea, soda and mineral water for the speciation of Se(IV) and

47 f fragments of two other housekeeping genes, sodA and mutS, confirmed that the isolates are most clos

48 ctose-rich beverages such as sugar-sweetened soda and orange juice can increase serum uric acid level

49 nt association was found for sugar-sweetened soda and seronegative RA.

50 of phenazine methosulfate or by deletion of sodA and sodB, stimulates rugose biofilm formation in th

51 sodA was 92 and 76% identical to that of the SodA and SodM proteins of S. aureus, respectively.

52 Viability of the sodA and sodM sodA mutants but not the sodM mutant was d

53 and DNA sequencing of the housekeeping gene sodA and the putative virulence gene hylB differentiated

54 = 0.003), with +1.09 cent/oz (p < 0.001) for sodas and energy drinks, but a lower change in other cat

55 ule, reacted with T. cruzi mitochondrial (Fe-SODA) and cytosolic (Fe-SODB) SODs with second order rat

56 f cumulatively averaged sugar-sweetened (eg, sodas) and artificially sweetened (eg, diet sodas) bever

57 d for laboratory strains of S. aureus: SodM, SodA, and a hybrid composed of SodM and SodA.

58 mon interfering agents (bleach, rust, cherry soda, and coffee) to other blood detection methods.

59 different types of SCBs (i.e., fruit juice, soda, and concentrate).We included 3312 mother-child pai

60 O(3) , and NaOH, found in table salt, baking soda, and detergents, respectively.

61 otein was not detected in macrophages; sodC, sodA, and fbpB transcription showed no decrease; and acr

62 decreased for superoxide dismutase C (sodC), sodA, and fibronectin-binding protein B (fbpB).

63 ing 54 VGS strains for which MLSA, 16S gene, sodA, and gyrB data are available at the NCBI, showing t

64 We sequenced rpoB, sodA, and hsp65 genes from isolates previously identifie

65 foods (e.g., salty snack foods, candy bars, soda, and processed meats) were not affected by WIC auth

66 m each other and showed 100% 16S rRNA, rpoB, sodA, and recN gene sequence similarity.

67 Sequencing of the 16S rRNA, rpoB, sodA, and recN genes; comparative whole-genome analysis;

68 ciation of soda, including specific types of soda, and risk of hip fracture in postmenopausal women.

69 SoDA appears generally to find more likely recombination

70 SodA to the periplasm, ruling out export of SodA as a complex with a TAT substrate as a chaperone.

71 utant, we identified superoxide dismutase A (SodA) as a protein dependent on SecA2 for secretion.

72 The diet soda association was not hypothesized and deserves furth

73 he gene encoding for superoxide dismutase A (sodA, bb0153), an enzyme mediating the dismutation of su

74 sodas) and artificially sweetened (eg, diet sodas) beverage intake with incident fatal and nonfatal

75 resulted in nitration and inactivation of Fe-SODA but not Fe-SODB, suggesting that these enzymes play

76 Regular consumption of sugar-sweetened soda, but not diet soda, is associated with increased ri

77 and weight gain by intake of sugar-sweetened sodas, but these trials are few.

78 The consumption of regular sodas by NHB children was somewhat lower than among MA a

79 n of other caffeinated products (caffeinated soda, caffeinated tea, decaffeinated coffee or chocolate

80 The dried raisin, the crushed soda can, and the collapsed bicycle inner tube exemplify

81 requency of consumption of unhealthy snacks (soda, candy, and potato chips) from 2001 to 2008 in Norw

82 respectively, more in BMI than the juice and soda cluster across the study interval in a multivariate

83 ly distributed between ribotypes or hylB and sodA clusters.

84 ndicates that M. catarrhalis strains lacking SodA constitutively express immunogenic OMPs previously

85 Information on sugar-sweetened soda consumption (including regular cola, caffeine-free

86 /d (18.4, 27.3 g/d), resulted from decreased soda consumption (P-linear trend <0.001 for both).

