ライフサイエンスコーパス: sodium

1 nd shellfish, sugar-sweetened beverages, and sodium).

2 ce treated with azoxymethane-dextran sulfate sodium.

3 enerate grazing lawns: dependable sources of sodium.

4 ast to other alkali ions such as lithium and sodium.

5 gy storage owing to the natural abundance of sodium.

6 ared with those found for RMs generated with sodium 1,4-bis-2-ethylhexylsulfosuccinate (Na-AOT).

7 Following addition of sodium 1-(13) C-acetate, parahydrogen bubbling within a

8 50 x 103/muL (1.92 [1.02-3.60]), lower serum sodium (1.12 [1.02-1.23 per 1 mmol/L decrease), and intr

9 ate, sodium chloride, gallic acid and pH 5.0 sodium acetate buffer system.

10 (i) preconditioning the soil to increase the sodium adsorption ratio, (ii) extracting colloids/NPs fr

11 (larger) and d(43) 1.2-2.7 um (smaller), and sodium alginate (0.5%, w/w) induced water gelation on cr

12 The method is also compatible with sodium amides, with the latter showing excellent promise

13 Hyperreabsorption of sodium and chloride induces tubuloglomerular feedback fr

14 nd to determine the content of protein, fat, sodium and density was demonstrated.

15 itors attenuate the proximal reabsorption of sodium and glucose, normalize tubuloglomerular feedback

16 Increased sodium and magnesium and decreased albumin and lactate l

17 nsequence of the properties of voltage-gated sodium and potassium channels.

18 importance is being seriously challenged by sodium and potassium, as the alkali-metal mediation of o

19 opose a mechanism where the coupling between sodium and proton translocation is facilitated by a seri

20 dy, we present a suite of hypotheses for how sodium and sugary exudate availability should vary for a

21 or alpha(1) -m, and suggest that reduced PT sodium and water reabsorption in Fanconi syndrome may co

22 appetite circuits for major nutrients-water, sodium, and food-operate on unique driving and quenching

23 that precise control of ionic flux (calcium, sodium, and potassium) contributes to in utero developme

24 pital admission, serum levels of albumin and sodium, and white blood cell count, to identify metaboli

25 However, in normal conditions, sodium appetite is suppressed to prevent homeostatic dev

26 ibition of these neurons specifically drives sodium appetite, even during euvolemic conditions.

27 achial nucleus (LPBN(Htr2c) neurons) inhibit sodium appetite.

28 c expressed by LPBN neurons is to disinhibit sodium appetite.

29 yered growth, which correlates to the use of sodium as an inorganic structure-directing agent, wherea

30 nificant increase in fractional excretion of sodium at day 3, which was absent at week 6 (mean differ

31 Auranofin, sodium aurothiomalate and aurothioglucose inhibited 48 c

32 lthio)-4-(het)arylidene-but-1-en-3-ones with sodium azide at higher temperatures.

33 1,3-bis(het)arylmonothio-1,3-diketones with sodium azide in the presence of IBX catalyst, has been r

34 over 9 weeks in PBS + 0.02% Tween 80 + 0.02% sodium azide pH 7.4 (PBST) at 37 degrees C.

35 man epidermal keratinocytes at 4(o)C or with sodium azide prevented SNA uptake, suggesting active end

36 synthesized in high yield by the reaction of sodium azide with 2H-azirine-2-carbonyl chlorides, gener

37 oxyl radicals (mannitol) and singlet oxygen (sodium azide) and carbon-centered radicals (DMPO) were t

38 he presence of sorbic acid, isopropanol, and sodium azide.

39 c esters via a copper-mediated reaction with sodium azide.

40 sterone was measured in participants in high sodium balance with suppressed renin activity.

41 calated into the interlayers of hierarchical sodium bentonite.

