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1 wn direct nitration of a benzimidazole using sodium nitrite.
2 ant extracts as an alternative to commercial sodium nitrite.
3 s were assessed for intentional exposures to sodium nitrite.
4 n center reports of intentional exposures to sodium nitrite.
5 ized in 4 steps from methyl vinyl ketone and sodium nitrite.
6 oked sausages produced with reduced level of sodium nitrite.
7 ity phase 1 dose-escalation trial of topical sodium nitrite.
8 s was completely inhibited by a low level of sodium nitrite.
9 rite and reacts with hemoglobin similarly to sodium nitrite.
10 s highly susceptible to killing by acidified sodium nitrite.
11 dium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium nitrite, 0.075 uL/g TP and 0.075 uL/g PM; T5: 50
12  randomized to receive intracoronary (10 mL) sodium nitrite (1.8 mumol) or NaCl (placebo) before ball
13                                           IV sodium nitrite (10 mug/kg/min) for 1 hour.
14            Our results indicate that topical sodium nitrite 2% cream is suitable for additional clini
15    Active treatment was divided as low dose (sodium nitrite, 3%, with citric acid, 4.5%, creams appli
16 of reperfusion in the absence or presence of sodium nitrite (30 nmol) administered topically 1 min be
17 ry bypass graft patients treated with either sodium nitrite (30-min infusion of 10 mumol/min) or vehi
18                Intravenous administration of sodium nitrite (48 nmol) 5 minutes before infusion of mu
19  system by mixing a solution of KY jelly and sodium nitrite (5% weight/volume), with a solution of KY
20 %, creams applied twice daily), middle dose (sodium nitrite, 6%, with citric acid, 9%, creams applied
21 cebo applied in the morning), and high dose (sodium nitrite, 6%, with citric acid, 9%, creams applied
22                                       Use of sodium nitrite, 6%, with citric acid, 9%, twice daily is
23       Systemic intravenous administration of sodium nitrite (8.7 mumol/min) dilated the radial artery
24                                Intrabrachial sodium nitrite (8.7 mumol/min) increased radial artery d
25 cise before and after treatment with inhaled sodium nitrite (90 mg) or placebo.
26                                      Topical sodium nitrite, a known nitric oxide donor, enhances blo
27           In this study, the effects of 1 mM sodium nitrite, a reactive nitrogen species (RNS) genera
28 rein, is investigated the effects of topical sodium nitrite administration in a rat model of renal is
29  were given inhaled NO gas (20 ppm), inhaled sodium nitrite aerosol (0.87 mol/L), or an intravascular
30                            Administration of sodium nitrite after severe subarachnoid hemorrhage diff
31 ts of decedents where the cause of death was sodium nitrite and (2) poison center reports of intentio
32 .075 uL/g TP and 0.075 uL/g PM; T5: 50 mg of sodium nitrite and 0.150 uL/g PM.
33 e; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL/g TP; T4: 50 mg of sodium ni
34 lators, phosphodiesterase type 5 inhibitors, sodium nitrite and endothelin receptor antagonists.
35 th metabolic syndrome after 12 weeks of oral sodium nitrite and nitrate treatment (IND#115926) displa
36 n spectra of wastewater solutions containing sodium nitrite and nitrate were measured in the concentr
37 unctionality of TPS/PBAT (50/50) blends with sodium nitrite and sodium erythorbate (1-5%) via blown f
38                                              Sodium nitrite and sodium erythorbate functionalized TPS
39                                              Sodium nitrite and sodium erythorbate improved processin
40 es, followed by their alpha-nitrosation with sodium nitrite and subsequent base mediated intramolecul
41 ch as potassium ferricyanide, hydroxylamine, sodium nitrite, and 4-dimethylaminophenol (DMAP), added
42                                              Sodium nitrite as either cause of death (medical examine
43 nts, and quantification of hydroxylamine and sodium nitrite as end reaction products.
44 alide source and tert-butyl nitrite (TBN) or sodium nitrite as the diazotization reagent.
45  of most strains exposed to either acidified sodium nitrite (ASN) or hydrogen peroxide (H(2)O(2)) was
46 bacterium tuberculosis isolates to acidified sodium nitrite (ASN) were compared.
