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1 esent in the lipopolysaccharide of Ralstonia solanacearum.
2 ty was with Ralstonia eutropha and Ralstonia solanacearum.
3 or (VEF) produced by wild-type strains of R. solanacearum.
4 h includes R. eutropha, R. pickettii, and R. solanacearum.
5 operon is the major virulence factor for R. solanacearum.
6 es virulence gene expression in wild-type R. solanacearum.
7 ributes substantially to the virulence of R. solanacearum.
8 ts efficacy and biosafety profile against R. solanacearum.
9 e effector from the plant pathogen Ralstonia solanacearum.
10 ial species that were antagonistic toward R. solanacearum.
11 by the plant-pathogenic bacterium Ralstonia solanacearum.
12 ibits bacterial niche competitors but not R. solanacearum.
13 ve as sole carbon or nitrogen sources for R. solanacearum.
14 seudomonas syringae and PopP2 from Ralstonia solanacearum.
15 in another P. syringae strain and Ralstonia solanacearum.
16 ulation of flagellum-mediated motility in R. solanacearum.
17 (bp) genes, as well as their orthologs in R. solanacearum.
18 ytopathogenic Xanthomonas spp. and Ralstonia solanacearum.
19 tii, as well as the plant pathogen Ralstonia solanacearum.
20 ulence genes in the plant pathogen Ralstonia solanacearum.
21 unknown function, and 13% were unique to R. solanacearum.
22 nas campestris pv. vesicatoria and Ralstonia solanacearum.
23 liana (Arabidopsis), Resistance to Ralstonia solanacearum 1 (RRS1-R) and Resistance to Pseudomonas sy
26 Our findings suggest that Brg11 may give R. solanacearum a competitive advantage and uncover a role
27 ineages, to suppress the bacterium Ralstonia solanacearum, a global phytopathogen capable of infectin
29 ntly showed that, in Ralstonia (Pseudomonas) solanacearum, a phytopathogenic bacterium, acyl-HSL prod
30 Expression of virulence genes in Ralstonia solanacearum, a phytopathogenic bacterium, is controlled
31 tomato (Solanum lycopersicum) with Ralstonia solanacearum, a soilborne pathogen that causes bacterial
35 red for a wild-type level of virulence in R. solanacearum although its individual role in wilt diseas
43 vity in plant-pathogenic bacteria (Ralstonia solanacearum) and fungi (Cochliobolus heterostrophus) re
44 dule, a fucose-binding lectin from Ralstonia solanacearum, and human norovirus VA387 P particle (24-m
46 ering from wilt disease (caused by Ralstonia solanacearum) as source for potential prebiotic metaboli
48 ercomonas sp.) affect the pathogen Ralstonia solanacearum, both on individual beneficial bacteria and
49 bacteria (not positively correlated with R. solanacearum) but not efficiently used by the pathogen i
50 plantarum ZPZ inhibited the growth of R. solanacearum by 72.46 +/- 14.42% based on OD(600) measur
54 ly-related bacteria 'Candidatus Liberibacter solanacearum' (CLso), associated with vegetative disorde
55 ologs from Brucella abortus and Liberobacter solanacearum complement the RF auxotrophy of the Sm1021D
56 e of ipx genes suggests that in its host, R. solanacearum confronts and overcomes a stressful and nut
57 In the phytopathogen Ralstonia (Pseudomonas) solanacearum, control of many virulence genes is partly
59 d L-phenylalaninamide were tested against R. solanacearum, E. coli, Staphylococcus sp. and B. subtili
64 py revealed that during tomato infection, R. solanacearum forms biofilm-like masses in xylem vessels.
65 dge, this is the first demonstration that R. solanacearum forms biofilms in plant xylem vessels, and
71 proteins encoded by 'Candidatus Liberibacter solanacearum' haplotype B, CKC_05770 and CKC_00930, whic
75 s of the bacterium, 'Candidatus Liberibacter solanacearum', have been identified worldwide, several o
76 o plants against the phytopathogen Ralstonia solanacearum in a T4BSS-dependent manner, suggesting tha
77 n resistance to the plant pathogen Ralstonia solanacearum in microcosms and in tomato plant rhizosphe
79 tor produced by the plant pathogen Ralstonia solanacearum, in complex with inositol hexaphosphate (In
91 Expression of virulence factors in Ralstonia solanacearum is controlled by a complex regulatory netwo
92 vessels of infected plants, we found that R. solanacearum is essentially nonmotile in planta, althoug
93 cyl-HSL-dependent autoinduction system in R. solanacearum is part of a more complex autoregulatory hi
97 ormed by oligomerization as in the Ralstonia solanacearum lectin and not by sequential domains like t
98 em-limited bacterium Candidatus Liberibacter solanacearum (Lso), the causative agent of economically
99 is associated with "Candidatus Liberibacter solanacearum" (Lso), which is transmitted by the tomato/
101 m limited bacterium 'Candidatus Liberibacter solanacearum' (Lso) is associated with disease in Solana
104 an extracellular factor that complements R. solanacearum mutants deficient in production of the 3-OH
106 To locate and infect host plant roots R. solanacearum needs taxis, the ability to move toward mor
107 tal variability, the biocontrol of Ralstonia solanacearum, one of the most destructive plant pathogen
108 bition zones were produced against Ralstonia solanacearum only when grown in the presence of the indi
113 type rescued DeltaspeC growth, indicating R. solanacearum produced and exported putrescine to xylem s
114 phy, and mass spectroscopy indicated that R. solanacearum produces staphyloferrin B rather than schiz
116 oea stewartii, Erwinia carotovora, Ralstonia solanacearum, Pseudomonas syringae, Pseudomonas aerugino
117 phores present in culture supernatants of R. solanacearum, R. metallidurans, and Bacillus megaterium
119 of the phytopathogen Ralstonia (Pseudomonas) solanacearum, requires the products of at least seven re
121 he gene encoding the catalytic subunit of R. solanacearum's sole assimilatory nitrate reductase, did
126 (IVET), we screened a library of 133 200 R. solanacearum strain K60 promoter fusions and isolated ap
130 olog of HrpB, the master regulator of the R. solanacearum T3SS (T3SS(rso)) and its secreted effectors
131 fector from the bacterial pathogen Ralstonia solanacearum, targets the plant E3 ubiquitin ligase PUB4
136 e against Pseudomonas syringae and Ralstonia solanacearum through activation of elicitor-mediated def
137 syringae and the vascular pathogen Ralstonia solanacearum Thus, the GFP strand system can be broadly
138 ntributes significantly to the ability of R. solanacearum to locate and effectively interact with its
141 ted by the bacterial wilt pathogen Ralstonia solanacearum, undergoes phosphorylation at specific resi
143 s syringae pv. pisi and PopP2 from Ralstonia solanacearum via an integrated WRKY domain in RRS1-R.
144 uggest that nitrate assimilation promotes R. solanacearum virulence by enhancing root attachment, the
145 nvestigate the role of these acyl-HSLs in R. solanacearum virulence gene expression, transposon mutan
147 not a sole carbon or nitrogen source for R. solanacearum, was enriched 76-fold to 37 microM in R. so
148 the growth of a bacterial invader, Ralstonia solanacearum, when introduced into communities comprised
153 ovora, Pectobacterium carotovorum, Ralstonia solanacearum, Xanthomonas campestris, Xanthomonas oryzae