42 Sodium benzoate (SBA) is a widely-used additive for prev

43 Oral sodium bicarbonate (NaHCO(3)) may preserve kidney functi

44 PS was treated with acetic acid and sodium bicarbonate to improve pallatability.

45 bis(fluorosulfonyl)imide (FSI) anions, with sodium bis(trifluoromethanesulfonyl)imide (NaTFSI) as a

46 r a range of -0.36 to 0.76 using the aqueous sodium bisulfate system.

47 Nondenaturing sodium bisulfite treatment catalyzes the conversion of u

48 when activated with a hydride source such as sodium borohydride, cleanly and selectively dehalogenate

49 The rapid and simple quantification of sodium by the TT-IR and TIE showed the possibility of a

50 how FHF2 orchestrates the interdependency of sodium, calcium, and gap junctional conductances to safe

51 modulates cardiomyocyte contractility via a sodium-calcium exchanger (NCX) mediated pathway.

52 assium (K(+)) currents (~30%), and increased sodium/calcium exchanger (NCX) activity (~52%).

53 Sodium carbonate addition increased pH and caused a cons

54 Mixing 4 mM sodium carbonate with the 2 mM tungstate was enough to p

55 gum, guar gum, gellan gum, xanthan gum, and sodium carboxymethyl cellulose.

56 DEs were produced with combined use of sodium caseinate, diacetyl tartaric acid esters of mono-

57 Overexpression in the epithelial sodium channel (ENaC) in membrane platelets can be relat

58 -sensing ion channels (ASICs) and epithelial sodium channel (ENaCs), these channel families display v

59 Voltage-gated sodium channel (VGSC) beta1 subunits are multifunctional

60 indirectly reduces principal cell epithelial sodium channel abundance and function.

61 orption, transepithelial voltage, epithelial sodium channel activity, and pendrin abundance and subce

62 e reduced chloride absorption and epithelial sodium channel activity, despite principal cell mineralo

63 bited increased expression of the epithelial sodium channel alpha-subunit, largely abolished basolate

64 of antiarrhythmic action and concluded that sodium channel block alone is responsible for flecainide

65 ect on arrhythmia burden, despite comparable sodium channel block.

66 ytes-lacking intact sarcolemma and devoid of sodium channel contribution-flecainide, but not its anal

67 (2020) report that the epithelial sodium channel ENaC, which serves as the salty receptor,

68 Reduced cardiac sodium channel expression is a known causal mechanism in

69 Cardiac sodium channel expression, I(Na) and atrial action poten

70 Missense variants in the SCN8A voltage-gated sodium channel gene are linked to early-infantile epilep

71 Sodium channel inhibition by GS-967 and eleclazine has u

72 GS-967 and eleclazine (GS-6615) are novel sodium channel inhibitors exhibiting antiarrhythmic effe

73 Nav1.6 is the primary voltage-gated sodium channel isoform expressed in mature axon initial

74 The voltage-gated sodium channel isoform Na(V)1.7 is highly expressed in d

75 er than 200-fold selectivity over off-target sodium channel isoforms, Na(V)1.1-1.6 and Na(V)1.8.

76 sed by de novo gain-of-function mutations of sodium channel Na(v) 1.6 that result in neuronal hyperac

77 Voltage-gated sodium channel Na(v)1.5 generates cardiac action potenti

78 Voltage-gated sodium channel Na(V)1.7 is a genetically validated targe

79 ted the effect of LITAF on the voltage-gated sodium channel Nav1.5, which is critical for cardiac dep

80 Our data show that TTX-S sodium channel Nav1.6 is involved in the functional chan

81 Importantly, the expression of the sodium channel protein NaV1.5 was altered in AV nodal ce

82 hysiological regulator of the heart-specific sodium channel, Nav1.5.

83 ) are proton-gated members of the epithelial sodium channel/degenerin (ENaC/DEG) superfamily of ion c

84 X), a neurotoxin that binds to voltage-gated sodium channels (Na(v) proteins), arresting electrical a

85 ytes pretreated with tetrodotoxin to inhibit sodium channels and isolate the effect of flecainide on

86 arrhythmogenic Ca release even when cardiac sodium channels are blocked.

