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1 hydrogen bonding network that is mediated by sodium ion.
2 a small cation-friendly cavity occupied by a sodium ion.
3 two protons that are later exchanged for one sodium ion.
4 o solid-state electrochemical reactions with sodium ions.
5 nd had a more organised structure around the sodium ions.
6 accessible ligand-binding pocket that lacked sodium ions.
7 e, they are susceptible to interference from sodium ions.
8 r ~800 water molecules and for magnesium and sodium ions.
9 3 in the presence of potassium ions but not sodium ions.
10 yte battery, which involves the insertion of sodium ions.
11 the synapse, assisted by the co-transport of sodium ions.
12 rom singly charged precursor ions with bound sodium ions.
13 esting the disruption of hydrogen-bonding by sodium ions.
14 phate group, together with flanking zinc and sodium ions.
15 ysiology, including the balance of water and sodium ions.
16 us phase was correlated with the mobility of sodium ions.
17 nable allosteric inhibition by extracellular sodium ions.
18 of the pore domain and ensuing permeation of sodium ions.
19 e, with a high preference for potassium over sodium ions.
20 , 0.34% vs. 0.31%; silicon, 0.36% vs. 0.37%; sodium ion, 0.21% vs. 0.18%; and sulfate, 0.35% vs. 0.38
21 ltaenhC mutants showed a hypersensitivity to sodium ion, a phenotype associated with dysfunction of t
25 ree distinct GA dimeric species, detected as sodium ion adduct ions [2GA + 2Na](2+), and these are as
26 opropanol lead to a significant reduction in sodium ion adduction but are not as effective as acetoni
27 he effectiveness of this method for reducing sodium ion adduction is related to the low proton affini
29 bF(6), can significantly lower the extent of sodium ion adduction to the molecular ions of proteins a
30 ction remains wide enough for the passage of sodium ions, aided by a continuous bridge of approximate
31 , mesitylenic acid, and solvent molecules on sodium ion all are critical in identifying the most favo
32 through a dual mechanism of intercalation of sodium ions along the x axis of the phosphorene layers f
33 ith cardiovascular hospitalizations, whereas sodium ion, aluminum, and magnesium, components abundant
34 gs differ from the conventional thought that sodium ions always lead to more severe fractures in the
35 d, the M3 receptor is bound by an allosteric sodium ion and confined mostly in the inactive state wit
37 increased some of the allosteric effects of sodium ions and amiloride, whereas orthosteric ligand bi
38 nist and antagonist affinity, allosterism by sodium ions and amilorides, and receptor functionality w
39 small but significant decrease in hemolymph sodium ions and an increase in calcium ions after 24 h p
40 ynamics occur in the absence and presence of sodium ions and aspartate, but stall in sodium alone, pr
41 amate from synapses are driven by symport of sodium ions and counter-transport of a potassium ion.
43 of one glutamate to the cotransport of three sodium ions and one proton and the countertransport of o
44 sists of cotransport of glutamate with three sodium ions and one proton, followed by countertransport
45 forms gated paracellular channels and allows sodium ions and other small positively charged ions to c
46 formation, the enzyme is able to capture two sodium ions and transport them to the external side of t
47 serve electroneutrality and osmotic balance, sodium ions and water also flow into the intestinal lume
48 ging rechargeable sodium-ion storage systems-sodium-ion and room-temperature sodium-sulfur (RT-NaS) b
49 , we show that this cation is a stably bound sodium ion, and although it is not a transported substra
51 otential role of structured water molecules, sodium ions, and lipids/cholesterol in GPCR stabilizatio
52 ed and reduced states, Na(+)-NQR binds three sodium ions, and that the affinity for sodium is the sam
53 A core domain of six helices harbours two sodium ions, and the remaining four helices pack in a ro
59 as active electrode materials of lithium or sodium ion batteries, catalysts for water splitting, and
63 and development efforts on room-temperature sodium-ion batteries (NIBs) have been revitalized, as NI
64 a promising negative electrode candidate for sodium-ion batteries (NIBs) owing to its easy scalabilit
65 u of 0.1 V in PIBs, slightly higher than for sodium-ion batteries (SIBs) (0.01 V), and well above the
70 to develop high-energy-density cathodes for sodium-ion batteries (SIBs), low-cost, high capacity Na(
73 ncreasing interest in the development of new sodium-ion batteries and new analytical methods to non-i
74 formance when used in lithium-ion batteries, sodium-ion batteries and supercapacitors, respectively.