47 ro than other clinical isolates to acidified sodium nitrite (ASN), a generator of reactive nitrogen a
48 at higher rates in the presence of acidified sodium nitrite (ASN), a model system used to generate ni
49 isolate 97A 2472) was resistant to acidified sodium nitrite (ASN).
50 sis of electrochemical sensing platforms for sodium nitrite, B-nicotinamide adenine dinucleotide (NAD
51 ow in a time-dependent manner, with low-dose sodium nitrite being most effective.
52           To test this, we delivered inhaled sodium nitrite by aerosol to newborn lambs with hypoxic
53            Raising metHb levels with dietary sodium nitrite can further decrease daytime core body te
54 f-hospital cardiac arrest, administration of sodium nitrite, compared with placebo, did not significa
55                                Mean residual sodium nitrite concentration was 76.5 mg kg(-1), ranging
56 oncentration, sulfanilic acid concentration, sodium nitrite concentration, reaction time, sulfanilic
57                         Using these methods, sodium nitrite concentrations in 700 processed meat prod
58      Here, we investigate long-term residual sodium nitrite content in processed meat products in Cos
59 as treated with a different concentration of sodium nitrite cream (cohort 1: 0.5%, cohort 2: 1.0%, co
60                 Increasing concentrations of sodium nitrite cream were applied twice weekly for 4 wee
61 n 2 New York medical examiner jurisdictions, sodium nitrite deaths increased yearly, and the medical
62            Medical examiner determination of sodium nitrite deaths was considered the criterion stand
63  scavenger carboxy PTIO completely abolished sodium nitrite-dependent ischemic tissue blood flow and
64     These findings do not support the use of sodium nitrite during resuscitation from out-of-hospital
65                      Therapeutic delivery of sodium nitrite during resuscitation improved survival in
66             Nevertheless, higher addition of sodium nitrite elevated the residual nitrite content, re
67 TICIPANTS: This retrospective case series of sodium nitrite exposures and deaths was conducted in 2 u
68                                              Sodium nitrite failed to improve mitochondrial metabolic
69  Overall, 205 patients (41%) in the 45 mg of sodium nitrite group and 212 patients (43%) in the 60 mg
70 arge for 66 patients (13.2%) in the 45 mg of sodium nitrite group and 72 patients (14.5%) in the 60 m
71 group and 212 patients (43%) in the 60 mg of sodium nitrite group compared with 218 patients (44%) in
72 roup and 72 patients (14.5%) in the 60 mg of sodium nitrite group compared with 74 patients (14.9%) i
73                            Administration of sodium nitrite had little effect in normoxia, but produc
74 lly converted to 4 in 45% yield by employing sodium nitrite in aqueous tartaric acid at 0-5 degrees C
75 olerability, and pharmacokinetics of topical sodium nitrite in patients with sickle cell disease and
76 catalyse nitric oxide generation from benign sodium nitrite in the presence of modest electric fields
77                                        Acute sodium nitrite infusion favorably attenuates hemodynamic
78 ency ratio was measured before and during IV sodium nitrite infusion.
79 tron-rich molecules present in the lumen and sodium nitrite ingested in the diet.
80 ver; combined infusion of acetazolamide with sodium nitrite inhalation did not further increase exhal
81                          Here we report that sodium nitrite (intraperitoneal, inhaled, or oral) limit
82                                              Sodium nitrite is a curing agent increasingly used for s
83 ntervention, including inhaled nitric oxide, sodium nitrite, L-arginine, phosphodiesterase-5 inhibito
84  45 mg of sodium nitrite (n = 500), 60 mg of sodium nitrite (n = 498), or placebo (n = 499), which wa
85  were randomized (1:1:1) to receive 45 mg of sodium nitrite (n = 500), 60 mg of sodium nitrite (n = 4
86 ble myocardial ischemia, saline and low-dose sodium nitrite (NaNO(2)) (1.5 mumol/min for 20 min) were
87 or the nitric oxide (NO)-generator acidified sodium nitrite (NaNO(2)) during aerobic growth.