87 Enrichment of sodium channels at nodes of Ranvier, a hallmark of myeli

88 Previous studies have shown that sodium channels cluster together in specific cellular su

89 Voltage-gated sodium channels comprise an ion-selective alpha-subunit

90 Through this mechanism, sodium channels effectively measure the frequency of act

91 e unbinding rate of these two compounds with sodium channels.

92 possible mechanism for CBD interactions with sodium channels.

93 ), in 64% yield, by atomic manipulation on a sodium chloride bilayer on Cu(111) at 5 K, and imaged by

94 wounding, specifically the concentration of sodium chloride in the immediate vicinity of the wound.

95 vaporates from the droplets, solutes such as sodium chloride in the media become more concentrated.

96 f GBM cell culture medium was adjusted using sodium chloride or water.

97 R)-IO sensor was suspended in a sterile 0.9% sodium chloride solution and placed in a water bath at 3

98 crystals isostructural to caesium chloride, sodium chloride, aluminium diboride and K(4)C(60) are se

99 stilled water, polyglycerol polyricinoleate, sodium chloride, gallic acid and pH 5.0 sodium acetate b

100 were reducing, with high pH (>7.5) and high sodium/chloride (Na/Cl) ratios resulting from cation exc

101 hieved at pilot-scale by the extraction with sodium citrate and purification with membrane separation

102 ties, similar to the already known inhibitor sodium citrate.

103 The majority (71%) of US dietary sodium comes from restaurant and packaged foods.

104 follow-up, relative to control, a dialysate sodium concentration of 135 mmol/L did not change the le

105 e a simple formula relating AIS geometry and sodium conductance density to the somatic voltage thresh

106 To address these challenges, a novel high sodium content (0.85) and plateau-free P2-type cathode-N

107 vity of these neurons is regulated by bodily sodium content, and their activation can rapidly suppres

108 x) is formed by intercalation of chlorophyll sodium copper salt into a melamine-based supramolecular

109 Here, the application of sodium correction significantly reduced overestimations

110 NaCT (SLC13A5; mINDY), a sodium-coupled citrate transporter, is the mammalian ort

111 A T-tubules sodium current could, however, not be demonstrated.

112 he density of Nav1.5-generated voltage-gated sodium current I (Na) and Nav1.5 surface protein levels

113 s and mouse models have demonstrated altered sodium current properties.

114 ved from lithium responders while increasing sodium currents and reducing fast potassium currents.

115 ivity (infiltration with sodium vanadate and sodium cyanide; plant exposure to carbon monoxide) can r

116 Our lead-free composition of sodium-deficient sodium niobate contains only three elem

117 SLC22A15 transport of several substrates was sodium-dependent and exhibited a higher Km for ergothion

118 itated-glucose transporters (GLUTs), not for sodium-dependent glucose cotransporters (SGLTs), which h

119 The synthesis of sodium diazeniumdiolates has proven to be challenging du

120 -) mice by administration of dextran sulfate sodium; disease severity was determined based on body we

121 ture of ball-milled samples, underlining the sodium disorder and showing that a local tetragonal fram

122 Extensive use of Sodium Dodecyl Sulfate (SDS) in households, agricultural

123 s were investigated, including the amount of sodium dodecyl sulfate (SDS), ethanol, and ionic strengt

124 PFOS); perfluorohexanesulfonic acid (PFHxS); sodium dodecyl sulfate (SDS); and sodium tetradecyl sulf

125 eral biotherapeutic proteins were studied in sodium dodecyl sulfate capillary gel electrophoresis (SD

126 or their capability to distinguish among PE, sodium dodecyl sulfate, and stearates.

127 Sodium dodecyl sulfate, which is commonly applied in mic

128 ice were more susceptible to dextran sulfate sodium (DSS)-induced colitis, manifested by increased we

129 se compared to free 6-S in a dextran sulfate sodium (DSS)-induced mouse model of colitis.

