ライフサイエンスコーパス: somatodendritic

1 An increase in somatodendritic 5-HT(1A) autoreceptor density in the dor

2 fect postulated to result from activation of somatodendritic 5-HT(1A) autoreceptors in the DRN.

3 notion that CLN3 is involved in Kv4.2/KChIP3 somatodendritic A-type channel formation, trafficking, a

4 minopeptidase-like protein subunits comprise somatodendritic A-type channels in mammalian neurons.

5 the unitary conductance (gamma) of neuronal somatodendritic A-type K(+) channels composed of Kv4 por

6 s mediate most of the subthreshold-operating somatodendritic A-type K+ current in CNS neurons.

7 Subthreshold-activating somatodendritic A-type potassium channels have fundament

8 wly than responses mediated by postsynaptic (somatodendritic) A1Rs in cultured neurons.

9 equired excitation of DA neurons mediated by somatodendritic alpha4beta2 nAChRs, as well as enhanceme

10 rthermore, the MT network was reduced in the somatodendritic and AIS compartments, and both the heavy

11 binding protein calretinin (CR) in both the somatodendritic and axonal compartment.

12 ll as in membrane protein trafficking in the somatodendritic and axonal compartments of differentiate

13 regulate the trafficking of vesicles between somatodendritic and axonal compartments.

14 Plasma membranes of the somatodendritic and axonal domains of neurons are known

15 npolarized distribution of ATP7B between the somatodendritic and axonal domains.

16 hysiological and physical bridge between the somatodendritic and axonal domains.

17 a physiological and physical bridge between somatodendritic and axonal domains.

18 s are highly polarized cells having distinct somatodendritic and axonal domains.

19 d Kv3.1b, are differentially targeted to the somatodendritic and axonal membrane, respectively, the l

20 Although initially inserted into both somatodendritic and axonal membranes, VGSCs are concentr

21 aneous transmission was due to activation of somatodendritic and axonal receptors while the depressio

22 ed to soma and dendrites and postulated that somatodendritic and axonal/presynaptic isoforms of N-typ

23 tion by forming at least two steps involving somatodendritic and ciliary sorting decisions.

24 This inhibition targeted both proximal somatodendritic and distal apical dendritic domains of g

25 eling was detected in many axonal as well as somatodendritic and glial profiles.

26 of elevated extracellular DA levels, altered somatodendritic and presynaptic D2 DA receptor (D2R) fun

27 ain bundle axonal tracts connecting midbrain somatodendritic and striatal axonal compartments of dopa

28 ansported between spatially distant midbrain somatodendritic and striatal axonal compartments.

29 tion was observed in male mouse dopaminergic somatodendritic and terminal regions.

30 e like calcium channels localize to both the somatodendritic and the axonal compartment of larval cra

31 stic frequency in the theta range across the somatodendritic arbor and specific STA measurements were

32 along with limited DAT endocytic activity in somatodendritic areas.

33 All of these subunits were localized to somatodendritic as well as axonal cell compartments.

34 and VGLUT2 in its subcellular location, with somatodendritic as well as axonal expression.

35 PAT-labeled 5-HT(1a) binding in pre-synaptic somatodendritic autoreceptors on dorsal raphe nucleus re

36 -layer pyramidal neurons across their entire somatodendritic axis for several months.

37 to a reconfiguration of inhibition along the somatodendritic axis of pyramidal cells, and enhances th

38 ion of T-type Ca2+ channels along the entire somatodendritic axis of sensory thalamocortical (TC) neu

39 e quantitative changes in boosting along the somatodendritic axis suggest that inputs from different

40 angential current flow (perpendicular to the somatodendritic axis) modulates synaptic efficacy acutel

41 (2) Radial current flow (parallel to the somatodendritic axis) modulates synaptic efficacy consis

42 the degree of boosting may differ along the somatodendritic axis.

43 nterneurons, this plasticity was observed at somatodendritic basket cell synapses, but not at distal

44 lar matrix, and NF-186 overexpression caused somatodendritic brevican clustering.

45 Finally, somatodendritic but not axonal application of GABA evoke

46 2-LO regulates LTP by enhancing postsynaptic somatodendritic Ca(2+) influx through L-type channels du

47 s provide a molecular platform for localized somatodendritic Ca(2+) signals in mammalian brain neuron

48 y combined in situ patch clamp recordings of somatodendritic calcium currents in an identified adult

49 a(v)2 homolog, Dmca1A, underlies HVA and LVA somatodendritic calcium currents in the same neuron.