88 only marginally dilating resistance vessels, sodium nitrite (NaNO2) infusion would increase cardiac o
89                         During contractions, sodium nitrite (NITRITE) or sodium chloride (SALINE) was
90                                          The sodium-nitrite NQR frequency shifts with temperature as
91 ham and acquire new knowledge on the role of sodium nitrite on the formation of its aroma.
92 ects of local and systemic administration of sodium nitrite on the radial artery (a muscular conduit
93                        Treatment with either sodium nitrite or L-citrulline did not reduce adiposity
94 ich we treated with two different NO donors (sodium nitrite or L-citrulline).
95 efore and 15 min after treatment with either sodium nitrite or matching placebo.
96 ous therapeutic doses (8.25-3,300 mug/kg) of sodium nitrite or PBS were administered.
97 A subset of HFpEF subjects (n = 52) received sodium nitrite or placebo therapy in a 1:1 double-blind,
98                                              Sodium nitrite (or saline as control) was then infused i
99 2 wk, then supplemented with sodium nitrate, sodium nitrite, or sodium chloride (1 g/L) in drinking w
100 adrupole resonance (NQR) of room-temperature sodium nitrite powder.
101 ings demonstrate that topically administered sodium nitrite protects the rat kidney against I/R injur
102 illance underestimates confirmed deaths from sodium nitrite; public health authorities should rely on
103              Acute administration of inhaled sodium nitrite reduces biventricular filling pressures a
104 ence of any NO generation, whereas nebulized sodium nitrite reduces HPV by NO formation; however; com
105                     A single nebulization of sodium nitrite reduces HPV, but this action wanes in the
106                                              Sodium nitrite significantly restored ischemic hind-limb
107 and their nano-entities was compared against sodium nitrite (SN)-added and without antioxidants-added
108 rototype toxic bait (HOGGONE 2 containing 5% sodium nitrite [SN]), though, revealed that wild pigs sp
109 , naloxone, pralidoxime, sodium bicarbonate, sodium nitrite, sodium thiosulfate, vasodilators, and va
110                   Neither sodium nitrate nor sodium nitrite supplementation altered mitochondrial cou
111 Five batches were produced: T1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg o
112 1: 100 mg/kg of sodium nitrite; T2: 50 mg of sodium nitrite; T3: 50 mg of sodium nitrite and 0.150 uL
113 h hydrogen peroxide (H(2)O(2)) and acidified sodium nitrite than were the other strains tested.
114 ceived the highest concentrations of topical sodium nitrite (the 1.8% and 2% cream).
115                                              Sodium nitrite therapy also increased ischemic tissue ni
116                          We examined whether sodium nitrite therapy altered ischemic revascularizatio
117    Here, we test the hypothesis that chronic sodium nitrite therapy can selectively augment angiogeni
118                                              Sodium nitrite therapy completely restored ischemic hind
119                        We have reported that sodium nitrite therapy exerts cytoprotective effects aga
120          These data demonstrate that chronic sodium nitrite therapy is a recently discovered therapeu
121                   Remarkably, the effects of sodium nitrite therapy were evident within 3 days of the
122 strating the potency and efficacy of chronic sodium nitrite therapy.
123                     Here we show that adding sodium nitrite to cobinamide yields a stable derivative
124 lm with or without the addition of 10 microM sodium nitrite to the perfusion medium.
125           Meat samples were pre-treated with sodium nitrite to transform oxymyoglobin and deoxymyoglo
126 o a cerebral nitric oxide donor (intravenous sodium nitrite) to explore whether this correlates with
127 , reaction time, sulfanilic acid volume, and sodium nitrite volume were identified using a Plackett-B
128                       Application of topical sodium nitrite was associated with a significant increas
129 n energy for the reaction of morpholine with sodium nitrite was found as 101 kJ/mol.
130 oked pork sausages produced with 50 mg/kg of sodium nitrite was investigated.
131  analysis showed that systemic absorption of sodium nitrite was very low.
132                                 Solutions of sodium nitrite were administered in the setting of hepat
133 vinyl alcohol (PVA) films, incorporated with sodium nitrite were developed, characterized and applied
134 ed by exhaled gas concentration from inhaled sodium nitrite were not increased by acetazolamide durin
135                                  Intravenous sodium nitrite, which is converted to nitric oxide in vi

 
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