130 ed by oral administration of dextran sulfate sodium (DSS).

131 effective treatment, dipping fillets into a sodium erythorbate solution, yielding a shelf-life of 15

132 y drugs (NSAIDs) such as ibuprofen, naproxen sodium, etodolac, diclofenac, and ketorolac in this prot

133 Dietary sodium restriction reduced sodium excretion from 160 to 64 mmol per day.

134 sitron emission tomography (PET) using (18)F-sodium fluoride ((18)F-fluoride) to detect microcalcific

135 valuate the interobserver agreement in (18)F-sodium fluoride (NaF) PET/CT for the detection of bone m

136 (1) (18)F-fluorodeoxyglucose (FDG) and (18)F-sodium fluoride (NaF) uptake in culprit versus nonculpri

137 nriched in magnesium flanking a core rich in sodium, fluoride and carbonate ions; this sandwich core

138 at plant exudates are an important source of sodium for grassland consumers.

139 Sodium-free 10% dextrose was used as the DSR solution.

140 The sodium glucose cotransporter 2 (SGLT2) inhibitor empagli

141 as successfully targeted by Dapagliflozin, a sodium glucose cotransporter 2 inhibitor, in clinical tr

142 e variable effects on cardiovascular events, sodium glucose cotransporter 2 inhibitors and glucagon-l

143 ger 3 and SGLT1, which regulate transport of sodium, glucose, and water, compared with tissues from c

144 Lowering blood sugar by the sodium-glucose co-transporter 2 inhibitor empagliflozin

145 Sodium-glucose co-transporter-2 inhibitors and the risk

146 lucagon-like peptide-1 receptor agonists and sodium-glucose co-transporter-2 inhibitors as combinatio

147 Sodium-glucose cotransport 2 inhibitors (SGLT2i) lower p

148 wth of the proximal tubule, which (alongside sodium-glucose cotransport) further limits urinary gluco

149 Sodium-glucose cotransporter 2 (SGLT2) inhibition reduce

150 The sodium-glucose cotransporter 2 inhibitor empagliflozin r

151 Sodium-glucose cotransporter 2 inhibitors (SGLT2i), a ne

152 e emergence of new glucose-lowering agents - sodium-glucose cotransporter 2 inhibitors and incretin t

153 Sodium-glucose cotransporter 2 inhibitors improve outcom

154 clinical trials established the benefits of sodium-glucose cotransporter 2 inhibitors in patients wi

155 death, adding further support for the use of sodium-glucose cotransporter 2 inhibitors in primary and

156 Sodium-glucose cotransporter 2 inhibitors reduce the ris

157 Sodium-glucose cotransporter 2 inhibitors reduce the ris

158 tial significance of ketosis associated with sodium-glucose cotransporter 2 inhibitors.

159 SGLT2 (sodium-glucose cotransporter 2) inhibitors have been sho

160 ar effects of ertugliflozin, an inhibitor of sodium-glucose cotransporter 2, have not been establishe

161 ndated cardiovascular outcome trials for all sodium-glucose cotransporter type 2 inhibitors and the r

162 en though important, may not be critical for sodium-glucose cotransporter type 2 inhibitors in view o

163 Sodium-glucose cotransporter-2 (SGLT-2) inhibitors reduc

164 Inhibition of the sodium-glucose cotransporter-2 (SGLT2i) improves outcome

165 Sodium-glucose cotransporter-2 inhibitors and the risk f

166 type 2 diabetes, despite dual inhibition of sodium-glucose transporter-2 and the renin-angiotensin s

167 Sodium/glucose cotransporter 2 (SGLT2) inhibitors were d

168 receptor-neprilysin inhibitors [ARNIs], and sodium/glucose cotransporter 2 [SGLT2] inhibitors) reduc

169 aged >18 years old, had a predialysis serum sodium >=135 mM, and were receiving hemodialysis at home

170 ogether this clarifies TMC-1's importance in sodium hedonics and offer molecular insight into salt ch