50 ition, we show that Dmca1A underlies the HVA somatodendritic calcium currents in vivo.

51 nt somatic APs, it must scale with the local somatodendritic capacitance.

52 s, the AIS location is finely tuned with the somatodendritic capacitive load, serving as a homeostati

53 nd flattened (dendrites limited to lamina I) somatodendritic categories.

54 normally localized to highly phosphorylated somatodendritic clusters in neurons.

55 d Kv2.2 heteromultimers did not aggregate in somatodendritic clusters observed with expression of Kv2

56 ly phosphorylated, localized in high-density somatodendritic clusters, and has a relatively depolariz

57 umption that mislocalization of tau into the somatodendritic compartment (6) and accumulation of fibr

58 rs that immunocytochemically highlight their somatodendritic compartment and brush, respectively.

59 al segment (AIS) separates the axon from the somatodendritic compartment and controls the microtubule

60 SLITRK1 is further enriched in the somatodendritic compartment and cytoplasmic vesicles of

61 n growth) became selectively targeted to the somatodendritic compartment and excluded from axons by p

62 nsistent with an adaptation occurring in the somatodendritic compartment and independent of a circuit

63 trast, CaV3.1 channels were localized to the somatodendritic compartment and proximal axon, but were

64 products traverse different pathways in the somatodendritic compartment before axonal entry.

65 of both sexes is distributed throughout the somatodendritic compartment but is particularly enriched

66 client, was rerouted from the axonal to the somatodendritic compartment by dominant-negative SEC24D.

67 n together, our results demonstrate that the somatodendritic compartment directly inhibits myelinatio

68 tion of mouse tau, its redistribution to the somatodendritic compartment in cortical and hippocampal

69 py revealed abundant ErbB4 expression in the somatodendritic compartment in which it accumulates at,

70 ressed with the pore-forming subunits in the somatodendritic compartment of CNS neurons.

71 Our study shows that the somatodendritic compartment of DA neurons matures before

72 at MAP1B light chain (LC) accumulates in the somatodendritic compartment of hippocampal neurons, wher

73 geted Channelrhodopsin-2 specifically to the somatodendritic compartment of neurons in mice in vivo.

74 l significance of glial interaction with the somatodendritic compartment of neurons.

75 nking results in aberrant myelination of the somatodendritic compartment of neurons.

76 xons of L4-L2/3 synapses, rather than on the somatodendritic compartment of presynaptic L4 neurons.

77 emonstrate the mislocalization of tau in the somatodendritic compartment of RGCs subjected to high in

78 cure, but shows a restricted invasion of the somatodendritic compartment of the cell.

79 icroscopic analyses, which revealed that the somatodendritic compartment was the principal target of

80 tial segment (AIS) electrically connects the somatodendritic compartment with the axon and converts t

81 , axons and dendrites (or more properly, the somatodendritic compartment) are radically different.

82 ly, misfolded Tau can be internalized at the somatodendritic compartment, or the axon terminals and i

83 atRA increased translation in the somatodendritic compartment, similar to brain-derived ne

84 addition the AIS separates the axon from the somatodendritic compartment, where it controls protein t

85 ed by the complexity and excitability of the somatodendritic compartment.

86 he microtubule-associated protein 2-positive somatodendritic compartment.

87 mposed on the proximal end of the AIS by the somatodendritic compartment.

88 l plate neurons, localizing primarily to the somatodendritic compartment.

89 levels, and electronic propagation from the somatodendritic compartment.

90 throcyte membrane skeleton structure, in the somatodendritic compartment.

91 not induce autophagy in either the axonal or somatodendritic compartment.

92 subcellular distribution for ZNF804A within somatodendritic compartments and a nanoscopic organizati

93 n occur in the absence of pyramidal neuronal somatodendritic compartments and are temporally correlat

94 Endogenous ZNF804A protein localized to somatodendritic compartments and colocalized with the pu

95 as restricted to C1 neurons and filled their somatodendritic compartments and efferent axons 7-28 day

96 by formation in axons by days 4-7, spread to somatodendritic compartments by days 7-10 and neuron dea

97 he co-existence of MOR and CB1r-ir in common somatodendritic compartments of catecholaminergic neuron

98 ors located on the presynaptic terminals and somatodendritic compartments of cortical GABAergic inter

99 We find that GSK3beta in somatodendritic compartments of hippocampal neurons beco

100 an exacerbated NMDAR-DeltaCa(2+) response in somatodendritic compartments of MNCs of RVH rats, and (2

101 4.2) mRNAs and regulates their expression in somatodendritic compartments of neurons.

