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1 ounterpart Na-exposed rats (pair-fed 216 ppm sodium acetate).
2 cetonitrile, followed by a cleanup step with sodium acetate.
3 fiber diet, or normal chow supplemented with sodium acetate.
4 ophoresis in solutions containing 0.01-1.0 M sodium acetate.
5 thanebis(diazene-N-oxide-N'-hydroxylate) and sodium acetate.
6 degrees C to produce methane by pyrolysis of sodium acetate.
7 thanebis(diazene-N-oxide-N'-hydroxylate) and sodium acetate.
8 es from 9:1 DMF/water mixtures buffered with sodium acetate.
9 ses for both Whatman 1PS paper and 30% TlNO3/sodium acetate.
10 s concentrations of potassium, rubidium, and sodium acetate.
11 ot to a normal meal or one supplemented with sodium acetate.
12 in calli supplemented with [U-(13)C]glucose, sodium acetate (1,2-(13)C2) and sodium octanoate (1,2,3,
13                            At pH 5.4 in 20mM sodium acetate, 100mM NaCl the temperature of maximum st
14 ere administered lead acetate, 100 mg/kg, or sodium acetate, 36 mg/kg (control), by i.p. for 7 days.
15 urbit[6]uril analogue (1) in aqueous buffer (sodium acetate, 50 mM, pH 4.74, 25 degrees C) toward a v
16                                  (Ecofix and sodium acetate-acetic acid-formalin), and one each from
17 n, unfixed, or fixed in 5 or 10% formalin or sodium acetate-acetic acid-formalin).
18  the literature conditions (acetic anhydride/sodium acetate) affords both the indole 6 and the indoli
19 phaE328A was sensitive to chemical rescue by sodium acetate, an effect that was not reproduced by pho
20  product at 100 degrees C in the presence of sodium acetate and 1% sodium dodecyl sulfate generates f
21                                              Sodium acetate and ammonium chloride (NH4Cl) were used t
22                    The spray deposition of a sodium acetate and carbonate buffer mixture at pH 10.3 o
23 tified via intravenous infusion of [1-(13)C] sodium acetate and mass isotopomer distribution analysis
24  of eicosanoids were poorly resolved by IMS, sodium acetate and metal adducts (e.g., Li, Na, Ag, Ba,
25 creased levels of select components, notably sodium acetate and sodium bicarbonate, restored the leve
26 l cellulose (MC) core with anthocyanidin and sodium acetate, and chitosan/gallotannin-based shell.
27 l cellulose (MC) core with anthocyanidin and sodium acetate, and chitosan/gallotannin-based shell.
28 expression included NaCl, sucrose, mannitol, sodium acetate, and choline chloride but not urea.
29 dium formate, [1-(13)C]glycine, and [2-(13)C]sodium acetate, and our technique is immediately extenda
30 r of salts (sodium sulfate, sodium fluoride, sodium acetate, and sodium chloride) on the thermodynami
31 nditions, using solid potassium carbonate or sodium acetate as a base.
32 ent, 3.5% (wt%) DCl in D(2)O as the solvent, sodium acetate as an internal standard, and heating at 9
33 ed with both Whatman 1PS paper and 30% TlNO3/sodium acetate as solid matrixes.
34 in was inhibited by acetylsalicylic acid and sodium acetate as well as by N-acetylated glucosamine an
35  situ from partial transformation of aqueous sodium acetate at 300 degrees C and 2.4-3.5 GPa and that
36 , potassium phosphate, potassium acetate and sodium acetate) at varying concentrations (0.8 to 1.6 M)
37                 Inexpensive reagents, namely sodium acetate, benzophenone, water, and acetonitrile, a
38 ry of the lipid A released by treatment with sodium acetate buffer (pH 4.5).
39 molar mixtures of Pb(2+) and Fe(2+) in 0.1 M sodium acetate buffer (pH 5) were detected.
40 al and electrochemical parameters used were: sodium acetate buffer (pH = 4.3), an accumulation potent
41 u were labeled with DOTATATE by heating in a sodium acetate buffer and 4-(2-hydroxyethyl)-1-piperazin
42               Our findings reveal that while sodium acetate buffer is suitable for LaNiO(3), alternat
43 r a period of 180 days, when stored in 0.01M sodium acetate buffer pH 5.5 at 4 degrees C.
44 ate, sodium chloride, gallic acid and pH 5.0 sodium acetate buffer system.
45 PHF suspended in Tris-buffered saline (TBS), sodium acetate buffer, and water.
46     A solution of AlCl(3).6H(2)O in a pH 4.0 sodium-acetate buffer was mixed with an aqueous solution
47  acidified 5 M NaCl solution directly into a sodium acetate-buffered solution containing a DOTA (1,4,
48  experiments, different acids or acetic acid/sodium acetate buffers at different pHs were used to evo
49 he vast majority of studies rely on standard sodium acetate buffers or commercially sourced reagents
50 is conjugated with an amide, the addition of sodium acetate catalyzed the isomerization.
