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1 issolution in a mixture of sulfuric acid and sodium bisulfite.
2  96-residue-long C-terminal part of D2) with sodium bisulfite.
3     Sodium metabisulfite, potassium sorbate, sodium bisulfite and sorbic acid showed inhibitory effec
4             Various reducing conditions with sodium bisulfite and with or without tin(II) chloride in
5                                   The use of sodium bisulfite as an electron donor to quench chlorami
6 atment of the palindrome-containing DNA with sodium bisulfite at high temperature results in deaminat
7            Isolated fragments are exposed to sodium bisulfite before library amplification and sequen
8                                   The use of sodium bisulfite (BS) treatment followed by hybridizatio
9                                              Sodium bisulfite conversion followed by sequencing (BS-S
10 an be measured at the single CpG level using sodium bisulfite conversion of genomic DNA followed by s
11 om primary chicken erythroid cells using the sodium bisulfite conversion technique and found all CpGs
12  from the Illumina Genome Analyzer following sodium bisulfite conversion.
13 assay entails initial modification of DNA by sodium bisulfite, converting all unmethylated, but not m
14                 Methylation-specific PCR and sodium bisulfite DNA sequencing analyses indicate that t
15 dithiothreitol, ascorbic acid, cysteine, and sodium bisulfite) except ascorbic acid disrupted inter-s
16 EO) value would be comparable to the cost of sodium bisulfite for areas with low electricity costs.
17                        Using high-resolution sodium bisulfite genome sequencing, we detected distinct
18                                              Sodium bisulfite genomic sequencing established that a C
19 ns of the 30 CpG sites within this region by sodium bisulfite genomic sequencing.
20 on involve the treatment of genomic DNA with sodium bisulfite; however, this method cannot distinguis
21                Treatment of genomic DNA with sodium bisulfite is used to convert unmethylated Cytosin
22                    This approach is based on sodium bisulfite-mediated conversion of 5hmC to cytosine
23                                              Sodium bisulfite modification assay revealed significant
24 hylation-sensitive SSCP (Ms-SSCP) technique, sodium bisulfite modification of DNA converts unmethylat
25                                    Menadione sodium bisulfite (MSB) increased the oxygen consumption
26 a pro-oxidant vitamin K precursor [menadione sodium bisulfite (MSB)] suppresses PC progression in mic
27 all cytosines were converted to thymidine by sodium bisulfite mutagenesis, indicating that none of th
28 alysis relies on the chemical deamination by sodium bisulfite of unmodified cytosine to uracil to per
29                                              Sodium bisulfite reacts specifically with cytosine in si
30 rom HCT116 cells and analysis of genome-wide sodium bisulfite sequencing data sets from several other
31 ethylation status of proximal GC boxes using sodium bisulfite sequencing reveals differential methyla
32                                              Sodium bisulfite sequencing showed a gradually reduced m
33 active DNMT3L, shown by knockdown assays and sodium bisulfite sequencing.
34  demethylation to nearby CpG sites, shown by sodium bisulfite sequencing.
35  several adjuvants such as edetate disodium, sodium bisulfite, sodium chloride and so on.
36           Genomic DNA was first reacted with sodium bisulfite to convert unmethylated cytosine to ura
37 al reaction of gDNA with concentrated acidic sodium bisulfite to quantitatively convert C to uracil (
38 more, unbiased next-generation sequencing of sodium bisulfite treated total DNA from HCT116 cells and
39 dent sequence differences in PCR products of sodium bisulfite-treated DNA as described previously.
40 ymerase chain reaction and sequencing of the sodium bisulfite-treated DNA from HCC cell lines and HCC
41                                Sequencing of sodium bisulfite-treated DNA showed dense methylation of
42 lation-specific PCR and sequence analysis of sodium bisulfite-treated genomic DNA, we demonstrate a s
43 ion-specific PCR and by sequence analysis of sodium bisulfite-treated genomic DNA.
44                                              Sodium bisulfite treatment (SBT) causes unmethylated cyt
45                                 Here, we use sodium bisulfite treatment and targeted gene enrichment
46                                Nondenaturing sodium bisulfite treatment catalyzes the conversion of u
47             We show that techniques based on sodium bisulfite treatment of DNA are incapable of disti
48 oportions of the C/T polymorphism induced by sodium bisulfite treatment of DNA reflects the DNA methy
49 isoschizomer restriction enzyme pairs and/or sodium bisulfite treatment of genomic DNA.
50           Subsequently, sequencing following sodium bisulfite treatment revealed 18 methylated CpG si
51 ical fo r functional photosystem II (PS II), sodium bisulfite was utilized for in vitro random mutage
52 f methylated and unmethylated cytosines with sodium bisulfite, which exclusively converts unmethylate