102 might differentially influence inhibition in somatodendritic compartments of pyramidal neurons and af

103 tentials propagated reliably into axonal and somatodendritic compartments with conduction velocities

104 ion regarding stimulus-secretion coupling at somatodendritic compartments, and shed light into mechan

105 , with alpha7 targeted preferentially to the somatodendritic compartments, whereas alpha4beta2 was lo

106 cells with functionally distinct axonal and somatodendritic compartments.

107 itiation and the boundary between axonal and somatodendritic compartments.

108 caused HCN1 to be mistargeted throughout CA1 somatodendritic compartments.

109 t to depend on inward current originating in somatodendritic compartments.

110 nhibitory interneurons, thus bypassing their somatodendritic compartments.

111 e mutant protein specifically accumulates in somatodendritic compartments.

112 elated neural rhythms, but the importance of somatodendritic conductances in rhythm generation is sti

113 They have diverse somatodendritic configurations and form relatively small

114 We hypothesize that an inhibitory somatodendritic cue is necessary to prevent non-axonal m

115 hannel KCNB1 (Kv2.1), which conducts a major somatodendritic current in cortex and hippocampus, is kn

116 ed the magnitude of D2R-dependent inhibitory somatodendritic currents and blunted the impact of D2R a

117 neurons display prominent, non-desensitizing somatodendritic D2-autoreceptor responses that show pron

118 nd RyR-dependent ER Ca(2+) stores facilitate somatodendritic DA release in the SNc.

119 ), suggesting a mechanism for maintenance of somatodendritic DA release with limited Ca(2+) entry.

120 which couple to IP(3) production), increased somatodendritic DA release, whereas CPCCOEt, an mGluR1 a

121 ng a functional role for ER Ca(2+) stores in somatodendritic DA release.

122 ocalization and function of Kv2.1, the major somatodendritic delayed rectifier voltage-dependent K+ c

123 t glutamate triggers GABA release only after somatodendritic depolarization and action potential gene

124 regions and a shift from a nuclear to a more somatodendritic distribution by approximately P13.

125 n filaments had no significant effect on the somatodendritic distribution of BC1 RNA.

126 This likely relates to the differential somatodendritic distribution of mGluRs and mAChRs and ma

127 More generally, we conclude that the somatodendritic distribution of Na(V) channels is a majo

128 As expected, FMRP is expressed in the somatodendritic domain in virtually all neurons.

129 initial segment (AIS) along with the entire somatodendritic domain of adult male mouse dopaminergic

130 solateral domain of epithelial cells and the somatodendritic domain of neurons is mediated by recogni

131 brane of polarized epithelial cells, and the somatodendritic domain of neurons through interactions w

132 gic synapses on the axon initial segment and somatodendritic domain of pyramidal neurons, where it in

133 ter ATP7B and the vesicle-SNARE VAMP4 to the somatodendritic domain of rat hippocampal neurons is med

134 istribution in pyramidal neurons, across the somatodendritic domain, depends on ongoing cyclic adenos

135 the soma, autophagosomes are confined to the somatodendritic domain, facilitating cargo degradation a

136 of the AIS, but not its position within the somatodendritic domain, is the major causal determinant

137 logs FXR1P and FXR2P are well studied in the somatodendritic domain, recent evidence suggests that th

138 nter the soma and remain confined within the somatodendritic domain.

139 rity is established, how distinct axonal and somatodendritic domains are maintained, and how integral

140 ent of membrane molecules between axonal and somatodendritic domains is unclear.

141 teractions is supported by colocalization in somatodendritic domains of cortical neurons in culture a

142 rs formed alpha1/beta-containing clusters on somatodendritic domains of MNTB principal neurons, coloc

143 regulators that are selectively targeted to somatodendritic domains of neurons.

144 thesized in, and released from, postsynaptic somatodendritic domains that are readily accessible to w

145 d proteins, which are normally restricted to somatodendritic domains, redistribute into the former ax

146 ctrin is found in neurons in both axonal and somatodendritic domains, using proteomics, biochemistry,

147 RNA, proteins, and lipids into the axonal or somatodendritic domains.

148 ion barrier is formed between the axonal and somatodendritic domains.