87 investigate the associations between sugared soda consumption and caries.

88 uate the association between sugar-sweetened soda consumption and risk of RA in US women.

89 Our objective was to examine patterns of soda consumption and substitution of alternative beverag

90 High soda consumption at 3 y of age was associated with an ac

91 Infant obesity at 6 mo of age and soda consumption at 4 y of age mediated the relation in

92 puted RRs for hip fractures by the amount of soda consumption by using Cox proportional hazards model

93 Soda consumption has been associated with poor bone heal

94 Recent studies have examined sugar-sweetened soda consumption in relation to early markers of kidney

95 The absence of apparent effects of sugared soda consumption in younger people may also be related t

96 Sugar-sweetened soda consumption is consistently associated with an incr

97 Increased soda consumption of all types may be associated with inc

98 The frequency of soda consumption remains high in the United States.

99 he attributable risk in our cohort for total soda consumption was 12.5%.

100 In contrast, diet soda consumption was not associated with change in abdom

101 Diet soda consumption was not significantly associated with r

102 a, significant associations between DMFS and soda consumption were generally seen in persons over age

103 ledge, no study has examined the relation of soda consumption with risk of hip fractures.

104 nce, outdoor free play, neighborhood income, soda consumption, and child's birth weight.

105 07-2008, primarily because of a reduction in soda consumption, mean intakes continue to exceed recomm

106 No differences in DMFS, relative to soda consumption, were seen in persons under age 25, or

107 pants were randomly assigned to consume diet soda containing sucralose or carbonated water (control)

108 rranted, particularly of the precise role of sodas containing artificial sweeteners in bladder sensat

109 consumed >/=1 cup (1 cup = 237 mL) sweetened soda/d had 16.3% higher estradiol concentrations compare

110 who consumed >/=1 serving of sugar-sweetened soda/d had a 63% (HR: 1.63; 95% CI: 1.15, 2.30; P-trend

111 (RR for consumption of >/=1 serving of diet soda/d when the 2 cohorts were pooled: 1.42; 95% CI: 1.0

112 Compared with 1 serving of sugar-sweetened soda/d, 1 serving of decaffeinated coffee/d was associat

113 stroke for >/= 1 serving of sugar-sweetened soda/d, compared with none, was 1.16 (95% CI: 1.00, 1.34

114 otal stroke for >/= 1 serving of low-calorie soda/d, compared with none, was 1.16 (95% CI: 1.05, 1.28

115 development of metacyclic promastigotes, as SODA/DeltasodA cultures were strongly depleted in this i

116 itochondrial oxidative damage and failure of SODA/DeltasodA promastigotes to differentiate into axeni

117 The utility of the indicator of latent soda demand was evaluated by assessing its association w

118 ce by enabling cell growth in oxygen through SodA-dependent and independent mechanisms.

119 uit drinks) and artificially sweetened (diet sodas, diet drinks) beverages from food-frequency questi

120 we demonstrate that a bulk of B. burgdorferi SodA directly associates with manganese, and a smaller p

121 ncer risk in individuals who consume regular soda do not permit the ruling out of chance as an explan

122 nducing agent, methyl viologen, but the sarA sodA double mutant was more resistant to the same stress

123 Consistent with that, SodA, DoxX, and SseA form a membrane-associated oxidored

124 urine, plasma, tablet, green tea, energy and soda drink).

125 caries increment were higher consumption of soda drinks, older age of child, greater weight-for-age,

126 In the present study, sodA (encoding manganese-cofactored superoxide dismutase

127 In this issue of the JCI, Soda et al. have identified clathrin-mediated endocytosi

128 A mutant of E. coli was strongly reduced for SodA export.

129 SODA expression above a critical threshold was also requ

130 manganese, greatly reduced SOD activity and SodA expression, suggesting that manganese regulates the

131 ganese to the Chelex-treated BSK-II enhanced SodA expression.