78 ategies for rational design of materials for sodium-ion batteries are presented to provide an overvie
81 be a scalable, low-cost cathode material for sodium-ion batteries exhibiting high capacity, long cycl
82 The as-prepared sample used as an anode in sodium-ion batteries exhibits the best rate performance
83 ries, the application of these structures in sodium-ion batteries has attracted great attention in re
85 ough recent reports on cathode materials for sodium-ion batteries have demonstrated performances comp
89 nd the manufacturing feasibility of low cost sodium-ion batteries with existing lithium-ion battery i
90 ural and chemical evolution of tin anodes in sodium-ion batteries with in situ synchrotron hard X-ray
93 articular emphasis on lithium-ion batteries, sodium-ion batteries, catalysis of hydrogen evolution, o
94 mize the side reactions of red phosphorus in sodium-ion batteries, demonstrating stable electrochemic
95 re proposed to fabricate superior anodes for sodium-ion batteries, featuring high-rate capabilities a
96 lude electrodes in rechargeable lithium- and sodium-ion batteries, lithium-sulfur batteries, and supe
97 pplications including lithium-ion batteries, sodium-ion batteries, lithium-sulfur batteries, lithium-
98 owever, due to the difference in lithium and sodium-ion batteries, there are several issues that need
118 n, we design a fully recyclable rechargeable sodium ion battery with bipolar electrode structure usin
119 pacity retention per cycle to be 99.88% as a sodium-ion battery (SIB) and 99.70% as a potassium-ion b
120 form of CTF-HUST-4 as an anode material in a sodium-ion battery achieving an excellent discharge capa
123 molecular precursor for the next generation sodium-ion battery cathode material, Na(2)Mn(2)FeO(6), i
124 n, for the first time, we report a family of sodium-ion battery electrodes obtained by replacing step
127 he cathode material used in a lithium-ion or sodium-ion battery is alkali-rich, this can increase the
128 ility, and cycle life, providing a practical sodium-ion battery powering an electric vehicle in frigi
130 major scientific challenge for a competitive sodium-ion battery technology is to develop viable anode
131 a sustainable way has rekindled interest for sodium-ion battery technology, owing to the natural abun
132 tion enables the fabrication of a discharged sodium-ion battery with a non-sodium metal anode, and th
137 med to understand the mechanistic effects of sodium ion binding on dynamic activation of the M3 musca
140 ucted to have altered ion selectivities, the sodium ion binding site nearest the extracellular side i
141 e D2.50-protonated receptor does not exhibit sodium ion binding to the D2.50 allosteric site and samp
146 ve charge introduced at position 124 and the sodium ions bound at Na3' and Na1 underlies the protecti
149 validated the concept of full-titanium-based sodium ion cells through the assembly of symmetric cells
151 ncreased expression of certain voltage-gated sodium ion channel (NaV) isoforms in peripheral sensory
153 HN-associated VZV isolates induce changes in sodium ion channel currents known to be associated with
154 lular geometry, gap junctional coupling, and sodium ion channel distribution on propagation velocity
158 luates the delivery of dsRNA targeted to the sodium ion channel paralytic A (TcNav) gene in Tribolium
160 iscern the role of the cardiac voltage-gated sodium ion channel SCN5A in the etiology of dilated card
162 analogs that inhibit NaV1.7, a voltage-gated sodium ion channel that is a compelling target for impro
165 on of Nav 1.6 and Nav 1.7 genes all encoding sodium ion channels the dysregulation of which is associ
166 by mutations in skeletal muscle chloride and sodium ion channels with considerable phenotypic overlap
169 djustment for gaseous copollutants, nitrate, sodium ion, chloride ion, magnesium, and nickel remained
170 s mutant, residual active outward pumping of sodium ions competes with passive inward transport of po
171 fferences in sodium ion mobility and in free sodium ion concentration, leading to differences in in-m
172 a dehydrated iron hexacyanoferrate with high sodium-ion concentration enables the fabrication of a di
173 demonstrate that sustained low extracellular sodium ion concentrations ([Na(+)]) directly stimulate o
175 thesis and are found to irreversibly inhibit sodium ion conductance in recombinantly expressed wild-t
177 analysis, we find that the particles form a sodium-ion conductive film on the anode, which stabilize
179 reased ionic conductivity in an archetypical sodium-ion conductor Na(3)PS(4) are not fully understood
180 establish the molecular basis for allosteric sodium ion control in opioid signalling, revealing that
181 Notably, norbuprenorphine interacted with sodium ion-coordinating residues W293(6.48) and N150(3.3
183 in nature, is approximately 52% faster than sodium ion (DNa+ = 1.33, DCl- = 2.03[10(-9)m(2)s(-1)]).