149 Somatodendritic dopamine (DA) release in the substantia

150 his study examined the mechanisms underlying somatodendritic dopamine and noradrenaline transmission

151 or the active zone protein RIM in stimulated somatodendritic dopamine release in the midbrain.

152 re the time course and calcium dependence of somatodendritic dopamine release in the ventral tegmenta

153 he mechanisms and functional consequences of somatodendritic dopamine transmission in the VTA vary am

154 ratios and decreased extracellular levels of somatodendritic dopamine, consistent with a decrease in

155 us implicating endosomal trafficking through somatodendritic early endosomes in L1-mediated axon grow

156 Our data demonstrate that key somatodendritic electrical conduction properties are hig

157 preferential axonal targeting and selective somatodendritic endocytosis.

158 lation of L1/NgCAM occurs via nondegradative somatodendritic endosomes and subsequent anterograde axo

159 Early Endosomal Protein 21 kDa) localizes to somatodendritic endosomes, and downregulation of NEEP21

160 dependent pathway (transcytosis), traversing somatodendritic endosomes.

161 21 kD) as a regulator of L1/NgCAM sorting in somatodendritic endosomes.

162 -rectifier Kv2.1 potassium channels regulate somatodendritic excitability during periods of repetitiv

163 but how these circuits interact to shape the somatodendritic excitability of Purkinje cells during mo

164 likely to play a major role as modulators of somatodendritic excitability.

165 expression continues through P35, with peak somatodendritic expression at P21.

166 of NMDARs on axons, indicating an exclusive somatodendritic expression of functional NMDARs.

167 Somatodendritic expression of Kv7.2 or Kv7.3 subunits wa

168 omous activity, while synaptic activation of somatodendritic GABA(A) receptors regulates the axonal i

169 Overexpression of GIRK3 decreased somatodendritic GABA(B)R- and D(2)R-dependent signaling

170 -G (480-kDa ankyrin-G) promotes stability of somatodendritic GABAergic synapses in vitro and in vivo.

171 Somatodendritic Girk currents evoked by the GABA(B) rece

172 The acute cocaine-induced weakening of somatodendritic Girk signaling complements the previousl

173 We find that somatodendritic glial ensheathment regulates the morphol

174 lial layer leads to selective elimination of somatodendritic glial ensheathment, thus allowing us to

175 with a similar morphology but reversed I(h) somatodendritic gradient to that previously observed in

176 cede the appearance of SWDs and that altered somatodendritic HVA currents are not required for abnorm

177 ivation of mTORC1 in dopamine neurons causes somatodendritic hypertrophy, reduces intrinsic excitabil

178 The somatodendritic IA (A-type) K(+) current underlies neuro

179 ofoundly shape the trafficking and gating of somatodendritic iGluRs, is unknown.

180 Most of the somatodendritic inhibitory effect of GABA(B)R and D(2)R

181 ortical inhibition is recruited by classical somatodendritic integration rather than direct activatio

182 roperties and may be a critical component of somatodendritic ISA channels in the brain.

183 Kv4 channels are responsible for the somatodendritic ISA s.

184 and computational modeling showed that while somatodendritic K(v)7 channels are strongly activated by

185 Somatodendritic L-type Ca2+ channels have long been view

186 how that dopamine release occurs also at the somatodendritic level, providing a substrate for an ultr

187 n Alzheimer's disease (AD), we asked whether somatodendritic levels of human BC200 RNA are deregulate

188 On the basis of these findings plus the somatodendritic localization of RACK1, we hypothesize th

189 f SNc neurons, suggesting that activation of somatodendritic M5 increases the intrinsic excitability

190 Although activation of somatodendritic M5 receptors on SNc neurons leads to inc

191 and computer-assisted morphometry to provide somatodendritic measures of 652 neurons.

192 ique to provide qualitative and quantitative somatodendritic measures of gigantopyramidal neurons acr

193 Quantitatively, somatodendritic measures of typical pyramidal neurons in

194 density innervation of 5-HT terminals on the somatodendritic membrane and a complete absence on the A

195 the function of alpha7 nAChRs located on the somatodendritic membrane of hippocampal interneurons.

196 The somatodendritic membrane was immunopositive for mGluR1al

197 the distal axon is estimated as <1% that of somatodendritic membrane.

198 long been thought to be homogeneous in their somatodendritic morphology and physiology.