132 German chemical company Badische Anilin- und Soda-Fabrik.

133 foods consumed with the television off, less soda, fast food, fruit, and vegetables were consumed wit

134 We observed 3 dietary patterns: juice and soda; fast food and fruit drinks; and fruit, vegetable,

135 t, we cloned the homologous genes (glnA1 and sodA) from the rapid-growing, nonpathogenic Mycobacteriu

136 SSBs included soda, fruit drinks, sports and energy drinks, sweetened

137 atively averaged intakes of sugar-sweetened (sodas, fruit punches, lemonades, fruit drinks) and artif

138 MLSA, internal gyrB, sodA, full-length, and 5' 16S gene sequences were used t

139 include those encoding superoxide dismutase (sodA), fumarate dehydratase (fumC), bacterioferritin (bf

140 Inactivation of the sodA gene abolished L. lactis I-1631's beneficial effect

141 The S. epidermidis sodA gene expressed from a plasmid complemented a sodA m

142 The sodA gene was found to be a better target than the 16S-2

143 ive phenotype observed in the absence of the sodA gene.

144 primers based upon the superoxide dismutase (sodA) gene for development of a multiplex PCR.

145 d soda intake, particularly caffeinated diet soda, had higher symptom scores, urgency, and LUTS progr

146 Consumption of sugar-sweetened soda has been associated with an increased risk of cardi

147 Consequently, the metal cofactor for SodA has been postulated to be manganese.

148 Whereas B. burgdorferi SodA has evolved in a manganese-rich, iron-poor environm

149 In addition, the superoxide dismutase gene (sodA) has been identified as a potential target for spec

150 hioredoxin that either participate directly (SodA, HPI, and AhpC) or have key regulatory functions (F

151 nd of activity most likely represents a SodM-SodA hybrid protein.

152 gher consumption of regular, sugar-sweetened soda in men but not in women.

153 nd substitution of alternative beverages for soda in relation to stroke risk.

154 an indicator of area-level latent demand for soda in the Census Metropolitan Area of Montreal in 2012

155 to quantify SY in isotonic drinks and orange sodas, in the range of 7.8-39.7 mg L(-1), with relative

156 We examined the association of soda, including specific types of soda, and risk of hip

157 with limited power, neither regular nor diet soda increased risk of leukemia but were associated with

158 However, in men, >/=1 daily serving of diet soda increased risks of NHL (RR: 1.31; 95% CI: 1.01, 1.7

159 icular, Cys(83) mutation (C83S, absent in Fe-SODA) increased the Fe-SODB sensitivity toward peroxynit

160 SoDA inferred the correct gene segments more frequently

161 Caffeinated soda intake and green tea intake >/=1 cup/d (1 cup = 240

162 there was no significant association between soda intake and risks of NHL and multiple myeloma.

163 escents and (2) among adolescents, increased soda intake was twice as large when fast food was consum

164 At baseline, SSB and diet soda intake were assessed using a valid food frequency q

165 t) of Kool-aid, fruit juices, and nondietary soda intake, expressed as servings per week, and classif

166 Women with recently increased soda intake, particularly caffeinated diet soda, had hig

167 s were found for >/=1 cup cola/d and noncola soda intake.

168 1.02, 1.84; P for trend = 0.04) but not with soda intake.

169 de biochemical and genetic data showing that SodA is a manganese-dependent enzyme.

170 h previous work suggests that B. burgdorferi SodA is an iron-dependent superoxide dismutase (SOD), la

171 n at moderate consumption amounts, sweetened soda is associated with elevated follicular estradiol co

172 nsumption of aspartame- and sugar-containing soda is associated with risk of hematopoetic cancers.

173 ypanosoma brucei SODA ortholog suggests that SODA is essential for trypanosomatid survival.

