ライフサイエンスコーパス: soft palate

1 s, fuse at the midline, forming the hard and soft palate.

2 mations affecting the upper lip and hard and soft palate.

3 acids is conserved in ferret, pig and human soft palate.

4 vention and non-surgical correction of cleft soft palate.

5 ue-tissue interactions, resulting in a cleft soft palate.

6 imal models exhibit an isolated cleft in the soft palate.

7 ongue and 41% of the neurons innervating the soft palate.

8 ue and a 28% loss in neurons innervating the soft palate.

9 enitors to differentiate into muscles in the soft palate.

10 ng the lateral pharyngeal walls, tongue, and soft palate.

11 is one virus, we demonstrate that the ferret soft palate, a tissue not normally sampled in animal mod

12 uvula, with a short posterior border of the soft palate, abnormal tonsillar pillars, and velopharyng

13 show that loss of Dlx5 leads to a shortened soft palate and an absence of the levator veli palatini,

14 lso an increased cross-sectional area of the soft palate and an increased airway volume in men compar

15 follicle-associated epithelium of the dorsal soft palate and dorsal nasopharynx in persistently infec

16 f neurons innervated the anterior tongue and soft palate and each target contained the same number of

17 kness; (5) minimal changes were noted in the soft palate and tongue.

18 aryngeal walls are more "compliant" than the soft palate and tongue.

19 nflammatory changes in the epithelium of the soft palate and trachea, along with mucosal damage and m

20 and tissue burden of C. albicans in tongue, soft palate, and esophagus (P < 0.001).

21 he huICV replicated in the nasal turbinates, soft palate, and trachea but not in the lungs while swID

22 h, gingival sulcus, tongue, cheeks, hard and soft palates, and tonsils, which are colonized by bacter

23 erior vibrating line in different classes of soft palate angulation (House Classification), according

24 The House classification of the soft palate angulation was identified and recorded as Cl

25 Using the mouse soft palate as a model, we performed an unbiased single-

26 e Blue chloride into the anterior tongue and soft palate (AT and SP neurons) and applying FG crystals

27 vides for the first time an animal model for soft palate cleft and submucous cleft.

28 gfbr2 in the palatal epithelial cells causes soft palate cleft, submucosal cleft and failure of the p

29 le human congenital malformations, including soft palate cleft, we propose that TGF-beta mediated Irf

30 , Tbx1, and Tbx22, have been associated with soft palate clefting in humans and mice, suggesting that

31 Soft palate clefting leads to muscle misorientation and

32 ate, but study of the underlying etiology of soft palate clefts is an emerging field.

33 igating molecular and cellular mechanisms of soft palate clefts.

34 The frequency of soft palate cycles was lowest early in a feeding sequenc

35 portance of distinguishing specific types of soft palate defects in patients and developing relevant

36 palates and the most posterior border of the soft palate, despite phospho-SMAD2 expression in these r

37 Recent advances in the understanding of soft palate development suggest that it may be regulated

38 nd homeostatic cellular activities governing soft palate development to improve regenerative strategi

39 they play important regulatory roles during soft palate development.

40 understanding of the regulatory mechanism of soft palate development.

41 to investigate the regulatory mechanisms of soft palate development.

42 to pharyngeal pressure (PPH) measured at the soft palate during eupnoeic breathing was studied.

43 cordings showed that rhythmic motions of the soft palate during mastication were linked temporally to

44 ed significant posterior displacement of the soft palate during sleep as compared with wakefulness.

45 pothesis that the frequency and amplitude of soft palate elevation associated with mastication would

46 hing appears to have a significant effect on soft palate elevation in mastication.

47 Cyclic soft palate elevation is temporally associated with mast

48 These pitfalls include pneumoparotid, soft palate elevation that resembles a nasopharyngeal ma

49 We conclude that cyclic movement of the soft palate in feeding is temporally linked to jaw motio

50 l fibroblasts and muscle malformation in the soft palate in Osr2(Cre);Tgfbr1(fl/fl) mice.