171 osition and stripping of metallic lithium or sodium held within a large number of parallel hollow tub

172 hown to affect blood pressure by influencing sodium homeostasis, and the effects of another GWAS sign

173 controlled reduction of tertiary amides by a sodium hydride/sodium iodide composite, in situ treatmen

174 pical levels and diffuse subapical levels of sodium hydrogen exchanger 3 and SGLT1, which regulate tr

175 nto the perfusate: saline vehicle (control); sodium hydrogen sulphide (NaHS); NaHS plus glibenclamide

176 The membranes were also treated with sodium hydroxide (NaOH) to increase membrane selectivity

177 -5,5-dimethylimidazolidine-2,4-dione (DMDM), sodium hydroxymethylglycinate (SMG), 2-(hydroxymethyl)-2

178 ng noncoding variants in the Hcn4 (potassium/sodium hyperpolarization-activated cyclic nucleotide-gat

179 were analyzed for creatinine, bilirubin, and sodium in all transplant centers within United Network f

180 ication of an existing approach to determine sodium in food sauces, involving enthalpimetric reaction

181 repared meals, investigating optimal dietary sodium in heart failure comes with challenges, including

182 ificant decrease in the amount of sugars and sodium in several groups of packaged foods and beverages

183 The deficient sodium in the P2-type cathode easily induces the bad str

184 rizes evidence for policy progress to reduce sodium in the US food supply and the American diet.

185 recover from and succumb to dextran sulfate sodium-induced colitis due to prolonged intestinal infla

186 c wounding and recovery from dextran sulfate sodium-induced colitis.

187 gic flux, reduced cellular stress, decreased sodium influx into cells, and restoration of mitochondri

188 o use, alcohol, diet, physical activity, and sodium intake), metabolic factors (ie, lipids, blood pre

189 y elevated blood pressure, water intake, and sodium intake, while optogenetic inhibition produced the

190 t, and their activation can rapidly suppress sodium intake.

191 , perceived thirst or xerostomia, or dietary sodium intake.

192 ction of tertiary amides by a sodium hydride/sodium iodide composite, in situ treatment of the result

193 Cultured cells were treated or not with sodium iodoacetate (IAA; glycolysis inhibitor) plus 2,4-

194 this site blocks the transmembrane-spanning sodium ion translocation pathway, providing a molecular

195 es, which undergo redox reactions coupled to sodium ion uptake and release.

196 , we show that this cation is a stably bound sodium ion, and although it is not a transported substra

197 ment of both anode and cathode materials for sodium-ion batteries (SIBs).

198 ncreasing interest in the development of new sodium-ion batteries and new analytical methods to non-i

199 Sodium-ion batteries have captured widespread attention

200 lizing high-performance P2-type cathodes for sodium-ion batteries.

201 he cathode material used in a lithium-ion or sodium-ion battery is alkali-rich, this can increase the

202 vailable electrode materials, especially for sodium-ion layered oxides, motivating the exploration of

203 monstrated to access similar capacities as a sodium-ion or potassium-ion cathode.

204 n; in plant consumers, particularly animals, sodium is essential to running costly Na-K ATPases.

205 In plants, sodium is found in low concentrations and has little met

206 r interleukin-6 levels correlated with lower sodium levels (p = 0.017).

207 In exploratory analysis, the association of sodium levels and interleukin-6 levels (which has been l

208 very narrow, and more frequent monitoring of sodium levels could avoid toxicity.

209 tilation, and discharge home compared across sodium levels using Kruskal-Wallis and chi-square tests.

210 duced (beta-MnO(2)) to 1.18 +/- 0.01 W/m(2) (sodium manganese oxide).

211 a novel method to produce healthier reduced-sodium meat products.

212 nitored compared to those treated with 1.25% sodium metabisulphite (SMS-1.25) and the control (withou

213 beneficial for the growth of a stable SEI on sodium metal.