174 mption of sugar-sweetened soda, but not diet soda, is associated with increased risk of seropositive

175 s within mycobacterial superoxide dismutase (SodA), L-alanine dehydrogenase (AlaDH), and L-glutamine

176 SodA lacks a classical signal sequence for protein expor

177 Our study provides insight into soda lake ecology, as well as a template to guide effort

178 water from natural alkaline sources such as soda lakes and hydrothermal oceanic vents.

179 ng data of microbial mats from four Canadian soda lakes indicate the presence of > 2,000 species of B

180 ied arsenic and sulfide rich hot springs and soda lakes where it was discovered.

181 vironments, microbial mats from two alkaline soda lakes, and saliva from multiple individuals.

182 In alkaline soda lakes, concentrated dissolved carbonates establish

183 ioalkalivibrio ubiquitous in saline alkaline soda lakes.

184 inity are known to promote NH(3) losses from soda lakes.

185 The use of baking soda leads to the continuous effervescence of CO(2) duri

186 can be detoxified by cytoplasmic lactoccocal SodA led to the finding that host antimicrobial-mediated

187 ue by performing (11)B NMR measurements on a soda lime borate glass that has been pressure-quenched a

188 induced changes in macroscopic properties of soda lime borate glasses compressed up to ~0.6 GPa are n

189 and carbon monoxide (CO) may be generated in soda lime canisters and may be inhaled by patients.

190 tic susceptibilities such as borosilicate or soda lime glass beads.

191 rowires into glass membranes at the end of a soda lime or lead glass capillary.

192 d, include the degradation of both agents by soda lime under certain circumstances during closed circ

193 The use of prepulled soda-lime glass capillaries allows one to bypass the irr

194 this is because of Mg out-diffusion from the soda-lime glass substrates and is not disadvantageous to

195 ass sandwich chips made from fused silica or soda-lime glass.

196 shaping of silver nanoparticles embedded in soda-lime glass.

197 parable to transparent polymers, silica- and soda-lime glasses.

198 hanism of silver nanoparticles embedded in a soda-lime silicate glass matrix.

199 emical and physical properties of commercial soda-lime slides affect the ability of these slides to b

200 A survey of commercial soda-lime slides yielded the surprising result that slid

201 bust, and spontaneous graphene n-doping on a soda-lime-glass substrate via surface-transfer doping fr

202 onductor that itself has been deposited onto soda-lime-glass, via surface-transfer doping from Na ato

203 These data indicate that while SodA may be the major SOD activity in S. aureus througho

204 The unique properties of B. burgdorferi SodA may represent adaptation to expression in the manga

205 BpuR bound to sodA mRNA in live B. burgdorferi, and a specific BpuR-bi

206 ch that viability and growth of an S. aureus sodA mutant are maintained.

207 complete attenuation of infectivity for the sodA mutant compared with control strains at 21 days pos

208 n the in vitro growth characteristics of the sodA mutant compared with the control strains.

209 An isogenic M. catarrhalis sodA mutant was constructed in strain 7169 by allelic ex

210 The sodA mutant was more susceptible to the effects of activ

211 However, only the viability of the sodM sodA mutant was reduced when MV was added during the lat

212 The sodA mutant was sensitive to an oxidative stress-inducin

213 r percentage of cell death upon treatment of sodA mutant with superoxide generators compared with its

214 sistant to the same stressor than the single sodA mutant.

215 , while the two lower bands were absent in a sodA mutant.

216 Viability of the sodA and sodM sodA mutants but not the sodM mutant was drastically red

217 Mycobacterium smegmatis, generated glnA1 and sodA mutants of M. smegmatis by allelic exchange, and qu

218 Neither the ssaB nor the sodA mutation affected sensitivity to phagocytic killing

219 gene expressed from a plasmid complemented a sodA mutation in S. aureus, and the protein formed a hyb

220 transformed into sodium hydroxide or caustic soda (NaOH) and hydrochloric acid (HCl).