51 During swallowing, however, the soft palate invariably elevated during the intercuspal p

52 eply infiltrative oropharynx tumors, minimal soft palate involvement, and low suspicion for pathologi

53 oth by the adenoid and tonsils; however, the soft palate is also larger in this group, adding further

54 However, the soft palate is normally lowered during nasal breathing t

55 cles derived from the 4th PA mesoderm in the soft palate, likely via interactions between CNC-derived

56 aste cells of the rat fungiform papillae and soft palate maintained within the intact epithelium in a

57 Movements of radiopaque soft palate markers were recorded by videofluorography w

58 We tested the hypothesis that cyclic soft palate motion is temporally linked to cyclic jaw mo

59 Soft palate motion occurred in every recording but not i

60 The soft palate moved upward as the jaw opened, but the naso

61 These findings suggest that masticatory soft palate movement is diminished during inspiration.

62 The soft palate moves rhythmically during feeding, but the t

63 mysial markers (Aldh1a2 and Hic1) as well as soft palate muscle defects in Osr2-Cre;Runx2(fl/fl) mice

64 a signaling in the palatal epithelium led to soft palate muscle defects in Tgfbr2(fl/fl);K14-Cre mice

65 Along this axis, the dimensions of tongue, soft palate, nasopharyngeal airway, and adenoid increase

66 Clefting of the soft palate occurs as a congenital defect in humans and

67 eta-catenin signaling, is upregulated in the soft palate of Tgfbr2(fl/fl);K14-Cre mice, and WNT-beta-

68 iferation and differentiation defects in the soft palate of Tgfbr2(fl/fl);K14-Cre mice.

69 lic polyacrylamide gel was injected into the soft palate of the rabbits to induce OSAHS.

70 ecifically, muscle mass was decreased in the soft palates of Tgfbr2 mutant mice, following defects in

71 n were found to mediate attachment of GAS to soft palate or skin keratinocytes, but this interaction

72 e., the motoneuronal cell groups innervating soft palate, pharynx, and larynx as well as diaphragm, i

73 iguus innervating the dilator muscles of the soft palate, pharynx, and larynx, but abnormal respirato

74 eceptors (e.g., rostral-central nucleus); 2) soft palate, pharynx, larynx, and tracheobronchial tree

75 palatine tonsils (PRP, 98%; mean SUV, 3.48), soft palate (PRP, 96%; mean SUV, 3.13), and lingual tons

76 However, the tongue, soft-palate, pterygoid, and parapharyngeal fat pads were

77 ar and cellular mechanism of clefting of the soft palate remains unclear because few animal models ex

78 th of vulnerable airway as well as increased soft palate size.

79 osterior pharyngeal wall at the level of the soft palate (superior PC), tip of the epiglottis (middle

80 presumptive junction of the future hard and soft palate that defines anterior-posterior differences

81 leep resulted from posterior movement of the soft palate, thickening of the lateral pharyngeal walls,

82 gnaling and proper muscle development in the soft palate through tissue-tissue interactions, resultin

83 e of the following: tonsillar zones, larynx, soft palate, uvula, and nasal cavities.

84 pes of anterior-posterior obstruction of the soft palate, uvular (94%) and velar (6%), and three type

85 After voluntary closure of the soft palate (VCSP) to eliminate nasal NO, exhaled NO fro

86 In stage II transport, the soft palate was elevated less frequently during inspirat

87 Class II House classification of the soft palate was found to be the most prevalent among the

88 The mean normal-tissue SUV in the soft palate was higher in male than in female patients (

89 ue were similar in both groups; however, the soft palate was larger in subjects with OSAS (3.5 +/- 1.

90 parapharyngeal fat pads, lateral walls, and soft palate) was similar between subjects with OSAS and

91 infection and was remarkably enriched in the soft palate, where long-chain alpha2,6-linked sialic aci

92 n the palatine tonsils, lingual tonsils, and soft palate, whereas uptake in the major salivary glands