51 on the observations of different patterns of sodium acetate clusters that are characteristic for each
52 ensity, are quantified as functions of salt (sodium acetate) concentration for the interactions of th
53 nd as a function of dihydrogen phosphate and sodium acetate concentrations.
54 (-1) for CB7 and CB8, respectively, in 50 mM sodium acetate-d3 D2O solution (pD 4.7).
55       The detection of MTS using EESI with a sodium acetate doped solvent to generate the [MTS+Na](+)
56 riptase-polymerase chain reaction, following sodium acetate/ethanol precipitation.
57        Allograft mice receiving supplemental sodium acetate exhibited similar protection from rejecti
58                       The iron desorbed with sodium acetate (FeNaAc) yielded heavier delta(56)Fe comp
59 ling the fast, exothermic crystallization of sodium acetate from a metastable aqueous solution into t
60 matography using sodium hydroxide (NaOH) and sodium acetate gradients, coupled with integrated pulsed
61 ication was carried out using supersaturated sodium acetate heat packs and results compared to those
62 e phase modifiers (i.e., ammonium formate or sodium acetate); however, these ESI adduct signals are l
63 onenzymatic synthesis of OAADPR from NAD and sodium acetate in acetic acid.
64  deprotected and cyclized in the presence of sodium acetate in refluxing ethanol to afford (+/-)-abys
65            Successful additives were 6 mol % sodium acetate in the (t)BuOH system and 4 mol % salicyl
66 ion of aqueous solvents sodium hydroxide and sodium acetate, in contrast to conventional isocratic el
67 s 5.7 x 10(9) M(-1) at 25 degrees C in 50 mM sodium acetate medium.
68 is is associated with changes of glucose and sodium acetate metabolism, demonstrating HRMAS NMR as a
69 by a high-fiber diet or supplementation with sodium acetate modified alloimmunity in a kidney transpl
70 broader (+13 to +42) in solutions containing sodium acetate (NaAc), ammonium chloride (AmmCl), potass
71       The GSP was highly responsive to NaCl, sodium acetate (NaAc), ammonium chloride, and sucrose; N
72 ve to 500 mM concentrations of NaCl, Na2SO4, sodium acetate (NaAc), KCl, K2SO4, potassium acetate (KA
73 ylbutyrate (PBA), sodium butyrate (NaB), and sodium acetate (NaAC), on ZIKV replication and associate
74 0 mM histidine/240 mM sucrose buffer, 0.25 M sodium acetate (NaOAc) buffer containing 5 mg.mL(-1) n-a
75 ute application of SCFAs (sodium propionate, sodium acetate or butyric acid) dose-dependently (0.5-30
76    In contrast, the replacement of NaCl with sodium acetate, or pretreatment of cells with the Cl- ch
77                Treatment with either NaCl or sodium acetate (pH 7.0) increased expression of an rpoS:
78 ution conditions used (3.8 mM protein, 50 mM sodium acetate, pH 4.5, 20 degreesC), and that it tumble
79 s measurements on a sample containing 100 mM sodium acetate, pH 5.4.
80 solution containing guanidinium thiocyanate, sodium acetate, phenol and chloroform, followed by centr
81 m-proton antiporter genes also contribute to sodium acetate, potassium acetate, and ammonium acetate
82 370) potassium chloride (PubChem CID: 4873), sodium acetate (PubChem CID: 517045), loganic acid (PubC
83 s, replacement of 300 mosmol/kgH2O NaCl with sodium acetate resulted in down-regulation of the gamma
84 ly from microbial membranes using an aqueous sodium acetate (SA) buffer that can be used to identify
85                                              Sodium acetate (SA) has been used as a highly effective
86            Synthetic food preservatives like sodium acetate (SA), sodium benzoate (SB), potassium sor
87 its for sodium chloride, potassium chloride, sodium acetate, sodium fluoride, sodium dodecyl sulfate
88 ure for up to 1 month in the ethylene glycol-sodium acetate solvent system without the requirement fo
89                                              Sodium acetate supplementation counters the effects of A
90                                              Sodium acetate supplementation rescues glycolysis inhibi
91  under 300 nm irradiation in the presence of sodium acetate to afford 5,6-bis(methylthio)-5-hexenol i
92 ntiporter gene nhaA confers the elevated AcR sodium acetate tolerance phenotype.
93 Zymomonas mobilis mutant (AcR) demonstrating sodium acetate tolerance that has potential importance i
94           In phase 1, a ternary composite of Sodium-Acetate-Trihydrate, Glycerol, Paraffin, and water
95 itter was incubated with 100 mM of [2-(13)C] sodium acetate under thermophilic anaerobic conditions.
96 ocks, including chromium, iron, nitrate, and sodium acetate, were selected to represent the shocks of
97 romatic C-H activation was first promoted by sodium acetate with [Cp*MCl2]2 (M = Rh, Ir) at room temp