214 Dialysate cooling and sodium modelling may prevent haemodynamic instability an

215 ion potentials are initiated by an influx of sodium (Na(+)) ions via voltage-gated Na(+) channels.

216 Persistently depolarizing sodium (Na(+)) leak currents enhance electrical excitabi

217 were sensitive to the specific voltage-gated sodium (Na(V)) channel blocker tetrodotoxin.

218 Voltage-gated sodium (Na(V)) channels are a functional hallmark of the

219 Voltage-gated sodium (Na(V)) channels are pore-forming transmembrane p

220 se tetrodotoxin (TTX) to block voltage-gated sodium (Na(v)) channels as a chemical defense against pr

221 Voltage-gated sodium (Na(V)) channels drive neuronal excitability and

222 Rechargeable sodium (Na) based batteries have gained tremendous resea

223 d that relatively small changes in perfusate sodium ([Na(+)](o)) composition significantly affect car

224 ur lead-free composition of sodium-deficient sodium niobate contains only three elements (Na, Nb, and

225 ial and unsustained conversion of refractory sodium nitrate (NaNO(3)) was observed at the inlet tempe

226 Neither sodium nitrate nor sodium nitrite supplementation altere

227 .075 uL/g TP and 0.075 uL/g PM; T5: 50 mg of sodium nitrite and 0.150 uL/g PM.

228 e; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium ni

229 catalyse nitric oxide generation from benign sodium nitrite in the presence of modest electric fields

230 Neither sodium nitrate nor sodium nitrite supplementation altered mitochondrial cou

231 oked pork sausages produced with 50 mg/kg of sodium nitrite was investigated.

232 dium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium nitrite, 0.075 uL/g TP and 0.075 uL/g PM; T5: 50

233 oked sausages produced with reduced level of sodium nitrite.

234 Five batches were produced: T1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg o

235 1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL

236 l ester, while iontophoresis of the NO donor sodium nitroprusside eliminated the observed differences

237 Dialysate sodium of 135 mmol/L did not reduce left ventricular mas

238 (23) Na T(2) -contrast MRI of metallic sodium offers a clear, routine method for observing and

239 ed in electrolytes containing over 50 wt% of sodium or 30 wt% of potassium carbonate, or at electroly

240 Hedgehog signaling pathway, driven by either sodium or potassium gradients.

241 ction therapy with rabbit ATG, mycophenolate sodium, or mycophenolate mofetil and rapid withdrawal of

242 foods and beverages containing added sugars, sodium, or saturated fats that exceed set nutrient or ca

243 The interfacial site is selective for sodium over other cations, except for Li(+), which compe

244 f concern (total sugars, saturated fats, and sodium, per 100 g/100 mL) and the proportion of products

245 bstituted tetrahydro-beta-carbolines undergo sodium periodate oxidative ring expansion in the presenc

246 s in coupling paracellular and transcellular sodium permeability.

247 with oxoammonium cations in the presence of sodium persulfate as a terminal oxidant, the N-acyl pyra

248 ssary to evaluate the efficacy and safety of sodium phenylbutyrate-taurursodiol in persons with ALS.

249 Sodium phenylbutyrate-taurursodiol resulted in slower fu

250 nt solution conditions (14 uM Abeta42, 20 mM sodium phosphate, 200 uM EDTA, pH 6.8).

251 The sodium-phosphate cotransporter NPT2a plays a key role in

252 It is unclear if the (99m)Tc-sodium phytate ((99m)Tc-SP) is as reliable as the gold-s

253 th wide internal spaces that can accommodate sodium polysulfides and withstand volumetric expansion.

254 digested and separated using a high-density sodium polytungstate solution (SPT) and microplastic par

255 We report the detection of lithium, sodium, potassium, and calcium in the atmosphere of the

256 The dendrite-free sodium-potassium (Na-K) liquid alloy composed of two alk

257 d aniline derivatives, by using a mixture of sodium powder, crown ether, trimethylsilyl bromide and N

258 of the surface temperature generated in the sodium precipitation reaction and development of softwar

259 3-86] to 60% [95% CI 51-69]) and in "high in sodium" products (in savory spreads, cheeses, ready-to-e

260 s articulated with voltage-dependent neutral sodium-proton exchanger (NHE).