199 We analyzed the somatodendritic morphology and ultrastructure of these t

200 ouse cortex undergoes significant changes in somatodendritic morphology during the critical period fo

201 nomic groups could be discriminated based on somatodendritic morphology for both superficial and giga

202 uld be further divided on the basis of their somatodendritic morphology into four types: multipolar,

203 esent study characterized and quantified the somatodendritic morphology of neocortical neurons in pre

204 e evaluated potassium channel expression and somatodendritic morphology of projection neurons and the

205 we studied the developmental changes of the somatodendritic morphology of subplate neurons with spec

206 it robust projection-specific differences in somatodendritic morphology, cellular excitability, and l

207 characterized the neurochemical specificity, somatodendritic morphology, synaptic ultrastructure as w

208 elevated neuronal rigidity, and reshaping of somatodendritic morphology.

209 Altogether, the data show that somatodendritic MORs in POMC neurons inhibit neuronal ac

210 dhesion molecule 2 (JAM2) as an inhibitor of somatodendritic myelination in spinal cord neurons, ther

211 stribution of membrane proteins to axonal or somatodendritic neuronal compartments is fundamental to

212 ge results from a compromise between AIS and somatodendritic oscillators.

213 s respond with transient Ca(2+) increase and somatodendritic oxytocin release following neuropeptide

214 VPS35 expression induces robust tau-positive somatodendritic pathology throughout the brain as indica

215 M interneuron models that incorporated I(M), somatodendritic placement of Kv7 channels best reproduce

216 d a uniform distribution of receptors in the somatodendritic plasma membrane when imaged over a 1 min

217 gly expressed and distributed throughout the somatodendritic plasma membrane.

218 te for delivering signaling receptors to the somatodendritic plasma membrane.

219 events mediating receptor insertion into the somatodendritic plasma membrane.

220 erved a transient appearance of NgCAM on the somatodendritic plasma membrane.

221 ic micron-scale domains within extrasynaptic somatodendritic plasma membranes of pyramidal neurons.

222 esicles, caused loss of TGN localization and somatodendritic polarity of ATP7B.

223 ab5 in rat hippocampal neurons abrogates the somatodendritic polarity of the transferrin receptor and

224 M5R was located mainly to VTA somatodendritic profiles (71%; n = 627), at least one-th

225 beling during P5-P10 was mainly localized in somatodendritic profiles but also was readily seen in ax

226 localized to endomembranes in DAT-containing somatodendritic profiles but showed a more prominent, si

227 mmunogold labeling was predominately seen in somatodendritic profiles throughout the PPT/LTD complex.

228 CB1r immunoreactivity (-ir) in somatodendritic profiles was more often localized to the

229 densities of NK3Rs in PVN AVP- or OC-labeled somatodendritic profiles were measured by quantitative i

230 Somatodendritic profiles with CB1r-ir typically received

231 the CB1r-immunoreactive structures, 66% were somatodendritic profiles, 22% were axon terminals, and t

232 he cytoplasm and near asymmetric synapses on somatodendritic profiles.

233 s population, striatal MSNs have dichotomous somatodendritic properties that mirror differences in th

234 n, functions in the neuronal soma to exclude somatodendritic proteins from axonal transport carriers.

235 ion of axonal retrograde carriers containing somatodendritic proteins toward the soma.

236 rom the brain and colocalizes with Cav1.2 in somatodendritic puncta of cortical neurons in culture.

237 and whether it plays a role in axonal versus somatodendritic receptor localization.

238 shy cells of the cochlear nucleus, expressed somatodendritic receptors (alpha1/beta heteromers) and s

239 retrieval for the polarized distribution of somatodendritic receptors at steady state.

240 e that small endosomal carriers derived from somatodendritic recycling endosomes can serve to redistr

241 phin, which is known to be secreted from the somatodendritic region and has been shown previously to

242 nterneurons and GluR6-containing KARs in the somatodendritic region of both interneurons and pyramida

243 Shh signal transduction originates from the somatodendritic region of the neurons and occurs in neur

244 w fluorescent protein-SERT from axons to the somatodendritic region.

245 ity and form strong synapses on the proximal somatodendritic region.

246 related to calmodulin (CaM) and localized in somatodendritic regions of principal neurons throughout

247 targeted mitochondria are accumulated in the somatodendritic regions where mature lysosomes are predo

248 th approaches demonstrated that, in midbrain somatodendritic regions, HA-DAT was present in the plasm

249 dritic with scattered puncta in neuropil and somatodendritic regions.