184 into the cell, driven by the co-transport of sodium ions down their transmembrane concentration gradi
185 trides, (3) an electrostatically stabilizing sodium ion during nitride installation, (4) selecting th
186 co-deintercalation of the hydrated water and sodium-ion during the high potential charging process re
188 ered birnessite (Na(0.27)MnO(2)) for aqueous sodium-ion electrochemical storage with a much-enhanced
191 erization of the cocrystalline solid-organic sodium ion electrolyte NaClO4 (DMF)3 (DMF=dimethylformam
195 tion light-driven H(+)/Na(+) pumps, ejecting sodium ions from cells in the presence of sodium and pro
199 upled with an Sb-based anode, the fabricated sodium-ion full-cells also exhibit excellent rate and cy
203 rate, and Na1 and Na2 for two co-transported sodium ions) have been resolved, we still lack a mechani
204 aling the presence and fundamental role of a sodium ion in mediating allosteric control of receptor f
207 titative assessment of the metabolic role of sodium ions in cellular processes and their malfunctions
209 was used to study the molecular mobility of sodium ions in model cheeses through measurements of the
210 ic, mucoadhesive thickener, the retention of sodium ions in the mouth is prolonged due to the mucoadh
211 for the first time the detailed locations of sodium ions in the selectivity filter of a sodium channe
212 ke potassium ions in potassium channels, the sodium ions in these channels appear to be hydrated and
214 in some prokaryotes, complex I may transport sodium ions instead, and three subunits in the membrane
219 reveals that the presence of binding-pocket sodium ions is necessary to stabilize the locked-occlude
221 vailable electrode materials, especially for sodium-ion layered oxides, motivating the exploration of
224 pid/protein ratios, 0% and 1% added NaCl) on sodium ion mobility ((23)Na NMR), in-mouth sodium releas
225 se composition, thus inducing differences in sodium ion mobility and in free sodium ion concentration
228 rmness and perceived hardness, and increased sodium ion mobility, in vivo sodium release and both sal
229 e cheeses, perceived hardness, and decreased sodium ion mobility, in vivo sodium release, saltiness a
242 ane with high selectivity for potassium over sodium; ion passage is blocked by specific chemical modi
243 and disrupt a continuous internal water and sodium ion pathway, preventing transitions to an active-
245 introduction of the R109Q mutation into the sodium ion pump of Dokdonia eikasta (KR2) results in pas
247 Sodium is globally available, which makes a sodium-ion rechargeable battery preferable to a lithium-
248 uptake of neurotransmitter with one or more sodium ions, removing neurotransmitter from the synaptic
250 ges that may illuminate the pathway by which sodium ions return to the endoneurial space after they h
251 the ion movements or to the pathway taken by sodium ions returning to their original endoneurial loca
253 tran, and altered apical side tight junction sodium ion selectivity, compared with wild-type mice.
255 that single-particle mass spectra with weak sodium ion signals can be produced by the desorption of
256 ne)amiloride 2 (HMA) supposedly bind in this sodium ion site and can influence orthosteric ligand bin
260 A receptor (hA2AAR), in which the allosteric sodium ion site was elucidated, makes it an appropriate
263 he seven-transmembrane bundle core, with the sodium ion stabilizing a reduced agonist affinity state,
265 ral disordering and structural water improve sodium-ion storage in a layered electrode and open up an
268 The energy required to remove calcium and sodium ions subsequent to nerve excitation was estimated
269 fied new materials design rules for emerging sodium-ion systems that do not apply to lithium-ion syst
271 allosteric ENaC inhibition by extracellular sodium ions, thereby increasing the probability of chann
272 om-centric Pt sites are formed by binding to sodium ions through -O ligands, the ensemble being equal
273 s reveal that the GLIC channel is open for a sodium ion to transport, but presents a approximately 11
274 al stimulation, the channel opens and allows sodium ions to enter the cell, inducing a fast upstroke
278 this site blocks the transmembrane-spanning sodium ion translocation pathway, providing a molecular
279 ria, a close model for the human enzyme, and sodium ion transport across the mitochondrial inner memb
281 show that the surface diffusion barrier for sodium ion transport is a sensitive function of the chem
283 te dense and homogeneous solid-liquid hybrid sodium-ion transportation channels through and along the
284 functional theory calculations suggest that sodium ions undergo occupancy-dependent stepwise inserti
287 ence of binding-pocket leucine substrate and sodium ions, we have sampled plausible conformational st
288 ile range increases in OCM, EC, silicon, and sodium ion were associated with estimated increases in m
293 th manganese oxides capable of intercalating sodium ions when their potentials were prepoised prior t
295 suggest specific localization sites for the sodium ions, which correspond with experimentally determ
296 D52A(2.50) directly affected the mobility of sodium ions, which readily migrated to another pocket fo
297 cations would strongly favor the passage of sodium ions while hindering translocation of chloride io
298 consumption during the diffusion process of sodium ions, while the carbon-coated structure can incre
299 h pHLIP and the bilayer; the coordination of sodium ions with the C-terminus of pHLIP led to localize
300 ges in helicity, whereas the coordination of sodium ions with the phosphate moiety of the phosphochol