221 ions of Egypt and the Levant used evaporitic soda (natron) mixed with Nile-derived sands.

222 Neither artificially sweetened soda nor fruit juice intake >/=1 cup/d was significantly

223 sis Our inability to generate L. amazonensis SODA null mutants and the lethal phenotype observed foll

224 ne SOD activity, which migrated similarly to SodA of S. aureus.

225 he manganese/iron-type superoxide dismutase (SodA) of Rhizobium leguminosarum bv.

226 cific BpuR-binding site was mapped 5' of the sodA open reading frame.

227 es of total SCBs and fruit juice, but not of soda or concentrate, were associated with a higher FMI [

228 d with those who consumed no sugar-sweetened soda or who consumed <1 serving/mo.

229 on is generally recommended (fried products, sodas or sugary drinks, fatty sweet products, processed

230 mediated silencing of the Trypanosoma brucei SODA ortholog suggests that SODA is essential for trypan

231 .001), fried foods (P(trend)=0.02), and diet soda (P(trend)=< 0.001) also were adversely associated w

232 Subjects in the juice and soda pattern had higher energy intakes and lowest BMI.

233 ble models, each additional serving of total soda per day was associated with a significant 14% incre

234 Thus, we hypothesized that lactococcal SodA played a role in attenuating colitis.

235 This study demonstrates that M. catarrhalis SodA plays a critical role in the detoxification of endo

236 cycling compound that produces ROS, and that SodA plays a protective role against the streptonigrin.

237 These studies indicate that SodA plays an important role in combating oxidative stre

238 On average, a phospho-soda preparation provided significantly less residual fl

239 lectrolyte solution preparation or a phospho-soda preparation the day prior to CT colonography.

240 The marRAB and sodA promoters could both be saturated by MarA and SoxS

241 Collectively, our data suggest that SODA promotes Leishmania virulence by protecting the par

242 proteins of S. aureus and the S. epidermidis SodA protein exist as dimers.

243 d protein with 75% identity to the S. aureus SodA protein.

244 oth hybrid SOD forms as well as the SodM and SodA proteins of S. aureus and the S. epidermidis SodA p

245 and produced lower levels of NapA (Dps) and SodA, proteins involved in the oxidative stress response

246 crobial-mediated lysis is a prerequisite for SodA release and SodA's extracytoplasmic O2 (-) scavengi

247 soda (RR: 1.19; 95% CI: 1.02, 1.38) and diet soda (RR: 1.12; 95% CI: 1.03, 1.21) and also did not sig

248 cantly elevated in consumers of both regular soda (RR: 1.19; 95% CI: 1.02, 1.38) and diet soda (RR: 1

249 However, in all three cases (whisky and club soda; rum with cola; gin and tonic water), MEC was quick

250 obials may play a critical role in mediating SodA's bioaccessibility.

251 lysis is a prerequisite for SodA release and SodA's extracytoplasmic O2 (-) scavenging.

252 erarchical Bayesian spatial model to data on soda sales from 1,097 chain retail food outlets.

253 nce, full-length and 5' partial 16S gene and sodA sequence analyses failed to correctly assign all st

254 howing that gyrB is superior to 16S gene and sodA sequence analyses for VGS species identification.

255 (i) EcoRI ribotyping, combined with hylB and sodA sequencing, provides a discriminatory subtype analy

256 B. burgdorferi SodA shows strong overall homology with other members of

257 endogenous level of H2S is reduced in fur or sodA sodB cells but restored after the addition of an ir

258 d rescued an Escherichia coli double mutant (sodA sodB) under conditions that are otherwise lethal.

259 ons were 0.2, 0.3, 0.8, and 1.9 mM for arsB, sodA sodB, crc, and gor mutants, respectively, and were

260 e aerobic growth defects of E. coli QC774, a sodA sodB-deficient mutant, demonstrated the functional

261 emented it as web-accessible software called SoDA (Somatic Diversification Analysis).