250 in translocation predominantly occurs in the somatodendritic regions; such distribution is associated

251 ction potentials failed to evoke significant somatodendritic release detected via D2 receptor-mediate

252 We conclude that somatodendritic release employs molecular scaffolds to e

253 However, it is unclear whether somatodendritic release employs specialized sites for re

254 for release, and the molecular machinery for somatodendritic release is not understood.

255 Here, we provide the first report of somatodendritic release of CCK in the brain in male Spra

256 lts in enhancement of inhibition through the somatodendritic release of CCK.

257 This activity is regulated in part by somatodendritic release of dopamine and subsequent feedb

258 mediated synaptic current that resulted from somatodendritic release of dopamine in brain slices take

259 The somatodendritic release of dopamine within the ventral t

260 vesicles that mediate the activity-dependent somatodendritic release of multiple retrograde signals i

261 Despite early convincing evidence for somatodendritic release of neurohypophysial peptides in

262 entricular (i.c.v.) NPS evoked a significant somatodendritic release of OXT within the PVN as assesse

263 Here, we provide the first evidence for somatodendritic release of the satiety peptide cholecyst

264 tterns and frequencies on activity-dependent somatodendritic release of vasopressin from paraventricu

265 Somatodendritic release of vasopressin was rarely observ

266 Somatodendritic release of VP was rarely observed in res

267 release, NMDAR activation was necessary for somatodendritic release to occur at physiological firing

268 ) and oxytocin (OT) undergo Ca(2+)-dependent somatodendritic release within the supraoptic and parave

269 hers ATP7A at the trans-Golgi network in the somatodendritic segments of motor neurons and that alter

270 up the Kv4.2 potassium channels (involved in somatodendritic signal integration and attenuation of de

271 Significance statement: Somatodendritic signaling using endocannabinoids or nitr

272 uggest that the M3 subtype is present on the somatodendritic site of glycinergic neurones and is main

273 -HT(1A) receptor; Rh-CT(5-HT1A) localizes to somatodendritic sites and is efficiently trafficked to d

274 from both terminals in projection areas and somatodendritic sites within the ventral midbrain.

275 the primary recognition event that underlies somatodendritic sorting and contribute to the evolving v

276 Herein we show that somatodendritic sorting of various transmembrane recepto

277 is distributed in a gradient throughout the somatodendritic space.

278 keleton, exclusion of both axon-specific and somatodendritic-specific cell surface proteins, and accu

279 that these properties underpin a whole-cell somatodendritic spike generation mechanism that makes th

280 'shadow') and identified on the basis of its somatodendritic structure.

281 s of the voltage-gated K+ channel underlying somatodendritic subthreshold A-type currents (I(SA)) in

282 Kv4.2 is a major pore-forming subunit in somatodendritic subthreshold A-type potassium current (I

283 Kv4 channels mediate most of the somatodendritic subthreshold operating A-type current (I

284 EEP21 leads to missorting of L1/NgCAM to the somatodendritic surface as well as to lysosomes.

285 VGSCs in hippocampal neurons to limit their somatodendritic surface expression, although exerting li

286 MPA-type glutamate receptors (AMPARs) to the somatodendritic surface of rat hippocampal pyramidal neu

287 es differentially wrap around the axonal and somatodendritic surface of the polymodal dendritic arbor

288 phas (alpha) 1, 2, 3 subunits was located on somatodendritic surfaces of neurochemically distinct mye

289 fects occurred without altering AIS Na(+) or somatodendritic T-type channel activity and could be med

290 can direct apical targeting in epithelia and somatodendritic targeting in neurons.

291 d tau hyperphosphorylation in the absence of somatodendritic tau inclusions.

292 levels in the brain and a greater extent of somatodendritic tau redistribution by three months of ag

293 RN) were examined with whole-cell recording, somatodendritic three-dimensional reconstructions and mo

294 v) channel alpha subunit responsible for the somatodendritic transient or A-type current I(SA) that a

295 Differences in somatodendritic transmission would be expected in vivo t

296 two corresponding timescales throughout the somatodendritic tree.

297 Syt10 colocalized with IGF-1 in somatodendritic vesicles of olfactory bulb neurons, and

298 t that there is a decrease in the content of somatodendritic vesicular dopamine in the Lep(ob/ob) mic

299 Rapidly activating and inactivating somatodendritic voltage-gated K(+) (Kv) currents, I(A),

300 ining pattern for PICK1-immunoreactivity was somatodendritic with scattered puncta in neuropil and so