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1 l genome that is merely bloated with AT-rich spacer DNA.
2 tes containing two CGG triplets separated by spacer DNA.
3 wo hexameric half-sites separated by 3 bp of spacer DNA.
4 ependent on conformational variations in the spacer DNA.
5 A show that the protein does not contact the spacer DNA.
6 as weaker than HS2 and more sensitive to the spacer DNA.
7 contact is seen between the proteins and the spacer DNA.
8 x to be the minimal machinery that catalyses spacer DNA acquisition and explain the significance of C
9 heterogeneity in telomere addition sites for spacer DNA also resembles the situation found for telome
10  that the most inactive promoters have short spacer DNA and the NtrC sites on the opposite side of th
11 t of CGG triplets, in contacting a TA in the spacer DNA, and in making direct dimer contacts between
12 ial (cox1) and nuclear (internal transcribed spacer) DNA data from the Schistosoma eggs or miracidia
13 rter gene when juxtaposed, and separation by spacer DNA has little effect on their function.
14 formational and/or dynamic properties of the spacer DNA in a process termed "indirect readout".
15 bstrate displayed protection in non-specific spacer DNA in addition to the recognition elements.
16                            Acquiring foreign spacer DNA into the CRISPR locus is an essential primary
17 esence of a nick or gap in one strand of the spacer DNA is presented in this paper.
18 mere addition, we sought to determine if the spacer DNAs might also undergo telomere addition prior t
19 ally interchangeable and require an internal spacer DNA of minimal length between 238 and 325 bp to b
20 d dimerization helix specifies the length of spacer DNA recognized.
21              The Mira gene is located in the spacer DNA region (SDR) separating Hoxa6 and Hoxa7, tran
22 s are typically separated from each other by spacer DNA segments, which are eliminated following chro
23 on both the sequence and the position of the spacer DNA separating the RBS and TRS motifs.
24  DNA binding and (v) determine that flexible spacer DNA sequences enhance Gsx2 cooperativity on dimer
25  DNA elements differing in the length of the spacer DNA that separates two conserved recognition moti
26             These elements are separated by 'spacer' DNA, the sequence of which is generally consider
27 ng factor that reshapes the suboptimal 19-bp spacer DNA to enable optimal promoter recognition, susta
28                               We deleted the spacer DNA to induce a potential heterochronic gain of f
29  with a specific geometry is required in the spacer DNA, to bring the Sin dimers at sites I and II to
30 ithin and outside those complementary to the spacer, DNA topology, target length, presence of non-spe
31 action approach, we found that at least some spacer DNAs undergo de novo telomere addition.
32                           The observation of spacer DNAs with telomeric repeats makes it unlikely tha
33 pacer' DNA, but how they avoid targeting the spacer DNA within the encoding CRISPR locus itself is un