262 is GSs (in the glnA1 strain) or SODs (in the sodA strain), in contrast to previous observations in wi

263 ture, from chimneys at hydrothermal vents to soda straws in caves.

264 detrimental effect of a constituent of diet soda, such as aspartame, on select cancers, the inconsis

265 high manganese is necessary to activate the SodA superoxide dismutase (SOD) essential for virulence.

266 Among these was the SodA superoxide dismutase, which is essential for mammal

267 ermidis and determined to be more similar to sodA than to sodM of S. aureus.

268 by one isolate due to a missense mutation in sodA that produced a premature stop codon.

269 hypothesis, we examined the specific role of SODA, the mitochondrial SOD isoform in Leishmania amazon

270 than 1 serving per month of sugar-sweetened soda, the multivariate relative risk of gout for 1 servi

271 The ability of recombinant SodA to rescue the aerobic growth defects of E. coli QC7

272 to deliver antioxidant, colitis-attenuating SodA to the inflamed intestinal mucosa, and host antimic

273 Export of the R. l. bv. viciae SodA to the periplasm was not limited to the genus Rhizo

274 A tatC mutant of R. l. bv. viciae exported SodA to the periplasm, ruling out export of SodA as a co

275 Susceptibility of T. cruzi Fe-SODA toward peroxynitrite was similar to that reported p

276 Reporter gene studies indicated that sodA transcription could be variably induced in iron-sta

277 y MarA resulted in greater marRAB and lesser sodA transcription than did saturation by SoxS, implying

278 r aerobic conditions, the levels of sodM and sodA transcription, in particular the sodM transcript, a

279 isolates), ATPase (types IA1, IA2, and IC), sodA (types IA2 and IB), atpD (type II), and recA (type

280 We cloned the MnSOD gene, sodA, using the expression vector pBAD, overexpressed th

281 us: -32% (RIRR, 0.68 [CI, 0.65 to 0.72]) for soda versus -11% (RIRR, 0.89 [CI, 0.82 to 0.97]) for ene

282 ed amino acid sequence of the S. epidermidis sodA was 92 and 76% identical to that of the SodA and So

283 c conditions, transcription of both sodM and sodA was considerably enhanced in the sarA mutant compar

284 Iron starvation induction of sodA was greatest in the wild-type strain and least in t

285 of Saccharomyces cerevisiae, B. burgdorferi SodA was inactive.

286 Increasing intake of sugar-sweetened soda was independently associated with increasing risk o

287 ral differences, the crystal structure of Fe-SODA was solved at 2.2 A resolution.

288 The export of SodA was unaffected in a secB mutant of E. coli, but its

289 nsumption of sugar-sweetened and low-calorie sodas was associated with a significantly higher risk of

290 e cola, and other sugar-sweetened carbonated soda) was obtained from a validated food-frequency quest

291 d the gene responsible for the SOD activity, sodA, was isolated from a recent pediatric clinical isol

292 he manganese-dependent superoxide dismutase, SodA, was significantly less virulent than wild-type in

293 s well as high-fructose corn syrup-sweetened soda, we see the pervasive influence of corn as an ingre

294 ed meat, processed meat, and sugar-sweetened soda were computed with responses to a 120-item food-fre

295 4, P = 0.02) and the frequent consumption of soda were positively associated with visceral obesity (O

296 The 10 N-terminal amino acid residues of SodA were sufficient to target the reporter protein alka

297 subset of its target mRNAs (fdoG, nuoA, and sodA), whereas the sodB and sdhC targets are barely affe

298 s the need for import and storage of caustic soda, which typically represents a cost and a hazard.

299 icantly differ between colas and noncolas or sodas with or without caffeine.

300 om low-symmetry enzyme sites such as that of SodA, with a minority pool of LMW Mn(2+) complexes that