261 the shapes of a prokaryotic and a eukaryotic sodium/proton antiporter homologue.

262 e strictly structural role fulfilled by this sodium provides new context to understand the structures

263 surface plasmon polaritons supported at the sodium-quartz interface can reach 200 micrometres at nea

264 conducting an RCT of the oft-cited issue of sodium reduction on cardiovascular outcomes and then pro

265 and other stakeholders made some progress on sodium-reduction actions.

266 ration made no progress in setting mandatory sodium-reduction standards, industry made some progress

267 Sodium removal was substantially higher with DSR (4.5+/-

268 lorothiazide (5 mg/50 mg daily) with dietary sodium restriction (60 mmol per day).

269 P (SBP; from 138 to 124 mm Hg) compared with sodium restriction (from 134 to 129 mm Hg), as well as a

270 distal diuretics are noninferior to dietary sodium restriction in reducing BP in patients with CKD s

271 Dietary sodium restriction reduced sodium excretion from 160 to

272 The mechanism of sodium retention and its location in kidney tubules may

273 s a major determinant of the tubular site of sodium retention in nephrotic mice.

274 Instead, I suggest that sodium's critical role in limiting herbivore performance

275 MI was induced in animals and borax, a sodium salt of boron, was administered for 7 days, p.o.,

276 oriented channels, a chromophore (resorufin sodium salt) was successfully embedded into the channels

277 owing to the relatively lower solubility of sodium salts compared to its alkaline cousins (Li, K, an

278 evels of nutrients of concern (i.e., sugars, sodium, saturated fat, or energy) according to Chilean n

279 The structure presents a chemical basis for sodium selectivity, and a constricted gate suggests a cl

280 Such a decrease was not observed in sodium (SS- or SN-PB) droplets, and no pH differences we

281 d hypomagnesaemic, with reduced interstitial sodium stores determined by (23)Na-magnetic resonance im

282 pecifically in mice with infectious, dextran sodium sulfate (DSS)-, and T-cell-induced colitis.

283 of Gal2 in colitis, we employed the dextran sodium sulfate (DSS)-induced acute colitis model in mice

284 n was measured; some mice were given dextran sodium sulfate to induce colitis and/or gavage with an a

285 t-free (control) diet and then given dextran sodium sulfate to induce colitis; we also studied Il10(-

286 epithelial cell-derived IL-33 during dextran sodium sulfate-induced colitis.

287 given oral Salmonella Typhimurium or dextran sodium sulfate.

288 esulting alpha-bromo o-arylacetophenone with sodium sulfinate (RSO(2)Na), and (iii) the CuBr(2)-media

289 Neurotransmitter:sodium symporters (NSS) are conserved from bacteria to m

290 d (PFHxS); sodium dodecyl sulfate (SDS); and sodium tetradecyl sulfate (TDS).

291 sinter mineralogy were dominated by borates, sodium, thiosulfate, sulfate, sulfite, sulfide, bicarbon

292 The promising P2-layered sodium transition metal oxides (P2-Na(x)TmO(2)) often su

293 ly asymmetric, revealing probable proton and sodium translocation pathways.

294 hagic flux and by exerting direct effects on sodium transport and inflammasome activation.

295 the possibility of a selective reaction for sodium, using aluminum nitrate, potassium and ammonium f

296 g stem metabolic activity (infiltration with sodium vanadate and sodium cyanide; plant exposure to ca

297 The sodium was also quantified by Thermal Infrared Enthalpim

298 In HIV-infected patients, higher plasma sodium was uniformly associated with good prognosis, whe

299 a reduction in paracellular permeability to sodium, whereas decreased claudin-8 abundance was associ

300 Because both diets reduced urinary sodium without adverse safety or quality of life signals